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1 nemia and severe cryoglobulinemia-associated membranoproliferative glomerulonephritis.
2 recurrent bacterial infections and to type 1 membranoproliferative glomerulonephritis.
3 tis, IgA nephropathy, Alport's syndrome, and membranoproliferative glomerulonephritis.
4 ed with diseases such as IgA nephropathy and membranoproliferative glomerulonephritis.
5 Two patients developed membranoproliferative glomerulonephritis.
6 athological diagnoses on renal biopsies were membranoproliferative glomerulonephritis (23%) followed
7 as been associated with de novo or recurrent membranoproliferative glomerulonephritis and acute trans
8 d-stage renal disease in 12; one patient had membranoproliferative glomerulonephritis and another pat
9 een reported to be typically associated with membranoproliferative glomerulonephritis and less freque
11 nty-one patients with biopsy-proven, diffuse membranoproliferative glomerulonephritis and significant
12 al was seen with recurrent and de novo FSGS, membranoproliferative glomerulonephritis, and HUS/TTP.
13 hey developed splenomegaly, lymphadenopathy, membranoproliferative glomerulonephritis, and lymphocyti
14 nephritides (e.g. Alport's Syndrome, type II membranoproliferative glomerulonephritis, and membranous
15 vere kidney disorders including mesangial or membranoproliferative glomerulonephritis, and/or Henoch
16 gy, whereas overexpression of TSLP induced a membranoproliferative glomerulonephritis, as previously
17 ciated with hemolytic uremic syndrome (HUS), membranoproliferative glomerulonephritis (dense deposit
18 By 6 months of age, ABCA1-/- animals develop membranoproliferative glomerulonephritis due to depositi
19 (FSGS), 57; immunoglobulin A nephritis, 22; membranoproliferative glomerulonephritis (GN), 18; and m
20 enal (membranous glomerulonephritis [GN] and membranoproliferative glomerulonephritis); hematologic (
21 ulting from other glomerular diseases (FSGS, membranoproliferative glomerulonephritis, IgA nephropath
22 malities in factor H have been implicated in membranoproliferative glomerulonephritis in both humans
23 ch was before the development of spontaneous membranoproliferative glomerulonephritis in some Cfh(-/-
24 in a mouse model of cryoglobulin-associated membranoproliferative glomerulonephritis induced by over
25 riably demonstrating severe neonatal anemia, membranoproliferative glomerulonephritis, liver fibrosis
30 opathies, such as IgA nephropathy (IgAN) and membranoproliferative glomerulonephritis (MPGN) remains
32 , a model of cryoglobulinemia and associated membranoproliferative glomerulonephritis (MPGN), in whic
35 tic nephropathy, membranous lupus nephritis, membranoproliferative glomerulonephritis, postinfectious
37 3 glomerulopathies/immune complex-associated membranoproliferative glomerulonephritis, serum-induced
38 ients had immune-complex glomerulonephritis: membranoproliferative glomerulonephritis type 1 (n = 12)
39 The predominant renal biopsy findings were membranoproliferative glomerulonephritis type 1 or type
40 ceased donor transplants were not present in membranoproliferative glomerulonephritis type I (adjuste
42 reviously published studies of patients with membranoproliferative glomerulonephritis type I are smal
43 allograft for end-stage renal disease due to membranoproliferative glomerulonephritis type I is prese
46 ypical hemolytic uremic syndrome (aHUS), and membranoproliferative glomerulonephritis type II (MPGN2)
47 Dense deposit disease (DDD; also known as membranoproliferative glomerulonephritis type II) is a p
50 plant FSGS was 2.25 (1.6-3.1), P<0.0001, for membranoproliferative glomerulonephritis was 2.37 (1.3-4
51 Renal involvement usually manifests as a membranoproliferative glomerulonephritis with marked mon
52 on with hepatitis C virus (HCV) infection is membranoproliferative glomerulonephritis, with or withou
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