ライフサイエンスコーパス: mesencephalic

1 of hESCs to near pure populations of ventral mesencephalic (A9-type) dopaminergic neurons has heighte

2 In vitro studies using primary cultures of mesencephalic and cerebellar granule neurons (CGN) and/o

3 , translational strategy that involved mouse mesencephalic and human induced pluripotent stem cells-d

4 AMPAR, mTOR and BDNF signaling in both mouse mesencephalic and human induced pluripotent stem cells-d

5 The mesencephalic and metencephalic region (MMR) of the vert

6 , pretectal area, nucleus of Darkschewitsch, mesencephalic and pontine reticular formation and pontin

7 coursed in the optic tectum and through the mesencephalic and rhombencephalic basal areas.

8 l investigations have implicated a number of mesencephalic and telencephalic regions in mediating the

9 nfected neurons were detected in a number of mesencephalic and telencephalic regions, >50% of such ne

10 on reaggregate cultures derived from ventral mesencephalic and their striatal target neurons, as well

11 ular nuclei, and several other diencephalic, mesencephalic, and rhombencephalic regions.

12 nal progenitors (NPs) arise from the ventral mesencephalic area by the combined actions of secreted f

13 Mesencephalic astrocyte-derived neurotrophic factor (MAN

14 fusion and induced numerous genes, including mesencephalic astrocyte-derived neurotrophic factor (MAN

15 Mesencephalic astrocyte-derived neurotrophic factor (MAN

16 growth factor (PDGF)-like signaling induced mesencephalic astrocyte-derived neurotrophic factor (MAN

17 Mesencephalic astrocyte-derived neurotrophic factor (MAN

18 determined that ER stress inducible protein Mesencephalic Astrocyte-derived Neurotrophic Factor (MAN

19 s, resulting in the reduced transcription of mesencephalic astrocyte-derived neurotrophic factor (MAN

20 Mesencephalic astrocyte-derived neurotrophic factor (MAN

21 he endoplasmic reticulum (ER) stress protein mesencephalic astrocyte-derived neurotrophic factor (MAN

22 es, which have previously been identified in mesencephalic astrocyte-derived neurotrophic factor, a C

23 cytoprotection by maintaining expression of mesencephalic astrocyte-derived neutrotrophic factor (MA

24 led family are involved in the patterning of mesencephalic boundaries through a mechanism classically

25 how that the positioning of the diencephalic-mesencephalic boundary (DMB) requires Engrailed paracrin

26 se (+) cells and dopamine (DA) uptake in rat mesencephalic cell cultures.

27 5 cells) and dopaminergic neurons in primary mesencephalic cell cultures.

28 nd the dorsal raphe nucleus (DRN), adjoining mesencephalic cell groups that are strategically positio

29 53T alphaS with microsomal membranes in both mesencephalic cells and transgenic mouse brains.

30 and also prevented by MT in MT(trans) fetal mesencephalic cells compared to control(wt) cells.

31 study, we transplanted murine foetal ventral mesencephalic cells into rats rendered hemiparkinsonian

32 Indeed, transplantation of fetal ventral mesencephalic cells into the DA-deficient striatum was f

33 dissociated telencephalic, diencephalic, and mesencephalic cells of E14 mouse embryos with a targeted

34 ore, Fgf8 from the MHB may signal the nearby mesencephalic cells to impart distinct cell surface char

35 se gene (lacZ) indicate that transduction of mesencephalic cells with a helper virus-free HSV amplico

36 icity in MT overexpressing (MT(trans)) fetal mesencephalic cells.

37 AT's postendocytic itinerary in immortalized mesencephalic cells.

38 X from the nidopallium and arcopallium, the mesencephalic central gray, and the dorsolateralis anter

39 disease tremor is localized to pontine- and mesencephalic-cerebellar-thalamic circuits, with abnorma

40 s input from the tectum mesencephali and the mesencephalic command-associated nucleus (MCA).

41 ioid antagonists administered into misplaced mesencephalic control placements ventral and lateral to

42 gic neuronal damage (iv) in vitro in primary mesencephalic cultures and (v) in vivo in a zebrafish sy

43 The addition of PTN to ventral mesencephalic cultures augmented DA neuron survival and

44 tex, or mesencephalon were treated with LPS, mesencephalic cultures became sensitive to LPS at a conc

45 We tested this hypothesis in primary mesencephalic cultures by adding 200 microM L-dopa with

46 n of 5 mum HNE-modified oligomers to primary mesencephalic cultures caused marked neurotoxicity becau

47 In this study, using primary mesencephalic cultures from NADPH oxidase--null (gp91pho

48 on protected dopaminergic neurons in ventral mesencephalic cultures from Parkinson's disease-relevant

49 Consistent with in vivo observations, mesencephalic cultures had fourfold to eightfold more mi

50 Kappa opioid tolerance in mesencephalic cultures has been established as a model s

51 inergic cell line 1RB3AN27 (N27) and primary mesencephalic cultures in vitro and in adult mice in viv

52 Mesencephalic cultures maintained in the presence of DAR

53 Mesencephalic cultures treated with anti-DARP-36aa conta

54 Mesencephalic cultures were also incubated with polyclon

55 Primary ventral mesencephalic cultures were exposed to ziram, and cell t

56 Treatment of mesencephalic cultures with alpha-amino-3-hydroxy-5-meth

57 Treatment of mesencephalic cultures with DARP-36aa, a synthetic pepti

58 In embryonic rat ventral mesencephalic cultures, serum withdrawal led to 80% loss

59 nsfected dopamine neurons from embryonic rat mesencephalic cultures, we find distinct sorting and fat

60 In this study, using primary mesencephalic cultures, we observed that nontoxic or min

61 ating peptide in both MN9D cells and ventral mesencephalic cultures, whereas knockdown of endogenous

62 n death in either Ndufs4(+/+) or Ndufs4(-/-) mesencephalic cultures.

63 reduced expression of these genes in ventral mesencephalic cultures.

64 -mediated neuron survival and development in mesencephalic cultures.

65 DARP-36aa selectively affected mesencephalic cultures; diencephalic and C6 glioma cells

66 describe a robust method for producing human mesencephalic DA neurons from hiPSCs.

67 ves the survival and phenotype expression of mesencephalic DA neurons in culture while simultaneously

68 ival and/or phenotypic expression of ventral mesencephalic DA neurons is dependent on GDNF.

69 Other mesencephalic DA neurons of the ventral tegmental area a

70 such as the anti-apoptotic protein bcl-2, to mesencephalic DA neurons prior to transplantation (ex vi

71 evidence for reduced growth and survival of mesencephalic DA neurons, associated with a decrease in

72 tamatergic cophenotype in the development of mesencephalic DA neurons, opening new perspectives into

73 he type 2 vesicular glutamate transporter in mesencephalic DA neurons, we hypothesized that this coph

74 otentially increase neurotoxicity in ventral mesencephalic DA neurons.

75 differentiation of DA progenitors within the mesencephalic DA system.

76 uts from the precommand nucleus (PCN) at the mesencephalic-diencephalic border and the ventroposterio

77 y factor in the preferential degeneration of mesencephalic dopamine (DA)-synthesizing neurons in PD.

78 rmine if L1 can prevent anoikis in grafts of mesencephalic dopamine neurons after transplantation.

79 Mesencephalic dopamine neurons are central to many aspec

80 nsofar as previous studies implicate ventral mesencephalic dopamine neurons as an essential component

81 ase have shown that transplants of embryonic mesencephalic dopamine neurons form new functional conne

82 Mesencephalic dopamine neurons form synapses with acetyl

83 e measured tyrosine hydroxylase (TH) mRNA in mesencephalic dopamine neurons in human brain and found

84 Individual mesencephalic dopamine neurons therefore have the potent

85 sion of Nurr1 increases the vulnerability of mesencephalic dopamine neurons to dopaminergic toxins.

86 better understand the development of ventral mesencephalic dopamine neurons, we performed subtractive

87 on either does not occur or it is nominal in mesencephalic dopamine neurons.

88 ribe a fourth major pathway originating from mesencephalic dopamine neurons: a mesothalamic system.

89 ge a specific neural circuitry including the mesencephalic dopamine system and its target areas, the

90 sfunction due to progressive degeneration of mesencephalic dopaminergic (DA) neurons in the substanti

91 ophic factor that promotes survival of adult mesencephalic dopaminergic (mDA) neurons and regulates t

92 FGFR1 as essential factors in development of mesencephalic dopaminergic (mDA) neurons.

93 hysiological and neurochemical properties of mesencephalic dopaminergic (mesDA) neurons derived from

94 The mesencephalic dopaminergic (mesDA) system controls movem

95 vented MMT-induced apoptosis in immortalized mesencephalic dopaminergic cells.

96 Exposure of a mesencephalic dopaminergic neuronal cell line (N27 cells

97 In mesencephalic dopaminergic neuronal cells, dieldrin indu

98 f Akt/mTor signaling in surviving endogenous mesencephalic dopaminergic neurons by viral vector trans

99 To pursue these questions, mesencephalic dopaminergic neurons derived from C57BL/6

100 kinson's disease has been questioned because mesencephalic dopaminergic neurons do not degenerate dur

101 1 is required for the development of ventral mesencephalic dopaminergic neurons in mice.

102 atment, strongly suggest that dysfunction of mesencephalic dopaminergic neurons in Pink1(-/-) mice is

103 heless, we have previously demonstrated that mesencephalic dopaminergic neurons in primary monolayer

104 at nicotine-induced structural plasticity at mesencephalic dopaminergic neurons involves alpha4beta2

105 minergic SH-SY5Y cells and fetal rat ventral mesencephalic dopaminergic neurons protected against 6-h

106 We found that mutant primary cortical and mesencephalic dopaminergic neurons were more susceptible

107 Ventral mesencephalic dopaminergic neurons were used to assess t

108 nologically detectable within MPP(+)-treated mesencephalic dopaminergic neurons, dopaminergic nigral

109 s, in both transfected cells and rat primary mesencephalic dopaminergic neurons, suggesting heteromer

110 nifies a differential effect of E on ventral mesencephalic dopaminergic neurons.

111 blation of dopamine (DA) D2 autoreceptors in mesencephalic dopaminergic neurons.

112 issue with use of primary cultures of mouse mesencephalic dopaminergic neurons.

113 nicotinic acetylcholine receptors (nAChR) in mesencephalic dopaminergic neurons.

114 ryonic primary cerebral cortical and ventral mesencephalic (dopaminergic) neurons.

115 active hybridization screens to find ventral mesencephalic genes expressed at rat embryonic day 10 wh

116 n with PD received bilateral putaminal fetal mesencephalic grafts as part of an NIH-sponsored double-

117 culture results, tenascin- and L1 Ab-treated mesencephalic grafts did not yield an increase in the nu

118 There are defined medullary, mesencephalic, hypothalamic, and thalamic functions in r

119 e spinal cord, as well as various medullary, mesencephalic, hypothalamic, thalamic, and telencephalic

120 At mesencephalic levels fibers reach the deep layers of the

121 The mesencephalic locomotor region (MLR) has been shown to b

122 ation increases following stimulation of the mesencephalic locomotor region (MLR) in an in vitro isol

123 We found that electrical stimulation of the mesencephalic locomotor region (MLR) inhibited the disch

124 Because electrical stimulation of the mesencephalic locomotor region (MLR) is known to elicit

125 In vertebrates, stimulation of the mesencephalic locomotor region (MLR) on one side evokes

126 comotor movements.SIGNIFICANCE STATEMENT The mesencephalic locomotor region (MLR) plays a crucial rol

127 The mesencephalic locomotor region (MLR) plays a crucial rol

128 s believed to be organized serially from the mesencephalic locomotor region (MLR) to reticulospinal n

129 l ganglia, which in turn project down to the mesencephalic locomotor region (MLR), a brainstem region

130 and indirect pathway-mediated control of the mesencephalic locomotor region (MLR), a brainstem target

131 ch overlaps with the physiologically defined mesencephalic locomotor region (MLR), is necessary for t

132 The mesencephalic locomotor region (MLR), which includes the

133 ne nucleus is one of the major nuclei of the mesencephalic locomotor region and has neurons related t

134 ore, they receive functional inputs from the mesencephalic locomotor region and have electrophysiolog

135 However, the existence of a mesencephalic locomotor region and of an arousal center

136 Pathways derived from the mesencephalic locomotor region and pontomedullary medial

137 ed from the brainstem (by stimulation of the mesencephalic locomotor region, MLR) with locomotion evo

138 ught to be initiated by projections from the mesencephalic locomotor region, the latter through a dis

139 ise induced by electrical stimulation of the mesencephalic locomotor region.

140 sal to the PPN corresponds to the descending mesencephalic locomotor region.

141 Using Lund Human Mesencephalic (LUHMES) cells, we show that NRG/ErbB sign

142 Serotonergic neurons in the mesencephalic median raphe nucleus (MnR) also give rise

143 Trigeminal mesencephalic (Mes V) neurons are critical components of

144 the neural tube which eventually defines the mesencephalic/ metencephalic boundary (MMB).

145 volving FGF and Wnt proteins produced at the mesencephalic/metencephalic boundary.

146 ains to develop longer, we detect changes in mesencephalic morphology.

147 In contrast to trunk neural crest cells, mesencephalic neural crest cells apparently fail to expr

148 Thus, a subset of mesencephalic neural crest cells fails to express dHAND

149 Here, we show that migrating quail mesencephalic neural crest cells grafted into the trunk

150 ts suggest that a subpopulation of migrating mesencephalic neural crest cells is refractory to catech

151 Mesencephalic neural crest cells normally form cholinerg

152 We now show that, in vitro, mesencephalic neural crest cells respond to exogenous FG

153 Furthermore, the proportion of quail mesencephalic neural crest cells that is TH+ in the symp

154 uman neuroblastoma cell line and rat primary mesencephalic neuron cultures, the present study examine

155 The results demonstrated that in a primary mesencephalic neuron-glia culture system, extracellular

156 rotected dopaminergic neurons in rat primary mesencephalic neuron-glia cultures against lipopolysacch

157 lpyridinium (MPP+) -treated C57 mice primary mesencephalic neuron-glia cultures as an in vitro Parkin

158 In this study using mesencephalic neuron-glia cultures as an in vitro PD mod

159 Our study using primary mesencephalic neuron-glia cultures showed that 3-HM prov

160 ively toxic to dopaminergic neurons in mixed mesencephalic neuron-glia cultures through nicotinamide

161 We report that mesencephalic neuron-glia cultures treated with diesel e

162 fects on dopaminergic neurons in rat primary mesencephalic neuron-glia cultures treated with LPS.

163 In mesencephalic neuron-glia cultures, DM significantly red

164 Using mesencephalic neuron-glia cultures, we address the novel

165 In this study, using rat primary mesencephalic neuron-glia cultures, we report that both

166 cMMP-3 caused DA cell death in mesencephalic neuron-glia mixed culture of wild-type (WT

167 oluble aggregates, we probed the cytosols of mesencephalic neuronal (MES) cells, normal and alpha S-t

168 gh a systematic centrifugal fractionation of mesencephalic neuronal cell lines and transgenic mouse b

169 s) enhanced alphaS oligomerization in intact mesencephalic neuronal cells.

170 opaminergic neuronal degeneration in primary mesencephalic neuronal cultures.

171 ease in Ser19 phosphorylation in rat primary mesencephalic neuronal cultures.

172 Here we show that treatment of primary mesencephalic neurons (48 h) or subchronic treatment of

173 report here that benomyl exposure in primary mesencephalic neurons (ii) inhibits ALDH and (iii) alter

174 n of differentiating neuroblastoma cells and mesencephalic neurons and axon outgrowth and sprouting o

175 d increased biotinylated DAT levels, in both mesencephalic neurons and cotransfected cells.

176 DAT was colocalized with alpha-synuclein in mesencephalic neurons and cotransfected Ltk- cells.

177 s in C2-ceramide-treated primary cultures of mesencephalic neurons and differentiated pheochromocytom

178 ignificant accumulation of alpha-Syn in both mesencephalic neurons and in WT mice striatum.

179 the brains were processed to exhibit ventral mesencephalic neurons containing FG.

180 ene abolished complex I activity in midbrain mesencephalic neurons cultured from embryonic day (E) 14

181 Although LPS appeared to impact upon mesencephalic neurons in general, an extensive loss of d

182 Major sites of DA synthesis are the mesencephalic neurons originating in the substantia nigr

183 ing animals, we find that the DA signal from mesencephalic neurons projecting to the NAc shell is dom

184 , we treated cotransfected cells and primary mesencephalic neurons with either colchicine, vinblastin

185 lization of CRF-BP and CRF2R in cultured rat mesencephalic neurons, and the localization and interact

186 e rat brain tissue as well as in cultures of mesencephalic neurons, further excluding the possibility

187 sin increased dopamine uptake in rat primary mesencephalic neurons, suggesting that DAT activity is a

188 f Parkinsonism (SH-SY5Y cotransfected cells, mesencephalic neurons, transgenic mice overexpressing al

189 f p-GSK-3beta in nuclei of SH-SY5Y cells and mesencephalic neurons.

190 d morphological development of embryonic rat mesencephalic neurons.

191 ely promotes the survival and development of mesencephalic neurons.

192 nd phenotypic expression of dopamine ventral mesencephalic neurons.

193 rally colocalize in the neurites of cultured mesencephalic neurons.

194 4) on superoxide levels in individual living mesencephalic neurons.

195 cts on glia and no effect on nondopaminergic mesencephalic neurons.

196 cell loss in a small subset of brainstem and mesencephalic nuclei and widespread aggregation of the a

197 licated the majority of the diencephalic and mesencephalic nuclei in electrosensory, visual, and acou

198 mediodorsal thalamus, dorsal raphe, and deep mesencephalic nuclei, and sparse projections go to prefr

199 al gray (VLPAG) and the adjacent dorsal deep mesencephalic nucleus (dDpMe) contain GABAergic neurons

200 deep layers of the superior colliculus/deep mesencephalic nucleus (deep SC/DpMe), and the lateral me

201 otransmitters is from the region of the deep mesencephalic nucleus (DpMe) just ventrolateral to the p

202 e primary afferent neurons in the trigeminal mesencephalic nucleus (Vmes) was examined.

203 midbrain, both the optic tectum and lateral mesencephalic nucleus contained numerous Gly-ir neurons.

204 l synapses between neurons of the trigeminal mesencephalic nucleus in rat brain slices are similarly

205 (PCRt) receives projection of the trigeminal mesencephalic nucleus neurons.

206 , the hippocampus, the substantia nigra, the mesencephalic nucleus of the trigeminal nerve, the cochl

207 r hypothalamus, precommissural nucleus, deep mesencephalic nucleus, and periaqueductal gray (PAG) of

208 um, the torus semicircularis and the lateral mesencephalic nucleus.

209 s; and midbrain superior colliculus and deep mesencephalic nucleus.

210 non-specifically toxic to neurons in ventral mesencephalic organotypic slice cultures, indicating tha

211 We show a common mesencephalic origin for these signals evident in overla

212 paminergic signaling is a hallmark of severe mesencephalic pathologies such as schizophrenia and psyc

213 by the emotional motor system, in which the mesencephalic periaqueductal gray (PAG) plays a central

214 activity within the brainstem, including the mesencephalic pontine reticular formation, and the anter

215 ralateral superior colliculus, dorsal raphe, mesencephalic, pontine and medullary reticular formation

216 withdrawal induce a rise in activity in the mesencephalic-pontine reticular formation (MPRF), an are

217 e demonstrate how a previously unappreciated mesencephalic population controls thalamic relay neuron

218 ), which receives dense projections from the mesencephalic precommand nucleus (PCN) and the adjacent

219 nvironment, we analyzed embryonic day 12 rat mesencephalic precursor cells in traditional cultures wi

220 orylation and activity in a cell line and in mesencephalic primary culture cells.

221 single amphioxus region, which we termed Di-Mesencephalic primordium (DiMes).

222 e importance of cortico-striatal and cortico-mesencephalic projections remains unclear, particularly

223 nuclei of the amygdala, ventral hippocampus, mesencephalic raphe nuclei, and novel localizations in t

224 al tegmental area, the rhabdoid nucleus, the mesencephalic raphe nuclei, and the dorsal tegmental nuc

225 trate that ex vivo gene transfer of bcl-2 to mesencephalic reaggregates is ineffective in increasing

226 xpressing HSV-TH9bcl-2 amplicon to transduce mesencephalic reaggregates.

227 mammillary, ventral lateral geniculate, deep mesencephalic, red, pedunculopontine and laterodorsal te

228 the midbrain are proposed to arise from the mesencephalic region of the neural tube.

229 pedunculopontine tegmental nucleus (PPTg), a mesencephalic region that provides input to several nucl

230 ll detectable by immunohistochemistry in the mesencephalic region, with particularly intense expressi

231 ring which op5 precursors are present in the mesencephalic region.

232 rug words in cocaine-addicted individuals in mesencephalic regions as possibly associated with dopami

233 ine users only, these increased drug-related mesencephalic responses were associated with enhanced ve

234 Neurones in the central mesencephalic reticular formation (cMRF) begin to discha

235 Since the central mesencephalic reticular formation (cMRF) is a major SC t

236 to the supraoculomotor area from the central mesencephalic reticular formation (cMRF), a region impli

237 deep layers of the superior colliculus/deep mesencephalic reticular formation (deep SC/Me) mediates

238 n the deep layers of the superior colliculus/mesencephalic reticular formation (deep SC/Me).

239 The mesencephalic reticular formation (MRF) is formed by the

240 alic nucleus (deep SC/DpMe), and the lateral mesencephalic reticular formation (MRF) that in turn pro

241 l mapping of the two major components of the mesencephalic reticular formation (MRF), namely the pedu

242 ve correlation between prefrontal cortex and mesencephalic reticular formation (RF) activity, and a w

243 entrolateral PAG, the cuneiform nucleus, the mesencephalic reticular formation, and the superior coll

244 ortex, hippocampal formation, colliculi, and mesencephalic reticular formation.

245 cular nucleus, retrorubral area, and lateral mesencephalic reticular nucleus), respiratory control (l

246 mic site received a projection from the deep mesencephalic reticular, pedunculopontine tegmental, dor

247 be identified in the brain as the trigeminal mesencephalic root, some Schnauzenorgan trigeminal affer

248 nd nucleus of the posterior tuberculum); (3) mesencephalic sensory structures (optic tectum, dorsal a

249 This study aimed to investigate the mesencephalic striatal and extrastriatal dopaminergic pr

250 es in HeLa and PAE cells, and in primary rat mesencephalic-striatal neuronal cocultures.

251 the telencephalon and diencephalon and some mesencephalic structures of A. burtoni.

252 of lower brainstem involvement accompanying mesencephalic synucleinopathy is often observed.

253 ranial neural crest, dorsal neural tube, and mesencephalic tectum, pretectum, and base, and at the mi

254 telencephalon, caudal preoptic area, dorsal mesencephalic tegmentum, and rostral rhombencephalon.

255 ptic tectum and torus semicircularis, in the mesencephalic tegmentum, in the cerebellar crest, in the

256 c areas, optic tectum, torus semicircularis, mesencephalic tegmentum, interpeduncular nucleus, superi

257 A network of activations in pons, midbrain (mesencephalic tegmentum, parabrachial nucleus, and peria

258 bulbs, pallium, basal ganglia, diencephalon, mesencephalic tegmentum, rhombencephalon, and spinal cor

259 enzyme form expressed in brain extracts from mesencephalic tegmentum, striatum, and hippocampus with

260 tectum and also course sparsely through the mesencephalic tegmentum.

261 BA neurons and show that they occupy ventral mesencephalic territory in a temporally and spatially sp

262 data indicate that the survival of embryonic mesencephalic TH-ir neurons is increased when Epo is adm

263 Clinical use of allografts of fetal ventral mesencephalic tissue as a treatment to replace dopaminer

264 Intrastriatal grafts of embryonic mesencephalic tissue can survive in the brains of patien

265 Ventral mesencephalic tissue from dopamine phosphatase and tensi

266 In the transplant recipients, cultured mesencephalic tissue from four embryos was implanted int

267 Intrastriatal transplants of human foetal mesencephalic tissue in Parkinson's patients have demons

268 that xenografts of embryonic porcine ventral mesencephalic tissue in the 6-hydroxydopamine-lesioned,

269 AF5 neural cells derived from fetal rat mesencephalic tissue were immortalized with a truncated

270 miparkinsonian rat, preincubation of ventral mesencephalic tissue with PD169316 prior to transplantat

271 intrastriatal grafts of human fetal ventral mesencephalic tissue, rich in dopaminergic neuroblasts,

272 ease is transplantation of embryonic ventral mesencephalic tissue.

273 side-effect of intrastriatal foetal ventral mesencephalic transplantation in patients with Parkinson

274 D) has been clearly shown with fetal ventral mesencephalic transplants, albeit inconsistently.

275 ide enhanced benefits in comparison to fetal mesencephalic transplants.

276 The mesencephalic trigeminal (MesV) nucleus, formed by the s

277 Stained mesencephalic trigeminal jaw-muscle spindle afferent axo

278 ive contacts between intracellularly stained mesencephalic trigeminal jaw-muscle spindle afferent bou

279 embrane oscillations and burst generation in mesencephalic trigeminal neurons (Mes V).

280 In order to determine whether the mesencephalic trigeminal neurons may receive a direct hy

281 ere observed traveling toward and contacting mesencephalic trigeminal neurons, some of which were mul

282 projected to the ventral paratrigeminal and mesencephalic trigeminal nuclei.

283 elevated transgene expression throughout the mesencephalic trigeminal nucleus (Me5) of the brain stem

284 n A immunoreactivity was examined in the rat mesencephalic trigeminal nucleus (MTN), using orexin A i

285 A direct projection from rat mesencephalic trigeminal nucleus (Vme) neurons to the hy

286 Jaw muscle spindle afferents (JMSA) in the mesencephalic trigeminal nucleus (Vme) project to the pa

287 the presence of the descending tract of the mesencephalic trigeminal nucleus in Alligator and other

288 ant reticular cells and large neurons in the mesencephalic trigeminal nucleus were immunoreactive for

289 muscle spindle afferent axons located in the mesencephalic trigeminal nucleus were physiologically id

290 ucleus of lateral lemniscus, pontine nuclei, mesencephalic trigeminal nucleus, external cuneate nucle

291 motor nuclei, as well as the proprioceptive mesencephalic trigeminal nucleus.

292 intense in sensory nerve tracts such as the mesencephalic trigeminal tract, vestibulospinal tract, o

293 /LacZ reporter in the sensory neurons of the mesencephalic trigeminal, but not other Brn3a midbrain n

294 hereas normal numbers were found for sensory mesencephalic trigeminal, facial, and trochlear motoneur

295 rpin RNAs at E16 into cells along the dorsal mesencephalic ventricle interferes with their normal mig

296 developing mandible, maxilla, intestine, and mesencephalic ventricle.

297 PC12-alphaSyn) cells and rat primary ventral mesencephalic (VM) cultures exposed to MPP+ neurotoxicit

298 Primary ventral mesencephalic (VM) cultures from E15 rats were grown for

299 Transplantation of embryonic ventral mesencephalic (VM) DA neurons into the striatum is a cur

300 of normal anatomic projections from ventral mesencephalic (VM) grafts placed in the substantia nigra