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1 8 also exhibit reduced hippocampus-dependent memory.
2 ross distant brain regions subserves working memory.
3 ngruence has a strong influence on long-term memory.
4 are known to play a key role in learning and memory.
5 PV+ cells that are required for learning and memory.
6 nderlie this form of translation-independent memory.
7 ile a quantum simulator requires only finite memory.
8  Imitation after delays implicates preverbal memory.
9 r reward, motivation, emotion, learning, and memory.
10 in novelty related modulation of hippocampal memory.
11                ADCs also induced immunologic memory.
12 rve top-down attentional control and working memory.
13 roteins in the brain central to learning and memory.
14  current from future search goals in working memory.
15 o provide the cellular basis of learning and memory.
16 erm potentiation, resulting in protection of memory.
17 ding attention, working memory, and episodic memory.
18 ibitor specifically and completely disrupted memory.
19  cell composition, and impaired learning and memory.
20  is part of a brain-wide network for working memory.
21 vely studied cellular model for learning and memory.
22 neurons to reconsolidate the original reward memory.
23  been demonstrated to play a crucial role in memory.
24 the representation of information in working memory.
25 pisode; and (iv) patients with gaps in their memories.
26 ng could be of interest for magnetoresistive memories.
27 eralized memories without affecting adaptive memories.
28 ted eFSE rats to encode and retrieve spatial memories.
29  participate in the formation and storage of memories about events in the environment that predict th
30 s, S1 is critically involved in updating the memory about the perturbation that is essential for fore
31 n is better suited to support integration of memories across experiences during consolidation.SIGNIFI
32 ma4 cells function in adaptive immunological memory against B. pertussis.
33 n part, by endowing RL systems with episodic memory, allowing them to (a) efficiently approximate val
34                              ABSTARCT: Shape memory alloys (SMAs) have the ability to show large reco
35                            This same optimum memory also maximizes geometric mean fitness, in steady
36 ysical platform for the realization of smart memories and machine learning and for operation of the c
37  (n = 23) with a control group (n = 24) in a memory and a visual perceptual task.
38 iversity of California, San Francisco (UCSF) Memory and Aging Center (collected between 1999-2015) an
39 on to include object information relevant to memory and behavior.
40 res the different contributions of different memory and decision-making systems thought to contribute
41 ency, inability to generate long-term immune memory and decreased activities against tumour-cell subp
42 soluble factors BAFF, IL-2, and IL-21 induce memory and DN B cell activation and differentiation has
43 ince the DLPFC and the ACC are implicated in memory and emotional regulation, and the ACC has motor a
44 r than expected effect of ageing on episodic memory and motor function with advanced stages of HIV in
45   In both models an abnormal distribution of memory and naive CD4 T cells occurred, and peripheral CD
46 in enhanced cognitive performance in working memory and object recognition paradigms at baseline and
47 h placebo is associated with improved verbal memory and other cognitive functions in older men with l
48 tio and n-3 predicted performance on working memory and planning tasks in children 7-12 y old.
49 tive symptoms in CD, and with poorer working memory and probabilistic category learning performance i
50 /-) mice exhibited impairments in contextual memory and spent less time than did controls interacting
51 e as a potent and selective means to enhance memory and synaptic plasticity.
52 d inversely with midlife visual and episodic memory and visuospatial associative learning (-0.140 sta
53 itive function, including attention, working memory, and episodic memory.
54 on, verbal speed, processing speed, auditory memory, and fine motor dexterity.
55 icity is the cellular basis for learning and memory, and it is crucial for the refinement of neuronal
56     The hippocampus is critical for episodic memory, and synaptic changes induced by long-term potent
57  exposures and mood, emotion, cognition, and memory; animal studies to determine epigenetic changes t
58 petitive and aversive consequences, parallel memories are established in a way that appetitive and av
59  suggest that the contents of human episodic memory are often constructed in the service of the expli
60 the MTL has a specific role in topographical memory as assessed in tasks of scene memory where the vi
61  subjective memory ("How would you rate your memory at the present time?").
62 ed rare affinity-matured human NANP-reactive memory B cell antibodies elicited by natural Pf exposure
63 ic infections with regard to dynamics of the memory B cell subsets point to their role in the pathoge
64 similar B-blasts can differentiate to become memory B cells (MBC), in which EBV persistence is establ
65 01), but higher proportions of IgM+CD21-/low memory B cells (P < .05), CD4+IFNgamma+ cells (P < .01),
66 ed by loss of naive B cells, loss of resting memory B cells due to their redistribution to the gut, i
67                                  Analysis of memory B cells from the immunized macaque suggests that
68 ymphomas, was specifically seen among IgM(+) memory B cells of the patients.
69 mmune systems of infected hosts to recall of memory B cells that recognized the lateral patch, the pr
70  revealed that frequencies of class-switched memory B cells were increased in the patients, whereas f
71 ctivated B cells and circulating tissue-like memory B cells, and expansion of the B regulatory cells
72 ll dysfunction (defined by loss of total and memory B cells, increased B regulatory cell [Breg] count
73 matic hypermutation, and differentiated into memory B cells.
74 i.e., activated memory cells and tissue-like memory B cells.
75                    These findings indicate a memory-based "autopilot role" for the default mode netwo
76 s now required for extinction of the updated memory but the retrosplenial cortex is no longer require
77                          This system, termed memory by engineered mutagenesis with optical in situ re
78                     Alternatively, retrieved memory can be maintained, following a period of reconsol
79               The number of items in working memory can be regulated by external excitation, enabling
80                                              Memory can inform goal-directed behavior by linking curr
81 ulation of correlated learning and trainable memory capability with strong tolerances to input faults
82                                Activated and memory CD4 T cells, macrophages, and dendritic cell (DC)
83                   Here we show blood central memory CD4 T-cell responses specific to Mtb dormancy rel
84 te antiretroviral therapy, HIV-1 persists in memory CD4(+) T cells, creating a barrier to cure.
85     The major activated reservoir cells were memory CD4+ T cells in vivo.
86                                        These memory CD8 T cells remain poised to rapidly elaborate ef
87  cytomegalovirus (CMV), and compared to bulk memory CD8 T cells.
88 okine-driven subset interconversion of human memory CD8 T cells.
89 bers and activity of H1N1- and H3N2-specific memory CD8(+) T cells, including tissue-resident cells,
90                                     Multiple memory CD8(+) T-cell subsets with distinct functional an
91 , and reduced naive and effector and central memory CD8+ T cells.
92 ly early (1 d) after activation of naive and memory cells and that demethylation is the predominant c
93 ed," virus-specific B cells, i.e., activated memory cells and tissue-like memory B cells.
94 s to adapt and differentiate into long-lived memory cells has added further complexity to this field.
95 ntum memory with 225 individually accessible memory cells in a macroscopic atomic ensemble.
96 n-free mice revealed that differentiation to memory cells was coupled to erasure of de novo methylati
97 ted that these cells were neither plasma nor memory cells.
98 art of their routine work-up at the Imperial Memory Centre, a tertiary referral clinic in the UK Nati
99 l progression in a sample of 393 nondemented memory clinic patients.
100 put elements are transformed into meaningful memory codes relies on the ability to integrate them wit
101           Verb and phonemic fluency, working memory, cognitive flexibility, immediate and delayed rec
102 deficit remained when accounting for working memory (Cohen d = 0.89; P = 2.21 x 10-17).
103 f mesoscopic particles-using a single shared memory commodity workstation.
104 tional response but led to reliably improved memory compared with control images when patients were g
105 iteria for AAMI based on baseline subjective memory complaints and objective memory performance.
106 sed risk, as did lower education, subjective memory concerns, poorer baseline cognitive performance,
107 rowth in galactose, GAL gene transcriptional memory confers a strong fitness benefit in Saccharomyces
108 mmobility and rest has been linked with both memory consolidation and navigational planning.
109 functions in brain development, learning and memory consolidation by selectively eliminating and main
110 w oscillations-is thought to be critical for memory consolidation during sleep, the role spindles pla
111 ngrams and circuits that support neocortical memory consolidation have thus far been unknown.
112       These results indicate that successful memory consolidation requires coherent hippocampal-neoco
113 C inhibition of PV+ interneurons blocks fear memory consolidation.
114 nt (NREM) sleep has been proposed to support memory consolidation.
115 otions and in procedural motor and emotional memory consolidation.
116 rmed stable attractors and was predictive of memory content.
117                                   The labile memory could then be weakened by an extinction protocol
118 in later life, contributing to cognitive and memory decline, is unknown.
119 elucidating the causative factors underlying memory defects in Alzheimer's patients.
120                    In schizophrenia, working memory deficit was mostly accounted for by processing sp
121 nts the loss of dendritic spines and rescues memory deficits after TBI.
122                                          The memory deficits we observed in mouse prion disease were
123 e (JNK) activation, which is associated with memory deficits.
124 implicated in the processing of fine-grained memory detail, supporting functional specialization of h
125  a concept for monolayer MoS2 optoelectronic memory devices using artificially-structured charge trap
126 ction CD8(+) T cell terminal effector versus memory differentiation are incompletely understood.
127 t to be important for balancing effector and memory differentiation; however, the epigenetic regulato
128 allows quantitative assessments of the shape-memory effect (SME) and can be utilized in any material
129     The discovery of the unexpected anomeric memory effect is further supported by IR-MS action spect
130    By combining superomniphobicity and shape memory effect, metamorphic superomniphobic (MorphS) surf
131 ed for all the phenotypic characteristics of memory-effector T cells such that with acute inactivatio
132 ng Dyson's original spin chain with the most memory-efficient classical algorithm known requires infi
133  gene-expression profiles in naive, effector memory (EM), and terminally differentiated EM (TEMRA) ce
134 in the mushroom body that controls long-term memory encoding.
135   One mechanism that may support prospective memory enhancements is the carry-over of emotional brain
136 was preserved across a night of sleep, while memory for both feature types declined over a day awake.
137 ally mapped infants' categorical recognition memory for hue onto a stimulus array used previously to
138                 We question whether implicit memory for language can and should be equated with lingu
139                             In Experiment 2, memory for shared properties improved across a nap, but
140                             In Experiment 1, memory for shared properties improved and memory for uni
141 1, memory for shared properties improved and memory for unique properties was preserved across a nigh
142 he hippocampus is the main locus of episodic memory formation and the neurons there encode the spatia
143 imentally induced theta rhythms and episodic memory formation in humans.
144  known to be powerful negative regulators of memory formation, but it is not clear whether this funct
145 m potentiation (LTP) are thought to underlie memory formation.
146  but not after, training is required for the memory formation.
147  of dopamine are essential for dlPFC working memory function, with many beneficial actions arising fr
148 direct influence of the respiratory cycle on memory function.
149 estyle has beneficial effects on hippocampal memory function.
150 e important for learning, but their specific memory functions remain unclear.
151  from apoptosis and expression of activation/memory genes during infection with the intracellular par
152  time and accuracy of eye-movements during a memory guided saccade task are related to fluctuations i
153 ake SWRs support accurate memory storage and memory-guided behavior, whereas sleep SWR reactivation i
154                                     Episodic memory has been analyzed in a number of different ways i
155  higher-order circuit supporting associative memory has not been previously available.
156 E STATEMENT Recent neurobiological models of memory have argued that large-scale neural oscillations
157 ation and reactivation, respectively, of the memory-holding cells (engram cells) by a common set of n
158 lculation as well as a measure of subjective memory ("How would you rate your memory at the present t
159 nt experimental findings on sleep-associated memory (i.e., neural activity patterns in sleep that ref
160 men with low testosterone and age-associated memory impairment (AAMI).
161                                  This threat memory impairment is also reflected in increased behavio
162 men with low testosterone and age-associated memory impairment, treatment with testosterone for 1 yea
163 eliorate the gliovascular damage and working memory impairments after hypoperfusion possibly via endo
164 tion, as well as contextual fear and spatial memory impairments.
165 habituation paradigms, assuming that babies' memories in laboratory contexts are best constructed aft
166 We studied the modularity in long-term motor memories in the context of locomotor adaptation using re
167 urrent study, we investigated visual working memory in a more dynamic setting, and assessed the follo
168 that this PDE4D inhibitor is able to enhance memory in AD transgenic mice and concomitantly rescues t
169  two primary approaches to studying episodic memory in an unparalleled manner.
170 sruptions in circadian timing impair spatial memory in humans and rodents.
171 e metarepresentational structure of episodic memory in terms of its role in communicative interaction
172 d, had a procognitive profile as it improved memory in the novel object recognition test but had no a
173  purely experience-based fear processing and memory in the right amygdala, thereby making a direct li
174  In addition, we find evidence of epigenetic memory in the transdifferentiated cells, with reminiscen
175 les promote the consolidation of motor skill memory in young adults.
176 es, but existing approaches are both CPU and memory-intensive, limiting their application to small, s
177 ice architecture for fast organic transistor memory is developed, based on a vertical organic transis
178 gs demonstrate that, while transgenerational memory is observed in one of six traits examined, they a
179             The formation and retrieval of a memory is thought to be accomplished by activation and r
180  and experimental tests, we found that these memory-like phenomena arise from a confluence of rapid,
181  We found that our patients with psychogenic memory loss fell into four distinct groups, which we cat
182 ol during an olfactory long-term associative memory (LTAM) in Caenorhabditis elegans hermaphrodites.
183 te counts peaked around 1 year, whereas most memory lymphocyte subsets more gradually increased durin
184  proteasome activity conferred attributes of memory lymphocytes.
185       To better understand the mechanisms of memory maintenance in CD8(+) T cells, we performed genom
186 at encoding operations were preserved, while memory maintenance processes were abnormal.
187                                  Their shape memory, material and magnetic properties (e.g. transform
188                                        Shape memory materials have the ability to recover their origi
189  experience are transiently reactivated from memory, movement- and immobility-associated activity pat
190 The aversive valence of these new extinction memories neutralizes previously learned odour preference
191 s neuropeptide in face perception and social memory, no prior research has tested the relationship be
192  neocortex and on the limitations of working memory, not on the MTL.
193  reactivation during sleep renders long-term memories of negative experiences more negative in SAD pa
194 ges the idea that ants use egocentric visual memories of the scene for guidance [1, 2, 6].
195  content (i.e., shares common elements), and memories of these events are organized in an intricate n
196         Faithful resetting of the epigenetic memory of a somatic cell to a pluripotent state during c
197 sed to interpersonal violence exhibited poor memory of contexts paired with angry faces and atypical
198 to the desired load and to clear the working memory of currently held items to make room for new ones
199 n Award Lecture is presented in honor of the memory of Edwin L.
200  food preference (STFP), mice form long-term memory of food odors presented by a social partner.
201               Our observations show that the memory of subtle differences in earlier deformation path
202  mice spontaneously form a spatially precise memory of the location of shelter, which is laid down qu
203 E-treated rats continued to display accurate memory of the shock-context association.
204                                    Intrusive memories often take the form of distressing images that
205 ith placebo was not associated with improved memory or other cognitive functions.
206                                 How are fear memories organized?
207 o others that proposed that with the loss of memory, people with dementia may also experience loss of
208 gence, executive function, processing speed, memory, perceptual reasoning, and verbal comprehension i
209 ability of this signature is associated with memory performance (P=0.0003) in 3,346 young and elderly
210 rom prefrontal cortex can disrupt short-term memory performance and is reminiscent of Alzheimer's dis
211 roups exhibited significantly better working memory performance in adulthood relative to sucrose SA c
212 iluzole, which was recently shown to improve memory performance in aged rats, prevented many of the h
213 ctations had a profound influence on working memory performance, leading to faster access times as we
214 SN/VTA), medial temporal lobe, or subsequent memory performance.
215 emory, whereas NR3C2 polymorphisms predicted memory performance.
216 he brain characteristics supporting superior memory performance.
217 e subjective memory complaints and objective memory performance.
218 s significantly accounted for variability in memory performance.
219 se found in the periphery, having a resident-memory phenotype (CD69(+)CD122(-)PD1(+)CD44(+)CD62L(-))
220 es, including incidence of chronic diseases, memory, physical functioning, and mental health, among p
221 nt antigen; yet, excessive expansion reduces memory potential and impairs antitumor immunity.
222 lization also tends to increase over time as memory precision for recent events gives way to more gis
223 ronounced in terminal effector cells than in memory precursor cells and was regulated by antigenic st
224 typic and epigenetic characterization of the memory-precursor effector subset of virus-specific CD8 T
225 n millions of atoms in a solid-state quantum memory prepared by the heralded absorption of a single p
226 ave been shown to underlie many learning and memory processes, little is known about these properties
227 d prefrontal cortex, structures critical for memory processes.
228 criptional factor involved with learning and memory processes.
229 and waning of interest, the role of sleep in memory processing remains controversial and elusive.
230 ural activity patterns in sleep that reflect memory processing) and review computational approaches t
231 zed Mini-Mental State Examination (MMSE) and memory, processing speed, language, and executive functi
232     Awake and sleep SWRs are associated with memory reactivation and are important for learning, but
233  We demonstrated that experimentally induced memory reactivation during sleep renders long-term memor
234                                              Memory reactivation was detected rapidly ( approximately
235 emporarily renew flexibility of consolidated memories, referred to as reconsolidation.
236 coefficients for associations with global or memory-related cognitive function were non-significant a
237 of disinhibition may include reduced working memory-related cortical activity associated with the dow
238 , our findings suggest that MYT1L influences memory-related processes by controlling a neuronal proli
239 versive pavlovian conditioning and that this memory requires cap-dependent initiation, a primary poin
240 that has thus far been largely overlooked in memory research.
241 atency antigen specific, regulatory, central memory response is therefore a novel functional componen
242 ity and systemic B cell- and T cell-mediated memory responses.
243  and among MBC subsets and how this leads to memory responses.
244 he inhibitor fasudil enhances action-outcome memory, resulting in goal-directed behavior in mice that
245 lations are reinstated to support successful memory retrieval.
246 for the role of oscillatory reinstatement in memory retrieval.SIGNIFICANCE STATEMENT Recent neurobiol
247 f global cognitive function, verbal episodic memory, semantic fluency, and calculation as well as a m
248 f reconsolidation or at a 6 h delay when the memory should be restabilized had no effect on reinstate
249                                              Memory skills strongly differ across the general populat
250  neural circuit supporting the trace-trained memory, so that it better reflects the circuit supportin
251 be a critical mechanism supporting long-term memory stabilization.
252 ngs suggest that awake SWRs support accurate memory storage and memory-guided behavior, whereas sleep
253 ght to be detrimental for the specificity of memory storage.
254 adults, enrolled in the Ginkgo Evaluation of Memory study at a university center from January 1, 2000
255  kinetics and structure of murine CD4 T cell memory subsets by measuring the rates of influx of new c
256 ng distinct expression patterns in naive and memory subsets.
257 ted for at least 6 years, and induced immune memory, suggesting possible protection against HPV vacci
258 o depend on the amygdala's interactions with memory systems.
259  A distinct subpopulation of CD4(+) effector memory T (TEM) cells that secrete IL-17A, but not IFN-ga
260 latory elements of the cytokine genes in the memory T cells are marked by activating histone modifica
261                                              Memory T cells sustain effector T-cell production while
262 g to generation of CD4 effector and effector memory T cells that contribute to protection.
263 of human resting naive, central and effector memory T cells using ChIP-Seq and found that unlike the
264  frequency of Pf-specific polyfunctional CD4 memory T cells was associated with protection.
265  cell subsets, that is, naive, effector, and memory T cells.
266 oxic features that is distinct from effector memory T cells.
267 tribute to the maintenance of viral-specific memory T cells.
268                        TH1, TH17, and CD8(+) memory T-cell differentiation was significantly reduced,
269 e significantly less accurate on the working memory task and their neuronal dynamics indicated that e
270        Participants performed an associative memory task during hr-fMRI in which they encoded and lat
271 ham' groups did not differ in online working memory task performance, but the transcranial direct cur
272 onship between hippocampal activity during a memory task using fMRI and subsequent longitudinal chang
273 elevated coupling with each other during the memory task, which correlated with the global reduction
274 an (SD) = 22.12 (2.16)] during a declarative memory task.
275 er BOLD PSC across three levels of a working memory task.
276 e present during the delay period of working memory tasks and may therefore reflect the representatio
277 e supportive for decision-making and working memory tasks.
278 dual-task paradigm and a verbal learning and memory test during and out of symptomatic allergy period
279 mages when patients were given a recognition-memory test the next day.
280 were given cued recollection and recognition memory tests designed to assess recollection and familia
281                             TH1/TH17 central memory (TH1/TH17CM) cells were selectively increased in
282 crabs build separate appetitive and aversive memories that compete during retrieval but not during ac
283 ecture for feature binding in visual working memory that employs populations of neurons with conjunct
284  relate to general processes of learning and memory, the review discusses how aging affects the perce
285 ial information, the creation and storage of memory traces for spatial information, and the use of sp
286 ion to slowly degrade molecular and cellular memory traces.
287 favorable conditions and six transcriptional memory types.
288                Here, we show that trace fear memory undergoes a protein synthesis-dependent reconsoli
289 rements associated with recall such as rapid memory updating and retrieval-driven instinctive fear re
290 ebo group, but not in the propranolol group, memory vividness significantly decreased from pretest to
291 e amygdalar hemodynamic response to positive memories was associated with changes in amygdalar respon
292                                    Word-list memory was modeled via inverse-probability weighted long
293 V-1 acquisition, but the efficacy and immune memory were inadequate.
294 aphical memory as assessed in tasks of scene memory where the viewpoint shifts from study to test.
295 C1 variation predicted attention and working memory, whereas NR3C2 polymorphisms predicted memory per
296  classical algorithm known requires infinite memory, while a quantum simulator requires only finite m
297 hat realizes a multiplexed DLCZ-type quantum memory with 225 individually accessible memory cells in
298 hallenging to target maladaptive generalized memories without affecting adaptive memories.
299              SIGNIFICANCE STATEMENT: Working memory (WM) is a key component of cognition.
300 ate the effectiveness of an adaptive working memory (WM) training (WMT) program, the corresponding ne

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