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1 Only 28% of augmented sinuses did not show membrane thickening.
2 itochondrial proliferation and mitochondrial membrane thickening.
3 te loss, acellular capillaries, and basement membrane thickening.
4 vely correlated with subepithelial reticular membrane thickening.
5 aired lung expansion, and alveolar-capillary membrane thickening.
6 over are considered to contribute to Bruch's membrane thickening.
7 to 10.1%, P < .001), subepithelial basement membrane thickening (4.4 mum [25th-75th IQR, 4.0-4.7 mum
9 ial matrix accumulation, glomerular basement membrane thickening, albuminuria, and podocyte dropout.
10 derate or severe diffuse glomerular basement membrane thickening and 2/14 had nodular glomerulosclero
12 associated with a higher degree of basement membrane thickening and edematous changes within the ves
13 tor involved in the pathogenesis of basement membrane thickening and extracellular matrix accumulatio
15 tron microscopy revealed glomerular basement membrane thickening and podocyte effacement in eNOS(-/-)
16 1 h after CAR injection attenuated pulmonary membrane thickening and polymorphonuclear leukocyte infi
17 ntin expression correlated with the basement membrane thickening and with CT score, as well as with t
18 drusen, and basal laminar deposits, Bruch's membrane thickening, and choroidal neovascularization.
20 angial matrix expansion, glomerular basement membrane thickening, and podocyte loss, whereas Pod-ETRK
21 ar and linear-like deposit material, Bruch's membrane thickening, and progressive RPE and choroidal a
23 angial matrix expansion, glomerular basement membrane thickening, and renal insufficiency that closel
24 mesangial expansion, and glomerular basement membrane thickening as determined by electron microscopy
25 ation, goblet cell hyperplasia, and basement membrane thickening compared to A. fumigatus-sensitized
26 inases exacerbated strial capillary basement membrane thickening, demonstrating that alterations in b
28 involving increased cell death and basement membrane thickening in the proximal convoluted tubules.
29 bleomycin administration, including basement membrane thickening, interstitial fibrin deposition and
30 including podocyte loss, glomerular basement membrane thickening, mesangial expansion, and proteinuri
31 ion, glomerulosclerosis, glomerular basement membrane thickening, mitochondrial DNA deletions, reduct
32 tion, eosinophilic infiltration and basement membrane thickening of the airways, both characteristic
33 lerosis, cast formation, glomerular basement membrane thickening, podocyte effacement, and albuminuri
34 ced mesangial expansion, glomerular basement membrane thickening, podocyte foot-process fusion, and t
35 al matrix expansion, mesangiolysis, basement membrane thickening, podocyte loss) and functional (prot
36 glomerular deposits, no glomerular basement membrane thickening, reduced levels of circulating dsDNA
38 consistent with early AMD, including Bruchs membrane thickening, retinal pigment epithelium cell los
39 shedding, goblet cell hyperplasia, basement membrane thickening, subepithelial fibrosis, airway smoo
40 view, we describe changes involving basement membrane thickening, tissue remodeling, gap junctions, i
41 tic type characterized by capillary basement membrane thickening was not demonstrated in any of the h
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