ライフサイエンスコーパス: melanotroph

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1 s neither Rb expression nor proliferation of melanotrophs.

2 had no hyperplasia of the intermediate lobe melanotrophs.

3 f membrane capacitance changes in single rat melanotrophs.

4 Targeted Rb1 deletion in melanotrophs ablates the entire pituitary intermediate l

5 nhibit aberrant proliferation of Rb1-deleted melanotrophs but induces their apoptotic death.

6 ormation, the earliest stages of spontaneous melanotroph carcinogenesis in Rb+/- heterozygous mice ha

7 Interestingly, abrogation of melanotroph carcinogenesis results in accelerated progre

8 toma (Rb) gene is sufficient for spontaneous melanotroph carcinogenesis to occur in almost all Rb+/-

9 e function of this gene affects the onset of melanotroph carcinogenesis, likely by controlling prefer

10 as Cdkn1b) in Sox2 mutants does not restore melanotroph emergence.

11 e-cell level, basal and regulated release by melanotrophs from fetal and early postnatal ages.

12 etal rats, the secretory capability of fetal melanotrophs has not yet been examined directly.

13 Melanotrophs have IP3-sensitive calcium stores, but no c

14 liferation, and subsequently, acquisition of melanotroph identity.

15 Secretion in melanotrophs is modulated by protein kinase activators.

16 To prevent melanotroph neoplasia while preserving spontaneous carci

17 re results in complete downregulation of the melanotroph pioneer factor PAX7, and subsequently a swit

18 g a period corresponding to the cessation of melanotroph proliferation between 35 and 60 days after b

19 Atypical melanotrophs remain competent for dopamine D2 receptor s

20 r directs expression of the human RB gene to melanotrophs (TgPOMC-RB).

21 , and subsequently a switch of identity from melanotrophs to ectopic corticotrophs.

22 f cancer more closely, spontaneous pituitary melanotroph tumors arising in immunocompetent Rb +/- mic

23 eltaN transgenes failed to prevent pituitary melanotroph tumors but delayed tumor formation or progre

24 crossed to Rb(+/-) background are devoid of melanotroph tumors but develop the usual spectrum of oth

25 sually succumb to fast-growing, Rb-deficient melanotroph tumors of the pituitary intermediate lobe, w

26 timulating hormone, which is overproduced by melanotroph tumors, attenuates neoplastic progression by

27 In contrast to surrounding mature melanotrophs with the wild-type Rb gene, Rb-negative cel

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