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1 fter heart operation (P = 0.03 compared with mesenteric ischemia).
2 atients with intestinal failure secondary to mesenteric ischemia.
3 ies that report on current outcome for acute mesenteric ischemia.
4 lood flow in this model of endotoxin-induced mesenteric ischemia.
5 is a promising test for diagnosis of chronic mesenteric ischemia.
6 se patients may predict the onset of chronic mesenteric ischemia.
7 e asymptomatic and 3 had symptoms of chronic mesenteric ischemia.
8 atient eventually died because of subsequent mesenteric ischemia.
9 ment of operative risk (standard deviations: mesenteric ischemia 20.2% vs 23.2%, P = 0.01; gastrointe
10 compared with surgeons in the control group [mesenteric ischemia: 43.7% vs 64.6%, P < 0.001 (RCV = 25
12 osses were sustained by the hospital for the mesenteric ischemia and infected aortic graft groups amo
17 laparotomy confirmed extensive nonocclusive mesenteric ischemia, and the patient rapidly died of mul
18 and/or preemptively diagnosis postcardiotomy mesenteric ischemia are necessary to decrease its associ
19 at 5 g once daily in a patient with chronic mesenteric ischemia (CMI) for chronic loose, frequent, a
20 consecutive series of patients with chronic mesenteric ischemia (CMI) who were treated with percutan
22 in vivo, can be used to diagnose and monitor mesenteric ischemia due to hemorrhagic shock in a canine
23 nts undergoing revascularization for chronic mesenteric ischemia, elective thoracoabdominal aortic an
24 nesthesia, rats were subjected to 60 mins of mesenteric ischemia followed by 60 mins of reperfusion.
25 od flow by 60% in this model of nonocclusive mesenteric ischemia (from 168 +/- 41 to 269 +/- 76 mL/mi
26 767) with four detailed clinical vignettes (mesenteric ischemia, gastrointestinal bleed, bowel obstr
29 y be an effective treatment for nonocclusive mesenteric ischemia, it has also been advocated to incre
30 ficits, altered mental status, myocardial or mesenteric ischemia, kidney failure, hypotension, cardia
32 cm (OR, 6.04; 95% CI, 2.87-12.73; P<0.001), mesenteric ischemia (OR, 9.03; 95% CI, 3.49-23.38; P<0.0
33 on, arterial thromboembolism, renal failure, mesenteric ischemia, or hepatic insufficiency occurred.
36 sm repair, with its requisite intraoperative mesenteric ischemia-reperfusion, often results in the de
37 2-deficient (Cr2(-/-)) mice are resistant to mesenteric ischemia/reperfusion (I/R) injury because the
38 tural Abs have been implicated in initiating mesenteric ischemia/reperfusion (I/R)-induced tissue inj
40 nhibited repair of damaged mucosa induced by mesenteric ischemia/reperfusion in the small intestine a
41 trates that the mechanism of U74389F against mesenteric ischemia/reperfusion includes a delay and red
42 mplement activation prevents and/or reverses mesenteric ischemia/reperfusion-induced injury in mice.
45 se and six patients with symptomatic chronic mesenteric ischemia, the same measurements were obtained
46 small bowel enteroscopy; early diagnosis of mesenteric ischemia; the use of polymerase chain reactio
49 he most frequent serious GI complication was mesenteric ischemia, which developed in 31 (67%) patient
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