ライフサイエンスコーパス: meloxicam

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1 acoxib is proposed compared to celecoxib and meloxicam.

2 A versus shams, with a significant effect of meloxicam.

3 bserved and were dose-dependently reduced by meloxicam.

4 ib (10 mg/kg BW), mavacoxib (4 mg/kg BW) and meloxicam (1 mg/kg BW) were determined following single

5 ly intraperitoneal injection of 0.9% NaCl or meloxicam 3 mg/kg, respectively, for 7 consecutive days.

6 Animals treated with meloxicam (5 mg/kg) exhibited a dose-dependent decrease

7 Meloxicam (5), an NSAID in the enol-carboxamide class, w

8 trasted with selective COX-2 inhibition with meloxicam, administered alone and in combination with AL

9 This study investigated whether the use of meloxicam alters bone repair via downregulation of VEGF

10 xpression of CD11a, CD11b, and CD18, whereas meloxicam and etanercept did not.

11 ion in CSCs we utilized the COX-2 inhibitors meloxicam and mavacoxib.

12 Two selective COX-2 inhibitors, meloxicam and NS398, decreased HIF-1alpha levels and nuc

13 t of nonselective (Piroxicam), preferential (Meloxicam), and selective COX-2 inhibitors (Parecoxib).

14 Notably, aspirin, meloxicam, and etanercept did not change retinal vascula

15 n given with rofecoxib, celecoxib, sulindac, meloxicam, and indometacin but not when given with ibupr

16 eri and either treated with dexamethasone or meloxicam (anti-inflammatory drugs) or left untreated.

17 crystal complexes of COX-2 with isoxicam and meloxicam at 2.0 and 2.45 angstroms, respectively, and a

18 ctively, and a crystal complex of COX-1 with meloxicam at 2.4 angstroms.

19 Aspirin and meloxicam both lowered retinal TNF-alpha levels.

20 In addition, a detailed study of meloxicam.COX-2 interactions revealed that mutation of V

21 In rat alveolar bone repair, meloxicam did not affect VEGF expression but downregulat

22 lective inhibitor of COX-1 and COX-2 whereas meloxicam displays some selectivity for COX-2.

23 4 to Ile significantly reduces inhibition by meloxicam due to subtle changes around Phe-518, giving r

24 High-dose aspirin, etanercept, and high-dose meloxicam each reduced leukocyte adhesion and suppressed

25 High-dose aspirin, etanercept, and high-dose meloxicam each reduced retinal ICAM-1 expression.

26 High-dose aspirin, etanercept and high-dose meloxicam, each suppressed the retinal expression of eNO

27 Only Meloxicam in histidine-tryptophan-ketoglutarate was demo

28 oxib, diclofenac, indomethacin, lumiracoxib, meloxicam, naproxen, rofecoxib, sodium salicylate, and S

29 those animals receiving analgesia (20 mg/kg meloxicam or 5 mg/kg bupivacaine) or saline post-operati

30 roach vasectomy) and either received saline, meloxicam or bupivacaine.

31 pirin, a cyclooxygenase 2 (COX-2) inhibitor (meloxicam), or a soluble tumor necrosis factor alpha (TN

32 pirin, celecoxib, indomethacin, lumiracoxib, meloxicam, or SC560, against COX-1 (human platelets) inc

33 contrast to celecoxib (T1/2el = 0.88 h) and meloxicam (T1/2el = 0.90 h), mavacoxib has a prolonged e

34 hown, with Parecoxib the least effective and Meloxicam the most effective treatment.

35 ided into a control group (CG; n = 60) and a meloxicam-treated group (TG; n = 60) that received eithe

36 d pain behavior in animals, without and with meloxicam treatment.

37 stration of the preferential COX-2 inhibitor Meloxicam via histidine-tryptophan-ketoglutarate showed

38 In contrast, the F% of meloxicam was low (F% = 11%).

39 In contrast, selective COX-2 inhibition with meloxicam was without effect on MNCV, NBF, or MI content

40 eroidal antiinflammatory drugs (NSAIDs), and meloxicam, were prescribed to a larger proportion of ost

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