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1 to inhibition or promotion, respectively, of mesangial cell proliferation.
2 hanism translates into similar regulation of mesangial cell proliferation.
3 monstrate that PDGF-D is a major mediator of mesangial cell proliferation.
4 pivotal role in the mediation of glomerular mesangial cell proliferation.
5 ess mesangial extracellular matrix (ECM) and mesangial cell proliferation.
6 et of PDGF-B signaling and a key mediator of mesangial cell proliferation.
7 of hypertrophy, vascular pole sclerosis, and mesangial cell proliferation.
8 , as well as low density lipoprotein-induced mesangial cell proliferation.
9 onstrictor properties, is known to stimulate mesangial cell proliferation.
10 was found to be most effective in inhibiting mesangial cell proliferation.
11 ix accumulation and is often associated with mesangial cell proliferation.
12 y, these atherogenic lipoproteins stimulated mesangial cell proliferation.
13 iated with lipoprotein-mediated induction of mesangial cell proliferation.
14 HDL at 1000 microg/ml and greater inhibited mesangial cell proliferation.
15 SPARC was correlated with the resolution of mesangial cell proliferation.
16 ates and heparin and heparan sulfate inhibit mesangial cell proliferation.
17 ne) is a matricellular protein that inhibits mesangial cell proliferation and also affects production
18 agement of PDGF receptor-beta in transducing mesangial cell proliferation and demonstrate that PDGF-D
19 IgA nephropathy (IgAN) is characterized by mesangial cell proliferation and extracellular matrix ex
20 tissue remodeling and is thought to regulate mesangial cell proliferation and extracellular matrix pr
23 xylin-eosin (HE) staining revealed increased mesangial cell proliferation and matrix formation compar
24 ritis model, roscovitine treatment decreased mesangial cell proliferation and matrix proteins [1].
25 nd platelet-derived growth factor-stimulated mesangial cell proliferation and promotes IL-6 productio
27 udy show that PF4 is a negative regulator of mesangial cell proliferation and suggest an interference
29 PGN with massive glomerular immune deposits, mesangial cell proliferation, extensive mesangial matrix
31 r of platelet-derived-growth-factor-mediated mesangial cell proliferation in glomerulonephritis and t
32 ment and progression of ECM accumulation and mesangial cell proliferation in this model of diabetic n
38 uria and glomerulosclerosis characterized by mesangial cell proliferation, sclerosis, lipid accumulat
40 The effect of thrombin on DNA synthesis and mesangial cell proliferation was inhibited by standard h
41 r B (PDGF-B) signaling has a pivotal role in mesangial cell proliferation, we examined the regulatory
42 s that podocyte injury frequently results in mesangial cell proliferation, whereas mesangial cell inj
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