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1 te that modulation by rmGluR2 and rmGluR3 is membrane delimited.
2 bimolecular FRET pairs that are not entirely membrane-delimited.
3                   However, the nature of the membrane-delimited ABA signal transduction steps remains
4                                              Membrane-delimited abscisic acid (ABA) signal transducti
5                              PKCdelta exerts membrane-delimited actions in cells activated by agonist
6 ltant structural picture is compatible with 'membrane delimited' activation of GIRK channels by G pro
7 hat the pathway leading to inhibition is not membrane delimited and that inhibited CFTR channels rema
8                        Both regulations were membrane-delimited and G protein-dependent, requiring bo
9                      One pathway is fast and membrane-delimited and inhibits N- and P/Q-type channels
10                                          The membrane-delimited and voltage-dependent inhibition of N
11 gma receptor-mediated signal transduction is membrane delimited, and requires neither G-protein activ
12 H/CG R activation leads to the activation of membrane-delimited ARF6.
13 ARF6-mediated mechanism to release a pool of membrane-delimited arrestin to bind GPCRs may be a wides
14 ates GIRK current inhibition was found to be membrane-delimited because bath application of ACh did n
15 t that endogenous RGS proteins contribute to membrane-delimited Ca(2+) channel modulation by regulati
16 tochastically from a single bacterium into a membrane-delimited, capsule-embedded cluster of progeny
17                                          How membrane-delimited channels penetrate cell walls is unkn
18 loop of the LH/CG receptor via activation of membrane delimited endogenous ARNO.
19 ing that some cellular effects can occur via membrane delimited events.
20 ing that some cellular effects can occur via membrane delimited events.
21 le-stranded DNA genome occurs in cytoplasmic membrane-delimited factories.
22 n of Gbetagamma-activated GIRK currents in a membrane-delimited fashion.
23 atodendritic regions of MNCs, possibly via a membrane-delimited G-protein-dependent pathway.
24  on pre-existing OR activity and mediated by membrane-delimited Gbetagamma subunits in a voltage-inde
25 g sites for Gbetagamma proteins that mediate membrane-delimited GIRK activation.
26 ignal-accepting network of 10 amino acids in membrane-delimited HAMP proteins.
27 tner that functions downstream of the plasma membrane-delimited heterotrimeric G-protein (GPA1) in a
28 E release at terminals closely parallels the membrane-delimited inhibition of N-type Ca2+ currents in
29 example, by the important G-protein-coupled, membrane-delimited inhibitory pathway.
30                  These results demonstrate a membrane-delimited interaction between 1/2 A-type protei
31                        Our data suggest that membrane-delimited interaction between MG53 and PTRF con
32                  Furthermore, AITC acts in a membrane-delimited manner and induces a shift of the vol
33 downmodulates the NMDA receptor channel in a membrane-delimited manner, mediated by G proteins, but n
34 ists eliminate I(AHV) fast inactivation in a membrane-delimited manner, suggesting a Kv3.4 channel si
35 sting that BAA can activate the channel in a membrane-delimited manner.
36 el was activated by mu opioid receptors in a membrane-delimited manner.
37 e alternative cation permeation pathway in a membrane-delimited manner.
38 o protein kinase blockade; consistent with a membrane delimited mechanism.
39 se results define a previously unrecognized, membrane-delimited mechanism for EGFR transactivation vi
40 ta2-adrenergic effects are consistent with a membrane-delimited mechanism involving Galphas.
41  its activation kinetics through an internal membrane-delimited mechanism.
42 ositol-4,5-bisphosphate (PIP2) is a possible membrane-delimited messenger that activates cardiac sodi
43                                    No direct membrane-delimited pathway for Ca2+ channel regulation b
44 date have focused primarily on the canonical membrane-delimited pathway for PKD1 activation by G prot
45 transmitters acting through G proteins via a membrane-delimited pathway independently of soluble intr
46 ing through G protein-coupled receptors in a membrane-delimited pathway involving Gbetagamma subunits
47 esults suggest that G proteins may act via a membrane-delimited pathway to regulate calcium channels
48 educes N- and P-type Ca2+ currents through a membrane-delimited pathway using a Gi/o-class G-protein.
49 se in Ca(2+)sensitivity of the channel via a membrane-delimited pathway.
50 ng through G protein-coupled receptors via a membrane-delimited pathway.
51 urrents, consistent with the activation of a membrane-delimited pathway.
52        In conclusion, hIK1 is activated by a membrane delimited PKA when endogenously expressed.
53  GTP uptake to GAP-catalyzed hydrolysis is a membrane-delimited process, and exchange of G alpha(t) b
54  heterologous systems and shown that it is a membrane-delimited process.
55 pothesis that transactivation can occur by a membrane-delimited process: direct increase in the activ
56 select G protein-coupled receptors and other membrane-delimited proteins.
57 nsitive, gateable, and reversible sensor for membrane-delimited reactive species.
58 lar proteins other than G proteins, creating membrane-delimited signal transduction complexes similar
59         Thus, LH/CGR appears to activate two membrane delimited signaling cascades via two types of G
60 rotein-coupled estrogen receptor to initiate membrane delimited signaling, which enhances kinase sign
61 2+) nanodomains because it participates in a membrane-delimited signaling complex that forms after st
62                       Our data indicate that membrane-delimited signaling involving cross-talk betwee
63                              Therefore, this membrane-delimited signaling pathway plays a critical ro
64 of G-protein-coupled receptors that generate membrane-delimited signals, yet these signals have not b
65                                       Plasma membrane-delimited steps in the GPCR-mediated activation
66 diomyocytes, suggesting the development of a membrane-delimited stimulatory pathway mediated through
67                       Peroxisomes are single membrane-delimited subcellular organelles that carry out
68 nstrate for both opsins the repetitive fast, membrane-delimited, ultra light-sensitive, and wavelengt

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