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1 causing bacteria, Neisseria meningitidis (N. meningitidis).
2 a, Mycobacterium tuberculosis, and Neisseria meningitidis.
3 amic transmission model of group A Neisseria meningitidis.
4 hracis, Neisseria gonorrhoeae, and Neisseria meningitidis.
5 ations frequently fail to identify Neisseria meningitidis.
6 l disease caused by infection with Neisseria meningitidis.
7  explain adolescent/adult colonization by N. meningitidis.
8 ctedly versatile Cas9 protein from Neisseria meningitidis.
9 ance, and virulence in the human pathogen N. meningitidis.
10 mary human meningothelial cells to Neisseria meningitidis.
11 ence factor and vaccine antigen of Neisseria meningitidis.
12  studies on PglL, the O-OTase from Neisseria meningitidis.
13 GI has also been found in some strains of N. meningitidis.
14 tion caused by the human pathogen, Neisseria meningitidis.
15 interactions between 11 Pil proteins from N. meningitidis.
16 d by three DsbA oxidoreductases in Neisseria meningitidis.
17 bA-catalysed oxidative protein folding in N. meningitidis.
18 revalent outer membrane protein in Neisseria meningitidis.
19 land (GGI), as do a few strains of Neisseria meningitidis.
20 or the lack of -35 consensus sequences in N. meningitidis.
21 ar with current recommendations regarding N. meningitidis.
22 s (phasevarions), have been identified in N. meningitidis.
23  inhibit the CRISPR-Cas9 system of Neisseria meningitidis.
24 loyment and assessment of vaccines against N meningitidis.
25 lactamica prevents colonization by Neisseria meningitidis.
26 tive as conventional methods in detecting N. meningitidis (13.2% versus 5.7%; P < 0.0001).
27 species (58.0%), followed by GBS (18.1%), N. meningitidis (13.9%), H. influenzae (6.7%), and L. monoc
28 86) of bacterial meningitis cases: Neisseria meningitidis (1350 cases, 22%), Streptococcus pneumoniae
29  regulation mechanism observed for Neisseria meningitidis 3-deoxy-d-arabino-heptulosonate 7-phosphate
30 umoniae (84% of positive CSF isolates) and N.meningitidis (4%).
31 rofiles to characterize strains of Neisseria meningitidis, a major cause of bacterial meningitis worl
32 mbilical vein endothelial cells or Neisseria meningitidis after incubation with human serum was compl
33 and Prevention for the analysis of Neisseria meningitidis and Bordetella bronchiseptica genomes.
34         PorB is located on the surface of N. meningitidis and can be recognized by receptors of the h
35 obactam, cefepime, and gentamicin, Neisseria meningitidis and ceftriaxone, and Haemophilus influenzae
36 shared function of fHbp in N. cinerea and N. meningitidis and cross-reactive responses elicited by Be
37  resulted in loss of functional traits in N. meningitidis and E. coli Our study indicates that the ex
38               We screened 126 isolates of N. meningitidis and found the GGI in 17.5% of strains, with
39 lysialic acid expressed on the surface of N. meningitidis and in the absence of specific antibody ser
40 ies cellular TNF secretion in response to N. meningitidis and may influence susceptibility to meningo
41 and compares it to studies done on Neisseria meningitidis and Moraxella catarrhalis; the two other or
42 cal isolate described here expresses both N. meningitidis and N. gonorrhoeae 16S rRNA genes, as shown
43 the lipooligosaccharide (LOS) from Neisseria meningitidis and N. gonorrhoeae engages the TLR4-MD-2 co
44 ggest that phosphoryl moieties of LA from N. meningitidis and N. gonorrhoeae LOSs play an important r
45    Comparison of PG fragment release from N. meningitidis and N. gonorrhoeae showed that meningococci
46 P activation on diverse strains of Neisseria meningitidis and N. gonorrhoeae specifically using nativ
47 eisseria contains two pathogenic species (N. meningitidis and N. gonorrhoeae) in addition to a number
48 ons that allow for discrimination between N. meningitidis and N. gonorrhoeae.
49 ld include the beta-proteobacteria Neisseria meningitidis and Neisseria gonnorhoeae, in which the cbb
50  against the Gram-negative species Neisseria meningitidis and Neisseria gonorrheae and improved activ
51                               Both Neisseria meningitidis and Neisseria gonorrhoeae recruit the alter
52       NrrF, a trans-acting sRNA in Neisseria meningitidis and Neisseria gonorrhoeae, has been shown i
53 nts is due largely to interaction between N. meningitidis and other members of the upper respiratory
54 umoniae compared with responses to Neisseria meningitidis and that in each case, the bacterial subcap
55 thogens, Neisseria gonorrhoeae and Neisseria meningitidis, and at least 13 species of commensals that
56 tly increases detection of S. pneumoniae, N. meningitidis, and H. influenzae in CSF, and that applica
57 hat were qPCR positive for S. pneumoniae, N. meningitidis, and H. influenzae, only 10 were culture po
58  against Streptococcus pneumoniae, Neisseria meningitidis, and H. influenzae.
59 athogens Streptococcus pneumoniae, Neisseria meningitidis, and Haemophilus influenzae, but the mechan
60 f pathogenic Neisseria gonorrheae, Neisseria meningitidis, and Haemophilus influenzae.
61 qPCR for Streptococcus pneumoniae, Neisseria meningitidis, and Haemophilus influenzae.
62  against Streptococcus pneumoniae, Neisseria meningitidis, and Hemophilus influenzae type b induce fu
63 er jejuni, Haemophilus influenzae, Neisseria meningitidis, and Pasteurella multocida.
64 f conjugate vaccines against H influenzae, N meningitidis, and S pneumoniae in England.
65 ction with Haemophilus influenzae, Neisseria meningitidis, and Streptococcus pneumoniae causes substa
66          Neisseria gonorrhoeae and Neisseria meningitidis are closely related organisms that cause th
67 r detecting pharyngeal carriage of Neisseria meningitidis are complex.
68 tal colonization and urethritis caused by N. meningitidis are possible across a range of meningococca
69 Several outer membrane proteins of Neisseria meningitidis are subject to phase variation due to alter
70 nses of human tonsil-derived DC to Neisseria meningitidis as a model organism.
71 face outer membrane protein 85 (OMP85) of N. meningitidis as an immobilized selective layer.
72 ate complement-mediated killing of Neisseria meningitidis as they enter the bloodstream from the orop
73        This was particularly relevant for N. meningitidis, as only 1/13 cases was culture positive.
74 a bacterial homologue of ASBT from Neisseria meningitidis (ASBT(NM)) at 2.2 A.
75 tructure of an ASBT homologue from Neisseria meningitidis (ASBT(NM)) in detergent was reported recent
76 isease-associated and carried isolates of N. meningitidis at the level of individual nucleotide varia
77 ation to respond to an outbreak of Neisseria meningitidis B at a U.S. university.
78  cases of meningococcal disease caused by N. meningitidis B were reported among vaccinated students.
79                                    Neisseria meningitidis binds the complement downregulating protein
80            This isolate was identified as N. meningitidis by biochemical identification methods but g
81 ulfment of Neisseria gonorrheae or Neisseria meningitidis by human cells and can offer deep understan
82 ive bacterial meningitis caused by Neisseria meningitidis can be prevented by active immunization wit
83                                    Neisseria meningitidis can be transmitted via asymptomatic nasopha
84 heat-killed cells of Gram-negative Neisseria meningitidis, capsular serogroup C (MenC) or Gram-positi
85                      At baseline, natural N. meningitidis carriage in the control group was 22.4% (36
86 ures grown overnight doubled the yield of N. meningitidis carriage isolates compared with conventiona
87 show that these proteins bind directly to N. meningitidis Cas9 (NmeCas9) and can be used as potent in
88          Cas9 orthologs (including Neisseria meningitidis Cas9 [NmeCas9]) have also been adopted for
89                                    Neisseria meningitidis causes 500 000 cases of septicemia and meni
90                                    Neisseria meningitidis causes bacterial meningitis and septicemia.
91                       The pathogen Neisseria meningitidis causes disease amongst infants and adolesce
92                                    Neisseria meningitidis changes its capsular phenotype through caps
93               Among 25 serogroup B Neisseria meningitidis clinical isolates, we identified four (16%)
94 several lines of supporting evidence that N. meningitidis colonization is correlated with propionic a
95  distinct protospacer adjacent motif, the N. meningitidis CRISPR-Cas machinery increases the sequence
96 real-time PCR assays for detection of (i) N. meningitidis ctrA, H. influenzae hpd, and S. pneumoniae
97  PCR assays have been developed to detect N. meningitidis ctrA, H. influenzae hpd, and S. pneumoniae
98 osphoethanolamine transferase from Neisseria meningitidis, determined to 2.75-A resolution.
99 ards a promising application in the field of meningitidis diagnosis.
100 rategies for outbreaks of invasive Neisseria meningitidis disease are informed by serogroup assays th
101 sed biosensor for the detection of Neisseria meningitidis DNA employing Kretschmann configuration.
102 or exhibits a linear response towards target meningitidis DNA over the concentration range from 10 to
103 idence of laboratory-confirmed serogroup A N meningitidis dropped significantly to 0.01 per 100 000 i
104                                    Neisseria meningitidis employs redundant heme acquisition mechanis
105                                    Neisseria meningitidis encodes up to five TpsA proteins that are s
106 c nature and the high diversity of Neisseria meningitidis, epidemiological surveillance incorporating
107 nts with Streptococcus pneumoniae, Neisseria meningitidis, Escherichia coli, and Pseudomonas aerugino
108  raised against sheaths presenting Neisseria meningitidis factor H binding protein (fHbp) antigen wer
109                        Recombinant native N. meningitidis FBA was purified and used in a coupled enzy
110 a are at odds with this proposal and that N. meningitidis fits the criteria that we have proposed for
111              In the human pathogen Neisseria meningitidis for example, 23 proteins are dedicated to T
112 keleton linker, were more pronounced when N. meningitidis formed larger colonies on HBMEC under physi
113  to hospital and identification of Neisseria meningitidis from a sterile site.
114 (fHbp), a virulence factor which protects N. meningitidis from innate immunity by binding the human c
115 ty in the Campylobacter jejuni and Neisseria meningitidis genomes encoded hypothetical proteins.
116 e class III Fic protein NmFic from Neisseria meningitidis gets autoadenylylated in cis, thereby auton
117 itis incidence and carriage due to Neisseria meningitidis group A (MenA).
118 s launched using a newly developed Neisseria meningitidis group A (NmA) polysaccharide-tetanus toxoid
119 ning the N-propionyl derivative of Neisseria meningitidis group B (MenB) capsular polysaccharide (NPr
120 sular polysaccharides from E. coli K1 and N. meningitidis group B and the heparosan-like capsular pol
121 ng protein, are essential for survival of N. meningitidis group B strain H44/76 in normal human serum
122 is (Haemophilus influenzae type b, Neisseria meningitidis group C and seven serotypes of Streptococcu
123                   Vaccines against Neisseria meningitidis group C are based on its alpha-2,9-linked p
124 s that can be protective against MenB and N. meningitidis group C strains.
125 athogens such as Escherichia coli, Neisseria meningitidis, Haemophilus influenzae, and Pasteurella mu
126                                    Neisseria meningitidis, Haemophilus influenzae, and Streptococcus
127 ingitis, which is caused mainly by Neisseria meningitidis, Haemophilus influenzae, and Streptococcus
128 ections (Streptococcus pneumoniae, Neisseria meningitidis, Haemophilus influenzae, S suis) and O tsut
129                            Group A Neisseria meningitidis has been a major cause of bacterial meningi
130                                           N. meningitidis has been considered a paradigmatic case of
131                                    Neisseria meningitidis has several strategies to evade complement-
132 eria, including the human pathogen Neisseria meningitidis, have evolved means to preferentially take
133 protein (fHbp) is a lipoprotein of Neisseria meningitidis important for the survival of the bacterium
134 e, GBS, Listeria monocytogenes, or Neisseria meningitidis in cerebrospinal fluid or other normally st
135 ach for fast instrument-free diagnosis of N. meningitidis in resource-limited settings.
136 k so far and shows the continued threat of N meningitidis in sub-Saharan Africa.
137 p genes provides a metabolic advantage to N. meningitidis in the adult oral cavity, which is rich in
138 vel aspects of the methylcitrate cycle in N. meningitidis include a propionate kinase which was purif
139 vity potentiator (rMIP) protein of Neisseria meningitidis induces significant serum bactericidal anti
140                   The incidence of Neisseria meningitidis infection decreased from 0.721 per 100 000
141  understanding the epidemiology of Neisseria meningitidis infection.
142                                    Neisseria meningitidis inhibits the alternative pathway (AP) of co
143 ake via GltT-GltM plays multiple roles in N. meningitidis internalization into HBMEC.
144        We previously reported that Neisseria meningitidis internalization into human brain microvasoc
145                                    Neisseria meningitidis is a commensal microbe that colonizes the h
146                                    Neisseria meningitidis is a commensal of humans that can colonize
147                                    Neisseria meningitidis is a frequent colonizer of the human nasoph
148                                    Neisseria meningitidis is a human commensal that can also cause li
149                                    Neisseria meningitidis is a human pathogen causing bacterial menin
150                                    Neisseria meningitidis is a human-specific bacterium that varies i
151                                    Neisseria meningitidis is a human-specific pathogen and leading ca
152                         GNA2091 of Neisseria meningitidis is a lipoprotein of unknown function that i
153                                    Neisseria meningitidis is a major cause of bacterial meningitis an
154                                    Neisseria meningitidis is a major cause of bacterial meningitis wo
155                                    Neisseria meningitidis is a major global pathogen causing invasive
156   The capsular polysaccharide surrounding N. meningitidis is a major virulence factor.
157                                    Neisseria meningitidis is a strict human pathogen that closely int
158                 The limit of detection of N. meningitidis is about 3 copies per LAMP zone within 45 m
159 that the closely related bacterium Neisseria meningitidis is also polyploid, while the commensal orga
160                                    Neisseria meningitidis is an encapsulated pathogen, and antibodies
161                                    Neisseria meningitidis is an important cause of invasive bacterial
162                                    Neisseria meningitidis is an important human pathogen that is capa
163                                    Neisseria meningitidis is an obligate human nasopharyngeal commens
164                 However, most of the time N. meningitidis is carried as a commensal not associated wi
165 or factor H (fH) to the surface of Neisseria meningitidis is critical for evasion of innate host defe
166 lthough the opportunistic pathogen Neisseria meningitidis is enriched for colonization in the throat,
167                           Although Neisseria meningitidis is naturally competent and porB genetic mos
168                                    Neisseria meningitidis is one of the main agents of bacterial meni
169 stems of Neisseria gonorrhoeae and Neisseria meningitidis is presented.
170                 An apparently rare Neisseria meningitidis isolate containing one copy of a Neisseria
171                                       The N. meningitidis isolate described must have obtained N. gon
172 Probe Aptima assays cross-react with this N. meningitidis isolate.
173 re, we analyzed the genomes of 39 diverse N. meningitidis isolates associated with urethritis, collec
174                Almost all invasive Neisseria meningitidis isolates express capsular polysaccharide.
175  report a 1.44 A crystal structure of the N. meningitidis major pilin PilE and a approximately 6 A cr
176 isease-associated and carried isolates of N. meningitidis may provide critical insight into mechanism
177                                           N. meningitidis MC58 NMB0419 encodes a Sel1-like repeat (SL
178 h glyco-conjugate capsular group C Neisseria meningitidis (Men C) vaccines in infancy.
179 vel protection against serogroup A Neisseria meningitidis (MenA) are unknown.
180                        Serogroup B Neisseria meningitidis (MenB) is a major cause of severe sepsis an
181  an effective vaccine, serogroup B Neisseria meningitidis (MenB) remains a major cause of invasive di
182                                    Neisseria meningitidis (meningococcus) is a symbiont of the human
183 entify the recognition site for three key N. meningitidis methyltransferases: ModA11 (exemplified by
184 y, PilE is structurally similar to Neisseria meningitidis minor pilins PilXNm and PilVNm, recently su
185 lipopolysaccharide-null mutants in Neisseria meningitidis, Moraxella catarrhalis, and most recently i
186 neumoniae, Haemophilus influenzae, Neisseria meningitidis, Mycoplasma pneumoniae, Mycobacterium tuber
187 on pathogen detected (n = 17) followed by N. meningitidis (n = 13).
188                                    Neisseria meningitidis (N. meningitidis), Streptococcus pneumoniae
189  main meningitis-causing bacteria, Neisseria meningitidis (N. meningitidis).
190    Last, we revisit the species status of N. meningitidis, N. gonorrheae, and N. lactamica in the lig
191 within the obligate human pathogen Neisseria meningitidis, NApe and NExo, are important for survival
192                  The three species Neisseria meningitidis, Neisseria gonorrheae, and Neisseria lactam
193                                    Neisseria meningitidis (Nm) clonal complex 11 (cc11) lineage is a
194  an important survival strategy of Neisseria meningitidis (Nm) during colonization and infection.
195                                    Neisseria meningitidis (Nm) is a Gram-negative diplococcus that no
196                 The human pathogen Neisseria meningitidis (Nm) is a leading cause of bacterial mening
197                                    Neisseria meningitidis (Nm) is a leading cause of bacterial mening
198                 The human pathogen Neisseria meningitidis (Nm) is known to possess several mechanisms
199                                    Neisseria meningitidis (Nm) strains infecting these patients are p
200 due to a large epidemic of group A Neisseria meningitidis (NmA) meningitis.
201 jugate vaccine against serogroup A Neisseria meningitidis (NmA), MenAfriVac, was first introduced in
202     The Cas9 RNA-guided endonuclease from N. meningitidis (NmCas9) recognizes a 5'-NNNNGATT-3' protos
203 e HO from the pathogenic bacterium Neisseria meningitidis (NmHO) possesses a crystallographically und
204 enase (HO), from the pathogenic bacterium N. meningitidis(NmHO), which secures host iron, shares many
205 e HO from the pathogenic bacterium Neisseria meningitidis, NmHO, possesses C-terminal His207, Arg208,
206                               Serogroup X N. meningitidis (NmX) dominated in both vaccinated and unva
207 nactivated, unencapsulated, intact Neisseria meningitidis nor Streptococcus agalactiae inhibited the
208 amica is not associated with disease, but N. meningitidis occasionally invades the host, causing meni
209  of the upper respiratory tract by Neisseria meningitidis occurs despite elicitation of adaptive immu
210 lonized by Neisseria lactamica and Neisseria meningitidis One HGT event resulted in the acquisition o
211  properdin do not bind directly to either N. meningitidis or N. gonorrhoeae but play a crucial role i
212 th S. pneumoniae, S. agalactiae, E. coli, N. meningitidis, or H. influenzae in combination with cefot
213 mmatory responses of lower magnitude than N. meningitidis organisms and N. meningitidis PorB (publish
214                       Carriage for Neisseria meningitidis (P < 0.05) and Neisseria lactamica (P < 0.0
215                                    Neisseria meningitidis phasevarions regulate genes including virul
216 nitude than N. meningitidis organisms and N. meningitidis PorB (published elsewhere as Nme PorB).
217 en switched between N. lactamica PorB and N. meningitidis PorB, we identified residues in loop 5 and
218 eriments demonstrate that the E. coli and N. meningitidis protein homologs are functionally conserved
219 erial pathogenic strains including Neisseria meningitidis, Pseudomonas aeruginosa and Escherichia col
220 urified polysialyltransferase from Neisseria meningitidis (PST(Nm)) to the extracellular environment.
221                             We found that N. meningitidis recycles PG fragments via the selective per
222  of comparative sequence analyses against N. meningitidis reference genome sequences of known serogro
223 st time, that PorB2-expressing strains of N. meningitidis regulate the AP of baby rabbits and rats.
224   The decreased release of PG monomers by N. meningitidis relative to N. gonorrhoeae is partly due to
225 us diseases over the past century, Neisseria meningitidis remains a major causative agent of meningit
226 ion of a few DNA copies per LAMP zone for N. meningitidis, S. pneumoniae and Hib were achieved within
227 alciparum infections, and virulent Neisseria meningitidis samples.
228 te sequences were obtained; 92 (76%) were N. meningitidis sequences, and 29 (24%) were N. gonorrhoeae
229 l correlates of protection against Neisseria meningitidis serogroup A (NmA) in Burkina Faso before th
230                          To combat Neisseria meningitidis serogroup A epidemics in the meningitis bel
231  An affordable, highly immunogenic Neisseria meningitidis serogroup A meningococcal conjugate vaccine
232                                    Neisseria meningitidis serogroup B (MnB) is a leading cause of bac
233 recently licensed vaccines against Neisseria meningitidis serogroup B (NmB) will depend partly on dis
234 neumoniae, Listeria monocytogenes, Neisseria meningitidis serogroup B, Candida albicans, and P. bergh
235 luding 1147 (71.5%) that were positive for N meningitidis serogroup C (NmC).
236                               Because the N. meningitidis serogroup L capsule polymer consists of a t
237 yze the synthesis of the complex trimeric N. meningitidis serogroup L capsule polymer repeating unit.
238 of the pathophysiologically less relevant N. meningitidis serogroup L, is one of the smallest known S
239 South Africa, and Israel caused by Neisseria meningitidis serogroup Y (NmY) was greater than the worl
240 ingococcal disease (IMD) caused by Neisseria meningitidis serogroup Y has increased in Europe, especi
241  i.p.-injected intact, heat-killed Neisseria meningitidis, serogroup C (MenC), a gram-negative bacter
242 ombinant capsular polymerases from Neisseria meningitidis serogroups A (CsaB) and X (CsxA) were chara
243                                    Neisseria meningitidis serogroups A and X are among the leading ca
244  consists of polyhexosamine phosphates in N. meningitidis serogroups A and X.
245 group-specific genes in the cap locus for N. meningitidis serogroups A, B, C, W135, X, and Y.
246  and S. pneumoniae lytA (NHS assay); (ii) N. meningitidis serogroups A, W135, and X (AWX assay); and
247 ups A, W135, and X (AWX assay); and (iii) N. meningitidis serogroups B, C, and Y (BCY assay).
248 nes are available to protect against four N. meningitidis serogroups, there is currently no commercia
249 ce determinants of disease causing Neisseria meningitidis species are their extracellular polysacchar
250 ynF showed 100% specificity for detecting N. meningitidis species, with high sensitivity (serogroup B
251 e resolution the primary transcriptome of N. meningitidis strain 8013.
252                                           N. meningitidis strain 8047 was subjected to serial passage
253 ive lipooligosaccharide (LOS) from Neisseria meningitidis strain 89I was analyzed by matrix-assisted
254 ect of purified PorB in vitro, a chimeric N. meningitidis strain expressing N. lactamica PorB induces
255                                    Neisseria meningitidis strain H44/76 was modified by expression of
256                   All four opa genes from N. meningitidis strain H44/76 were sequentially disrupted t
257 the transcriptome of adherent serogroup B N. meningitidis strain MC58 was determined at intervals dur
258 much lower than that within the wild-type N. meningitidis strain only upon HBMEC infection and was co
259  ratio of species-specific sequences, the N. meningitidis strain seems to have replaced one of its fo
260 tion against antigenically diverse Neisseria meningitidis strains and to compare this protection to a
261 to construct all possible combinations of N. meningitidis strains deficient in one, two, three, or al
262 allelic patterns in urethritis-associated N. meningitidis strains may reflect genetic diversity in th
263           Laboratory data on 4,735 Neisseria meningitidis strains was collected and reported by the N
264 s-like and distinguishing them from other N. meningitidis strains.
265 henotypes and hence invasive potential of N. meningitidis strains.
266                   Neisseria meningitidis (N. meningitidis), Streptococcus pneumoniae (S. pneumoniae),
267 nfluenzae, Listeria monocytogenes, Neisseria meningitidis, Streptococcus pneumoniae, Streptococcus ag
268 E6 thioesterase from the bacterium Neisseria meningitidis Structural analysis with X-ray crystallogra
269                                           N. meningitidis subverts immune responses by hijacking a ho
270 s how the important human pathogen Neisseria meningitidis subverts immune responses by mimicking the
271 rleukin 8 (IL-8) secretion than wild-type N. meningitidis, suggesting a role for PorB in induction of
272                     In all infected mice, N. meningitidis targeted the human vasculature, leading to
273               These data demonstrate that N. meningitidis targets human endothelial cells in vivo and
274 uenzae type b and capsular group C Neisseria meningitidis tetanus toxoid conjugate vaccine (Hib-MenC-
275  genomic island (the prp gene cluster) in N. meningitidis that enables this species to utilize propio
276  study of the AP endonuclease from Neisseria meningitidis that has allowed us to capture structural i
277 land is absent from the close relative of N. meningitidis, the commensal Neisseria lactamica, which c
278                            In E. coli and N. meningitidis, the predominant lipid is a lysophosphatidy
279 on has been studied extensively in Neisseria meningitidis, the specific subset of genes that CrgA tar
280 ely related opportunistic pathogen Neisseria meningitidis, there is an absence of adaptive cell-media
281 complement attack achieved by circulating N. meningitidis therefore depends on the relative levels of
282 ilable vaccine for serogroup B strains of N. meningitidis, this kind capsule-switching event could ha
283 ty of the human bacterial pathogen Neisseria meningitidis to cause invasive disease depends on surviv
284 ease-associated and 4 carried isolates of N. meningitidis to search for SNPs that show mutually exclu
285                                    Neisseria meningitidis, typically a resident of the oro- or nasoph
286  damage, are not required for survival of N. meningitidis under oxidative stress.
287                                    Neisseria meningitidis use Type IV pili (T4P) to adhere to endothe
288                                    Neisseria meningitidis utilizes capsular polysaccharide, lipooligo
289 r the direct quantification of two Neisseria meningitidis vaccine antigens, in mono- and multivalent
290                               Carriage of N. meningitidis was investigated by using three different m
291 ture of an ADEP-ClpP complex derived from N. meningitidis was solved.
292 thogens Haemophilus influenzae and Neisseria meningitidis We hypothesized that activation of compleme
293  a distinct CRISPR-Cas system from Neisseria meningitidis, we demonstrate efficient targeting of an e
294 c variation (Av) of two strains of Neisseria meningitidis were determined using an unbiased DNA seque
295 95.7% and 85.7%, and L. monocytogenes and N. meningitidis were not observed in the study.
296 number of microorganisms including Neisseria meningitidis, which can lead to permanent neurological d
297 ted genomes from the 4 carried genomes of N. meningitidis, which is far more than can be expected by
298          Hyperinvasive lineages of Neisseria meningitidis, which persist despite extensive horizontal
299  hexaacylated endotoxin (LOS) from Neisseria meningitidis with [(13)C]acetate allowed the use of NMR
300 s and 25 isolates from carriers of Neisseria meningitidis without disease.

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