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1 ant disease control and resulted in enhanced melanoma-specific survival.
2 The primary end point was melanoma-specific survival.
3 Recurrence, overall survival, and melanoma-specific survival.
4 s of this analysis were overall survival and melanoma-specific survival.
5 dissection was not associated with increased melanoma-specific survival among 1934 patients with data
6 prognostic information but did not increase melanoma-specific survival among patients with melanoma
7 ults from prior studies.OBJECTIVE To compare melanoma-specific survival between patients with histopa
10 to extend disease free survival and increase melanoma-specific survival for patients with early nodal
11 d prolongs distant disease-free survival and melanoma-specific survival for patients with nodal metas
12 mitotic rate was a significant predictor of melanoma-specific survival (Hazard Ratio [HR] = 1.2, 95%
13 tus was an independent prognostic factor for melanoma-specific survival (hazard ratio [HR] = 1.34; 95
14 tasis, 0.62; P=0.02) and the 10-year rate of melanoma-specific survival (hazard ratio for death from
15 mph-node biopsy is associated with increased melanoma-specific survival (i.e., survival until death f
16 whereas a marker in EBF1 was associated with melanoma-specific survival in advanced-stage patients (r
17 (P<0.0001) and significantly associated with melanoma-specific survival in both stage III (P<0.0001)
18 all six genes were significant in predicting melanoma specific survival (MSS) in a univariate analysi
22 d rates of 10-year overall survival (OS) and melanoma-specific survival (MSS) were 82% and 94%, respe
25 l lymph node (SLN) had significantly reduced melanoma-specific survival (MSS), DFS, regional node rec
28 sociated with poorer overall (OS; P < .001), melanoma-specific survival (MSS; P = .0025), and disease
31 in the recurrence rate, overall survival, or melanoma-specific survival of patients with MIS treated
34 ntermediate-thickness melanomas, the 10-year melanoma-specific survival rate was 62.1 +/- 4.8% among
35 treatment-related difference in the 10-year melanoma-specific survival rate was seen in the overall
42 sults suggest that the MAPK pathway mediates melanoma-specific survival signaling by differentially r
45 ol analysis, the mean (+/-SE) 3-year rate of melanoma-specific survival was similar in the dissection
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