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1 ase (MMP)2 are present and active in cushion mesenchymal tissue.
2 re but diverse malignant tumors derived from mesenchymal tissue.
3 een axons, Schwann cells and the surrounding mesenchymal tissue.
4 hese cells, luminal bacteria, and underlying mesenchymal tissue.
5 yclin D1 (n = 7; RQ 1 vs. 0.61, P < 0.05) in mesenchymal tissue.
6 arrest of epithelial invasion into the cecal mesenchymal tissue.
7 ate growth signals in various epithelial and mesenchymal tissues.
8 in for ALT immortalization in cell lines and mesenchymal tissues.
9 ute to the regeneration of multiple types of mesenchymal tissues.
10 cells (MSC) can differentiate into multiple mesenchymal tissues.
11 hat was dependent upon midline cartilaginous/mesenchymal tissues.
12 ustaining epithelial stem cells and adjacent mesenchymal tissues.
13 ion factor that regulates the development of mesenchymal tissues.
15 an increase in apoptosis in both condensing mesenchymal tissues and cartilage of the nasal region in
16 h due to a splicing switch express Fgfr2b in mesenchymal tissues and manifest Apert syndrome-like phe
17 can be induced to differentiate into various mesenchymal tissues and trans-differentiate into some no
18 gene that is expressed during development in mesenchymal tissues and ventrally derived structures wit
21 isms by which these undifferentiated cushion mesenchymal tissues are remodeled "post-EMT" into mature
23 the basement membrane lying between this and mesenchymal tissues, both of which express Fli1 at the t
26 ction in lung volume was due to loss of lung mesenchymal tissue correlating with a decrease in cell p
30 often called "laminopathies," mainly affect mesenchymal tissues (e.g., striated muscle, bone, and fi
31 tic value not only for the repair of damaged mesenchymal tissues following hematopoietic stem cell tr
32 nolytic activities required in collagen-rich mesenchymal tissues for extracellular matrix remodeling
33 this study we have recombined epithelial and mesenchymal tissues from normal and LEF-1-deficient embr
35 co-expressed at high levels in neuronal and mesenchymal tissues in the developing mouse, and at vari
36 differentiate into mature cells of multiple mesenchymal tissues including fat, bone, and cartilage.
37 in patients with genetic disorders affecting mesenchymal tissues, including bone, cartilage, and musc
38 he potential to differentiate to lineages of mesenchymal tissues, including bone, cartilage, fat, ten
39 d in vivo to differentiate into a variety of mesenchymal tissues, including bone, cartilage, tendon,
41 stinct mechanisms: direct differentiation to mesenchymal tissues, including skeletal and smooth muscl
42 e the capacity to differentiate into several mesenchymal tissues, including the components of the hem
43 ontext in the absence of direct contact with mesenchymal tissue, indicating that the program for bran
45 However, a bud of undifferentiated cecal mesenchymal tissue is maintained throughout development.
46 w stroma (hMSCs) differentiate into numerous mesenchymal tissue lineages and are attractive candidate
47 ced cells that can regenerate epithelial and mesenchymal tissues may potentially be utilized in limb
48 al allelic expression (DAE) of BMP5 in human mesenchymal tissues obtained from 16 donors undergoing j
49 embrane-type 3 MMP, MT3-MMP, is expressed in mesenchymal tissues of the skeleton and in peri-skeletal
50 er between the kidney and ureter forms where mesenchymal tissues originating in two different areas o
52 iver regeneration in host animals by forming mesenchymal tissue, progenitor cells, hepatocytes, and c
55 related processes in tumors originating from mesenchymal tissues, such as bone and soft-tissues sarco
56 (day 10.5-18.5), TIMP-2 mRNA was abundant in mesenchymal tissues that surrounded developing epithelia
57 ) is a tyrosine kinase receptor expressed in mesenchymal tissues, the ligand of which is fibrillar co
59 enetic protein (BMP) antagonist expressed in mesenchymal tissues whose function in development of the
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