ライフサイエンスコーパス: meliloti

1 the closely related bacterium Sinorhizobium meliloti.

2 eted in the incompatible interaction with S. meliloti.

3 d ChvI, and localizes to the periplasm of S. meliloti.

4 odel root nodulating bacterium Sinorhizobium meliloti.

5 em (PTS) have been found in the genome of S. meliloti.

6 of the mechanisms that govern motility in S. meliloti.

7 both free-living and symbiotic states of S. meliloti.

8 osynthesis of exopolysaccharides (EPS) by S. meliloti.

9 codes an LPS sulfotransferase activity in S. meliloti.

10 quorum sensing downregulates motility in S. meliloti.

11 architecture of the biofilm of Sinorhizobium meliloti.

12 y reduced nodulation when inoculated with S. meliloti.

13 with the diazotropic bacterium Sinorhizobium meliloti.

14 les following inoculation with Sinorhizobium meliloti.

15 novel lipid of R. leguminosarum 3841 and S. meliloti.

16 otic nitrogen fixing bacterium Sinorhizobium meliloti.

17 d) genes in the soil bacterium Sinorhizobium meliloti.

18 has not been characterized previously in S. meliloti.

19 an important role in manganese uptake in S. meliloti.

20 )-polymerase activator DctD of Sinorhizobium meliloti.

21 ication in the legume symbiont Sinorhizobium meliloti.

22 tween alfalfa and its symbiont Sinorhizobium meliloti.

23 tages during invasion of M. truncatula by S. meliloti.

24 odulation of alfalfa (Medicago sativa) by S. meliloti.

25 ther possible quorum sensing system(s) in S. meliloti.

26 the symbiotic interaction with Sinorhizobium meliloti.

27 influenced the activity of NCR247 against S. meliloti.

28 on of NCR247 lowered its activity against S. meliloti.

29 exhibited antimicrobial activity against S. meliloti.

30 open reading frames, podJ1 and podJ2, in S. meliloti.

31 diated regulation of stress adaptation in S. meliloti.

32 ream responses to its symbiont Sinorhizobium meliloti.

33 ylphosphonate in the bacterium Sinorhizobium meliloti 1021 is proposed based on the analysis of the g

34 e plant intracellular symbiont Sinorhizobium meliloti 1021 is required for conjugal transfer of DNA.

35 Previous studies had shown that S. meliloti 1021 mutants that produce increased levels of s

36 ogen-fixing rhizobial symbiont Sinorhizobium meliloti 1021 produces acidic symbiotic exopolysaccharid

37 Transfer of pMJK-1 into S. meliloti 1021 results in heterologous expression of 1-ph

38 otal lipids from R. leguminosarum 3841 or S. meliloti 1021 were added.

39 t, enhances the symbiotic productivity of S. meliloti 1021 with the host plant Medicago truncatula cv

40 tal mating of these genes into Sinorhizobium meliloti 1021, a strain that lacks these particular GalA

41 ologous expression of RgtA in Sinorhizhobium meliloti 1021, a strain that normally lacks GalA modific

42 guanylate (c-di-GMP) levels in Sinorhizobium meliloti 8530, a bacterium that does not carry known cel

43 Sinorhizobium meliloti, a legume symbiont and Brucella abortus, a phyl

44 Sinorhizobium meliloti, a legume symbiont, and Brucella abortus, a phy

45 S. meliloti, a symbiont of leguminous plants, synthesizes m

46 termined that BacA specifically protected S. meliloti against oxidized NCR247.

47 new avenues for understanding the complex S. meliloti-alfalfa interactions which occur during symbios

48 ld more in this interaction compared with S. meliloti alone.

49 S. meliloti also produces sulfated cell surface polysacchar

50 Strikingly, Sinohizobium meliloti, an intracellular symbiont of legume plants, ca

51 plays in the symbiosis between Sinorhizobium meliloti and alfalfa has been studied for over a decade,

52 e symbiosis that forms between Sinorhizobium meliloti and alfalfa requires biosynthesis of Nod factor

53 In the symbiosis between Sinorhizobium meliloti and alfalfa, mutations in GlnD, the major bacte

54 that have been inoculated with Sinorhizobium meliloti and are developing root nodules.

55 The lipid A molecules of S. meliloti and B. abortus are unusually modified with a ve

56 crucial for the chronic infection of both S. meliloti and B. abortus.

57 le-dependent regulons of CtrA and DnaA in S. meliloti and C. crescentus.

58 o related alphaproteobacteria, Sinorhizobium meliloti and Caulobacter crescentus, serve as models for

59 ere that two of these species, Sinorhizobium meliloti and Caulobacter crescentus, simply lack any ext

60 analyses of the SOE SorT from Sinorhizobium meliloti and its cognate electron acceptor SorU.

61 symbiotic relationship between Sinorhizobium meliloti and its host Medicago sativa (alfalfa) depends

62 he establishment of the symbiosis between S. meliloti and its host Medicago sativa.

63 rogen-fixing symbiosis between Sinorhizobium meliloti and its legume host alfalfa (Medicago sativa) d

64 o study the coordinate differentiation of S. meliloti and its legume partner during nodule developmen

65 rogen-fixing symbiosis between Sinorhizobium meliloti and its leguminous host plant Medicago truncatu

66 Symbiosis between S. meliloti and its plant host alfalfa is dependent on bact

67 nt of a nitrogen-fixing symbiosis between S. meliloti and its plant hosts.

68 Using the Sinorhizobium meliloti and Medicago truncatula symbiotic system, we pr

69 uch as the interaction between Sinorhizobium meliloti and Medicago, bacteroid differentiation is driv

70 In addition, MtFNSII-2 was inducible by S. meliloti and methyl jasmonate treatment, whereas MtFNSII

71 r the regulation of core glycosylation in S. meliloti and other bacteria containing LpcC orthologs.

72 tative proteins, SMc02148 from Sinorhizobium meliloti and PA3455 from Pseudomonas aeruginosa, reveale

73 ommonalities between symbiotic Sinorhizobium meliloti and pathogenic Brucella bacteria in terms of ex

74 s that have been classified as Sinorhizobium meliloti and Sinorhizobium medicae.

75 nvolved in more than carbon regulation in S. meliloti and suggest that the phenotypes observed occur

76 n the requirements for RpoH1 and RpoH2 in S. meliloti and that there must be other crucial targets.

77 truncatula, its symbiosis with Sinorhizobium meliloti, and on soil microbial community structure.

78 Our data indicate that in S. meliloti, and possibly in other Rhizobiaceae species, th

79 Coexpression of S. meliloti aqpS and arsC in a strain of E. coli lacking th

80 d the role of aqpS in arsenic resistance, S. meliloti aqpS and arsC were disrupted individually.

81 rter], and arsC (arsenate reductase), the S. meliloti ars operon includes an aquaglyceroporin (aqpS)

82 gest that AqpS is the only protein of the S. meliloti ars operon that facilitates transport of arseni

83 This pathway is likely not limited to S. meliloti as suggested by the presence of homologous gene

84 es and the rhizobial bacterium Sinorhizobium meliloti as well as with the pathogenic oomycete Aphanom

85 successfully predict sRNAs in Sinorhizobium meliloti, as well as in multiple and poorly annotated sp

86 The Medicago truncatula-Sinorhizobium meliloti association is an excellent model for dissectin

87 to wild-type and succinoglycan-deficient S. meliloti at the early time point of 3 days postinoculati

88 Sinorhizobium meliloti bacA mutants are symbiotically defective, deoxy

89 We show that, when starved, dividing S. meliloti bet hedge by forming two daughter cells with di

90 The S. meliloti bluB mutant is unable to grow in minimal media

91 a greater role in the overall fitness of S. meliloti both during the free-living stage and in its as

92 n the closely related bacteria Sinorhizobium meliloti, Brucella abortus, and Ochrobactrum anthropi.

93 The AAA+ domain of Sinorhizobium meliloti C4-dicarboxylic acid transport protein D (DctD)

94 Sinorhizobium meliloti can live as a soil saprophyte and can engage in

95 Sinorhizobium meliloti can live as symbionts inside legume root nodule

96 approach, we identified a homolog of the S. meliloti carbohydrate sulfotransferase, LpsS, in Mesorhi

97 In Sinorhizobium meliloti, catabolite repression is influenced by a nonca

98 chanisms governing the alterations of the S. meliloti cell cycle in planta are largely unknown.

99 To achieve polyploidy, the S. meliloti cell cycle program must be altered to uncouple

100 The predicted S. meliloti cell cycle regulon of CtrA, but not that of Dna

101 ll growth that enabled global analysis of S. meliloti cell cycle-regulated gene expression.

102 Only 28% of the 462 S. meliloti cell cycle-regulated genes were also transcript

103 out the small 83 amino acid protein FeuN, S. meliloti cells are unable to grow, and this phenotype is

104 In addition, S. meliloti cells constitutively producing Nod factors were

105 nderstanding of the physiological changes S. meliloti cells go through during the early stages of sym

106 Moreover, adding Nod Factor antibody to S. meliloti cells inhibits biofilm formation, while chitina

107 Sinorhizobium meliloti cells store excess carbon as intracellular poly

108 d lsrB genes are expressed in free-living S. meliloti cells, but they are not required for cell growt

109 ation with the use of flavone pre-treated S. meliloti cells, completely restored nodulation in flavon

110 e HK-two RR motif found in the Sinorhizobium meliloti chemotaxis pathway and measuring the resulting

111 Our discovery of S. meliloti chemotaxis to plant-derived QACs adds another r

112 ipid A is important, but not crucial, for S. meliloti chronic infection and that BacA must have an ad

113 their microscopic nodule phenotype using S. meliloti constitutively expressing lacZ.

114 f three quorum sensing systems present in S. meliloti, controls the production of the symbiotically a

115 We show that S. meliloti CpdR1 has a polar localization pattern and a ro

116 The S. meliloti cpdR1-null mutant can invade the host cytoplasm

117 In Sinorhizobium meliloti, CuxR stimulates transcription of an EPS biosyn

118 Sinorhizobium meliloti dctA encodes a transport protein needed for a s

119 ted selection procedure, four independent S. meliloti dctA mutants were isolated that retained some a

120 Sinorhizobium meliloti DctD is an activator of sigma(54)-RNA polymeras

121 Within specialized host cells, S. meliloti differentiates into highly polyploid, enlarged

122 -dependent activator DctD from Sinorhizobium meliloti, displayed an altered DNase I footprint at its

123 loti engages in a symbiosis with legumes: S. meliloti elicits the formation of plant root nodules whe

124 minosarum and the lpsB gene of Sinorhizobium meliloti encode protein orthologs that are 58% identical

125 ntracellular regulatory pathways to drive S. meliloti endoreduplication and differentiation during sy

126 dwelling alpha-proteobacterium Sinorhizobium meliloti engages in a symbiosis with legumes: S. melilot

127 Deletion of hprK in S. meliloti enhanced catabolite repression caused by succin

128 Sinorhizobium meliloti enters into a symbiotic relationship with legum

129 The alpha-proteobacterium Sinorhizobium meliloti establishes a chronic intracellular infection d

130 The production of the Sinorhizobium meliloti exopolysaccharide, succinoglycan, is required f

131 Sinorhizobium meliloti ExoR regulates the production of succinoglycan

132 In S. meliloti, exoR and the exoS-chvI two-component system re

133 The Sinorhizobium meliloti ExoS/ChvI two-component signaling pathway is re

134 In pathogenic bacteria closely related to S. meliloti, exoS-chvI homologues are required for virulenc

135 In S. meliloti, ExoS/ChvI is a key regulator of gene expressio

136 ula plants inoculated with the Sinorhizobium meliloti exoY mutant, and the M. truncatula vapyrin-2 mu

137 ecule also inhibits the expression of the S. meliloti exp genes, responsible for the production of EP

138 microscopy after staining with X-Gal for S. meliloti expressing a constitutive GUS gene, by confocal

139 When tested for nodulation by Sinorhizobium meliloti, flavonoid-deficient roots had a near complete

140 the alpha-proteobacteria (e.g. Sinorhizobium meliloti), for dephosphorylating CheY-P.

141 Sinorhizobium meliloti forms symbiotic, nitrogen-fixing nodules on the

142 However, in S. meliloti, free-living cells of the cpdR1-null mutant sho

143 erial BacA protein is critical to prevent S. meliloti from being hypersensitive toward NCR AMPs.

144 Recently, we discovered that a Sinorhizobium meliloti gene, bluB, is necessary for DMB biosynthesis.

145 tem also seems to regulate a multitude of S. meliloti genes, including genes that participate in low-

146 tom Affymetrix GeneChip with the complete S. meliloti genome and approximately 10,000 probe sets for

147 The S. meliloti genome encodes 14 of these alternative sigma fa

148 were identified in the recently sequenced S. meliloti genome.

149 oxide in roots inoculated with Sinorhizobium meliloti greatly increased our understanding of the role

150 Expression profiling of free-living S. meliloti grown with the plant signal molecule luteolin i

151 otic nitrogen-fixing bacterium Sinorhizobium meliloti harbors a gene, SMc02396, which encodes a predi

152 including Brucella; however, its role in S. meliloti has not been investigated.

153 S. meliloti has three quorum-sensing systems (Sin, Tra, and

154 ymology and genetics of PHB metabolism in S. meliloti have been well characterized, phasins have not

155 S. meliloti Hfq is involved in controlling the population d

156 ument complex transcriptional profiles of S. meliloti in different environments.

157 pression in the soil bacterium Sinorhizobium meliloti in response to the plant-secreted flavonoid lut

158 its a reduced transcriptional response to S. meliloti, indicating that the machinery responsible for

159 structure-related genes in both PhiM9 and S. meliloti-infecting T4 superfamily phage PhiM12, which co

160 This phylogenetic and structural study of S. meliloti-infecting T4 superfamily phage PhiM9 provides n

161 on microscopy structure of the Sinorhizobium meliloti-infecting T4 superfamily phage PhiM9.

162 lotillin-like genes (FLOTs) in Sinorhizobium meliloti infection of its host legume Medicago truncatul

163 d that protein exudation in the M. sativa-S. meliloti interaction caused an increase in the secretion

164 This report shows that M. truncatula-S. meliloti interactions involve ecotype-strain specificity

165 ferentiation of the bacterium, Sinorhizobium meliloti into a nitrogen-fixing bacteroid within the leg

166 The S. meliloti iolA (mmsA) gene product seems to be involved i

167 Sinorhizobium meliloti is a gram-negative soil bacterium capable of fo

168 Sinorhizobium meliloti is a gram-negative soil bacterium found either

169 Sinorhizobium meliloti is a gram-negative soil bacterium found either

170 Sinorhizobium meliloti is a gram-negative soil bacterium, capable of e

171 Sinorhizobium meliloti is a member of the Alphaproteobacteria that fix

172 Sinorhizobium meliloti is a nitrogen-fixing bacterial symbiont of alfa

173 The bacterium Sinorhizobium meliloti is attracted to seed exudates of its host plant

174 These results imply that, when S. meliloti is exposed to environmental arsenate, arsenate

175 hat the ExpR/Sin quorum-sensing system in S. meliloti is involved in the regulation of genes responsi

176 Growth of S. meliloti is modelled in three ecological niches (bulk so

177 onally, during early stages of symbiosis, S. meliloti is presented with an oxidative burst that must

178 We show that GroEL1 of Sinorhizobium meliloti is required for efficient infection, terminal d

179 on the lipid A in the free-living form of S. meliloti, is essential for the chronic infection.

180 ferent species of rhizobia in a strain of S. meliloti lacking endogenous NodD activity.

181 Our studies of an S. meliloti loss-of-function hfq mutant have revealed that

182 After inoculation with compatible S. meliloti, LYK3:GFP puncta are relatively stable.

183 l role during key steps of the Sinorhizobium meliloti-M. truncatula symbiotic interaction.

184 studies indicate that, in contrast to the S. meliloti-Medicago model symbiosis, bacteroids in the S.

185 The Sinorhizobium meliloti megaplasmid pSymA has previously been implicate

186 iate GalA donor substrate is available in S. meliloti membranes.

187 dioxygenases and hydrolases in Sinorhizobium meliloti, Mesorhizobium loti, and Bradyrhizobium japonic

188 The S. meliloti ML beta-glucan participates in bacterial aggreg

189 a inoculated with succinoglycan-deficient S. meliloti more strongly express an unexpectedly large num

190 d with wild-type, succinoglycan-producing S. meliloti more strongly express genes encoding translatio

191 S. meliloti MsbA2 is required for the invasion of nodule ti

192 In planktonic S. meliloti, MucR properly coordinates a diverse set of bac

193 he host plant Medicago sativa, Sinorhizobium meliloti must overcome an oxidative burst produced by th

194 ium meliloti or to the invasion defective S. meliloti mutant, exoA.

195 d this by constructing and characterizing S. meliloti mutants in the lpxXL and acpXL genes, which enc

196 We have constructed S. meliloti mutants that lack HPrK or that lack key amino a

197 two-part screen, we identified Sinorhizobium meliloti mutants that were both sensitive to hydrogen pe

198 Mapping of S. meliloti mutations conferring resistance to PhiM12, N3,

199 ute to the release of free fatty acids in S. meliloti, neither one can use phospholipids as substrate

200 ensures that a cascade of the Sinorhizobium meliloti nitrogen fixation genes is induced as the conce

201 S. meliloti Nod factor is a lipochitooligosaccharide that u

202 the synthesis of an inducer of Sinorhizobium meliloti nod genes, as well as a gene associated with No

203 , 4'-dihydroxyflavone, a major inducer of S. meliloti nod genes, completely restored nodulation.

204 n sulfated fungal Myc-LCOs and Sinorhizobium meliloti Nod-LCOs, having very similar structures, each

205 D, we assayed the DNA binding activity of S. meliloti NodD1 treated with the flavonoid inducer luteol

206 Sinorhizobium meliloti nodL and nodF mutations additively reduce infec

207 S. meliloti nodulation factor treatments of MtROP9i led to

208 A17, the Fix phenotypes are reversed with S. meliloti NRG185 and NRG34, and there is a correlation be

209 S. meliloti NRG185 produces oligosaccharides that are almos

210 a A17, and the phenotype is reversed with S. meliloti NRG185.

211 Sinorhizobium meliloti NRG247 has a Fix(+) phenotype on Medicago trunc

212 ass spectrometry, and the analysis of the S. meliloti NRG247 oligosaccharides showed that this strain

213 fixing root nodules induced by Sinorhizobium meliloti on the model plant Medicago truncatula, tubules

214 first characterization of the Sinorhizobium meliloti open reading frame SMc01113.

215 plants exposed for 24 h to WT Sinorhizobium meliloti or to the invasion defective S. meliloti mutant

216 Sinorhizobium meliloti participates in a nitrogen-fixing symbiosis wit

217 ExoR(c20) was isolated directly from S. meliloti periplasm to identify its N-terminal amino acid

218 genomic and structural levels, PhiM9 and S. meliloti phage PhiM12 have a small number of open readin

219 he recently characterized cyanophage-like S. meliloti phages of the PhiM12 group.

220 e gram-negative soil bacterium Sinorhizobium meliloti plays an important role in the establishment of

221 As in C. crescentus, the S. meliloti PodJ1 protein appears to act as a polarity beac

222 We leveraged synchronized S. meliloti populations to determine how treatment with a s

223 Sinorhizobium meliloti produces an exopolysaccharide called succinogly

224 discovered that in the absence of VLCFAs, S. meliloti produces novel pentaacylated lipid A species, m

225 ng SMc01113/YbeY expression in Sinorhizobium meliloti produces symbiotic and physiological phenotypes

226 An S. meliloti relA mutant which cannot produce ppGpp was prev

227 The symbiotic lifestyle of Sinorhizobium meliloti requires a drastic cellular differentiation tha

228 Symbiotic infection by S. meliloti requires an osmosensory two-component system co

229 Sinorhizobium meliloti requires exopolysaccharides in order to form a

230 Sinorhizobium meliloti requires ExoS/ChvI two-component signalling to

231 herefore, like E. coli and other species, S. meliloti requires only one groEL gene for viability, and

232 of the whole-genome expression profile of S. meliloti reveals that the ExpR/Sin quorum-sensing system

233 ysaccharide produced by strain Sinorhizobium meliloti Rm1021 contributes to symbiosis with Medicago s

234 The Sinorhizobium meliloti Rm1021 Delta glnD-sm2 mutant, which is predicte

235 ions of A. tumefaciens C58 and Sinorhizobium meliloti Rm1021 genomes.

236 trogen stress response (NSR) of wild type S. meliloti Rm1021, and isogenic strains missing both PII p

237 st identity to SinI (71%) from Sinorhizobium meliloti Rm1021.

238 As populations of Sinorhizobium meliloti Rm2011 were similar in bulk/dissolved and ENM t

239 ns involve ecotype-strain specificity, as S. meliloti Rm41 and NRG247 are Fix+ (compatible) on M. tru

240 o compatibility; most importantly, (v) an S. meliloti Rm41 derivative, carrying exo genes from an M.

241 whereas the oligosaccharides produced by S. meliloti Rm41 include many nonsuccinylated subunits, as

242 ibility with the microsymbiont Sinorhizobium meliloti Rm41.

243 The inferred S. meliloti RpoH promoter consensus sequences share feature

244 ze the global transcriptional response of S. meliloti rpoH1, rpoH2, and rpoH1 rpoH2 mutants during he

245 r, luteolin, the plant-derived inducer of S. meliloti's nod genes, is not required for mature biofilm

246 Contrary to S. meliloti, S. fredii HH103 showed little or no sensitivit

247 The nitrogen-fixing symbiont Sinorhizobium meliloti senses and responds to constantly changing envi

248 ere we report evidence that in Sinorhizobium meliloti, sensing of AHLs with acyl chains composed of 1

249 Proline utilization A from Sinorhizobium meliloti (SmPutA) is a 1233-residue bifunctional enzyme

250 Sinorhizobium meliloti stores carbon and energy in poly-3-hydroxybutyr

251 biont of one of these species (Sinorhizobium meliloti strain Rm1021) and an opportunistic bacterial p

252 es this type of specificity pertaining to S. meliloti strain Rm41.

253 Nearly all Ensifer meliloti strains completely lack ebpA, tcrA, tcsA and fs

254 eric oligosaccharides (STOs) from the two S. meliloti strains were compared by chromatography and mas

255 Analysis of free-living S. meliloti strains with mutations in genes related to nitr

256 lycan oligosaccharide populations between S. meliloti strains; (iv) the structural nature of the succ

257 one of two exopolysaccharides produced by S. meliloti (succinoglycan or EPS II) is required for nodul

258 acteria with similar ecological niches as S. meliloti, suggesting that the CtrA cell cycle regulatory

259 p2) showed no transcriptional response to S. meliloti, suggesting that the encoded proteins are requi

260 in mutant nodules, we assayed Sinorhizobium meliloti symbiosis gene promoters (nodF, exoY, bacA, and

261 SMc01113 gene is absolutely required for S. meliloti symbiosis with alfalfa and also for the protect

262 er, in the Medicago truncatula-Sinorhizobium meliloti symbiosis, incompatibility between symbiotic pa

263 In the Medicago truncatula/Sinorhizobium meliloti symbiosis, the plant undergoes a series of deve

264 (Fix+) in Medicago truncatula-Sinorhizobium meliloti symbiosis.

265 S. meliloti synthesizes an unusual sulfate-modified form of

266 nal LPS sulfotransferase activity(ies) in S. meliloti that can compensate for the loss of LpsS.

267 two cpdR homologues, cpdR1 and cpdR2, of S. meliloti that encode single-domain response regulators.

268 c model of the legume symbiont Sinorhizobium meliloti that is integrated with carbon utilization data

269 tic nitrogen-fixing bacterium, Sinorhizobium meliloti The LDSS-P profiles that overlap with the 5' se

270 e study of trehalose uptake by Sinorhizobium meliloti, the demonstrated approach is applicable to man

271 of Sma0114 from the bacterium Sinorhizobium meliloti, the first such characterization of a receiver

272 y do not stimulate nod gene expression in S. meliloti, the flavonoids naringenin, eriodictyol, and da

273 First discovered in Sinorhizobium meliloti, the main response regulator CheY2-P shuttles i

274 Here we identify in Sinorhizobium meliloti, the Medicago symbiont, a cAMP-signaling regula

275 Quorum sensing (QS) in Sinorhizobium meliloti, the N-fixing bacterial symbiont of Medicago ho

276 ce of the compatible bacterium Sinorhizobium meliloti, the nip mutant showed nitrogen deficiency symp

277 Sinorhizobium meliloti, the nitrogen-fixing symbiont of alfalfa, has t

278 ales order: the plant symbiont Sinorhizobium meliloti, the plant pathogen Agrobacterium tumefaciens,

279 In Sinorhizobium meliloti, the production of exopolysaccharides such as s

280 In particular, in Sinorhizobium meliloti there are four groESL operons and one groEL gen

281 In Sinorhizobium meliloti there are two rpoH genes, four groESL operons,

282 n LPS structure can affect the ability of S. meliloti to form an effective symbiosis.

283 We utilized the genomic sequence of S. meliloti to identify an open reading frame, SMc04267 (wh

284 at ExoR binds to ExoS in the periplasm of S. meliloti to inhibit ExoS/ChvI activity, and that ExoR re

285 obacterial pathogens and might be used by S. meliloti to sense host cues during infection.

286 play an important role in the ability of S. meliloti to successfully invade its host, also helps the

287 rminating seeds, we assayed chemotaxis of S. meliloti towards betonicine, choline, glycine betaine, s

288 This peptide affects Sinorhizobium meliloti transcription, translation, and cell division a

289 A SMc01003-deficient mutant of S. meliloti transiently accumulates diacylglycerol, suggest

290 We recently identified an S. meliloti two-component sensory histidine kinase, CbrA, w

291 S. meliloti unable to produce the exopolysaccharide succino

292 hat a FadD-deficient mutant of Sinorhizobium meliloti, unable to convert free fatty acids to their co

293 ng symbiosis with host plants, Sinorhizobium meliloti undergoes a profound cellular differentiation,

294 When they are available, Sinorhizobium meliloti utilizes C(4)-dicarboxylic acids as preferred c

295 ne sma0113 as needed for strong SMCR when S. meliloti was grown in succinate plus lactose, maltose, o

296 The CbcX orthologue ChoX from Sinorhizobium meliloti was similar to CbcX in these binding properties

297 Searching the genome of S. meliloti, we identified a potential lysophospholipase (S

298 ula and its rhizobial symbiont Sinorhizobium meliloti, which includes more than 23,000 proteins, 20,0

299 ccharides is unclear, although mutants of S. meliloti with reduced LPS sulfation exhibit symbiotic ab

300 Medicago truncatula roots and Sinorhizobium meliloti would identify regulated plant genes that likel