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1 ithin the rostral PAG (rPAG) and surrounding mesencephalon.
2 wing intrastriatal grafting of fetal ventral mesencephalon.
3 (GFRalpha1) and GFRalpha2 within the ventral mesencephalon.
4 lating growth and polarity in the developing mesencephalon.
5 sory ganglia and in structures caudal to the mesencephalon.
6  caudal to rostral gradient in the posterior mesencephalon.
7 H, particularly in the telencephalon and the mesencephalon.
8 nervous system, such as the diencephalon and mesencephalon.
9 nsula, anterior cingulate, hypothalamus, and mesencephalon.
10 t the neuraxis from frontal neocortex to the mesencephalon.
11 ry ganglia adjacent to the developing dorsal mesencephalon.
12 ncovered exquisite ventral patterning in the mesencephalon.
13 the DRN and periaqueductal gray (PAG) of the mesencephalon.
14 Lmx1b/RCAS) maintains Wnt1 expression in the mesencephalon.
15  actions in the brainstem, hypothalamus, and mesencephalon.
16 tract at the level of the pretectum/anterior mesencephalon.
17 tosidase expression in the telencephalon and mesencephalon.
18 ned following transection through the caudal mesencephalon.
19  suspensions of embryonic day 15 rat ventral mesencephalon.
20 jecting neurons that remained in the ventral mesencephalon after these lesions, 54% did not co-locali
21                          Imaging covered the mesencephalon and basal ganglia.
22 bomeres 3 and 5 and ventricular zones in the mesencephalon and diencephalon.
23             Wnt-1 is expressed in the caudal mesencephalon and Fgf8 in the most rostral metencephalon
24 axons of the dopaminergic cell groups of the mesencephalon and hypothalamus, whereas the D2 long is m
25 en together, these studies indicate that the mesencephalon and metencephalon develop as one independe
26 ecting the molecular and cellular control of mesencephalon and metencephalon development in vertebrat
27 ns are generated at the dorsal border of the mesencephalon and metencephalon, the rhombic lip.
28 was found in discrete zones of the embryonic mesencephalon and myelencephalon.
29  who suffered severe injury to the tegmental mesencephalon and paramedian thalamus showed widely pres
30 amic nuclei, dopaminergic cell groups in the mesencephalon and pons, the principal nucleus of the tri
31 ves and cerebellar plate, suggests that both mesencephalon and rhombomere 1 (r1) are delected, with t
32                          Cells in the caudal mesencephalon and rostral metencephalon become organized
33           Similarly, the compartments of the mesencephalon and the main rhombencephalic regions, incl
34 ue was divided between the caudal end of the mesencephalon and the rostral end of the rhombencephalon
35 gnaling within the embryonic mouse midbrain (mesencephalon) and cerebellum (rhombomere 1) by misexpre
36 as of parenchymal inflammation and necrosis (mesencephalon) and in leptomeningeal and white matter pe
37 ms were found in the thalamus, diencephalon, mesencephalon, and brainstem.
38 ptic area and the isthmal gray region of the mesencephalon, and found labeled fibers throughout the p
39  archistriatum, hypothalamic regions, dorsal mesencephalon, and in and around the brainstem nucleus t
40  the secondary prosencephalon, diencephalon, mesencephalon, and isthmus showed some deviation from th
41 were lower in hippocampus, septum, thalamus, mesencephalon, and pons in knock-out mice than wild-type
42 the cranial neural crest, facial mesenchyme, mesencephalon, and pontine reticular nucleus.
43 on zones in the telencephalon, diencephalon, mesencephalon, and rhombencephalon that were primarily a
44 elencephalon, preoptic region, hypothalamus, mesencephalon, and rhombencephalon.
45 sed in discrete regions of the diencephalon, mesencephalon, and rhombencephalon.
46 napses are abundantly present within grafted mesencephalon, and that these contacts are enriched in a
47 ed into control misplacements in the lateral mesencephalon, and the small hyperalgesia elicited by op
48 ing within the human lifetime to involve the mesencephalon, and thereby cause the substantia nigra pa
49                                           In mesencephalon, AptCB1R mRNA is expressed in deep layers
50           The transgene was expressed in the mesencephalon as early as embryonic day 10 and in the hi
51 reactivities in sections through the ventral mesencephalon at 3, 7, 10, 14 and 21 days after 6-hydrox
52 uclei, tectal nuclei, and other areas of the mesencephalon became more diffuse after maturity, wherea
53 lic cell density were lost, the diencephalon/mesencephalon boundary was unclear and the third ventric
54  Pax-6 expression defines the prosencephalon-mesencephalon boundary, and mutant embryos lack this mor
55 tyrosine hydroxylase activity in the ventral mesencephalon, but did not affect striatal activity of c
56 expression of Sonic Hedgehog (Shh) in dorsal mesencephalon can recapitulate this patterning in its en
57                 Interestingly, the remaining mesencephalon cells develop into anterior midbrain, indi
58                                       In the mesencephalon, cells retrogradely labeled from the RVM w
59 h of the turkey telencephalon, diencephalon, mesencephalon, cerebellum, pituitary, and pineal gland.
60 much of the rat telencephalon, diencephalon, mesencephalon, cerebellum, spinal cord, and dorsal root
61 as detected in developing cerebellum, dorsal mesencephalon, cerebral cortex, and epithalamus, but not
62 lateral spiriform nucleus (SpL) in the chick mesencephalon contains functional nicotinic receptors an
63 t integrated within the diencephalon and the mesencephalon continued to express a telencephalic marke
64 ell line - the large T-antigen immortalized, mesencephalon-derived 1RB3AN27 (N27).
65                The commonality of regions of mesencephalon, diencephalon and limbic/paralimbic areas
66 rin beta-1 (CA*beta1) in the embryonic chick mesencephalon, enhances neurogenesis and increases the n
67 evious microinjection mapping studies in the mesencephalon established a significant association betw
68 , we examined four subregions of the ventral mesencephalon for differential expression of NR subunit
69 s using cells derived from embryonic ventral mesencephalon have shown that transplanted dopaminergic
70                                       In the mesencephalon, immunoreactive cell bodies were found onl
71     Increased gray matter mainly in the left mesencephalon in 31 TS patients.
72 g neuronal migration and connectivity in the mesencephalon in normal as well as diseased brains.
73  words, but not neutral words, activated the mesencephalon in the cocaine users only.
74 h leads to transformation of the presumptive mesencephalon into an extended rhombomere 1 (r1).
75 precursor cells derived from E12 rat ventral mesencephalon into dopaminergic neurons.
76 ion of dopamine cells derived from the fetal mesencephalon is associated with a potentially disabling
77  lost (Pax-6, Lim-1, Gsh-1) and a marker for mesencephalon is expanded rostrally into the prosencepha
78            Ectopic expression of Fgf8 in the mesencephalon is sufficient to activate expression of En
79 ing rostral-to-caudal gradient in the dorsal mesencephalon is the earliest known marker for polarity
80 ax7 specifically labeled cells in the dorsal mesencephalon, mainly in the optic tectum, and Pax6 cell
81 P) and dorsal-ventral (DV) patterning of the mesencephalon (mes) and rhombomere 1 (r1) is instrumenta
82            Brain structures derived from the mesencephalon (mes) and rhombomere 1 (r1) modulate disti
83 rising two distinct territories known as the mesencephalon (mes) and rostral metencephalon (met; rhom
84 veal aspects of mouse gene regulation in the mesencephalon/metencephalon that have been difficult to
85  in initiating normal Pax5 expression in the mesencephalon/metencephalon.
86 he obscurus (NRO) and pallidus (NRP)] or the mesencephalon [n. raphe dorsalis (DRN)] was recorded in
87 eceived embryonic VM grafts into the rostral mesencephalon near the host SN, and injections of adeno-
88 ere used as substrates for embryonic ventral mesencephalon neurons that were plated on their surfaces
89 tentially toxic levels in vulnerable ventral mesencephalon neurons.
90 e hypothesis that ectopically placed ventral mesencephalon not only produces, but maintains the relea
91 es) throughout the forebrain and part of the mesencephalon of A. burtoni.
92 k of orthodenticle homeobox 2 in the ventral mesencephalon of orthodenticle homeobox 2 conditional kn
93 ied out in primary cultures derived from the mesencephalon of rat embryos.
94 ducible ErbB4 virus (AAV-ErbB4.DIO) into the mesencephalon of TH-Cre;ErbB4(f/f) mice, which selective
95    Dopamine cells collected from the ventral mesencephalon of the cloned fetuses 42 to 50 days post-c
96 ntation with enriched microglia derived from mesencephalon or cortex rendered LPS-insensitive cortica
97 ultivation with either fetal porcine ventral mesencephalon or porcine fetal lateral ganglionic eminen
98                                       In the mesencephalon, PITX2-labeled nuclei also appear in diffe
99 r colliculus (SC)/optic tectum of the dorsal mesencephalon plays a major role in responses to visual
100 olliculus, red nucleus, periaqueductal gray, mesencephalon, pons, and several nuclei involved in ocul
101 erved in multiple areas in the diencephalon, mesencephalon, pons/medulla, and spinal cord.
102 e found that primary cultures of rat ventral mesencephalon responded to PDNF by increasing the number
103 n the diencephalon (pretectum and thalamus), mesencephalon (reticular formation and nucleus ruber), r
104 dal portion of the neural tube, reaching the mesencephalon rostrally.
105 droxylase (TH) immunostaining in the ventral mesencephalon showed two subpopulations of dopaminergic
106                    The developing vertebrate mesencephalon shows a rostrocaudal gradient in the expre
107                                       In the mesencephalon, sonic hedgehog (Shh) specifies a ventral
108 asal regions of the diencephalon and rostral mesencephalon (substantia nigra/ventral tegmental area);
109 oxins of dopaminergic neurons in the ventral mesencephalon that project to the core and medial shell
110 fter grafting with non-DA cells (from dorsal mesencephalon), the DA binding ratio remained reduced to
111    Using organotypic cultures of rat ventral mesencephalon, the effects of chronic (12-15 day) exposu
112 ry projections from the DLPFC to the ventral mesencephalon, the location of dopamine cells projecting
113  reduction causes cell death in the anterior mesencephalon, the region furthest from the source of FG
114 ing mDA progenitors from rat primary ventral mesencephalon through flow cytometry.
115 fying effects; and (4) implantation of fetal mesencephalon tissue and other grafts may be effective i
116                    Grafting of fetal ventral mesencephalon to the dorsal striatum of MPTP-treated mon
117 ing transplantation of the embryonic ventral mesencephalon to the striatum have focused upon the part
118 ng the effect of transplanting fetal ventral mesencephalon to the striatum of 1-methyl-4-phenyl-1,2,3
119 ity of these neurons remained in the ventral mesencephalon until at least 18 months of age.
120 ltures of rat embryonic day 14 (E14) ventral mesencephalon (VM) and E21 striatum, we have described t
121                                      Ventral mesencephalon (VM) and lateral ganglionic eminence cells
122 from embryonal tissue dissected from ventral mesencephalon (VM) and spinal cord (SC).
123  were then exposed to E11 developing ventral mesencephalon (VM) in explant culture.
124 ntrated on the implantation of fetal ventral mesencephalon (VM) into the striatum in attempts to rest
125 rosine hydroxylase expression in the ventral mesencephalon (VM) neuron.
126 eceived implants of embryonic monkey ventral mesencephalon (VM), or sham implants, aimed at the rostr
127 xpressing progenitors located in the ventral mesencephalon (vMes) during embryogenesis.
128 dbrain dopamine (mDA) neurons in the ventral mesencephalon (vMes).
129                          Tissue from ventral mesencephalon was transplanted into a single site in dop
130 riched and neuron/glia cultures from the rat mesencephalon, we discovered an extraordinary feature fo
131  from embryonic day 14.5 (E14.5) rat ventral mesencephalon, we show that Shh is also trophic for dopa
132 , primary cultured dopaminergic neurons from mesencephalon were more sensitive to rotenone-induced ce
133 eurons in grafts, strands of E15 rat ventral mesencephalon were pretreated with a combination of GDNF
134 derived from the rat hippocampus, cortex, or mesencephalon were treated with LPS, mesencephalic cultu
135 eal that FGF8 is a potent mitogen within the mesencephalon: when ectopically expressed, neural precur
136 opaminergic (DAergic) neurons in the ventral mesencephalon where it converts 3,4-dihydroxyphenylaceta
137 ne abusers showed: (1) hypoactivation in the mesencephalon, where dopamine neurons are located, as we
138 velopment, remains largely unexplored in the mesencephalon, where dopaminergic (DA) and GABA neurons
139 -related words can trigger activation in the mesencephalon, where dopaminergic cells are located.
140  receptors in regions of the feline pons and mesencephalon which are involved in the generation of RE
141 lectrical stimulation to target areas in the mesencephalon which inhibit RO.
142 brid library from developing mouse embryonic mesencephalon with the Nurr1 ligand-binding domain (NLBD
143 iently expressed in the rat embryonic dorsal mesencephalon within a restricted region between prolife

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