ライフサイエンスコーパス: membrane conductance

1 the status of a voltage-dependent, intrinsic membrane conductance.

2 +/- S.E.M.), without appreciable effects on membrane conductance.

3 IL-2 (0.01-500 ng/ml) alone had no effect on membrane conductance.

4 rebound and regular spikes and an increased membrane conductance.

5 mbrane potential and an increase in apparent membrane conductance.

6 m changes in neurotransmitter release and/or membrane conductance.

7 ed by a decrease or no significant change in membrane conductance.

8 ude, accompanied by a pronounced increase in membrane conductance.

9 fect was not associated with changes in cell membrane conductance.

10 iated with either an increase or decrease in membrane conductance.

11 by cellular hyperpolarization and increased membrane conductance.

12 sed to measure the voltage dependency of the membrane conductance.

13 was typically associated with a decrease in membrane conductance.

14 urrent (Iacpd) associated with a decrease of membrane conductance.

15 d a reversible voltage-dependent decrease in membrane conductance.

16 econds to seconds, depending on the level of membrane conductance.

17 esence of the appropriate mRNA, protein, and membrane conductance.

18 ontacts with vestibular afferent nerves, and membrane conductance.

19 tral and its activation had little effect on membrane conductance.

20 ) channels and the Abeta-induced increase in membrane conductance.

21 at include a substantial increase in overall membrane conductance.

22 solved monitoring (</=30 ms) of IC-modulated membrane conductance.

23 l dendrites and contributed substantially to membrane conductance.

24 (P)10 because of a maturational increase in membrane conductance.

25 ate, whereas serotonin decreases the overall membrane conductance.

26 ng substantial (21 nS on average) changes in membrane conductance.

27 , on deoxygenation, showed a further rise in membrane conductance.

28 IL-2 (0.01-10 ng/ml) alone had no effect on membrane conductance.

29 everalfold increase in the rate of change of membrane conductance.

30 pette seal that is in parallel with the true membrane conductance.

31 y (HEK 293) cells and significantly increase membrane conductance.

32 ited spike firing through activation of K(+) membrane conductance.

33 y, followed by exponentiation through active membrane conductances.

34 etween excitatory and inhibitory neurons and membrane conductances.

35 ad effects on cells including alterations in membrane conductances.

36 lex neurons, together with voltage-dependent membrane conductances.

37 annels involved in the control of background membrane conductances.

38 ses the activation of at least two different membrane conductances.

39 imescale dynamics that arise from correlated membrane conductances.

40 ent sets of synaptic strengths and intrinsic membrane conductances.

41 may develop slowly by continuous changes in membrane conductances, a discontinuous change in axonal

42 th pentobarbitone and propofol increased the membrane conductance, although the benzodiazepine ligand

43 trations of toxin resulted in an increase of membrane conductance and a decrease in membrane stabilit

44 Lc/+ Purkinje cells have a very high membrane conductance and a depolarized resting potential

45 AHP that was associated with an increase in membrane conductance and a rightward shift in the discha

46 this mechanism may contribute to the resting membrane conductance and basal Ca2+ influx in this parti

47 hannels are thought to contribute to resting membrane conductance and basal Ca2+ influx in vascular m

48 siological setup allowed for measurements of membrane conductance and capacitance.

49 lls and arrested the maturational changes in membrane conductance and coupling coefficients.

50 t human RBCs display circadian regulation of membrane conductance and cytoplasmic conductivity that d

51 It was not accompanied by evident changes in membrane conductance and had a decay time constant simil

52 ged inositol 1,4,5-triphosphate (InsP(3)) on membrane conductance and intracellular Ca(2+) concentrat

53 eters (e.g., channel properties and density, membrane conductance and leak) and will apply to most sp

54 urrent; this change was due to a decrease in membrane conductance and may reflect the suppression of

55 he mutants' altered function with respect to membrane conductance and Na(+) sensitivity.

56 s characterized by a significant decrease in membrane conductance and reversed at a potential close t

57 g current was associated with a reduction in membrane conductance and reversed near Ek.

58 ons is associated with reductions in passive membrane conductance and the amplitude of the slow after

59 and ROS) and a specific effect on the plasma membrane conductance and the reduced ascorbate.

60 (K1), at hyperpolarized potentials decreases membrane conductance and thereby potentiates the ability

61 n of potassium channels increased the cell's membrane conductance and thus had a shunting effect on G

62 not associated with a significant change in membrane conductance and was relatively independent of m

63 s current was associated with an increase in membrane conductance and was still seen in the presence

64 h other, were associated with an increase in membrane conductance and were attenuated by the applicat

65 I(K-LVA) contributed strongly to the resting membrane conductance and, during trains of simulated EPS

66 s both to their own intrinsic pacemaker-like membrane conductances and excitatory synaptic inputs.

67 mechanisms involving direct control of both membrane conductances and gene expression in the MC4R PV

68 f halothane, isoflurane and enflurane on the membrane conductances and ion channels of cultured corti

69 gical processes by modulating both intrinsic membrane conductances and synaptic transmission.

70 otoreceptor neurotransmitter expression, and membrane conductances and synaptic vesicle release prope

71 clear separation of changes in capacitance, membrane conductance, and access resistance.

72 o produces an inward current, an increase in membrane conductance, and an elevation of [Ca2+]i.

73 /44 neurones) associated with an increase in membrane conductance, and an inward current (I5-HT,inwar

74 arrages (namely, depolarization, increase in membrane conductance, and increase in membrane potential

75 ization of the membrane potential, decreased membrane conductance, and increased discharge of action

76 ctivity (by depolarizing neurons, increasing membrane conductance, and introducing fluctuations) stro

77 onic', synaptic input, which increases their membrane conductance, and so modifies the spatial and te

78 gest that neurons experience periods of high membrane conductance, and that action potentials are oft

79 depolarization was associated with decreased membrane conductance, and this current had a reversal po

80 Their intrinsic membrane conductances, and their remaining excitatory sy

81 Small hyperpolarizing pulses used to measure membrane conductances appeared not to disturb major ioni

82 conductance of the channels and the overall membrane conductance are directly related to the overall

83 bilayer, characteristic spikelike changes in membrane conductance are observed.

84 Insulin also caused a parallel increase in membrane conductance as measured by whole-cell patch cla

85 stitution and channel blockers to reduce the membrane conductance as much as possible.

86 confirms activity-dependent co-regulation of membrane conductances as a mechanism underlying homeosta

87 ls (3 of 77) demonstrated clear increases in membrane conductance, associated with the activation of

88 HCN1 channels dominate the membrane conductance at rest, are not required for theta

89 hyperpolarization associated with decreased membrane conductance attributable to blockade of an inwa

90 Membrane conductance before electroporation was measurab

91 Membrane conductance between -50 and -60 mV was signific

92 nt clamp, GIRK activation increased the cell membrane conductance by 1- to 2-fold, hyperpolarized the

93 rial depolarization preceded changes in cell membrane conductance by 3-4 s.

94 wo other articles report magnetic control of membrane conductance by attaching ferritin to an ion cha

95 02 models that had varying densities of nine membrane conductances centered on a hand-tuned model tha

96 r metabotropic receptor-mediated currents or membrane conductance changes.

97 tial at -93 mV associated with a decrease of membrane conductance, closely resembling the effect of 1

98 ring and are distinguished by a lower "leak" membrane conductance compared with adjacent nonbursting

99 When inhibition was strong, the membrane conductance could be doubled or tripled.

100 Membrane conductance decreased by approximately 50% when

101 rane depolarization that were accompanied by membrane conductance decreases.

102 Although E(m) and total membrane conductance did not differ between MCF-7 and MC

103 LCO and its subdivisions, alveolar-capillary membrane conductance (DM) and pulmonary capillary blood

104 Functionally, the IPSC could increase membrane conductance during the decay of binaural glutam

105 A consistent reduction in membrane conductance during the IDAP was observed in all

106 How active membrane conductance dynamics tunes neurons for specific

107 pyramid-like arrangement enabled sampling of membrane conductance every 30 ms.

108 and diverse, and that the range of intrinsic membrane conductances expressed endow AD-SPN with the ab

109 teady-state inward current and instantaneous membrane conductance (fast current).

110 noceptors depolarize VMN neurons by reducing membrane conductance for K(+).

111 O-(3-thiotriphosphate) (GTPgammaS) increased membrane conductance from 10 to 260 picosiemens/picofara

112 Anoxia decreased resting membrane conductance from 322 to 131 pS.

113 ent, voltage-gated Na(+) conductance to leak membrane conductance (g(Na,P)/g(leak)) compared with adj

114 In contrast, the total membrane conductance (G(tot)) was well constrained, and

115 d ionic current (I(BK)) with two distinctive membrane conductances (g(m)).

116 disulfonic acid (DNDS)-sensitive basolateral membrane conductance (GDS) of cells expressing pNBC1, bu

117 n potentials causes changes in fibre resting membrane conductance (Gm) that reflect regulation of ClC

118 technique that permits measurement of plasma membrane conductance (Gm), membrane potential (Vm) and j

119 evidence that these differences are due to a membrane conductance gradient mediated by HCN and leak p

120 The curly bipolar cells had a higher membrane conductance, holding current and hyperpolarizat

121 nhancement of the widely expressed intrinsic membrane conductance Ih converts the potentiated synapti

122 amyloidogenic proteins and peptides increase membrane conductance in a conformation-specific fashion

123 unknown because of the inability to measure membrane conductance in human sperm.

124 de channel ClC-1 is the major contributor of membrane conductance in skeletal muscle and has been ass

125 om, 24-30 h), a synthetic glucocorticoid, on membrane conductance in the human airway epithelial cell

126 onicity (25 %) failed to evoke any change in membrane conductance in the majority of defolliculated o

127 In most cells the resting membrane conductances, including the H conductances, wer

128 ntegrity marker dye YO-PRO-1 (YP) and by the membrane conductance increase measured by patch clamp.

129 ), the membrane potential hyperpolarized and membrane conductance increased.

130 uced membrane depolarizations accompanied by membrane conductance increases.

131 Rather, membrane conductance induced by sPB1-F2 fluctuated and v

132 nt-voltage (I-V) relationship (passive) K(+) membrane conductance is a hallmark of mature hippocampal

133 Membrane conductance is also increased during shunting i

134 Impaired alveolar-capillary membrane conductance is the major cause for the reductio

135 short-term plasticity, how is the balance of membrane conductances maintained over long-term timescal

136 l produced pronounced suppression of resting membrane conductance measured with whole-cell recording

137 Membrane conductance (measured 200 microseconds after th

138 PC1 and TRPC4 are essential for an intrinsic membrane conductance mediating the plateau potential in

139 hough immature AIIs lacked the complement of membrane conductances necessary to generate bursting, ph

140 The increase in membrane conductance occurs without any evidence of disc

141 This yielded a lower limit for the membrane conductance of 158 pS.

142 The membrane conductance of a pyramidal neuron in vivo is su

143 be excluded that the changes observed in the membrane conductance of cortical astrocytes disturb the

144 The membrane conductance of every T. arvense leaf cell was d

145 In the face of the high membrane conductance of octopus cells, sodium and calciu

146 smembrane chloride movements via the lateral membrane conductance of the cell, GmetL, could serve to

147 However, factors other than active membrane conductances of AD-SPN must ultimately regulate

148 ng membrane potentials and voltage-activated membrane conductances of type B cells, but not type A ce

149 The high power dependence (up to 10) of the membrane conductance on the avicin concentration indicat

150 , each of which confers a distinctive plasma membrane conductance on transfected 293 cells.

151 We observed no activation of membrane conductance or Cx43-mediated dye uptake in astr

152 ole, cocaine or amphetamine were observed on membrane conductance or holding current (at holding pote

153 Increased membrane conductance, or shunting, does not simply reduc

154 EFO caused a 1.5-2 times greater increase in membrane conductance (p<0.05) than bipolar NEFO, along w

155 shown that a small and transient increase of membrane conductance parallels NP crossing of plasma mem

156 nous expression of GRA17 or GRA23 alters the membrane conductance properties of Xenopus oocytes in a

157 ultiple factors such as firing frequency and membrane conductance, raising doubts about their effecti

158 We find that increasing membrane conductance reduces the gain of the steady-stat

159 hat the active form of cystic fibrosis trans-membrane conductance regulator (CFTR) Cl(-) channel is a

160 6 with a cysteine in position 35 exhibited a membrane conductance sensitive to the thiol reagent male

161 on, by way of effects of GABAergic events on membrane conductance ('shunting' inhibition) and membran

162 membrane voltage in resting neurons with low membrane conductances than in active neurons with high c

163 t one mechanism for mediating the changes in membrane conductance that are essential for the cellular

164 Removal of external Ca2+ induced a membrane conductance that differed from MG channels in i

165 ivation of a rather large increase of apical membrane conductance that preceded significant activatio

166 arge non-selective (PK /PCl approximately 1) membrane conductance that was not blocked by 100 microM

167 Changes of membrane conductance through synaptic input or spiking a

168 73 mV through a relatively small increase in membrane conductance to Cl(-).

169 eceptors, relying on metabolically expensive membrane conductances to boost performance.

170 oupling synergically interacts with specific membrane conductances to promote synchronization of thes

171 Oxygenated normal red blood cells had a low membrane conductance, unaffected by deoxygenation.

172 cells as a model system, we investigated the membrane conductance underlying these oscillations.

173 ndentation failed to activate an increase in membrane conductance up to the point of causing visible

174 Here we show that, under increased membrane conductance, voltage fluctuations restore Na(+)

175 Fiber cell membrane conductance was a factor of 2.7 times larger in

176 ed to current in a nearly linear manner, and membrane conductance was found to be increased in the Up

177 ar application of MBB to intact oocytes, the membrane conductance was unaffected.

178 w IPSPs were reduced without any increase of membrane conductance, we conclude that 5-HT has in addit

179 The effects of VIP on membrane conductance were abolished by the hyperpolariza

180 Iacpd and the 1S,3R-ACPD-induced decrease of membrane conductance were diminished.

181 Gap junctional conductance (G(j)), and membrane conductance were evaluated by using frequency d

182 effects of insulin on membrane turnover and membrane conductance were inhibited by blockers of phosp

183 y at pH 6.5, only occasional fluctuations of membrane conductance were observed at pH 7.5.

184 Ca2+-signalling pathways and the associated membrane conductances were distinguished kinetically and

185 Membrane conductances were measured by frequency domain

186 by GABA or bicuculline; however, the resting membrane conductances were reduced by picrotoxin, zinc,

187 embrane potentials associated with increased membrane conductance when pulse widths are microseconds

188 ction of an inward current and a decrease in membrane conductance, whereas activation of group-II or

189 l produced a decrease in outward current and membrane conductance, whereas NaCl, KCl, NH(4)Cl, and HC

190 ses, such as modulation of voltage-dependent membrane conductances, which are expressed as changes in

191 altered by 25-fold by either manipulation of membrane conductance with optogenetic methods or generat

192 increased in the Up state, attributable to a membrane conductance with the same reversal potential as