ライフサイエンスコーパス: memory trace

1 in) storing and recalling memories (i.e., a memory trace).

2 tioning, a short-lived and branch-restricted memory trace.

3 nsistent with an expression of a stable fear memory trace.

4 hippocampus, experiences disappear without a memory trace.

5 between mossy fibers and DCN neurons in this memory trace.

6 , probably reflecting a fast decaying iconic memory trace.

7 es that are thought to form the basis of the memory trace.

8 memory reactivation, on strengthening of the memory trace.

9 C function, depending on the strength of the memory trace.

10 ) neurons and so possess the properties of a memory trace.

11 to indicate disruption of an existing motor memory trace.

12 destabilization and modification of the fear memory trace.

13 porating new contextual information into the memory trace.

14 t to explain in terms of access to a unitary memory trace.

15 hways to localize definitively the essential memory trace.

16 oral weighted voltage trace, also called the memory trace.

17 rfering with reconsolidation of the original memory trace.

18 proposed to act as a long-lasting molecular memory trace.

19 ion to slowly degrade molecular and cellular memory traces.

20 the formation and expression of new spatial memory traces.

21 s, indexing the ignition of their underlying memory traces.

22 hippocampal-association cortical transfer of memory traces.

23 rontal cortex (PFC), to "stamp in" posttrial memory traces.

24 emporal persistence of transient hippocampal memory traces.

25 critical for the establishment of permanent memory traces.

26 ecific synaptic modifications to consolidate memory traces.

27 retain odors by extending the decay of weak memory traces.

28 means of "updating" or "rewriting" existing memory traces.

29 for the hypothesized transfer of hippocampal memory traces.

30 s of reconsolidation which leads to modified memory traces.

31 he environment to determine when to form new memory traces.

32 thereby improving the signal:noise ratio of memory traces.

33 t is recruited first into dopamine-dependent memory traces.

34 ather than suppression of no-longer-relevant memory traces.

35 stabilization of hippocampal and neocortical memory traces.

36 ng in REM sleep rather than linking episodic memory traces.

37 l amygdala (LA) neurons are assigned to fear memory traces.

38 etting arises due to inhibition of competing memory traces.

39 the molecular components of the long-lasting memory trace?

40 e periodic clearance of outdated hippocampal memory traces after cortical memory consolidation, there

41 ism for consolidation is the reactivation of memory traces after their initial encoding during subseq

42 and is behaviorally relevant for stabilizing memory traces against interference.

43 d activation of amygdala neurons bearing the memory trace and increased the synaptic exchange from Ca

44 the sensory representation of the competitor memory trace and predicted the amount of retrieval-induc

45 ocampus could coordinate the reactivation of memory traces and direct their reinstatement in cortical

46 inks between intact identity-specific visual memory traces and later semantic face processing stages.

47 lter inhibiting the development of imprecise memory traces and reducing the false memory rate.

48 sing finding that DPMs contain odor-specific memory traces and send integrated information about the

49 estions about the fundamental limits of such memory traces and the properties required of dynamical s

50 Memory traces are believed to be ensembles of cells used

51 o the cerebellum), and putative higher-order memory traces are characterized in the hippocampus.

52 widely assumed to be the mechanism by which memory traces are encoded and stored in the central nerv

53 5) solved the long-standing puzzle of where memory traces are formed in the brain when the CS is ele

54 regions of these subtypes, we conclude that memory traces are guided to target regions of the activa

55 These studies suggest that distinct memory traces are located in the DG and in CA3 but that

56 These findings also suggest that fear memory traces are partially erased after extinction.

57 ultineuronal activity suggestive of episodic memory traces are reactivated during REM sleep.

58 authors point to findings that suggest that memory traces are susceptible to modification.

59 Over time, memory traces are thought to undergo a neural reorganiza

60 During these fast oscillations, memory traces are transferred from the hippocampus to th

61 xperiences are represented by collections of memory traces at the cellular level.

62 role in trace conditioning is to maintain a memory trace between the offset of the CS+ and the delay

63 memory, but rather led to disruption of the memory trace, breaking down the link between memory reac

64 ty are more likely to be integrated into the memory trace, but that competitive synaptic interactions

65 ity that occurs during the creation of a new memory trace can be observed using functional magnetic r

66 ade, a large body of research has shown that memory traces can become labile upon retrieval and must

67 , pathway tracing) to identify the essential memory trace circuit for a given form of learning and me

68 e responses as a model system, the essential memory trace circuit is identified, the basic memory tra

69 p participates in the consolidation of fresh memory traces come from a wide range of experimental obs

70 Such memory traces could be stabilised from short- to long-te

71 However, a model that elucidates how these memory traces could emerge through spike-timing-dependen

72 such SWR-directed reactivation of brain-wide memory traces could underlie memory consolidation.

73 nt synaptic reinforcement of the hippocampal memory traces created during initial learning.

74 hat maintenance of subsequent reconsolidated memory trace depends on CaMKII, and these results also s

75 ation of synaptic circuits to retain salient memory traces despite the noise of daily experience.

76 A cellular memory trace, detected as increased calcium influx into

77 However, the mechanisms by which motor memory traces develop during sleep remain controversial

78 el experiences, suggesting that a persistent memory trace develops with experience.

79 We suggest that reactivation of memory traces distributed across modality-specific brain

80 t SWS, suggesting a lack of strengthening of memory traces during REM sleep, at least in the case of

81 rack the reactivation of lateralized sensory memory traces during retrieval.

82 ggers the reactivation and reorganization of memory traces during sleep, a systems-level process that

83 proposed to be involved in the processing of memory traces during sleep.

84 rgeted memory reactivation (TMR) of specific memory traces during slow-wave sleep promotes the emerge

85 nce learning to investigate how newly-formed memory traces evolve dynamically over time.

86 The cellular memory traces first appear at 30 min after conditioning

87 Hippocampal memory traces followed by novelty were more dominant by

88 we have succeeded in localizing an essential memory trace for a basic form of associative learning an

89 fantile amnesia period is stored as a latent memory trace for a long time; indeed, a later reminder r

90 dimensional dynamical systems could retain a memory trace for past inputs in their current state.

91 ptive field plasticity, and could serve as a memory trace for stimuli or episodes that have acquired

92 t produced initial retention impairment, the memory trace for the aversive event was reactivated (i.e

93 ould result in a long-lasting and meaningful memory trace for the event but, at the same time, make i

94 ausal processes, and to create and reinforce memory traces for better recall and application over tim

95 An important feature of our task is that memory traces for contextual information were not access

96 elease may interfere with the laying down of memory traces for incidents of childhood abuse.

97 ial information, the creation and storage of memory traces for spatial information, and the use of sp

98 rtex engages in the formation and storage of memory traces for spatial information.

99 We predict that memory traces for various stimuli may "merge," such that

100 optical imaging have revealed that cellular memory traces form in different areas of the insect brai

101 These memory traces form in only one of the two branches of th

102 Notably, a memory trace formed in the APL neuron by pairing odor wi

103 an instructive role in the communication of memory traces from the hippocampus to the cerebral corte

104 ult dentate gyrus may serve to clear out old memory traces from the hippocampus, thus leaving the hip

105 ern classification, resolves why an enduring memory trace has proven elusive in previous human studie

106 nsistent with the idea that these particular memory traces have strengthened with time, and therefore

107 brain substrates of memory is the nature of memory traces, how memories are formed, stored, and retr

108 ing memory trace, reward creates a competing memory trace, impairing expression of the original rewar

109 mation and strengthening of neural long-term memory traces, improving discrimination skills, in parti

110 nd maintain synaptic specificity of a labile memory trace in a recurrent DPM and MB alpha'beta' neuro

111 e, whereas the capacity to form a short-term memory trace in the alpha'/beta' mushroom body neurons r

112 pendent behavioral changes to the underlying memory trace in this marine mollusk.

113 A new study of memory traces in an invertebrate challenges convention i

114 tic events required for the consolidation of memory traces in cortical networks.

115 and on consistent processing of associative memory traces in midline structures that are involved in

116 esized to allow for the transient storage of memory traces in neuronal networks.

117 excessive DA may prevent storage of lasting memory traces in PFC networks and impair executive funct

118 Together with few episodic memory traces in REM sleep, and REM sleep deprivation af

119 n, a process that enables updating of stored memory traces in response to novelty.

120 ression (LTD) underlie at least a portion of memory traces in the brain, but the exact cellular locus

121 imply simultaneous reactivation of coherent memory traces in the cortex and hippocampus during sleep

122 However, early memory traces in the MB remain elusive.

123 hat avoidance training produces two opposing memory traces in these regions.

124 ly recognition ('retrieval') and matching of memory traces in working memory.

125 cal response reliability, but also leaves a 'memory trace' in subsequent spontaneous activity.

126 he persistence and breadth of the DPM neuron memory trace influences the duration of behavioral memor

127 ry consolidation transforms initially labile memory traces into more stable representations.

128 , it remains unknown whether a given sensory memory trace is being maintained as a unitary item to as

129 This time window for the gamma neuron memory trace is displaced relative to the modifications

130 gh classical associative learning, but which memory trace is eligible for modification depends on a s

131 ing spatial learning an experience-dependent memory trace is formed in this structure.

132 emory trace circuit is identified, the basic memory trace is localized (to the cerebellum), and putat

133 in interacting with pre-existing reactivated memory traces is critical for successful modification of

134 ional imaging study has revealed a long-term memory trace manifested as an increase in the Ca(2+) act

135 tute memory recall, whereas consolidation of memory traces may be revealed and served by correlated f

136 The transient memory traces may support behavior across the time windo

137 gets suggest that the persistent form of the memory trace might be comprised of molecules that mainta

138 cation, according to which modification of a memory trace occurs through classical associative learni

139 ntion can reach into the past, acting on the memory trace of a stimulus that has disappeared before b

140 at CaMKIIalpha accumulation at synapses is a memory trace of past synaptic activity.

141 hypothesis is that the hippocampus stores a memory trace of the conditioned stimulus (CS) during the

142 Our analyses show that the intrinsic memory trace of the fractional derivative provides a neg

143 join neural networks, and may constitute the memory trace of the imprinted stimulus.

144 ments gives rise to a kinematically specific memory trace of the observed motions in M1.

145 biological systems must retain a short-term memory trace of their recent inputs.

146 and demonstrate that there are two parallel memory traces of a novel taste: a short-duration robust

147 t to reflect the activation of stored visual memory traces of known individual faces.

148 or if this phenomenon generalizes to working memory traces of other visual features.

149 quiring, storing, and retrieving associative memory traces of repeatedly co-occurring neural events i

150 This Bayesian analysis revealed the 'memory traces' of the chemical network.

151 s that retrieval can destabilize an existing memory trace, opening a time-dependent window during whi

152 to processes involved in the formation of a memory trace or, more probably, involves both trace form

153 y of a cocaine-cue association (the "cocaine memory trace" or "cocaine engram").

154 anges in neuronal physiology that encode the memory trace, or engram.

155 for initial encoding, for the expression of memory traces, or for both encoding and expression.

156 on in the study of long-term memory is how a memory trace persists for years when the proteins that i

157 honological encoding and/or by strengthening memory traces rather than by fundamentally subserving co

158 Although memory trace reactivation is correlated with low-voltage

159 To investigate the types of memory traces recovered by the medial temporal lobe (MTL

160 the influence of sleep discharge patterns on memory traces remains fragmentary.

161 Because detection and quantification of memory-trace replay depends critically on analysis metho

162 mpal memory space, with consolidation of the memory traces representing repeated paired associates in

163 amic compensatory response only when initial memory traces required consolidation.

164 t that, rather than reinforcing the existing memory trace, reward creates a competing memory trace, i

165 ing that the mPFC controls the expression of memory traces stored in the hippocampus biasing retrieva

166 that both factors modulate a unidimensional memory trace strength.

167 y when there is competition between opposing memory traces, such as that which occurs during the acqu

168 me-target pairs, a subliminal pair leaves no memory trace that can be observed in response to the nex

169 m of plasticity leaves a hidden postsynaptic memory trace that enables fast relearning of previously

170 alog of the spinal stretch reflex, creates a memory trace that includes changes in the spinal cord.

171 e activity of the Purkinje cell allows for a memory trace that is resistant to ongoing activity in th

172 yed, branch-specific, and amnesiac-dependent memory trace that may guide behavior after acquisition.

173 , but only some episodes will leave detailed memory traces that can be recollected after weeks and mo

174 formation of distinct yet flexible emotional memory traces that confer an ability to recall extinctio

175 Here we contrast the cellular memory traces that form in the dorsal paired medial (DPM

176 imaging of living flies have identified six memory traces that form in the olfactory nervous system

177 aging to monitor two different calcium-based memory traces that underlie olfactory classical conditio

178 ared that "consciousness arises instead of a memory-trace." The aim of reconsolidation, and of psycho

179 in the progressive strengthening of cortical memory traces through reactivation of cortical NMDA rece

180 zation; and second, by localizing a critical memory trace to neurons located outside the behavioral c

181 ippocampus has enabled real-time patterns of memory traces to be mathematically described, directly v

182 their ability to form and utilize transient memory traces to guide behavior.

183 use the integrated floral traits from their memory traces to mediate future foraging decisions.

184 literature on the potential vulnerability of memory traces to modification and on the effects of stre

185 deep sleep can underlie mapping hippocampal memory traces to persistent cortical representation.

186 lying the characteristic labilization of the memory trace triggered by retrieval.

187 These findings suggest that a memory trace undergoes rapid modifications, and that the

188 Our results demonstrate that the memory traces underlying cortical deviance detection for

189 Thus, the memory traces underlying cortical deviance detection may

190 as administered 24 h after training, when IA memory trace was already formed.

191 This memory trace was defective in all 26 of the LTM mutants.

192 Rather, the potentials appeared whenever a memory trace was observed behaviorally.

193 To visualize memory traces, we created a transgenic line that allows

194 he alpha/beta and the gamma neuron long-term memory traces were both blocked by expressing a represso

195 and may act by weakening previously encoded memory traces when new information is learned.

196 anges reflected the context attribute of the memory trace, which has been envisioned as an essential

197 leads to both associative and nonassociative memory traces, which can be preferentially accessed by e

198 ed reactivation of distributed components of memory traces while the cortex is "offline," i.e., not e

199 we tested the impact of reinforcing a skill memory trace with monetary reward following memory react

200 a nonuniform pattern of reactivation of fear memory traces, with the most robust reactivation during

201 ble inactivation can be used to localize the memory traces within this circuit.