ライフサイエンスコーパス: mesenteric

1 lved the lower extremity (58%) than visceral-mesenteric (31%) or upper extremity (10%).

2 refore, blocking IL-6 cytokine signaling in (mesenteric) adipocytes may be a novel approach to blunti

3 lso documents decreased lipogenic pathway in mesenteric adipose tissue after HFD and/or OVX, independ

4 tion and associated lymphatic leakage in the mesenteric adipose tissue deviates migratory dendritic c

5 perpermeable lymphatic collecting vessels in mesenteric adipose tissue that impair antigen and immune

6 r-HuBDNF alone could cause mesenteric afferent mechanical hypersensitivity independ

7 augmented discharges in isolated intestinal mesenteric afferent nerves.

8 nstrated a temporally associated increase in mesenteric and colonic vascularity with an increase in m

9 nstrated a temporally associated increase in mesenteric and colonic vascularity with an increase in m

10 ch were generally accompanied by significant mesenteric and hindquarters, but not renal, vasoconstric

11 a progressive myogenic tone augmentation in mesenteric and olfactory cerebral arteries; neither HFD

12 Inflammation of mesenteric and pancreatic lymph node cells was also eval

13 ve evidence that myogenic responses of mouse mesenteric and renal arteries rely on ligand-independent

14 theterized anteroposterior (celiac, superior mesenteric, and inferior mesenteric arteries) and mediol

15 , ameliorated hyperdynamic circulation, PSS, mesenteric angiogenesis, hepatic angiogenesis, and fibro

16 uated platelet accumulation in FeCl3-induced mesenteric arterial injury.

17 d flow volume, while the celiac and superior mesenteric arterial RI is increased.

18 11,12-epoxyeicosatrienoic acid (EET) induces mesenteric arterial vasodilation, which contributes to t

19 h-old) were tested for vascular functions in mesenteric arteries (MA) and ion channel activities in s

20 hesis that functional sensory innervation of mesenteric arteries (MAs) is impaired for Old (24 months

21 The dilatory role for sensory innervation of mesenteric arteries (MAs) is impaired in Old ( approxima

22 e placed in 692 renal arteries, 156 superior mesenteric arteries (SMA), and 50 celiac arteries.

23 olic blood flow in the superior and inferior mesenteric arteries and celiac trunk (CT) compared with

24 ified a mechanosensing mechanism in isolated mesenteric arteries and in the renal circulation that re

25 ion and function of the KCNE4 subunit in rat mesenteric arteries and to determine whether it has a fu

26 4) by means of surgical occlusion of distal mesenteric arteries and veins.

27 imary endothelial cells isolated from murine mesenteric arteries express functional Kir2.1 channels s

28 We tested this hypothesis in resistant mesenteric arteries from Adipo-MROE mice using myography

29 An increased response to acetylcholine of mesenteric arteries from rats with cirrhosis (50% effect

30 t ECs, but not smooth muscle cells, of small mesenteric arteries have Kir currents, which are substan

31 with enhanced HNO-mediated vasorelaxation in mesenteric arteries in vitro and arteriolar dilation in

32 vasodilatation (FIV) assayed in pressurized mesenteric arteries pre-constricted with endothelin-1.

33 Vascular function evaluated ex vivo in mesenteric arteries showed that in wild-type mice, CHF m

34 of EET production normalizes the response of mesenteric arteries to vasodilators, with beneficial eff

35 lial Kir channels contribute to FIV of mouse mesenteric arteries via an NO-dependent mechanism, where

36 Small resistance mesenteric arteries were connected to a pressure servo c

37 nt responses to acetylcholine in pressurized mesenteric arteries were reduced in KW versus HW (P<0.01

38 r (celiac, superior mesenteric, and inferior mesenteric arteries) and mediolateral (renal arteries) b

39 d with pulsed Doppler flow probes (renal and mesenteric arteries, and the descending abdominal aorta)

40 In mesenteric arteries, BMAL1 bound to the promoter of and

41 s expressed in a variety of arteries and, in mesenteric arteries, co-localizes with Kv7.4, which is i

42 In the mesenteric arteries, only ECs, but not smooth muscle cel

43 Interestingly, HB-EGF had no effect on mesenteric arteries, suggesting a possible mechanistic b

44 found in thoracic aortas but not in superior mesenteric arteries.

45 teries from TgNotch3(R169C) mice, but not in mesenteric arteries.

46 uced surface and total KV 1.5 protein in rat mesenteric arteries.

47 of S1P synthesis reduced vasoconstriction of mesenteric arteries.

48 esonance angiography, gastric tonometry, and mesenteric arteriography.

49 icroscopy to ferric chloride (FeCl3)-injured mesenteric arterioles and laser-induced injury of cremas

50 ording to their sources into simple inferior mesenteric artery (IMA), simple lumbar artery (LA), comp

51 senteric artery, CT (p < 0.04), and inferior mesenteric artery (p = 0.056) was correlated with the pr

52 obstruction by compression from the superior mesenteric artery (SMA) can be managed using minimally i

53 An aneurysm of the superior mesenteric artery (SMA) with a diameter of 2.2 cm was fo

54 epatic artery (HA) arising from the superior mesenteric artery (SMA), and increasing donor BMI were a

55 Mesenteric artery and aortic transcriptome was profiled

56 half of the nerve plexus around the superior mesenteric artery and celiac axis.

57 ng the aorta in continuity with the inferior mesenteric artery and portal vein in continuity with the

58 Superior mesenteric artery aneurysm (SMAA) is an uncommon vascula

59 less invasive option for rupture of superior mesenteric artery aneurysm.

60 of dissection of the celiac and/or superior mesenteric artery are rare; as far as we know, only 24 c

61 e result of a threefold increase in superior mesenteric artery BFV (P < .0001).

62 ons in gastric volume (P < 0.0001), superior mesenteric artery blood flow (P < 0.0001), and velocity

63 ume, small bowel water content, and superior mesenteric artery blood flow and velocity were measured

64 ) contact (r = -0.38), and post-CRT superior mesenteric artery contact (r = 0.34).

65 nd 71 cases of spontaneous isolated superior mesenteric artery dissection have been reported.

66 ntaneous isolated celiac artery and superior mesenteric artery dissections must be kept in mind in th

67 nfusion increased SBP (P<0.01) and decreased mesenteric artery endothelial function (P<0.01) in wild-

68 ance, reduced portal pressure (PP), superior mesenteric artery flow, mesenteric vascular density, por

69 njury by transient occlusion of the superior mesenteric artery for 30 min.

70 animals/group) by occlusion of the superior mesenteric artery for 90 min and subsequent reperfusion

71 are noteworthy: FMD limited to the inferior mesenteric artery has not been previously reported, FMD

72 ominant-negative Kv7.4 and Kv7.5 subunits in mesenteric artery myocytes reduced endogenous Kv7 curren

73 showed that KCNE4 co-localized with Kv7.4 in mesenteric artery myocytes.

74 hanism, reducing abundance at the surface of mesenteric artery myocytes.

75 expression was found in the membrane of the mesenteric artery myocytes.

76 Doppler ultrasonography of the superior mesenteric artery revealed a twofold increase in blood f

77 olino-induced knockdown of KCNE4 depolarized mesenteric artery smooth muscle cells and resulted in th

78 teromeric channels natively expressed in rat mesenteric artery smooth muscle cells.

79 c Kv7.4/7.5 channels in A7r5 cells or native mesenteric artery smooth muscle Kv7.4/7.5 channels were

80 ric vein thrombosis, and 4 (3%) had superior mesenteric artery stricture or spasm.

81 Seventy-four (65%) had a superior mesenteric artery thromboembolism, 25 (22%) had a superi

82 ion, as demonstrated with the FeCl3 model of mesenteric artery thrombosis.

83 ume were measured in celiac artery, superior mesenteric artery, and main portal vein (MPV).

84 rricane eye, small bowel behind the superior mesenteric artery, and right-sided anastomosis.

85 r-vessel interface was noted at the superior mesenteric artery, celiac artery, or common hepatic arte

86 n analysis, lower blood flow in the superior mesenteric artery, CT (p < 0.04), and inferior mesenteri

87 e patient died of thrombosis in the superior mesenteric artery.

88 limited to the distribution of the inferior mesenteric artery.

89 e patient died of thrombosis in the superior mesenteric artery.

90 V) lodged between the aorta and the superior mesenteric artery.

91 The mesenteric AT (MeAT) was collected on d 0, 4, 7 (early s

92 GLP-2 increases mesenteric blood flow and activates proabsorptive pathwa

93 sufficient to regulate vascular tone of the mesenteric blood vessels where the adult parasites resid

94 nally life-threatening consequences, such as mesenteric, bowel, ureteral, and/or bladder obstruction.

95 ted the migration of specific B cells to the mesenteric but not draining lymph nodes.

96 insulin's ability to suppress lipolysis from mesenteric but not epididymal adipocytes.

97 nd lymphatic vasculatures can be seen in the mesenteric circulation accounting for rapid and continuo

98 sis and ascites was markedly enhanced in the mesenteric circulation compared to the thoracic aorta.

99 s was assessed in the thoracic aorta and the mesenteric circulation of control rats and rats with cir

100 r injury with thrombin microinjection in the mesenteric circulation of mice, we have demonstrated tha

101 In inflamed vessels of the mesenteric circulation, VWF recruited S. lugdunensis to

102 ch as patent urachus, Meckel's diverticulum, mesenteric cyst, and accessory pancreas.

103 liac artery compression by the MAL including mesenteric duplex ultrasonography, computed tomography a

104 , while promoting tissue accumulation in the mesenteric fat and spleen.

105 niculitis (MP) is an inflammatory process of mesenteric fat considered to be of unknown etiology.

106 adrant, increased density of the surrounding mesenteric fat tissue, and mesenteric lymph nodes.

107 rease in the echogenicity of the surrounding mesenteric fat tissue.

108 to breast feeds, and increased postprandial mesenteric flow.

109 ement (odds ratio, 3.9; 95% CI: 1.3, 12) and mesenteric fluid (odds ratio, 3.6; 95% CI: 1.0, 12.8) we

110 in was injected into the celiac and superior mesenteric ganglia (CSMG) of rats.

111 onfidence interval [CI]: 2.6, 23.5), diffuse mesenteric haziness (odds ratio, 6.1; 95% CI: 2.5, 15.2)

112 cement, a closed-loop mechanism, and diffuse mesenteric haziness) can accurately predict strangulatio

113 llowing BAT is thought to occur secondary to mesenteric hematoma formation or mesenteric tear complic

114 mary ovarian mass(es), presence of definable mesenteric implants and infiltration, presence of other

115 to determine the aetiological factors for a mesenteric infarction and the effects of restoring bowel

116 ctive review of data on patients treated for mesenteric infarction from 2000 to 2010.

117 Patients who have a bowel resection for mesenteric infarction may require parenteral nutrition (

118 The presence of mesenteric infiltration and diffuse peritoneal involveme

119 Mesenteric infiltration at CT was associated with CLOVAR

120 Patients with mesenteric infiltration had shorter median progression-f

121 Our patient did not have any mesenteric injury or hematoma on initial abdominal CT.

122 gression-free survival than patients without mesenteric involvement (14.7 months vs 25.6 months accor

123 = 2.40) and left upper quadrant (OR = 1.19), mesenteric involvement (OR = 7.10), and lymphadenopathy

124 Mesenteric involvement at CT was an indicator of signifi

125 Mesenteric involvement at CT was associated with signifi

126 n, presence of PD in gastrohepatic ligament, mesenteric involvement, and supradiaphragmatic lymphaden

127 at 5 g once daily in a patient with chronic mesenteric ischemia (CMI) for chronic loose, frequent, a

128 cm (OR, 6.04; 95% CI, 2.87-12.73; P<0.001), mesenteric ischemia (OR, 9.03; 95% CI, 3.49-23.38; P<0.0

129 ment of operative risk (standard deviations: mesenteric ischemia 20.2% vs 23.2%, P = 0.01; gastrointe

130 clinician to identify patients in whom acute mesenteric ischemia develops.

131 Acute mesenteric ischemia is associated with high morbidity an

132 The diagnosis of acute mesenteric ischemia was proposed based on evidence of po

133 laparotomy confirmed extensive nonocclusive mesenteric ischemia, and the patient rapidly died of mul

134 767) with four detailed clinical vignettes (mesenteric ischemia, gastrointestinal bleed, bowel obstr

135 ue damage, we also studied its expression in mesenteric ischemia-reperfusion (I/R) injury.

136 Mesenteric ischemia-reperfusion injury was induced in ma

137 atient eventually died because of subsequent mesenteric ischemia.

138 atients with intestinal failure secondary to mesenteric ischemia.

139 nhibited repair of damaged mucosa induced by mesenteric ischemia/reperfusion in the small intestine a

140 f the gut barrier function after exposure to mesenteric ischemia/reperfusion.

141 compared with surgeons in the control group [mesenteric ischemia: 43.7% vs 64.6%, P < 0.001 (RCV = 25

142 Mesenteric lesions involved intense arterial remodeling,

143 Mesenteric LN stromal cells, stromal cell line HK, or CX

144 lactosylceramide activated NKT2 cells in the mesenteric LN, resulting in local IL-4 release.

145 quences in axillary, brachial, inguinal, and mesenteric LNs were virtually identical, and a substanti

146 ning the skin (subcutaneous LNs) or the gut (mesenteric LNs) or in Peyer's patches.

147 We identified three small mesenteric LNs, distinct from small intestinal LNs, whic

148 exclusively identified in skin-draining and mesenteric LNs, respectively.

149 t-derived inflammatory mediators carried via mesenteric lymph (ML).

150 Exosomes in postshock mesenteric lymph are key mediators of acute lung injury

151 henotypic composition of DCs were similar in mesenteric lymph from germ-free and conventionally house

152 NKT2 cells were abundant in the mesenteric lymph node (LN) of BALB/c mice and produced I

153 and TH17 cytokine, and Tgfbeta expression in mesenteric lymph node (MLN) CD4(+) T cells and jejunum w

154 They were exclusively found in the mesenteric lymph node after T cell-mediated colitis indu

155 ects were mediated through the limitation of mesenteric lymph node and intestinal DC accumulation and

156 Symptoms, intestinal inflammation, and mesenteric lymph node and intestine mucosal DCs were ass

157 In this report, we demonstrated that mesenteric lymph node CD103(-) DCs express, among other

158 d heightened IL-4 and IL-17A production from mesenteric lymph node CD4(+) cells.

159 Mesenteric lymph node CD4(+) FoxP3(+) regulatory T cells

160 Adhesion of mesenteric lymph node cells to mucosal addressin cell ad

161 ubsets present within the lamina propria and mesenteric lymph node compartments based on expression o

162 Likewise, plasmablast frequency in the mesenteric lymph node correlated with viremia.

163 Mesenteric lymph node cultures from VDR KO and B-VDR KO

164 activity of vitamin A-converting enzymes in mesenteric lymph node dendritic cells, along with increa

165 esponses against a soluble protein Ag and in mesenteric lymph node GC responses against gut-derived A

166 alities include intestinal lymphangiectasia, mesenteric lymph node lymphadenopathy, and lymphangiogen

167 ntestinal inflammation by activating gut and mesenteric lymph node myeloid cells.

168 Spleen and mesenteric lymph node were collected, processed, and ana

169 thereby preventing their accumulation in the mesenteric lymph node.

170 ion of DCs in rhesus macaque (Macaca mulata) mesenteric lymph node.

171 mphatics convey Ags and microbial signals to mesenteric lymph nodes (LNs) to induce adaptive immune r

172 hat impair antigen and immune cell access to mesenteric lymph nodes (LNs), which normally sustain app

173 e collected intestinal and other tissues and mesenteric lymph nodes (MLN) from SAMP mice.

174 ressing) L. monocytogenes recovered from the mesenteric lymph nodes (MLN) was extracellular within th

175 uction was impaired during T cell priming in mesenteric lymph nodes (MLN), which correlated with a re

176 propria dendritic cells (DCs) into draining mesenteric lymph nodes (MLN).

177 103, which is essential for migration to the mesenteric lymph nodes (MLN).

178 sed effector T cell induction in the CLN and mesenteric lymph nodes (MLN).

179 ne receptor (Cxcr)5(+) CD4(+) T cells in the mesenteric lymph nodes (MLNs) of transiently n-6-fed mic

180 elium from where they are transported to the mesenteric lymph nodes (MLNs) within migrating immune ce

181 populations in the peripheral blood, liver, mesenteric lymph nodes (MLNs), jejunum, and bronchoalveo

182 enhances alloantigen presentation within the mesenteric lymph nodes (mLNs), mediated by donor CD103(+

183 from the gut to systemic sites, such as the mesenteric lymph nodes (MLNs), via CD11b(+) migratory de

184 nt a resident memory (Trm) population in the mesenteric lymph nodes (MLNs).

185 patches, limited dendritic cell migration to mesenteric lymph nodes [mLNs] causing reduced T cell-med

186 seca et al. (2015) demonstrate disruption of mesenteric lymph nodes and associated lymphatics after Y

187 bacterial translocation from the gut to the mesenteric lymph nodes and exhibited reduced liver regen

188 tion and decreased the numbers of DCs in the mesenteric lymph nodes and lamina propria.

189 and enhances bacterial translocation to the mesenteric lymph nodes and liver, promoting the progress

190 ation of gut-homing effector T-cells in both mesenteric lymph nodes and Peyer's patches without obvio

191 Mesenteric lymph nodes and spleen were the most heavily

192 itic cells, and less Il10 gene expression in mesenteric lymph nodes and spleens.

193 However, whether GALT and/or mesenteric lymph nodes are required for intestinal Th17

194 from proximal and distal graft sections and mesenteric lymph nodes at 20 min, 12 hr, 7 day, and 6 mo

195 ositive PCR detection of M. canettii for 5/8 mesenteric lymph nodes at days 1 and 3 p.i. and 5/6 pool

196 RORgammat(+) T cells were induced in mesenteric lymph nodes early after SFB colonization and

197 Apoptotic IECs were trafficked to mesenteric lymph nodes exclusively by the dendritic cell

198 ry cells (CD25(+)CD127(-)) in the spleen and mesenteric lymph nodes in the mice treated with both Abs

199 ic lymphatic vessels and lymph drainage into mesenteric lymph nodes may be compromised.

200 SS2 was detected in mesenteric lymph nodes of 40% of challenged piglets.

201 of gammadelta-17 cells in the peripheral and mesenteric lymph nodes of adult IL-15Ralpha KO mice, but

202 rase chain reaction of intestinal mucosa and mesenteric lymph nodes of Duoxa(-/-) mice that lack func

203 regulate T cell homing, were also reduced in mesenteric lymph nodes of infected AMCase-deficient mice

204 required to maintain the Th2 response in the mesenteric lymph nodes of infected mice.

205 TLR4 conditioned the in vivo mobilization to mesenteric lymph nodes of intestinal migratory CD103(+)

206 om rectal biopsy specimens, bone marrow, and mesenteric lymph nodes of vaccinated infected, unvaccina

207 +) T cells preferentially home to spleen and mesenteric lymph nodes owing to increased expression of

208 Culture of mesenteric lymph nodes showed higher density of infectio

209 o initially differentiate in the GALT and/or mesenteric lymph nodes upon Ag encounter and subsequentl

210 s (cDC) in the intestinal lamina propria and mesenteric lymph nodes were GFP(+) However, in vitro inf

211 PA-TG, the concentrations of free MPA in the mesenteric lymph nodes were significantly enhanced (up t

212 aive lymphocytes traffic to the gut-draining mesenteric lymph nodes where they undergo antigen-induce

213 of the target genes in colonic biopsies and mesenteric lymph nodes which was accompanied with a dist

214 lls under inflammatory conditions in spleen, mesenteric lymph nodes, and colon tissue.

215 cruitment, and cytokine expression in ileum, mesenteric lymph nodes, and spleen.

216 CD8(+) T cells in the intestinal mucosa and mesenteric lymph nodes, express the cell adhesion molecu

217 enous increase of SIV RNA in superficial and mesenteric lymph nodes, spleen, and the gastrointestinal

218 ed lymphatic transport of dendritic cells to mesenteric lymph nodes, two features likely to actively

219 g of ILCs from the intestine to the draining mesenteric lymph nodes, which specifically for the LTi-l

220 -specific CD4 T cells in Peyer's patches and mesenteric lymph nodes, which was accompanied by increas

221 cell populations in the Peyer's patches and mesenteric lymph nodes, while 6'-sialyllactose also indu

222 l accumulation in the inflamed intestine and mesenteric lymph nodes.

223 of the small intestine, Peyer's patches, and mesenteric lymph nodes.

224 f the surrounding mesenteric fat tissue, and mesenteric lymph nodes.

225 and immature in the fetal thymus, spleen and mesenteric lymph nodes.

226 CII(+) macrophages in the lamina propria and mesenteric lymph nodes.

227 f Breg cells in the spleen as well as in the mesenteric lymph nodes.

228 re determined in duodenum, ileum, colon, and mesenteric lymph nodes.

229 -specific regulatory T cell induction in the mesenteric lymph nodes.

230 dritic cells became prominently activated in mesenteric lymph nodes.

231 om the draining lymph nodes/CNS route to the mesenteric lymph nodes/gut route, which ameliorated EAE

232 imals exhibited normal growth of the primary mesenteric lymphatic plexus but failed to form valves in

233 These observations suggest that downstream mesenteric lymphatic vessels and lymph drainage into mes

234 otch1 is expressed throughout the developing mesenteric lymphatic vessels at E16.5, and that, by E18.

235 Mesenteric lymphatic vessels from MetSyn or LPS-injected

236 orphologic features and functional status of mesenteric lymphatics in CD.

237 in isolated human vessels (thoracic duct and mesenteric lymphatics) maintained under isometric condit

238 ractile properties and valvular functions of mesenteric lymphatics, developed a surgical model for va

239 rectosigmoid mucosa associated with a large mesenteric mass of unknown nature.

240 Single perfused rat mesenteric microvessels were perfused with fluorescent e

241 issection and spinal (n = 4), renal (n = 7), mesenteric (n = 2), and/or iliofemoral (n = 9) malperfus

242 B4 did not overexpress VEGF or have signs of mesenteric neovascularization, and developed less-severe

243 al preparations with attached splanchnic and mesenteric nerves were used to study mechanosensory and

244 superior mesenteric (P = .358), and inferior mesenteric (P = .065) arteries.

245 guidance for the celiac (P = .755), superior mesenteric (P = .358), and inferior mesenteric (P = .065

246 Mesenteric panniculitis (MP) is a radiological finding a

247 Mesenteric panniculitis (MP) is an inflammatory process

248 proximately 60% of Nppb-/- females developed mesenteric polyarteritis-nodosa (PAN)-like vasculitis in

249 ctor (VEGF), phospho-VEGFR2, and phospho-Akt mesenteric protein expression.

250 e to acetylcholine were improved in isolated mesenteric resistance arteries of Plekha7 mutant rats co

251 Analyzing contraction of mesenteric resistance arteries supported the biological

252 and flow-mediated dilatations in third-order mesenteric resistance arteries were improved.

253 asodilatation via nitric oxide (NO) in mouse mesenteric resistance arteries.

254 asodilatation via nitric oxide (NO) in mouse mesenteric resistance arteries.

255 ffect on endothelium-dependent relaxation in mesenteric resistance artery.

256 arget cardiomyocytes, skeletal myocytes, and mesenteric resistance vessels and are sufficient to conf

257 genic tone (MT) in isolated, pressurized rat mesenteric small arteries, and Ca2+ signalling in primar

258 The best signs were mesenteric swirl (sensitivity and specificity, 86%-89% a

259 and sensitivity for diagnosis of IH included mesenteric swirl and SBO, the model with the highest spe

260 st overall accuracy and sensitivity included mesenteric swirl and SBO, with a diagnostic odds ratio o

261 owing previously established CT signs of IH: mesenteric swirl, small-bowel obstruction (SBO), mushroo

262 econdary to mesenteric hematoma formation or mesenteric tear complications.

263 Critically, TNFDeltaARE mice also present mesenteric tertiary lymphoid organs and have altered lym

264 rty-day mortality was greater after visceral-mesenteric than lower- or upper-extremity SEE (55%, 17%,

265 to characterize the lymphatic vasculature in mesenteric tissue from controls or patients with CD.

266 stricted intact arteries isolated from mouse mesenteric tissue.

267 Liver and mesenteric tissues were collected and analyzed in angiog

268 Adult rat mesenteric tissues were harvested and cultured for three

269 ssure (PP), superior mesenteric artery flow, mesenteric vascular density, portosystemic shunting (PSS

270 e features (with inflammatory bowel disease, mesenteric vascular diseases, or other conditions).

271 ographic interface between primary tumor and mesenteric vasculature.

272 ransit the lungs en route to the hepatic and mesenteric vasculature.

273 eceptor agonists able to induce systemic and mesenteric vasoconstriction have shown their usefulness

274 ich was associated with decreased aortic and mesenteric vasoconstriction in hypertensive Sphk1(-/-) m

275 increased cardiac contractility and restored mesenteric vasoreactivity.

276 Hypoglycemic detection at the portal-mesenteric vein (PMV) appears mediated by spinal afferen

277 evaluated images for two new signs, superior mesenteric vein (SMV) "beaking" and "criss-cross" of the

278 mallest axes (r = -0.39), change in superior mesenteric vein (SMV) and/or portal vein (hereafter, SMV

279 to the liver and atypically located superior mesenteric vein (SMV) joining with the splenic vein to f

280 y measurements were obtained in the superior mesenteric vein (SMV), splenic vein (SV), portal vein (P

281 ze with abutment of the portal vein-superior mesenteric vein confluence for less than 180 degrees .

282 ery thromboembolism, 25 (22%) had a superior mesenteric vein thrombosis, and 4 (3%) had superior mese

283 portal vein in continuity with the inferior mesenteric vein.

284 Glucosensory elements in the portal-mesenteric veins are dispensable with faster rates when

285 colonic ischemia, the presence of gas in the mesenteric veins but not in the portal vein.

286 ceptor 7/8 (TLR7/8)-mediated inflammation of mesenteric veins, platelet activation drives the rapid m

287 l cells also increased leukocyte adhesion in mesenteric venules and increased the frequency of neutro

288 showed reduced rolling in thrombin-activated mesenteric venules and inflamed brain microcirculation.

289 Intravital microscopy of mouse mesenteric venules demonstrated that PD1n-3 DPA and RvD5

290 m that triggers endothelial communication to mesenteric vessel muscle cells, leading to vasoconstrict

291 In ex vivo rings, aortic and mesenteric vessels from SHR treated with DHI exhibited s

292 A small bowel loop was exposed, and 3 to 4 mesenteric vessels were clipped in 6 pigs.

293 Furthermore, pretreatment of mesenteric vessels with a VEGF receptor 2-neutralizing a

294 Identical experiments were performed in rat mesenteric vessels with and without phosphodiesterase ty

295 ein (SMV) "beaking" and "criss-cross" of the mesenteric vessels.

296 odel of a tumor that commonly involves major mesenteric vessels.

297 A transcriptome analysis comparing mesenteric visceral AT (vAT) of HF and HF/DDE groups rev

298 okine-induced lipolysis may be restricted to mesenteric white adipose tissue and that it contributes

299 ivity of mast cells to fungi was tested with mesenteric windows (ex vivo) and the human mast cell lin

300 agonist) induced mast cell degranulation in mesenteric windows and HMC-1 cells responded to fungal a