ライフサイエンスコーパス: memory reconsolidation

1 (DH) critically controls contextual cocaine-memory reconsolidation.

2 ful psychotherapy exerts its effects through memory reconsolidation.

3 uroscience and translational aspects of fear memory reconsolidation.

4 zing exposure therapy based on principles of memory reconsolidation.

5 ral pathways that may underlie therapy-based memory reconsolidation.

6 ion provides a more plausible alternative to memory reconsolidation.

7 ct on which to build integrative models than memory reconsolidation.

8 ecent advances in our understanding of human memory reconsolidation.

9 y reactivation significantly attenuated fear memory reconsolidation.

10 ing how these findings relate to theories of memory reconsolidation.

11 induced synaptic enhancements mediating fear memory reconsolidation.

12 egulated and required in the amygdala for IA memory reconsolidation.

13 n during retrieval and shortly after, during memory reconsolidation.

14 facilitated extinction or disrupted drug-cue memory reconsolidation.

15 ption or modification through the process of memory reconsolidation.

16 diated translational control is required for memory reconsolidation.

17 anisms in hippocampus that are necessary for memory reconsolidation.

18 novo protein synthesis is not essential for memory reconsolidation.

19 During memory reconsolidation, a memory that is recalled become

20 Synaptic mechanisms underlying memory reconsolidation after retrieval are largely unkno

21 As such, disruption of memory reconsolidation after retrieval has received atte

22 y analyzing the role of specific pathways in memory reconsolidation after retrieval.

23 We question memory reconsolidation and emotional arousal as sufficie

24 ation of a conditioned fear stimulus induces memory reconsolidation and fear memory persistence, whil

25 am substrates are crucial in alcohol-related memory reconsolidation and highlight this pathway as a t

26 the hypothesis that memory retrieval induces memory reconsolidation and suggest that the hypothesis n

27 in the BLA is an important modulator of fear memory reconsolidation and that its effects on memory ar

28 NMDAR subtypes in the mechanisms underlying memory reconsolidation and to further understand the con

29 f adrenergic signaling in alcohol-associated memory reconsolidation, and suggest a pharmacological ta

30 omplex activity leads to impairments in fear memory reconsolidation, and that elevating histone acety

31 xplicit memory, emotion-memory interactions, memory reconsolidation, and the relationship between aut

32 Lane et al. view the process of memory reconsolidation as a main ingredient of psychothe

33 ar requirement for memory consolidation), in memory reconsolidation at two time points after training

34 ch has shown that extinction training during memory reconsolidation attenuates the recovery of fear m

35 relapse prevention in drug addiction through memory reconsolidation blockade.

36 een widely implicated as being necessary for memory reconsolidation, but gene transcription mechanism

37 eptors (NMDARs) is known to be important for memory reconsolidation, but while most studies show that

38 While initial demonstrations of memory reconsolidation came mainly from animal studies,

39 Therefore, memory reconsolidation can be both disrupted and enhance

40 Manipulations of memory reconsolidation can interfere with the ability of

41 The target article argues memory reconsolidation demonstrates how therapeutic chan

42 a selective enhancement and an impairment of memory reconsolidation dependent on amygdalar PKA.

43 However, avoidance memory reconsolidation depends on a set of still loosely

44 These results indicate that spatial memory reconsolidation depends on the functionality of L

45 what is known about fear-motivated avoidance memory reconsolidation derives from studies based on fea

46 of fear conditioning, dysregulated circuits, memory reconsolidation, epigenetics, and genetic factors

47 These data show that memory reconsolidation for a cocaine-paired stimulus is

48 Although disrupting the process of memory reconsolidation has a great potential for clinica

49 es in adolescents, building on principles of memory reconsolidation in adults.

50 , these results suggest that contextual drug-memory reconsolidation in Pavlovian and instrumental set

51 ound that one normal function of hippocampal memory reconsolidation in rats is to modify the strength

52 rgent forms of psychotherapy is a process of memory reconsolidation integrating new emotional experie

53 Authors postulate that memory reconsolidation is a key common neurobiological p

54 My commentary suggests that memory reconsolidation is an element within self-reorgan

55 psychotherapeutic change comes about through memory reconsolidation is compelling; however, the model

56 Memory reconsolidation is considered to be the process w

57 Here, the adaptive function of memory reconsolidation is explored in more detail, with

58 That memory reconsolidation is the process underlying decisiv

59 In the process of memory reconsolidation, it is now clear that every time

60 Memory reconsolidation may provide such a target for dis

61 Thus, applying HDACis during memory reconsolidation might constitute a treatment opti

62 uss the implications of the target article's memory reconsolidation model in the development of more

63 knowledge about the precise rules governing memory reconsolidation of aversive memories that might b

64 ockdown of Arc/Arg3.1 in the LA impairs fear memory reconsolidation of both a recent (1-d-old) as wel

65 indicate that the hippocampus is involved in memory reconsolidation only when reactivation results in

66 we found that the hippocampus is involved in memory reconsolidation only when the animals acquired th

67 heir reactivation through retrieval during a memory reconsolidation process that depends on protein s

68 results demonstrate how the consequences of memory reconsolidation processes are represented in the

69 look at the functional role of Arc/Arg3.1 in memory reconsolidation processes in the mammalian brain.

70 The functional role of Arc/Arg3.1 in memory reconsolidation processes, however, has not been

71 I suggest it is premature to assume memory reconsolidation provides a unifying model of psyc

72 y and its role in both implicit and explicit memory reconsolidation, rather than balances concerning

73 the hippocampus in fear-motivated avoidance memory reconsolidation remains controversial.

74 Memory reconsolidation requires the gamma2alpha'1 mushro

75 fore memory retrieval, but not after (during memory reconsolidation), reverses the cocaine-evoked pre

76 Memory reconsolidation that incorporates new emotional e

77 Lane et al. propose that memory reconsolidation through new emotional experiences

78 Here we show that blockade of drug memory reconsolidation, through infusion of Zif268 antis

79 ocedure to determine what was learned during memory reconsolidation under normal retrieval conditions

80 This reduction in relapse after disrupted memory reconsolidation was not only seen after several h

81 xtinction reflects a behavioural analogue of memory reconsolidation, we show that prevention of conte

82 consolidation, (c) brain plasticity, and (d) memory reconsolidation; we finish with a summary of the

83 articipation of the hippocampus in avoidance memory reconsolidation, which is associated with an incr

84 on of the molecular underpinnings of cocaine-memory reconsolidation will likely facilitate the develo

85 at timing extinction to coincide with threat memory reconsolidation would prevent the return of defen