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1 s to clarify the possible role of tension in membrane flow.
2 hanism to regulate the rate of intracellular membrane flow.
3 ess that has been proposed to occur via bulk membrane flow.
4 pathways in eukaryotic cells and coordinate membrane flow and cargo transport through the Golgi stac
6 er, in contrast to default recycling by bulk membrane flow, and is distinguishable in several ways fr
9 heir participation in opposite directions of membrane flow between the endoplasmic reticulum and Golg
10 ain membrane proteins does not occur by bulk membrane flow but is instead mediated by a specific endo
11 We conclude that bafilomycin A1 slows bulk membrane flow, but it causes additional inhibition of re
12 h upon CRISPR knockout led to reduced plasma membrane flow directionality despite increased actin flo
13 alysis explains the phenomenon in terms of a membrane flow driven by liberated reaction energy, leadi
15 direct effects, such as either by regulating membrane flow from other compartments or by modulating P
16 e or a transient manifestation of continuous membrane flow from specialized ER exit domains (ERES).
17 ow-temperature-sensitive step that regulates membrane flow from VTCs to the Golgi complex and back to
18 ind that, to a marked degree, the pattern of membrane flow in the cell is encoded and recapitulated b
19 of VHA-c1 and VHA-c3 in tissues with active membrane flow, including root cap, vascular strands, and
21 t, despite intense efforts to understand how membrane flow relates to Golgi form and function, this o
22 ipated by three viscous mechanisms including membrane flow, slip between the two monolayers that form
23 sorptive phenomena in which a tension-driven membrane flow supplements diffusive transfer of Golgi me
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