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1 embranoproliferative glomerulonephritis, and membranous glomerulonephritis).
2 ultrastructural, and clinical model of human membranous glomerulonephritis.
3 CD5Rg in a murine model of antibody-mediated membranous glomerulonephritis.
4 hich reproduces features of human idiopathic membranous glomerulonephritis.
6 tis, focal proliferative glomerulonephritis, membranous glomerulonephritis, and mesangial glomerulone
7 tanding the pathogenesis of human idiopathic membranous glomerulonephritis, and proposes future direc
8 sions that involved >or=50% of glomeruli +/- membranous glomerulonephritis (class III >or=50%+/-V) we
9 diffuse proliferative glomerulonephritis +/- membranous glomerulonephritis (class IV+/-V) was less co
10 overexpressed in human kidney epithelium in membranous glomerulonephritis, diabetes mellitus, IgA ne
11 [PCT], vitiligo, and lichen planus); renal (membranous glomerulonephritis [GN] and membranoprolifera
14 sed the earliest manifestations of recurrent membranous glomerulonephritis (MGN) in renal allografts.
16 nts with focal-segmental glomerulosclerosis, membranous glomerulonephritis, minimal change disease, I
17 = 3; psoriasis, n = 2; Graves disease, n 1; membranous glomerulonephritis, n = 2; rheumatoid arthrit
19 diagnosed as having transverse myelitis and membranous glomerulonephritis secondary to systemic lupu
20 us pseudotumor, sclerosing mesenteritis, and membranous glomerulonephritis should all be added to the
21 in mind, particularly in older patients with membranous glomerulonephritis where the possibility of m
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