ライフサイエンスコーパス: mesenteric artery

1 in but only anti-TRPC6 inhibited activity in mesenteric artery.

2 have compared the data to that from a small mesenteric artery.

3 -2d also elicited vasodilation effect in rat mesenteric artery.

4 niform patterns of branching at the superior mesenteric artery.

5 as significantly greater in coronary than in mesenteric artery.

6 rived from the adult rat (or mouse) superior mesenteric artery.

7 V) lodged between the aorta and the superior mesenteric artery.

8 increased in the Day28 low flow first order mesenteric artery.

9 tery arising independently from the superior mesenteric artery.

10 rminant of peripheral resistance - the small mesenteric artery.

11 from 1st to 7th order branches of guinea-pig mesenteric artery.

12 carba-UDP were studied in a model of the rat mesenteric artery.

13 rn endotoxemia on blood flow in the superior mesenteric artery.

14 se of the occluded (for 15 minutes) superior mesenteric artery.

15 e patient died of thrombosis in the superior mesenteric artery.

16 limited to the distribution of the inferior mesenteric artery.

17 and Doppler ultrasonography of the superior mesenteric artery.

18 e patient died of thrombosis in the superior mesenteric artery.

19 only in native rat renal arteries but not in mesenteric arteries.

20 roduction to increase myogenic tone in small mesenteric arteries.

21 uced surface and total KV 1.5 protein in rat mesenteric arteries.

22 s less in the endothelium of IH than in sham mesenteric arteries.

23 nd dilated intact or endothelium-denuded rat mesenteric arteries.

24 (SUR) 2-deficient [SUR2(-/-)] mouse myogenic mesenteric arteries.

25 of S1P synthesis reduced vasoconstriction of mesenteric arteries.

26 of the structure and function of resistance mesenteric arteries.

27 of a change in endothelial cell Ca2+ in rat mesenteric arteries.

28 thetic neurotransmission in rat second-order mesenteric arteries.

29 erwent stent placement in 79 stenotic (>70%) mesenteric arteries.

30 le for Cx40 in EDHF-mediated dilation of rat mesenteric arteries.

31 revealed expression of Kir6.1/SUR2B mRNAs in mesenteric arteries.

32 ed vasodilation of the isolated and perfused mesenteric arteries.

33 scle hyperpolarization and relaxation in rat mesenteric arteries.

34 found in thoracic aortas but not in superior mesenteric arteries.

35 teries from TgNotch3(R169C) mice, but not in mesenteric arteries.

36 MP (Epac) in mediating vasorelaxation in rat mesenteric arteries.

37 ulates the myogenic response in cerebral and mesenteric arteries.

38 ecrease in contractile response was found in mesenteric arteries.

39 ) in celiac arteries, five (25%) in inferior mesenteric arteries; 15 (75%) of first-order branching,

40 e maternal diabetic animals when assessed in mesenteric arteries 16 days postpartum.

41 ction of the distal branches of the superior mesenteric artery (60 minutes) and reperfusion for 90 mi

42 in (94%), hepatic artery (93%), and superior mesenteric artery (93%) in these patients.

43 nsport activity was investigated in VSMCs of mesenteric arteries after an NH4(+) prepulse.

44 relaxation of microvessels from the superior mesenteric artery after I/R was significantly attenuated

45 In small mesenteric arteries, alpha(1A)-subtype-specific antagoni

46 In rat mesenteric arteries, anandamide-induced vasodilation is

47 olic blood flow in the superior and inferior mesenteric arteries and celiac trunk (CT) compared with

48 ere studied on VSMs acutely dissociated from mesenteric arteries and HEK293 cells expressing Kir6.1/S

49 4) CaSR mRNA and protein were present in rat mesenteric arteries and in porcine coronary artery endot

50 ified a mechanosensing mechanism in isolated mesenteric arteries and in the renal circulation that re

51 GS5 caused augmented myogenic tone in intact mesenteric arteries and increased activation of protein

52 ent in VSMCs of medium-sized vessels such as mesenteric arteries and larger retinal arterioles.

53 tural and functional integrity of resistance mesenteric arteries and lowered blood pressure in low-re

54 C5 antibodies inhibited SOCs in coronary and mesenteric arteries and portal vein but anti-TRPC6 block

55 dispersed myocytes from rabbit coronary and mesenteric arteries and portal vein.

56 ion and function of the KCNE4 subunit in rat mesenteric arteries and to determine whether it has a fu

57 Mesenteric arteries and veins from female mice flown on

58 to induce rapid constriction in femoral and mesenteric arteries and veins in rats.

59 f this study was to test the hypothesis that mesenteric arteries and veins will exhibit diminished va

60 4) by means of surgical occlusion of distal mesenteric arteries and veins.

61 ith an ultrasonic flow probe on the superior mesenteric artery and a catheter into the superior mesen

62 ic flow probe was inserted into the superior mesenteric artery and a catheter into the superior mesen

63 tic flowprobe was placed around the superior mesenteric artery and an ileal tonometer was inserted.

64 Mesenteric artery and aortic transcriptome was profiled

65 half of the nerve plexus around the superior mesenteric artery and celiac axis.

66 Lower blood flow in the superior mesenteric artery and CT was correlated with HF severity

67 ce of [14C]lactate infused into the superior mesenteric artery and direct measurements of blood lacta

68 ng the aorta in continuity with the inferior mesenteric artery and portal vein in continuity with the

69 ity in coronary artery but inhibited SOCs in mesenteric artery and portal vein myocytes.

70 vation of SOCs in coronary artery similar to mesenteric artery and portal vein.

71 roperitoneal D3 located between the superior mesenteric artery and the aorta was seen on US in all pa

72 nduced in rats by clamping both the superior mesenteric artery and the celiac trunk for 45 min, follo

73 nduced in rats by clamping both the superior mesenteric artery and the celiac trunk for 45 min, follo

74 were placed around a branch of the superior mesenteric artery and the right femoral artery.

75 differentially enhanced in the PHT superior mesenteric artery and thoracic aorta during the developm

76 tructures, relative position of the superior mesenteric artery and vein.

77 given by the terminal branch of the superior mesenteric artery and venous outflow by a proximal segme

78 r (celiac, superior mesenteric, and inferior mesenteric arteries) and mediolateral (renal arteries) b

79 ncreatic adenocarcinoma, celiac and superior mesenteric arteries, and superior mesenteric and portal

80 Rabbit aorta, mesenteric arteries, and the combination of 15-LO and cy

81 d with pulsed Doppler flow probes (renal and mesenteric arteries, and the descending abdominal aorta)

82 ume were measured in celiac artery, superior mesenteric artery, and main portal vein (MPV).

83 rricane eye, small bowel behind the superior mesenteric artery, and right-sided anastomosis.

84 Superior mesenteric artery aneurysm (SMAA) is an uncommon vascula

85 less invasive option for rupture of superior mesenteric artery aneurysm.

86 Significant remodeling of resistance mesenteric arteries (approximately 100-microm outside di

87 olving both roots of the celiac and superior mesenteric artery are deemed unresectable by conventiona

88 of dissection of the celiac and/or superior mesenteric artery are rare; as far as we know, only 24 c

89 asoconstriction to NE was also determined in mesenteric arteries at 1, 5, and 7 d postlanding.

90 m efflux was increased in both the aorta and mesenteric artery at 24 hrs.

91 -sec reperfusion and reocclusion of superior mesenteric artery at the initiation of reperfusion.

92 e result of a threefold increase in superior mesenteric artery BFV (P < .0001).

93 ons in gastric volume (P < 0.0001), superior mesenteric artery blood flow (P < 0.0001), and velocity

94 tant with a significant decrease in superior mesenteric artery blood flow (Qsma) after 15 days in PHT

95 ume, small bowel water content, and superior mesenteric artery blood flow and velocity were measured

96 In mesenteric arteries, BMAL1 bound to the promoter of and

97 ) proteins modulate the myogenic response in mesenteric arteries, but involvement in other vascular b

98 thelium-denuded IPAs by 235 +/- 32 %, and in mesenteric arteries by 218 +/- 38 %.

99 evere hypoxia decreases tone in isolated rat mesenteric arteries by a mechanism which is independent

100 ort hairpin RNA (shRNA) were transduced into mesenteric arteries by chemical loading plus liposomes.

101 r-vessel interface was noted at the superior mesenteric artery, celiac artery, or common hepatic arte

102 postischemia reperfusion (IR) injury of the mesenteric artery, characterized by marked neutrophil ad

103 itol (osmotic control), followed by superior mesenteric artery clamping for 60 minutes and 30 minutes

104 ed in tissue superfusates upon EFS of canine mesenteric artery (CMA), canine urinary bladder, and mur

105 s expressed in a variety of arteries and, in mesenteric arteries, co-localizes with Kv7.4, which is i

106 ed to EDHF, was significantly reduced in OHF mesenteric arteries compared with controls.

107 ) contact (r = -0.38), and post-CRT superior mesenteric artery contact (r = 0.34).

108 ood pressure, heart rate, aortic function or mesenteric artery contractile function, at either 3 or 6

109 n analysis, lower blood flow in the superior mesenteric artery, CT (p < 0.04), and inferior mesenteri

110 rdial space until blood flow in the superior mesenteric artery decreased to half of baseline.

111 Pressure myography using mesenteric arteries demonstrated that relaxation respons

112 nd 71 cases of spontaneous isolated superior mesenteric artery dissection have been reported.

113 ntaneous isolated celiac artery and superior mesenteric artery dissections must be kept in mind in th

114 gh/low flow, the portal vein and first order mesenteric artery dynamically downregulate Tra2beta conc

115 TRPV4-C1-P2 complex in primary cultured rat mesenteric artery endothelial cells (MAECs) and HEK293 c

116 Finally, in pure cell populations of mouse mesenteric artery endothelial cells, we show that P2X(1)

117 sulin resistance, dyslipidaemia, obesity and mesenteric artery endothelial dysfunction in adult offsp

118 nfusion increased SBP (P<0.01) and decreased mesenteric artery endothelial function (P<0.01) in wild-

119 Small mesenteric artery endothelium-dependent dilation to acet

120 hich is similar to the pressure second-order mesenteric arteries experience in vivo, and that Ca(2+)

121 imary endothelial cells isolated from murine mesenteric arteries express functional Kir2.1 channels s

122 ance, reduced portal pressure (PP), superior mesenteric artery flow, mesenteric vascular density, por

123 e is used to provide inflow to the renal and mesenteric arteries followed by aortic relining with ste

124 I/R injury induced by clamping the superior mesenteric artery for 100 min with tissue analysis at 4

125 al ischemia (GI) was induced by clamping the mesenteric artery for 20 minutes and then reperfused for

126 al I/R was induced by occluding the superior mesenteric artery for 30 min followed by reperfusion for

127 sion by occlusion (clamping) of the superior mesenteric artery for 30 min, followed by unclamping and

128 njury by transient occlusion of the superior mesenteric artery for 30 min.

129 duced by temporary occlusion of the superior mesenteric artery for 30 mins, followed by 2 hrs of repe

130 inuously infused into the cranial (superior) mesenteric artery for 48 hours.

131 animals/group) by occlusion of the superior mesenteric artery for 90 min and subsequent reperfusion

132 Resistance mesenteric arteries from 6-month-old female rats fed the

133 We tested this hypothesis in resistant mesenteric arteries from Adipo-MROE mice using myography

134 w-dependent dilatation is impaired in distal mesenteric arteries from adult SHR compared with WKY con

135 w-dependent dilatation is impaired in distal mesenteric arteries from adult spontaneously hypertensiv

136 , Rho-kinase II, and MYPT1 were increased in mesenteric arteries from endotoxemic rats, but the phosp

137 Myogenic tone was greater in mesenteric arteries from IH than sham control rat arteri

138 Isolated renal and mesenteric arteries from Lewis rats were incubated with

139 mpared with those fed the depleted diet, and mesenteric arteries from male and female rats fed the is

140 xpressed in thoracic aortas, small renal and mesenteric arteries from mice and rats of both sexes, as

141 mented myosin light chain phosphorylation in mesenteric arteries from mice with smooth muscle-specifi

142 An increased response to acetylcholine of mesenteric arteries from rats with cirrhosis (50% effect

143 SK3 protein was elevated in small mesenteric arteries from SK3T/T mice compared with wild-

144 enhanced noradrenaline sensitivity in small mesenteric arteries from VHF rats (VHF vs. VC, P < 0.05)

145 ly with the evoked contractile response of a mesenteric artery from a healthy Sprague Dawley rat.

146 constrictors, and their aortic, femoral, and mesenteric arteries had reduced contractile responses to

147 are noteworthy: FMD limited to the inferior mesenteric artery has not been previously reported, FMD

148 t ECs, but not smooth muscle cells, of small mesenteric arteries have Kir currents, which are substan

149 neurovascular transmission in isolated small mesenteric arteries have used either isometric recording

150 sease models such as portal hypertension and mesenteric artery high/low flow, the portal vein and fir

151 In preconstricted rat mesenteric arteries, highly diluted sangre de grado (1:1

152 difference in contractility of medium-sized mesenteric arteries; however, responsiveness of the aort

153 ografts were transplanted below the inferior mesenteric arteries (IMA) into 6 rhesus monkeys.

154 acers were placed intraluminally in inferior mesenteric artery (IMA) or inferior mesenteric vein (IMV

155 ording to their sources into simple inferior mesenteric artery (IMA), simple lumbar artery (LA), comp

156 (i) vasorelaxation of rat mesenteric arteries in response to calcitonin gene-relat

157 with enhanced HNO-mediated vasorelaxation in mesenteric arteries in vitro and arteriolar dilation in

158 issues such as the rat portal vein and small mesenteric artery, in which E23 is spliced, as compared

159 the portal vein, small intestine, and small mesenteric artery, in which Mypt1 E23 is predominately i

160 In vitro, CRF injected into the inferior mesenteric artery increased distal colonic myoelectric a

161 e [14C]lactate was infused into the superior mesenteric artery, indicating increased first-pass clear

162 Using an in vivo mesenteric artery injury model and real-time continuous

163 d by the finding that the myogenic reflex of mesenteric arteries is absent in MT knockout mice (MT(-/

164 uced endothelium-dependent relaxation in OHF mesenteric arteries is due to impaired EDHF-mediated rel

165 ital and subjected to 30 minutes of superior mesenteric artery ischemia, followed by 4 hours of equia

166 gments in vitro was significantly reduced in mesenteric arteries isolated from VEGF-treated rats (P<0

167 on of P2Y receptor on the endothelium of rat mesenteric arteries leads to marked spreading dilatation

168 anal verge, CCI of 3 or more, high inferior mesenteric artery ligation (above left colic artery), in

169 h-old) were tested for vascular functions in mesenteric arteries (MA) and ion channel activities in s

170 The function of sympathetic nerves supplying mesenteric arteries (MA) and veins (MV) in rats was inve

171 nduced (P < 0.05) vasodilatation in isolated mesenteric arteries (MA) from protein-restricted pregnan

172 We studied mesenteric artery (MA) from transgenic mice expressing d

173 ere ligated so that the upstream first order mesenteric artery (MA1) is under chronic low flow and th

174 tion (KCl)-induced constriction of rat small mesenteric arteries (MAs) and veins (MVs) to the dilator

175 Abstract Mesenteric arteries (MAs) are studied widely in vitro bu

176 hesis that functional sensory innervation of mesenteric arteries (MAs) is impaired for Old (24 months

177 The dilatory role for sensory innervation of mesenteric arteries (MAs) is impaired in Old ( approxima

178 usion (35 min) and reopening of the superior mesenteric artery, MC3R-null mice displayed a higher deg

179 ylation of key proteins in denuded rat small mesenteric artery, midsized caudal artery and thoracic a

180 In the mesenteric artery, more than 20 myocytes are required fo

181 ion conductances in freshly dispersed rabbit mesenteric artery myocytes at the single-channel level u

182 ctivates two distinct cation conductances in mesenteric artery myocytes by stimulation of AT1 recepto

183 ominant-negative Kv7.4 and Kv7.5 subunits in mesenteric artery myocytes reduced endogenous Kv7 curren

184 channel activity in freshly dispersed rabbit mesenteric artery myocytes using patch clamp recording a

185 In contrast, mesenteric artery myocytes, which express both KCNQ4 and

186 P(2)-induced inhibition of TRPC6 activity in mesenteric artery myocytes.

187 e OAG activates a homomeric TRPC6 channel in mesenteric artery myocytes.

188 d no effect on OAG-induced TRPC6 activity in mesenteric artery myocytes.

189 man KCNQ5 currents) and freshly isolated rat mesenteric artery myocytes.

190 reversal potentials in coronary compared to mesenteric artery myocytes.

191 hanism, reducing abundance at the surface of mesenteric artery myocytes.

192 showed that KCNE4 co-localized with Kv7.4 in mesenteric artery myocytes.

193 expression was found in the membrane of the mesenteric artery myocytes.

194 type IA, n=1; type IB, n=1; type II inferior mesenteric artery, n=2; type II lumbar artery, n=28; typ

195 OS+/+ and iNOS-/- mice subjected to superior mesenteric artery occlusion (SMAO) in which bacterial tr

196 l ventilation (CMV) over 60 mins of superior mesenteric artery occlusion and 60 mins of reperfusion.

197 d to a sham operation or 30 mins of superior mesenteric artery occlusion followed by reperfusion.

198 ischemia-reperfusion groups, where superior mesenteric artery occlusion was maintained for 1 hr and

199 After superior mesenteric artery occlusion, intestinal permeability inc

200 gnal-regulated kinase (Erk) were assessed in mesenteric arteries of 3- (3M) and 9-month-old (9M) male

201 ed expression of HO-1 was found in aorta and mesenteric arteries of BDL rats in a close chronologic r

202 single myocytes freshly isolated from small mesenteric arteries of guinea-pig was used to investigat

203 and protein were significantly increased in mesenteric arteries of hypertensive animals, and pharmac

204 ompared IP3R expression and function between mesenteric arteries of normotensive and hypertensive ani

205 dothelial cells within isolated, pressurized mesenteric arteries of the rat.

206 onor spermine-NONOate (SPER-NO) in aorta and mesenteric arteries of WT mice.

207 or expression was determined in the superior mesenteric artery of sham and PHT rats by in situ autora

208 e (eNOS) protein expression in the aorta and mesenteric artery, on sodium and water excretion.

209 In the mesenteric arteries, only ECs, but not smooth muscle cel

210 owel are diverse, ranging from occlusions of mesenteric arteries or veins to complicated bowel obstru

211 ins of ischemia by occlusion of the superior mesenteric artery or 30 mins of ischemia followed by 60

212 e coil embolization (hypogastric or inferior mesenteric artery) (OR = 2.1, P = 0.008).

213 Small mesenteric arteries (outside diameter, 50-150 microm) we

214 senteric artery, CT (p < 0.04), and inferior mesenteric artery (p = 0.056) was correlated with the pr

215 plant hepatic artery, celiac trunk, superior mesenteric artery, portal vein, superior mesenteric vein

216 vasodilatation (FIV) assayed in pressurized mesenteric arteries pre-constricted with endothelin-1.

217 mmHg) decreased vessel tone in isolated rat mesenteric arteries precontracted with either high [K+]

218 uced contractile differences between ex vivo mesenteric artery preparations.

219 inase into Triton X-100-permeabilized rabbit mesenteric artery provoked a Ca(2+)-free contraction.

220 ation in response to acetycholine in control mesenteric arteries remained after inhibition of nitric

221 tation and hyperpolarization of VSM in small mesenteric arteries requires BK(Ca) channels.

222 elialized Pkd2(+/-) resistance (fourth-order mesenteric) arteries responded to PE with a stronger con

223 ells of rat cerebral (basilar), coronary and mesenteric arteries revealed transcripts for Kir2.1.

224 Doppler ultrasonography of the superior mesenteric artery revealed a twofold increase in blood f

225 iac RI (0.78 versus 0.73, P = 0.04) superior mesenteric artery RI (0.89 versus 0.84, P = 0.005), and

226 of isolated rat PMNs to thrombin-stimulated mesenteric artery segments in vitro was significantly re

227 intact, but not of endothelium-denuded, rat mesenteric artery segments, modulation of endothelial BK

228 s as a full agonist in relaxing rat isolated mesenteric artery segments.

229 min) of both canine and guinea-pig isolated mesenteric artery segments.

230 nts between renal arteries, and the inferior mesenteric arteries served as autografts.

231 Vascular function evaluated ex vivo in mesenteric arteries showed that in wild-type mice, CHF m

232 mathematical model of Ca(2+) dynamics in rat mesenteric arteries shows that a number of synchronizing

233 e placed in 692 renal arteries, 156 superior mesenteric arteries (SMA), and 50 celiac arteries.

234 Blood pressure, superior mesenteric artery (SMA) and skeletal muscle blood flow,

235 aorta by approximately 60 % and the superior mesenteric artery (SMA) by approximately 90 %.

236 obstruction by compression from the superior mesenteric artery (SMA) can be managed using minimally i

237 Vasoconstriction of the superior mesenteric artery (SMA) is the earliest hemodynamic even

238 he final bile duct, pancreatic, and superior mesenteric artery (SMA) margins.

239 rtery (CHA) arising from either the superior mesenteric artery (SMA) or the aorta.

240 t vasodilatation of both rat aorta and small mesenteric artery (SMA) segments and reduced Phe-induced

241 Superior mesenteric artery (SMA) syndrome describes vascular comp

242 preoperative embolization of graft superior mesenteric artery (SMA) to facilitate intestinal graft r

243 sion of isolated neutrophils to rat superior mesenteric artery (SMA) vascular segments stimulated wit

244 octreotide on vascular tone in the superior mesenteric artery (SMA) was studied in portal-hypertensi

245 An aneurysm of the superior mesenteric artery (SMA) with a diameter of 2.2 cm was fo

246 low probe was positioned around the superior mesenteric artery (SMA), and cannulation of the pericard

247 epatic artery (HA) arising from the superior mesenteric artery (SMA), and increasing donor BMI were a

248 ays was analyzed by western blot in superior mesenteric artery (SMA).

249 heter was placed selectively in the superior mesenteric artery (SMA).

250 , endothelium-dependent relaxations of small mesenteric arteries (SMAs).

251 olino-induced knockdown of KCNE4 depolarized mesenteric artery smooth muscle cells and resulted in th

252 the only SUR isoform expressed in SUR2(+/+) mesenteric artery smooth muscle cells, whereas SURs were

253 endent Cl- channel has been described in rat mesenteric artery smooth muscle cells.

254 ivated Ca(2+)-dependent Cl(-) channel in rat mesenteric artery smooth muscle cells.

255 teromeric channels natively expressed in rat mesenteric artery smooth muscle cells.

256 dogenous Kv7 currents in A7r5 rat aortic and mesenteric artery smooth muscle cells.

257 c Kv7.4/7.5 channels in A7r5 cells or native mesenteric artery smooth muscle Kv7.4/7.5 channels were

258 diet had catheters placed into the superior mesenteric artery so that the visceral adipose bed could

259 ric vein thrombosis, and 4 (3%) had superior mesenteric artery stricture or spasm.

260 Interestingly, HB-EGF had no effect on mesenteric arteries, suggesting a possible mechanistic b

261 fied the occurrence of an allograft superior mesenteric artery-superior mesenteric vein (SMA-SMV) AVF

262 mone; pancreatitis; cholelithiasis; superior mesenteric artery syndrome; ileus; pnemothorax; hemothor

263 le responses to these drugs were assessed in mesenteric arteries taken from animals at 24 hrs using w

264 In rat middle cerebral and mesenteric arteries the K(Ca)2.3 component of endotheliu

265 In isolated mesenteric arteries, the vasodilatation in response to t

266 Seventy-four (65%) had a superior mesenteric artery thromboembolism, 25 (22%) had a superi

267 ion, as demonstrated with the FeCl3 model of mesenteric artery thrombosis.

268 elium-dependent vasodilation in rat isolated mesenteric arteries through a G protein-coupled receptor

269 ethanandamide relax rings of rabbit superior mesenteric artery through endothelium-dependent and -ind

270 in portal vein but only weakly associated in mesenteric artery tissue lysates.

271 of EET production normalizes the response of mesenteric arteries to vasodilators, with beneficial eff

272 antly reduced the blood flow in the superior mesenteric artery to 53% of baseline.

273 5%) of 20 MR angiograms obtained in superior mesenteric artery trunks, 15 (75%) in celiac arteries, f

274 hronic low flow and the adjacent first order mesenteric artery under chronic high flow.

275 by angiotensin II (Ang II) in native rabbit mesenteric artery vascular smooth muscle cells (VSMCs).

276 lial Kir channels contribute to FIV of mouse mesenteric arteries via an NO-dependent mechanism, where

277 SRF controls vasoconstriction in mesenteric arteries via vascular SM cell phenotypic modu

278 ive single TRPC channels in acutely isolated mesenteric artery VSMCs from wild-type (WT) and TRPC1-de

279 sentery, and skin; plus aortic, carotid, and mesenteric artery walls.

280 and electrical field stimulation (P<0.05) in mesenteric arteries was also significantly increased in

281 otensin II-mediated constriction of isolated mesenteric arteries was blunted in OVE26EP1(-/-) mice, d

282 Vascular reactivity of the mesenteric arteries was not different.

283 Furthermore, NO-mediated relaxation in mesenteric arteries was significantly improved in DOCA-s

284 unaltered whereas the conductance of SOCs in mesenteric artery was increased fourfold.

285 administered 60 minutes before the superior mesenteric artery was occluded for 90 minutes and reperf

286 Small resistance mesenteric arteries were connected to a pressure servo c

287 lyte and hormone measurements, and aorta and mesenteric arteries were harvested for cGMP determinatio

288 thesis alternating pairs of rat second order mesenteric arteries were ligated so that the upstream fi

289 nt responses to acetylcholine in pressurized mesenteric arteries were reduced in KW versus HW (P<0.01

290 Mesenteric arteries were studied in a myograph.

291 The aorta and mesenteric artery were examined ex vivo for rubidium eff

292 and eNOS protein expression in the aorta and mesenteric artery were increased in CIR as compared with

293 This contrasts with the mesenteric artery, where Ca entry and ryanodine receptor

294 ocated at the branch points of the renal and mesenteric arteries, whereas lesions in this area were n

295 ium-dependent relaxation is impaired in T1DM mesenteric arteries, which is rescued by SOD mimetic tem

296 subunit mRNA increased significantly in the mesenteric artery while rubidium efflux was increased in

297 The response of rat mesenteric arteries with intact (+E) and denuded (-E) en

298 ETA relaxed endothelium-denuded rabbit small mesenteric arteries with maximum relaxations of 22.6 +/-

299 In the present study, treatment of rat mesenteric arteries with phenylephrine (PE) led to the i

300 the bleeding rate in the injured femoral and mesenteric arteries, with a complete hemorrhage arrest a