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1 cause of both uphill transport and increased membrane resistance.
2 erning signal dissipation through changes in membrane resistance.
3 d instead likely mediated by lowering of the membrane resistance.
4 s in spontaneous IPSCs (sIPSCs), sEPSCs, and membrane resistance.
5 t decrease in spontaneous AP firing rate and membrane resistance.
6 microM), with a concomitant net decrease in membrane resistance.
7 iring rate but as a secondary action reduces membrane resistance.
8 eus resulted in depolarization and increased membrane resistance.
9 ns by inducing depolarization and increasing membrane resistance.
10 rization and hence a decrease in basolateral membrane resistance.
11 llular resistivity, membrane capacitance, or membrane resistance.
12 eover, it did not produce sizable changes in membrane resistance.
13 cells a hyperpolarization and a reduction of membrane resistance.
14 able phase of the action potential with high membrane resistance.
15 le for these cannabinoid-produced changes in membrane resistances.
16 urons and a relative decrease in the somatic membrane resistance (0.7-8.1 M omega) was detected durin
17 sport resistance for P(m)>1 cm s(-1) whereas membrane resistance accounts for 50-75% of total transpo
18 e appears to depend upon both an increase in membrane resistance and a decrease in total cell surface
20 ride was associated with a decrease in ileal membrane resistance and an increase in inducible nitric
21 ne excitability, as indexed by a decrease in membrane resistance and an increase in the stimulus thre
22 ane region includes local models for passive membrane resistance and capacitance, nonlinear active so
26 filaments have to be bundled to overcome the membrane resistance and that the filopodial length is li
28 ere also associated with a small increase in membrane resistance, and in voltage-clamp recordings ore
29 s of membrane potential, severe reduction of membrane resistance, and influx of Na+, Ca2+, Cl- and wa
30 ntial (RMP), spontaneous AP firing rate, and membrane resistance are cyclically regulated as a functi
31 llate cells, a voltage-dependent increase in membrane resistance at sub-threshold voltages mediated b
32 ance of the nerve cell and by increasing its membrane resistance, but little is known about the latte
33 eurons (7.8 +/- 0.6 mV; n = 16), reduced the membrane resistance by 33 +/- 3%, and could convert the
34 rectification and steady-state components of membrane resistance by 37 and 38 %, respectively, in 66
35 dels was accompanied with decreased specific membrane resistance by approximately 25% and efficacy of
38 hannels in S-D muscle produced high specific membrane resistance, comparable to similarly treated con
39 olarized resting potentials and an increased membrane resistance compared with age-matched control ce
40 to L-glutamate without significant change in membrane resistance, consistent with the well-establishe
43 onstrates for the first time that increasing membrane resistance decreases the efficiency of this res
46 ee Hb due to transport resistances including membrane resistance, extra- and intra-cellular resistanc
47 a passive current-voltage relationship, low membrane resistance, high capacitance, and dye-coupling
48 e contrast is due to the decrease in tip and membrane resistance, in the vicinity of the pore opening
51 er of G-protein trimers required to overcome membrane resistance is 3 to 5, within a contact zone bet
52 1) for 10-45% Hct, respectively, below which membrane resistance is more significant and above which
53 d depend on the number of bundled filaments, membrane resistance, lamellipodial protrusion rate, and
60 P>0.05) in T-type current kinetics or in the membrane resistance of the thalamic cells between the tw
61 ing voltage clamp, we found that the passive membrane resistance of VS cells was reduced during fligh
62 ough strophanthidin did not alter either the membrane resistance or the Na(+) reversal potential, the
63 ated inhibitory postsynaptic potentials, the membrane resistance, or the holding current, whereas it
64 se effects on resting membrane potential and membrane resistance persisted in the presence of TTX.
65 n ORNs (4.4 +/- 0.4 pF) and a lower apparent membrane resistance (R(m)) (160 +/- 11 MOmega versus 664
69 in fibres of young HSA(LR) mice, the resting membrane resistance (Rm) at -90 mV is only slightly larg
70 resting membrane potential (Vm), IK,ADO, and membrane resistance (Rm) in rabbit isolated AV nodal myo
71 e inexcitable despite resting potentials and membrane resistances similar to those of control denerva
72 ices taken from MeA-trained birds had higher membrane resistances than did cells from water-trained c
73 ime constant is attributed to an increase in membrane resistance; the increase in input resistance ap
78 n = 18) and PNs (-48 +/- 1 mV, n = 23) while membrane resistance was significantly higher in TNs.
80 Image contrast also becomes independent of membrane resistance when an electrical shunt is used, al
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