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1 causing bacteria, Neisseria meningitidis (N. meningitidis).
2 a, Mycobacterium tuberculosis, and Neisseria meningitidis.
3 amic transmission model of group A Neisseria meningitidis.
4 hracis, Neisseria gonorrhoeae, and Neisseria meningitidis.
5 ations frequently fail to identify Neisseria meningitidis.
6 l disease caused by infection with Neisseria meningitidis.
7 explain adolescent/adult colonization by N. meningitidis.
8 ctedly versatile Cas9 protein from Neisseria meningitidis.
9 ance, and virulence in the human pathogen N. meningitidis.
10 mary human meningothelial cells to Neisseria meningitidis.
11 ence factor and vaccine antigen of Neisseria meningitidis.
12 studies on PglL, the O-OTase from Neisseria meningitidis.
13 GI has also been found in some strains of N. meningitidis.
14 tion caused by the human pathogen, Neisseria meningitidis.
15 interactions between 11 Pil proteins from N. meningitidis.
16 d by three DsbA oxidoreductases in Neisseria meningitidis.
17 bA-catalysed oxidative protein folding in N. meningitidis.
18 revalent outer membrane protein in Neisseria meningitidis.
19 land (GGI), as do a few strains of Neisseria meningitidis.
20 or the lack of -35 consensus sequences in N. meningitidis.
21 ar with current recommendations regarding N. meningitidis.
22 s (phasevarions), have been identified in N. meningitidis.
23 inhibit the CRISPR-Cas9 system of Neisseria meningitidis.
24 loyment and assessment of vaccines against N meningitidis.
25 lactamica prevents colonization by Neisseria meningitidis.
27 species (58.0%), followed by GBS (18.1%), N. meningitidis (13.9%), H. influenzae (6.7%), and L. monoc
28 86) of bacterial meningitis cases: Neisseria meningitidis (1350 cases, 22%), Streptococcus pneumoniae
29 regulation mechanism observed for Neisseria meningitidis 3-deoxy-d-arabino-heptulosonate 7-phosphate
31 rofiles to characterize strains of Neisseria meningitidis, a major cause of bacterial meningitis worl
32 mbilical vein endothelial cells or Neisseria meningitidis after incubation with human serum was compl
35 obactam, cefepime, and gentamicin, Neisseria meningitidis and ceftriaxone, and Haemophilus influenzae
36 shared function of fHbp in N. cinerea and N. meningitidis and cross-reactive responses elicited by Be
37 resulted in loss of functional traits in N. meningitidis and E. coli Our study indicates that the ex
39 lysialic acid expressed on the surface of N. meningitidis and in the absence of specific antibody ser
40 ies cellular TNF secretion in response to N. meningitidis and may influence susceptibility to meningo
41 and compares it to studies done on Neisseria meningitidis and Moraxella catarrhalis; the two other or
42 cal isolate described here expresses both N. meningitidis and N. gonorrhoeae 16S rRNA genes, as shown
43 the lipooligosaccharide (LOS) from Neisseria meningitidis and N. gonorrhoeae engages the TLR4-MD-2 co
44 ggest that phosphoryl moieties of LA from N. meningitidis and N. gonorrhoeae LOSs play an important r
45 Comparison of PG fragment release from N. meningitidis and N. gonorrhoeae showed that meningococci
46 P activation on diverse strains of Neisseria meningitidis and N. gonorrhoeae specifically using nativ
47 eisseria contains two pathogenic species (N. meningitidis and N. gonorrhoeae) in addition to a number
49 ld include the beta-proteobacteria Neisseria meningitidis and Neisseria gonnorhoeae, in which the cbb
50 against the Gram-negative species Neisseria meningitidis and Neisseria gonorrheae and improved activ
53 nts is due largely to interaction between N. meningitidis and other members of the upper respiratory
54 umoniae compared with responses to Neisseria meningitidis and that in each case, the bacterial subcap
55 thogens, Neisseria gonorrhoeae and Neisseria meningitidis, and at least 13 species of commensals that
56 tly increases detection of S. pneumoniae, N. meningitidis, and H. influenzae in CSF, and that applica
57 hat were qPCR positive for S. pneumoniae, N. meningitidis, and H. influenzae, only 10 were culture po
59 athogens Streptococcus pneumoniae, Neisseria meningitidis, and Haemophilus influenzae, but the mechan
62 against Streptococcus pneumoniae, Neisseria meningitidis, and Hemophilus influenzae type b induce fu
65 ction with Haemophilus influenzae, Neisseria meningitidis, and Streptococcus pneumoniae causes substa
68 tal colonization and urethritis caused by N. meningitidis are possible across a range of meningococca
69 Several outer membrane proteins of Neisseria meningitidis are subject to phase variation due to alter
72 ate complement-mediated killing of Neisseria meningitidis as they enter the bloodstream from the orop
75 tructure of an ASBT homologue from Neisseria meningitidis (ASBT(NM)) in detergent was reported recent
76 isease-associated and carried isolates of N. meningitidis at the level of individual nucleotide varia
78 cases of meningococcal disease caused by N. meningitidis B were reported among vaccinated students.
81 ulfment of Neisseria gonorrheae or Neisseria meningitidis by human cells and can offer deep understan
82 ive bacterial meningitis caused by Neisseria meningitidis can be prevented by active immunization wit
84 heat-killed cells of Gram-negative Neisseria meningitidis, capsular serogroup C (MenC) or Gram-positi
86 ures grown overnight doubled the yield of N. meningitidis carriage isolates compared with conventiona
87 show that these proteins bind directly to N. meningitidis Cas9 (NmeCas9) and can be used as potent in
94 several lines of supporting evidence that N. meningitidis colonization is correlated with propionic a
95 distinct protospacer adjacent motif, the N. meningitidis CRISPR-Cas machinery increases the sequence
96 real-time PCR assays for detection of (i) N. meningitidis ctrA, H. influenzae hpd, and S. pneumoniae
97 PCR assays have been developed to detect N. meningitidis ctrA, H. influenzae hpd, and S. pneumoniae
100 rategies for outbreaks of invasive Neisseria meningitidis disease are informed by serogroup assays th
101 sed biosensor for the detection of Neisseria meningitidis DNA employing Kretschmann configuration.
102 or exhibits a linear response towards target meningitidis DNA over the concentration range from 10 to
103 idence of laboratory-confirmed serogroup A N meningitidis dropped significantly to 0.01 per 100 000 i
106 c nature and the high diversity of Neisseria meningitidis, epidemiological surveillance incorporating
107 nts with Streptococcus pneumoniae, Neisseria meningitidis, Escherichia coli, and Pseudomonas aerugino
108 raised against sheaths presenting Neisseria meningitidis factor H binding protein (fHbp) antigen wer
110 a are at odds with this proposal and that N. meningitidis fits the criteria that we have proposed for
112 keleton linker, were more pronounced when N. meningitidis formed larger colonies on HBMEC under physi
114 (fHbp), a virulence factor which protects N. meningitidis from innate immunity by binding the human c
115 ty in the Campylobacter jejuni and Neisseria meningitidis genomes encoded hypothetical proteins.
116 e class III Fic protein NmFic from Neisseria meningitidis gets autoadenylylated in cis, thereby auton
118 s launched using a newly developed Neisseria meningitidis group A (NmA) polysaccharide-tetanus toxoid
119 ning the N-propionyl derivative of Neisseria meningitidis group B (MenB) capsular polysaccharide (NPr
120 sular polysaccharides from E. coli K1 and N. meningitidis group B and the heparosan-like capsular pol
121 ng protein, are essential for survival of N. meningitidis group B strain H44/76 in normal human serum
122 is (Haemophilus influenzae type b, Neisseria meningitidis group C and seven serotypes of Streptococcu
125 athogens such as Escherichia coli, Neisseria meningitidis, Haemophilus influenzae, and Pasteurella mu
127 ingitis, which is caused mainly by Neisseria meningitidis, Haemophilus influenzae, and Streptococcus
128 ections (Streptococcus pneumoniae, Neisseria meningitidis, Haemophilus influenzae, S suis) and O tsut
132 eria, including the human pathogen Neisseria meningitidis, have evolved means to preferentially take
133 protein (fHbp) is a lipoprotein of Neisseria meningitidis important for the survival of the bacterium
134 e, GBS, Listeria monocytogenes, or Neisseria meningitidis in cerebrospinal fluid or other normally st
137 p genes provides a metabolic advantage to N. meningitidis in the adult oral cavity, which is rich in
138 vel aspects of the methylcitrate cycle in N. meningitidis include a propionate kinase which was purif
139 vity potentiator (rMIP) protein of Neisseria meningitidis induces significant serum bactericidal anti
159 that the closely related bacterium Neisseria meningitidis is also polyploid, while the commensal orga
165 or factor H (fH) to the surface of Neisseria meningitidis is critical for evasion of innate host defe
166 lthough the opportunistic pathogen Neisseria meningitidis is enriched for colonization in the throat,
173 re, we analyzed the genomes of 39 diverse N. meningitidis isolates associated with urethritis, collec
175 report a 1.44 A crystal structure of the N. meningitidis major pilin PilE and a approximately 6 A cr
176 isease-associated and carried isolates of N. meningitidis may provide critical insight into mechanism
181 an effective vaccine, serogroup B Neisseria meningitidis (MenB) remains a major cause of invasive di
183 entify the recognition site for three key N. meningitidis methyltransferases: ModA11 (exemplified by
184 y, PilE is structurally similar to Neisseria meningitidis minor pilins PilXNm and PilVNm, recently su
185 lipopolysaccharide-null mutants in Neisseria meningitidis, Moraxella catarrhalis, and most recently i
186 neumoniae, Haemophilus influenzae, Neisseria meningitidis, Mycoplasma pneumoniae, Mycobacterium tuber
190 Last, we revisit the species status of N. meningitidis, N. gonorrheae, and N. lactamica in the lig
191 within the obligate human pathogen Neisseria meningitidis, NApe and NExo, are important for survival
194 an important survival strategy of Neisseria meningitidis (Nm) during colonization and infection.
201 jugate vaccine against serogroup A Neisseria meningitidis (NmA), MenAfriVac, was first introduced in
202 The Cas9 RNA-guided endonuclease from N. meningitidis (NmCas9) recognizes a 5'-NNNNGATT-3' protos
203 e HO from the pathogenic bacterium Neisseria meningitidis (NmHO) possesses a crystallographically und
204 enase (HO), from the pathogenic bacterium N. meningitidis(NmHO), which secures host iron, shares many
205 e HO from the pathogenic bacterium Neisseria meningitidis, NmHO, possesses C-terminal His207, Arg208,
207 nactivated, unencapsulated, intact Neisseria meningitidis nor Streptococcus agalactiae inhibited the
208 amica is not associated with disease, but N. meningitidis occasionally invades the host, causing meni
209 of the upper respiratory tract by Neisseria meningitidis occurs despite elicitation of adaptive immu
210 lonized by Neisseria lactamica and Neisseria meningitidis One HGT event resulted in the acquisition o
211 properdin do not bind directly to either N. meningitidis or N. gonorrhoeae but play a crucial role i
212 th S. pneumoniae, S. agalactiae, E. coli, N. meningitidis, or H. influenzae in combination with cefot
213 mmatory responses of lower magnitude than N. meningitidis organisms and N. meningitidis PorB (publish
216 nitude than N. meningitidis organisms and N. meningitidis PorB (published elsewhere as Nme PorB).
217 en switched between N. lactamica PorB and N. meningitidis PorB, we identified residues in loop 5 and
218 eriments demonstrate that the E. coli and N. meningitidis protein homologs are functionally conserved
219 erial pathogenic strains including Neisseria meningitidis, Pseudomonas aeruginosa and Escherichia col
220 urified polysialyltransferase from Neisseria meningitidis (PST(Nm)) to the extracellular environment.
222 of comparative sequence analyses against N. meningitidis reference genome sequences of known serogro
223 st time, that PorB2-expressing strains of N. meningitidis regulate the AP of baby rabbits and rats.
224 The decreased release of PG monomers by N. meningitidis relative to N. gonorrhoeae is partly due to
225 us diseases over the past century, Neisseria meningitidis remains a major causative agent of meningit
226 ion of a few DNA copies per LAMP zone for N. meningitidis, S. pneumoniae and Hib were achieved within
228 te sequences were obtained; 92 (76%) were N. meningitidis sequences, and 29 (24%) were N. gonorrhoeae
229 l correlates of protection against Neisseria meningitidis serogroup A (NmA) in Burkina Faso before th
231 An affordable, highly immunogenic Neisseria meningitidis serogroup A meningococcal conjugate vaccine
233 recently licensed vaccines against Neisseria meningitidis serogroup B (NmB) will depend partly on dis
234 neumoniae, Listeria monocytogenes, Neisseria meningitidis serogroup B, Candida albicans, and P. bergh
237 yze the synthesis of the complex trimeric N. meningitidis serogroup L capsule polymer repeating unit.
238 of the pathophysiologically less relevant N. meningitidis serogroup L, is one of the smallest known S
239 South Africa, and Israel caused by Neisseria meningitidis serogroup Y (NmY) was greater than the worl
240 ingococcal disease (IMD) caused by Neisseria meningitidis serogroup Y has increased in Europe, especi
241 i.p.-injected intact, heat-killed Neisseria meningitidis, serogroup C (MenC), a gram-negative bacter
242 ombinant capsular polymerases from Neisseria meningitidis serogroups A (CsaB) and X (CsxA) were chara
246 and S. pneumoniae lytA (NHS assay); (ii) N. meningitidis serogroups A, W135, and X (AWX assay); and
248 nes are available to protect against four N. meningitidis serogroups, there is currently no commercia
249 ce determinants of disease causing Neisseria meningitidis species are their extracellular polysacchar
250 ynF showed 100% specificity for detecting N. meningitidis species, with high sensitivity (serogroup B
253 ive lipooligosaccharide (LOS) from Neisseria meningitidis strain 89I was analyzed by matrix-assisted
254 ect of purified PorB in vitro, a chimeric N. meningitidis strain expressing N. lactamica PorB induces
257 the transcriptome of adherent serogroup B N. meningitidis strain MC58 was determined at intervals dur
258 much lower than that within the wild-type N. meningitidis strain only upon HBMEC infection and was co
259 ratio of species-specific sequences, the N. meningitidis strain seems to have replaced one of its fo
260 tion against antigenically diverse Neisseria meningitidis strains and to compare this protection to a
261 to construct all possible combinations of N. meningitidis strains deficient in one, two, three, or al
262 allelic patterns in urethritis-associated N. meningitidis strains may reflect genetic diversity in th
267 nfluenzae, Listeria monocytogenes, Neisseria meningitidis, Streptococcus pneumoniae, Streptococcus ag
268 E6 thioesterase from the bacterium Neisseria meningitidis Structural analysis with X-ray crystallogra
270 s how the important human pathogen Neisseria meningitidis subverts immune responses by mimicking the
271 rleukin 8 (IL-8) secretion than wild-type N. meningitidis, suggesting a role for PorB in induction of
274 uenzae type b and capsular group C Neisseria meningitidis tetanus toxoid conjugate vaccine (Hib-MenC-
275 genomic island (the prp gene cluster) in N. meningitidis that enables this species to utilize propio
276 study of the AP endonuclease from Neisseria meningitidis that has allowed us to capture structural i
277 land is absent from the close relative of N. meningitidis, the commensal Neisseria lactamica, which c
279 on has been studied extensively in Neisseria meningitidis, the specific subset of genes that CrgA tar
280 ely related opportunistic pathogen Neisseria meningitidis, there is an absence of adaptive cell-media
281 complement attack achieved by circulating N. meningitidis therefore depends on the relative levels of
282 ilable vaccine for serogroup B strains of N. meningitidis, this kind capsule-switching event could ha
283 ty of the human bacterial pathogen Neisseria meningitidis to cause invasive disease depends on surviv
284 ease-associated and 4 carried isolates of N. meningitidis to search for SNPs that show mutually exclu
289 r the direct quantification of two Neisseria meningitidis vaccine antigens, in mono- and multivalent
292 thogens Haemophilus influenzae and Neisseria meningitidis We hypothesized that activation of compleme
293 a distinct CRISPR-Cas system from Neisseria meningitidis, we demonstrate efficient targeting of an e
294 c variation (Av) of two strains of Neisseria meningitidis were determined using an unbiased DNA seque
296 number of microorganisms including Neisseria meningitidis, which can lead to permanent neurological d
297 ted genomes from the 4 carried genomes of N. meningitidis, which is far more than can be expected by
299 hexaacylated endotoxin (LOS) from Neisseria meningitidis with [(13)C]acetate allowed the use of NMR
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