ライフサイエンスコーパス: mercuric chloride

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1 395 nm upon addition of the thiol chelator, mercuric chloride.

2 -fold; this water transport was inhibited by mercuric chloride.

3 cetylcysteine, and DMSO inhibit injury after mercuric chloride administration.

4 Mercuric chloride also suppressed hypoxia- and Co-induce

5 esicles was partially inhibited (30%-39%) by mercuric chloride and phloretin, a non-specific channel

6 eon Sulfolobus solfataricus was sensitive to mercuric chloride, and low-level adaptive resistance cou

7 the mercury within [(N-acryloylamino)phenyl] mercuric chloride (APM) polyacrylamide gels, whereas mil

8 A comparison of formalin, PVA, (mercuric chloride based), and STF was done by aliquoting

9 Formalin and mercuric chloride-based low-viscosity polyvinyl alcohol

10 pounds, many laboratories have switched from mercuric chloride-based Schaudinn's and polyvinyl alcoho

11 This AqpB mutant was inhibited by mercuric chloride, confirming the presence of a cysteine

12 ies and are often presented as one-vial, non-mercuric chloride fixatives that aim at performing the s

13 with arylisothiocyanates in the presence of mercuric chloride (HgCl(2)) yielded the corresponding re

14 In susceptible mice treated with mercuric chloride (HgCl(2)), administration of a blockin

15 y astrocyte cultures were exposed to MeHg or mercuric chloride (HgCl2) for 1 or 6 h.

16 lly susceptible H-2s mice, subtoxic doses of mercuric chloride (HgCl2) induce a complex autoimmune sy

17 oablation could confer the ability to resist mercuric chloride (HgCl2)-induced autoimmunity.

18 eased from S-nitrosothiols by treatment with mercuric chloride in an acidic environment.

19 induced by 5 mg/kg subcutaneous injection of mercuric chloride in BALB/c mice.

20 SO acts in part as an antioxidant to inhibit mercuric chloride-induced acute renal injury.

21 DMSO partially prevented a mercuric chloride-induced decrease in glutathione peroxi

22 impairment and reduced tubular apoptosis in mercuric chloride-induced injury.

23 ATc1 expression in a PTC subpopulation after mercuric chloride-induced injury.

24 In contrast, mercuric chloride induces anti-fibrillarin Abs only in S

25 and their resealed ghosts in the dark via a mercuric chloride-inhibitable mechanism and with a speed

26 jected intraperitoneally at 0 and 24 h after mercuric chloride injection, or DMSO treatment was delay

27 ation agents (rho-hydroxymercuribenzoate and mercuric chloride) irreversibly inhibit the ROC1-CUL1 ub

28 yvinyl alcohol (PVA) containing the fixative mercuric chloride is considered the "gold standard" for

29 However, mercuric chloride is potentially hazardous to laboratory

30 in strains of mice, subtoxic doses of HgCl2 (mercuric chloride; mercury) induce a complex autoimmune

31 Neither mercuric chloride nor DMSO affected catalase activity si

32 ated products was optimized with a magnesium/mercuric chloride reagent system and afforded desulfonyl

33 e to hydrogen peroxide or menadione, but not mercuric chloride, resulted in significantly greater lac

34 fers resistance to erythromycin, gentamicin, mercuric chloride, streptomycin, tetracycline-minocyclin

35 Until now, mercuric chloride supported on carbon is used as the cat

36 ury in Balb/c mice and Nfatc1(+/-) mice with mercuric chloride; the PTCs of Nfatc1(+/-) mice demonstr

37 Mercuric chloride treatment of the enzyme results in con

38 The inhibition by mercuric chloride was readily reversible by 2-mercaptoet

39 id and severe injury after administration of mercuric chloride, with increased serum creatinine, incr

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