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1 m tuberculosis; and an antibody epitope from mengovirus.
2 al polyprotein amounts relative to wild-type mengovirus.
3 loops in the 3' untranslated region (UTR) of mengovirus.
4 cornaviruses such as human rhinovirus 14 and mengovirus.
6 Deletions of each stem were engineered in mengovirus cDNAs and also in mengovirus replicons, in wh
8 ingly, and doubly phosphorylated variants of Mengovirus L (L(M)) show both modifications act together
9 that poly(C) tract-dependent attenuation of mengovirus may be due in part to a viral replication def
11 When coxsackievirus B3, human rhinovirus 14, mengovirus, or hepatitis A virus (HAV) capsid proteins w
15 e engineered in mengovirus cDNAs and also in mengovirus replicons, in which part of the viral capsid
17 ells confirm the essential dispensability of mengovirus's poly(C) tract but reveal a subtle, measurab
19 , and deletions in this motif did not affect mengovirus virulence in the same manner as deletions in
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