ライフサイエンスコーパス: mesenteric lymph node

1 ression and bacterial translocation into the mesenteric lymph node.

2 lular leak initiates immune responses in the mesenteric lymph node.

3 e system interactions with LjN6.2 within the mesenteric lymph node.

4 hese sequences were compartmentalized in the mesenteric lymph node.

5 es, but only the wild-type virus reached the mesenteric lymph node.

6 ssin cell adhesion molecule-1 similar to the mesenteric lymph node.

7 thereby preventing their accumulation in the mesenteric lymph node.

8 ion of DCs in rhesus macaque (Macaca mulata) mesenteric lymph node.

9 on and exhibited limited colonization of the mesenteric lymph nodes.

10 ed by standard microbiological techniques on mesenteric lymph nodes.

11 y induced both IFN-beta and IFN-gamma in the mesenteric lymph nodes.

12 inflammatory cytokine production within the mesenteric lymph nodes.

13 as correlated to a Th17 cell bias within the mesenteric lymph nodes.

14 egs within the intestinal lamina propria and mesenteric lymph nodes.

15 dritic cells became prominently activated in mesenteric lymph nodes.

16 y measuring numbers of regulatory T cells in mesenteric lymph nodes.

17 Salmonella from the intestinal tract to the mesenteric lymph nodes.

18 intestines and their draining lymphatics and mesenteric lymph nodes.

19 splenomegaly and massive enlargement of the mesenteric lymph nodes.

20 ology, and production of interferon-gamma in mesenteric lymph nodes.

21 ential adoptive transfers of CD4+ cells from mesenteric lymph nodes.

22 ugh viable bacteria could be observed in the mesenteric lymph nodes.

23 ice devoid of intestinal Peyer's patches and mesenteric lymph nodes.

24 rs of activated (CD44(high)) CD4(+) cells in mesenteric lymph nodes.

25 mory CD4(+) T cell subset from the blood and mesenteric lymph nodes.

26 able to form spontaneous germinal centers in mesenteric lymph nodes.

27 l accumulation in the inflamed intestine and mesenteric lymph nodes.

28 of the small intestine, Peyer's patches, and mesenteric lymph nodes.

29 f the surrounding mesenteric fat tissue, and mesenteric lymph nodes.

30 and immature in the fetal thymus, spleen and mesenteric lymph nodes.

31 CII(+) macrophages in the lamina propria and mesenteric lymph nodes.

32 f Breg cells in the spleen as well as in the mesenteric lymph nodes.

33 re determined in duodenum, ileum, colon, and mesenteric lymph nodes.

34 -specific regulatory T cell induction in the mesenteric lymph nodes.

35 d levels of enteric bacterial genomic DNA in mesenteric lymph nodes.

36 amine the expression of meprins in the mouse mesenteric lymph node, 2) determine whether macrophages

37 nlarged germinal centers (GCs) in spleen and mesenteric lymph nodes; 3) enrichment in IRF4(+)CD138(-)

38 were detected in the Peyer's patches (4/5), mesenteric lymph nodes (4/5), spleens (4/10), livers (6/

39 In the mesenteric lymph node, a fourth subset expressed CCR9 an

40 These animals frequently had sterile mesenteric lymph nodes, a finding consistent with transl

41 tinal immune cells that traffic to liver and mesenteric lymph nodes, a process that occurs even durin

42 and CD30 x OX40-deficient OTII cells in the mesenteric lymph node after oral immunization.

43 They were exclusively found in the mesenteric lymph node after T cell-mediated colitis indu

44 lex host response in the Peyer's patches and mesenteric lymph nodes after oral infection with Y. ente

45 Compared with those in mesenteric lymph nodes, AI4 T cells entering PLNs underw

46 urther analyses showed that 55 +/- 7% of the mesenteric lymph node alpha4beta7+CD4 expressed L-select

47 ects were mediated through the limitation of mesenteric lymph node and intestinal DC accumulation and

48 Symptoms, intestinal inflammation, and mesenteric lymph node and intestine mucosal DCs were ass

49 iased cytokine response in both the mucosal (mesenteric lymph node and Peyer's patch) and systemic (s

50 memory CD4+ T cells persist in the draining mesenteric lymph node and protect mice against reinfecti

51 These cells colonize the spleen and mesenteric lymph node and secrete IL-17A in vitro follow

52 PSGL-1 was detected in venules of mesenteric lymph node and small intestine by immunohisto

53 on remained on endothelium, localized within mesenteric lymph node and small intestine.

54 rol animals had an inverted CD4:CD8 ratio in mesenteric lymph node and were depleted of both CD4(+) a

55 seca et al. (2015) demonstrate disruption of mesenteric lymph nodes and associated lymphatics after Y

56 's patches, mice deficient in LT beta retain mesenteric lymph nodes and cervical lymph nodes, suggest

57 node homing receptors vigorously expanded in mesenteric lymph nodes and colon by day 6 after transpla

58 /acquired Treg (iTreg) cells was observed in mesenteric lymph nodes and colon from both groups, the s

59 d WT mice migrated in fewer numbers into the mesenteric lymph nodes and colon of MMP3-/- mice than in

60 -cadherin(+) DCs accumulated in the inflamed mesenteric lymph nodes and colon, had high expression of

61 ion revealed commensal gut microflora in the mesenteric lymph nodes and elevated LPS levels in the se

62 ited increased Th2 cytokine responses in the mesenteric lymph nodes and elevated serum IgE and IgG1 l

63 bacterial translocation from the gut to the mesenteric lymph nodes and exhibited reduced liver regen

64 10 was suppressed in the Peyer's patches and mesenteric lymph nodes and IL-4 mRNA was suppressed in t

65 es and their distinct modes of action in the mesenteric lymph nodes and intestinal tissues.

66 -specific IL-17A and IL-22 production in the mesenteric lymph nodes and intestine.

67 tion and decreased the numbers of DCs in the mesenteric lymph nodes and lamina propria.

68 also ameliorated bacterial translocation to mesenteric lymph nodes and leakage of fluorescein isothi

69 and enhances bacterial translocation to the mesenteric lymph nodes and liver, promoting the progress

70 iated organized lymphoid tissues such as the mesenteric lymph nodes and Peyer patches.

71 ation of gut-homing effector T-cells in both mesenteric lymph nodes and Peyer's patches without obvio

72 Mesenteric lymph nodes and spleen were the most heavily

73 lied in all organs except for lungs and that mesenteric lymph nodes and spleen were the most heavily

74 n ligand in cutaneous lymph nodes but not in mesenteric lymph nodes and spleen, demonstrating that ex

75 C in the Peyer's Patches (PP) but not in the mesenteric lymph nodes and spleen.

76 DC subsets were depleted from peripheral and mesenteric lymph nodes and spleens in monkeys with AIDS,

77 itic cells, and less Il10 gene expression in mesenteric lymph nodes and spleens.

78 e early accumulation of donor T cells in the mesenteric lymph nodes and subsequently in the colon.

79 rential expansion in the Peyer's patches and mesenteric lymph nodes and subsequently in the epithelia

80 T lymphocytes is significantly increased to mesenteric lymph nodes and to the inflamed paw in a mode

81 rred within the spleen, intestinal tract, or mesenteric lymph nodes and were present at higher propor

82 eta are expressed in leukocytes of the mouse mesenteric lymph node, and meprin alpha, but not beta, d

83 on of murine CD4(+) T cells from the spleen, mesenteric lymph node, and Peyer's patch.

84 decrease in IL-12p35 expression in colon and mesenteric lymph nodes, and a substantial increase in th

85 s, CCR9 and alpha4beta7, on donor T cells in mesenteric lymph nodes, and augmented the accumulation o

86 lls under inflammatory conditions in spleen, mesenteric lymph nodes, and colon tissue.

87 cells were decreased in the Peyer's patches, mesenteric lymph nodes, and intestinal mucosa of transge

88 age populations in the spleen, kidney, skin, mesenteric lymph nodes, and liver are normal.

89 ess, CD25(+) CD4(+) T cells from the spleen, mesenteric lymph nodes, and Peyer's patches of orally to

90 D24(high) B cells in peripheral lymph nodes, mesenteric lymph nodes, and Peyer's patches.

91 reduced recruitment of MDSCs to the spleen, mesenteric lymph nodes, and primary tumor site.

92 nce of the bacterium in the Peyer's patches, mesenteric lymph nodes, and spleen, suggesting a role fo

93 F-beta transcription in the Peyer's patches, mesenteric lymph nodes, and spleen.

94 cruitment, and cytokine expression in ileum, mesenteric lymph nodes, and spleen.

95 T cells were assessed in the Peyer patches, mesenteric lymph nodes, and spleens by using flow cytome

96 7 in the granulomas, but not in the draining mesenteric lymph nodes, and with a markedly suppressed S

97 However, whether GALT and/or mesenteric lymph nodes are required for intestinal Th17

98 from proximal and distal graft sections and mesenteric lymph nodes at 20 min, 12 hr, 7 day, and 6 mo

99 ositive PCR detection of M. canettii for 5/8 mesenteric lymph nodes at days 1 and 3 p.i. and 5/6 pool

100 ol subjects, of the ileum wall thickness and mesenteric lymph nodes, at enrollment and 1 month later.

101 ignant cells were identified on laparoscopic mesenteric lymph nodes biopsies.

102 n of CD11b(+)CD103(-) dendritic cells in the mesenteric lymph nodes, both of which could be reversed

103 ipts in the ileal Peyer's patches (IPPs) and mesenteric lymph nodes but on average only approximately

104 IL-17F was mainly produced in the spleen and mesenteric lymph nodes, but IL-23 was unaltered in Il17a

105 resulted in increased viable bacteria in the mesenteric lymph nodes by day 3 postinfection.

106 eferentially infects the Peyer's patches and mesenteric lymph nodes, causing an acute inflammatory re

107 In this report, we demonstrated that mesenteric lymph node CD103(-) DCs express, among other

108 3SL directly stimulated mesenteric lymph node CD11c(+) dendritic cells and induc

109 d heightened IL-4 and IL-17A production from mesenteric lymph node CD4(+) cells.

110 Mesenteric lymph node CD4(+) FoxP3(+) regulatory T cells

111 Strikingly, neonatal mesenteric lymph node CD4(+) T cells produced Yersinia-s

112 Mesenteric lymph node CD4+ cells from the second group o

113 CSFE-labeled mesenteric lymph node CD4+ T lymphocytes from infected W

114 IL-33 augmented mesenteric lymph node cell secretion of IL-5, IL-13, IL-

115 t notably, suppression was transferable with mesenteric lymph node cells (MLNC) from infected animals

116 nt egg Ag-stimulated IFN-gamma production by mesenteric lymph node cells and hepatic egg granuloma si

117 erpes virus entry mediator, because LIGHT Tg mesenteric lymph node cells do not cause intestinal infl

118 Polyclonal activation of mesenteric lymph node cells from naive WSX-1 KO or wild-

119 Mesenteric lymph node cells from STAT6(-/-) mice with co

120 m-specific proliferative immune responses of mesenteric lymph node cells in C57BL/6J mice infected wi

121 ntal model for CD by adoptive transfer of Tg mesenteric lymph node cells into RAG(-/-) mice.

122 e treatment in supernatants from cultures of mesenteric lymph node cells of SAMP1/YitFc mice ( P < .0

123 SEA-stimulated bulk mesenteric lymph node cells or CD4 T cells from 7-week-i

124 Mesenteric lymph node cells produced lower levels of cyt

125 Analyses of spleen and mesenteric lymph node cells showed no differences in tot

126 nalysis of cytokine production by spleen and mesenteric lymph node cells showed production of IL-4, I

127 Adhesion of mesenteric lymph node cells to mucosal addressin cell ad

128 Stimulation of splenic or mesenteric lymph node cells with lactococci resulted in

129 L-10, and transforming growth factor beta in mesenteric lymph node cells, purified CD4 T cells, and i

130 secretion of interferon-gamma by stimulated mesenteric lymph node cells.

131 d TNF-alpha by schistosome egg Ag-stimulated mesenteric lymph node cells.

132 terial translocation, measured as numbers of mesenteric lymph node CFU of noninvasive Escherichia col

133 significant protection from both spleen and mesenteric lymph node colonization by M. avium subsp. pa

134 aled that a DeltasfpA strain is defective in mesenteric lymph node colonization.

135 ph nodes and IL-4 mRNA was suppressed in the mesenteric lymph nodes compared to noninfected controls,

136 ubsets present within the lamina propria and mesenteric lymph node compartments based on expression o

137 thiocyanate dextran, bacterial counts in the mesenteric lymph nodes complex, and gut water content we

138 Likewise, plasmablast frequency in the mesenteric lymph node correlated with viremia.

139 Mesenteric lymph node cultures from VDR KO and B-VDR KO

140 when exposed to oral Ag, and 4-1BB-deficient mesenteric lymph node DC displayed weak RALDH activity a

141 the TNFR family, together with CD103, marked mesenteric lymph node DC with the highest level of RALDH

142 describe that p38alpha signaling in CD103(+) mesenteric lymph node DCs reciprocally regulates the dif

143 The phenotypes of these DCs and of mesenteric lymph node DCs were assessed by flow cytometr

144 reover, we identify a population of CD103(+) mesenteric lymph node dendritic cells (DCs) that induce

145 activity of vitamin A-converting enzymes in mesenteric lymph node dendritic cells, along with increa

146 s, the production of IFN-gamma by spleen and mesenteric lymph node-derived CD4+ T cells was down-regu

147 d expansion of these cell populations is the mesenteric lymph nodes draining sites of infection.

148 Mesenteric lymph nodes draining sites of M. avium subsp.

149 response and IL-4 production in the draining mesenteric lymph nodes during infection with the enteric

150 RORgammat(+) T cells were induced in mesenteric lymph nodes early after SFB colonization and

151 Apoptotic IECs were trafficked to mesenteric lymph nodes exclusively by the dendritic cell

152 CD8(+) T cells in the intestinal mucosa and mesenteric lymph nodes, express the cell adhesion molecu

153 oduction also increased significantly in the mesenteric lymph nodes following exposure to viable Salm

154 In peripheral and mesenteric lymph nodes from animals with AIDS there was

155 Colonic mucosa and mesenteric lymph nodes from Ccl17-deficient mice produce

156 cipients, while no differences were found in mesenteric lymph nodes from each group.

157 esponses against a soluble protein Ag and in mesenteric lymph node GC responses against gut-derived A

158 Mesenteric lymph node GCs were more resistant but became

159 om the draining lymph nodes/CNS route to the mesenteric lymph nodes/gut route, which ameliorated EAE

160 YopJ-dependent apoptosis in mesenteric lymph nodes has also been demonstrated in a m

161 hanced C. jejuni invasion into the colon and mesenteric lymph nodes in Il10(-/-); Nod2(-/-) mice, com

162 of Foxp3(+) regulatory T cells increased in mesenteric lymph nodes in mice given MSCs.

163 enzymes were increased in the intestine and mesenteric lymph nodes in mice with active GVHD.

164 ry cells (CD25(+)CD127(-)) in the spleen and mesenteric lymph nodes in the mice treated with both Abs

165 Cellularity of local mesenteric lymph nodes, including T- and B-lymphocytes,

166 ed by a second wave of dissemination via the mesenteric lymph nodes (indirect pathway).

167 stitutively presented in both peripheral and mesenteric lymph nodes, leading to abortive activation a

168 of donor-type Treg cells accumulated in host mesenteric lymph node (LN) and spleen when CD4+CD25+CD62

169 NKT2 cells were abundant in the mesenteric lymph node (LN) of BALB/c mice and produced I

170 Mesenteric lymph nodes (LN) and spleen were differential

171 asma cells appeared first in the hepatic and mesenteric lymph nodes (LNs) and then at later times in

172 mphatics convey Ags and microbial signals to mesenteric lymph nodes (LNs) to induce adaptive immune r

173 from a reduction of CD103(+) DCs in draining mesenteric lymph nodes (LNs), which is associated with d

174 hat impair antigen and immune cell access to mesenteric lymph nodes (LNs), which normally sustain app

175 Lin(-)c-Kit(+) innate cell population in the mesenteric lymph node, lung, and liver.

176 alities include intestinal lymphangiectasia, mesenteric lymph node lymphadenopathy, and lymphangiogen

177 h evidence of bacterial translocation to the mesenteric lymph nodes, macrophage, and T-lymphocyte inf

178 emonstrates induction of PGE(2) synthesis in mesenteric lymph nodes, macrophages, and dendritic cells

179 ic lymphatic vessels and lymph drainage into mesenteric lymph nodes may be compromised.

180 During in vivo mesenteric lymph node migration assays, the absence of L

181 trate significant bacterial translocation to mesenteric lymph node (MLN) and liver following morphine

182 We show that mudeltaCSR occurs in mouse mesenteric lymph node (MLN) B cells and is AID-dependent

183 Here we report that mesenteric lymph node (MLN) B cells protect mice from co

184 Cytokine production of purified mesenteric lymph node (MLN) B cells was examined by flow

185 and TH17 cytokine, and Tgfbeta expression in mesenteric lymph node (MLN) CD4(+) T cells and jejunum w

186 nfected mice by intraperitoneal injection of mesenteric lymph node (MLN) cells alone from H. hepaticu

187 Splenocytes and mesenteric lymph node (MLN) cells from hamsters infected

188 IgG1, and IgG2a and cytokine secretion from mesenteric lymph node (MLN) cells were analyzed.

189 lamina propria mononuclear cells (LPMC) and mesenteric lymph node (MLN) cells.

190 of alpha4beta7 and CCR9 by Peyer's patch and mesenteric lymph node (MLN) dendritic cells (DCs) in a r

191 ype in the bone marrow (BM), spleen, and the mesenteric lymph node (MLN) of allergic and control mice

192 The impact of CT on DC subsets in the mesenteric lymph node (MLN) was assessed by flow cytomet

193 cellular apoptosis in Peyer's patches (PPs), mesenteric lymph node (MLN), and spleen.

194 rain 16M was consistently recovered from the mesenteric lymph node (MLN), spleen, and liver beginning

195 istinct immune responses concurrently in the mesenteric lymph nodes (MLN) and the spleen after i.p. a

196 The Peyer's patches (PP) and mesenteric lymph nodes (MLN) are structural components o

197 cell differentiation was investigated in the mesenteric lymph nodes (MLN) as well as in galectin-9-ex

198 We show that CCR9(+) T cells isolated from mesenteric lymph nodes (MLN) draining CD SB express a mo

199 was readily detectable in the intestine and mesenteric lymph nodes (MLN) for at least 1 week after i

200 e collected intestinal and other tissues and mesenteric lymph nodes (MLN) from SAMP mice.

201 ng those in adults arise very rapidly in the mesenteric lymph nodes (MLN) of neonates.

202 bone marrow and spleen of naive mice and in mesenteric lymph nodes (mLN) of Trichinella spiralis-inf

203 FN-gamma and TNF-alpha CSCs were detected in mesenteric lymph nodes (MLN) or spleen and Th2 (IL-4) CS

204 en for differentially expressed genes in pig mesenteric lymph nodes (MLN) responding to infection wit

205 ressing) L. monocytogenes recovered from the mesenteric lymph nodes (MLN) was extracellular within th

206 d RRV were detected extraintestinally in the mesenteric lymph nodes (MLN), livers, lungs, blood, and

207 Lymphocytes isolated from ileum, mesenteric lymph nodes (MLN), spleen and thymus were lab

208 uction was impaired during T cell priming in mesenteric lymph nodes (MLN), which correlated with a re

209 sed effector T cell induction in the CLN and mesenteric lymph nodes (MLN).

210 te antigens presented by cells isolated from mesenteric lymph nodes (MLN).

211 propria dendritic cells (DCs) into draining mesenteric lymph nodes (MLN).

212 103, which is essential for migration to the mesenteric lymph nodes (MLN).

213 n of large numbers of myeloid cells into the mesenteric lymph nodes (MLNs) and colon.

214 at CD172a(+)CD11c(+) cells accumulate in the mesenteric lymph nodes (mLNs) and inflamed intestinal mu

215 g lymphocytes were purified from SAMP1/YitFc mesenteric lymph nodes (MLNs) and their ability to induc

216 ing of luminescent Salmonella identified the mesenteric lymph nodes (MLNs) as a major reservoir of re

217 ient T reg cells failed to accumulate in the mesenteric lymph nodes (MLNs) at early time points (2-5

218 e distribution of T and B lymphocytes in the mesenteric lymph nodes (MLNs) by flow cytometry.

219 urium can persist for as long as 1 yr in the mesenteric lymph nodes (MLNs) of 129sv Nramp1(+)(/)(+) (

220 Compared with the mesenteric lymph nodes (MLNs) of AKR control mice, SAMP1

221 The pathogen was detected in the mesenteric lymph nodes (MLNs) of irradiated mice 1-4 d p

222 T cells purified from mesenteric lymph nodes (MLNs) of orally immunized WT mic

223 ne receptor (Cxcr)5(+) CD4(+) T cells in the mesenteric lymph nodes (MLNs) of transiently n-6-fed mic

224 T cells were cultured with purified DCs from mesenteric lymph nodes (MLNs) or intestines of wild-type

225 Migration of Foxp3+ T cells from the mesenteric lymph nodes (MLNs) to the lamina propria occu

226 elium from where they are transported to the mesenteric lymph nodes (MLNs) within migrating immune ce

227 ses lymphopenia, lymphocyte sequestration in mesenteric lymph nodes (MLNs), and immunosuppression.

228 nting translocation of commensal bacteria to mesenteric lymph nodes (mLNs), and limiting colitogenic

229 lated from the intestinal lamina propria and mesenteric lymph nodes (MLNs), and the following paramet

230 ack CD103+CD11b- DCs in the lung, intestine, mesenteric lymph nodes (MLNs), dermis, and skin-draining

231 populations in the peripheral blood, liver, mesenteric lymph nodes (MLNs), jejunum, and bronchoalveo

232 enhances alloantigen presentation within the mesenteric lymph nodes (mLNs), mediated by donor CD103(+

233 from the gut to systemic sites, such as the mesenteric lymph nodes (MLNs), via CD11b(+) migratory de

234 n the lamina propria (LP) and migrate to the mesenteric lymph nodes (MLNs), where they drive the diff

235 atory responses in Peyer's patches (PPs) and mesenteric lymph nodes (MLNs).

236 nt a resident memory (Trm) population in the mesenteric lymph nodes (MLNs).

237 he lumen to a key immune inductive site, the mesenteric lymph nodes (MLNs).

238 patches, limited dendritic cell migration to mesenteric lymph nodes [mLNs] causing reduced T cell-med

239 these cells in the liver, lung, spleen, and mesenteric lymph node, more than one week after transpla

240 ntestinal inflammation by activating gut and mesenteric lymph node myeloid cells.

241 Except for IgG3 in the IPPs and mesenteric lymph nodes, no stochastic pattern of Cgamma

242 it at high levels in the small intestine and mesenteric lymph node of young adult mice, suggesting a

243 SS2 was detected in mesenteric lymph nodes of 40% of challenged piglets.

244 of gammadelta-17 cells in the peripheral and mesenteric lymph nodes of adult IL-15Ralpha KO mice, but

245 population of DCs was also depleted from the mesenteric lymph nodes of B27-transgenic rats.

246 MAbs) produced from the Peyer's patches and mesenteric lymph nodes of BALB/c mice immunized intragas

247 ing with the population of CD4+ cells in the mesenteric lymph nodes of BM-->tg epsilon26 mice.

248 Colon tissues and mesenteric lymph nodes of Card9-null mice had reduced le

249 NF-alpha), in PBMCs, intestinal lesions, and mesenteric lymph nodes of cattle naturally infected with

250 persistence of Salmonella in the spleen and mesenteric lymph nodes of chronically infected mice.

251 CD4+ cells purified from the mesenteric lymph nodes of colitic BM-->tg epsilon26 mice

252 rase chain reaction of intestinal mucosa and mesenteric lymph nodes of Duoxa(-/-) mice that lack func

253 ency of CD4(+)CD25(+)Foxp3(+) T cells in the mesenteric lymph nodes of GF mice compared with SPF mice

254 regulate T cell homing, were also reduced in mesenteric lymph nodes of infected AMCase-deficient mice

255 ions is essential for their expansion in the mesenteric lymph nodes of infected mice.

256 required to maintain the Th2 response in the mesenteric lymph nodes of infected mice.

257 TLR4 conditioned the in vivo mobilization to mesenteric lymph nodes of intestinal migratory CD103(+)

258 ica infection within the Peyer's patches and mesenteric lymph nodes of mice.

259 ed mice readily accumulated in the colon and mesenteric lymph nodes of recipient mice, and they recon

260 Cells isolated from mesenteric lymph nodes of the transgenic mice had signif

261 om rectal biopsy specimens, bone marrow, and mesenteric lymph nodes of vaccinated infected, unvaccina

262 CCR9(high) memory-phenotype cells within the mesenteric lymph nodes of wild-type hosts.

263 as comparable to those found in the draining mesenteric lymph node or the spleen.

264 +) T cells preferentially home to spleen and mesenteric lymph nodes owing to increased expression of

265 or cells, which localized in T-cell zones of mesenteric lymph nodes, Peyer's patches, spleen, and thy

266 Culture of mesenteric lymph nodes showed higher density of infectio

267 D69 by CD4 T cells isolated from the spleen, mesenteric lymph nodes, small intestinal lamina propria,

268 the mouse and are particularly prevalent in mesenteric lymph nodes, spleen, and liver.

269 phimurium recovered from the lamina propria, mesenteric lymph nodes, spleen, and liver.

270 d in cervical lymph nodes at 8 weeks, in the mesenteric lymph nodes, spleen, and Peyer's patches at 1

271 enous increase of SIV RNA in superficial and mesenteric lymph nodes, spleen, and the gastrointestinal

272 develop organized lymphoid tissues including mesenteric lymph nodes, splenic follicles and gut-associ

273 The cavitating mesenteric lymph node syndrome (CMLNS) is a rare manifes

274 s were sacrificed on day 1 after injury, and mesenteric lymph node T cells were isolated.

275 f antigen-specific regulatory T cells in the mesenteric lymph nodes than mast cell-containing control

276 greater dissemination of the bacteria to the mesenteric lymph nodes than mice infected with wild-type

277 slocate, most likely via Peyer's patches and mesenteric lymph nodes, to the internal organs and trigg

278 stantially in their ability to expand in the mesenteric lymph nodes, trigger proinflammatory cytokine

279 ed lymphatic transport of dendritic cells to mesenteric lymph nodes, two features likely to actively

280 hese CD8(+) T(reg) undergo conversion in the mesenteric lymph nodes under the influence of recipient

281 o initially differentiate in the GALT and/or mesenteric lymph nodes upon Ag encounter and subsequentl

282 nces in survival observed between resectable mesenteric lymph nodes versus unresectable masses in the

283 Affymetrix porcine GeneChip analysis of pig mesenteric lymph nodes was used to identify 848 genes sh

284 Spleen and mesenteric lymph node were collected, processed, and ana

285 endogenous TGF-beta, DCs from gut-associated mesenteric lymph nodes were capable of differentiating F

286 ed that 1% to 2% of lymphocytes in the graft mesenteric lymph nodes were CD4/CD25(HIGH+)/Foxp3(+) cel

287 Spleens and mesenteric lymph nodes were collected and analyzed for T

288 s (cDC) in the intestinal lamina propria and mesenteric lymph nodes were GFP(+) However, in vitro inf

289 The Peyer's patches and mesenteric lymph nodes were markedly enlarged with expan

290 pha(-/-)) mice that lack Peyer's patches and mesenteric lymph nodes were orally infected with murine

291 Both the ileum and the mesenteric lymph nodes were persistently infected for mo

292 PA-TG, the concentrations of free MPA in the mesenteric lymph nodes were significantly enhanced (up t

293 bacteria across the intestinal epithelium to mesenteric lymph nodes were significantly greater in wil

294 aive lymphocytes traffic to the gut-draining mesenteric lymph nodes where they undergo antigen-induce

295 mRNA expression was highly inducible in the mesenteric lymph nodes, whereas COX-1 mRNA levels were c

296 of the target genes in colonic biopsies and mesenteric lymph nodes which was accompanied with a dist

297 g of ILCs from the intestine to the draining mesenteric lymph nodes, which specifically for the LTi-l

298 -specific CD4 T cells in Peyer's patches and mesenteric lymph nodes, which was accompanied by increas

299 cell populations in the Peyer's patches and mesenteric lymph nodes, while 6'-sialyllactose also indu

300 arterial rim enhancement around the necrotic mesenteric lymph nodes, without venous wash-out.