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1 te that modulation by rmGluR2 and rmGluR3 is membrane delimited.
2 bimolecular FRET pairs that are not entirely membrane-delimited.
6 ltant structural picture is compatible with 'membrane delimited' activation of GIRK channels by G pro
7 hat the pathway leading to inhibition is not membrane delimited and that inhibited CFTR channels rema
11 gma receptor-mediated signal transduction is membrane delimited, and requires neither G-protein activ
13 ARF6-mediated mechanism to release a pool of membrane-delimited arrestin to bind GPCRs may be a wides
14 ates GIRK current inhibition was found to be membrane-delimited because bath application of ACh did n
15 t that endogenous RGS proteins contribute to membrane-delimited Ca(2+) channel modulation by regulati
16 tochastically from a single bacterium into a membrane-delimited, capsule-embedded cluster of progeny
24 on pre-existing OR activity and mediated by membrane-delimited Gbetagamma subunits in a voltage-inde
27 tner that functions downstream of the plasma membrane-delimited heterotrimeric G-protein (GPA1) in a
28 E release at terminals closely parallels the membrane-delimited inhibition of N-type Ca2+ currents in
33 downmodulates the NMDA receptor channel in a membrane-delimited manner, mediated by G proteins, but n
34 ists eliminate I(AHV) fast inactivation in a membrane-delimited manner, suggesting a Kv3.4 channel si
39 se results define a previously unrecognized, membrane-delimited mechanism for EGFR transactivation vi
42 ositol-4,5-bisphosphate (PIP2) is a possible membrane-delimited messenger that activates cardiac sodi
44 date have focused primarily on the canonical membrane-delimited pathway for PKD1 activation by G prot
45 transmitters acting through G proteins via a membrane-delimited pathway independently of soluble intr
46 ing through G protein-coupled receptors in a membrane-delimited pathway involving Gbetagamma subunits
47 esults suggest that G proteins may act via a membrane-delimited pathway to regulate calcium channels
48 educes N- and P-type Ca2+ currents through a membrane-delimited pathway using a Gi/o-class G-protein.
53 GTP uptake to GAP-catalyzed hydrolysis is a membrane-delimited process, and exchange of G alpha(t) b
55 pothesis that transactivation can occur by a membrane-delimited process: direct increase in the activ
58 lar proteins other than G proteins, creating membrane-delimited signal transduction complexes similar
60 rotein-coupled estrogen receptor to initiate membrane delimited signaling, which enhances kinase sign
61 2+) nanodomains because it participates in a membrane-delimited signaling complex that forms after st
64 of G-protein-coupled receptors that generate membrane-delimited signals, yet these signals have not b
66 diomyocytes, suggesting the development of a membrane-delimited stimulatory pathway mediated through
68 nstrate for both opsins the repetitive fast, membrane-delimited, ultra light-sensitive, and wavelengt
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