ライフサイエンスコーパス: mesentery

1 l muscle, and is unlike other vascular beds (mesentery).

2 rfused venular microvessels of mouse and rat mesentery).

3 vasculature of the allergen-challenged mouse mesentery.

4 y, posterior to the descending colon and its mesentery.

5 n accumulated throughout the interior of the mesentery.

6 and in association with blood vessels in the mesentery.

7 or idiopathic inflammatory conditions in the mesentery.

8 ersus unresectable masses in the root of the mesentery.

9 ation of E28 pig pancreatic primordia in the mesentery.

10 penetrated through the muscle wall into the mesentery.

11 symmetries in the architecture of the dorsal mesentery.

12 es involving the pancreatic head and root of mesentery.

13 thin, elastic membranous tissue, the dorsal mesentery.

14 ital microscopy on IL-1beta-stimulated mouse mesentery.

15 l microscopy in the microvessels in five rat mesenteries.

16 iver (53%), small bowel (25%), kidney (17%), mesentery (14%), adrenal gland (8%), abdominal wall (8%)

17 dian, 83 v 63 mm; P = .003), and the area of mesentery (19,657 v 11,829 mm(2); P < .0001).

18 P < .001), resulting in a smaller amount of mesentery (8,309 v 17,957 mm(2), P < .001) and nodal yie

19 We used mouse mesentery, a tissue whose numerous microvessels are high

20 he major source of oxidants generated in the mesentery after abdominal irradiation.

21 ure, the large accumulations observed in the mesenteries and ascites fluid of tumor-bearing animals m

22 oth muscle of all major blood vessels in the mesenteries and gut.

23 carcinomas in the colon that spread into the mesentery and adjacent organs.

24 termediate region by surgical lesions of the mesentery and by application of guanethidine (3 microM)

25 and thoracic levels stop short of the dorsal mesentery and do not enter the gut.

26 ue lymphatic channels were identified in the mesentery and followed to the blue-stained SN(s), which

27 t cells from other axial levels to enter the mesentery and gut mesenchyme.

28 n kidneys, lymph nodes, blood vessels, bowel mesentery and intestine.

29 at vasohibin-1 and VEGF are up-regulated, in mesentery and liver, in cirrhotic and precirrhotic porta

30 ctin, with the largest responses observed in mesentery and lung.

31 denopathy, and lymphangiogenesis in both the mesentery and mucosa.

32 kidneys, liver, musculoskeletal system, and mesentery and pancreas were not altered by diaspirin cro

33 t, spleen, musculoskeletal system, skin, and mesentery and pancreas.

34 oneal cavity, such as abdominal lymph nodes, mesentery and peri-intestinal adipose tissues, demonstra

35 uation of the bowel lumen and wall, adjacent mesentery and soft tissues, as well as a variety of extr

36 opportunity to invade the mesenchyme of the mesentery and the gut, so that earlier arrival assures t

37 lete lymphangion) were isolated from the rat mesentery and tied to glass cannulae capable of independ

38 denopathy, thickening of the omentum and the mesentery, and ascites.

39 articularly advantageous for the peritoneum, mesentery, and bowel.

40 ic fragment to different sites (mediastinum, mesentery, and kidney capsule) of ATX B6 mice treated wi

41 terioles serving heart, brain, kidney, lung, mesentery, and skin; plus aortic, carotid, and mesenteri

42 mural striation, fatty proliferation of the mesentery, and soft-tissue infiltration of pericolonic f

43 The mesenchymal cells of the dorsal mesentery are more condensed on the left side than on th

44 tolic blood pressure (SBP) by telemetry, and mesentery artery endothelial function by pressurized myo

45 Using the mesentery as a model tissue, we show that Tbx1 is not re

46 vessel walls and to connective tissue in the mesentery as they migrate toward the gonadal ridges.

47 ed to positional changes of the colon in the mesentery, as opposed to true mobility of the polyp.

48 ion, very few, if any, PGCs found in the gut mesentery at E10.5 migrate into the genital ridges.

49 age dependency and development along the gut-mesentery-blood-brain course of infection, can be replic

50 cine tissue in either the mediastinum or the mesentery, but not in mice grafted under both kidney cap

51 ynovial capillaries, so the results for frog mesentery capillaries cannot be generalised.

52 he majority of cases involve the small bowel mesentery; colorectal MP is rare.

53 ripotent stem cell lines, gonadal ridges and mesenteries containing primordial germ cells (PGCs, 5-9

54 pport the physiological relevance of the rat mesentery culture model as a biomimetic tool for investi

55 Recently, our laboratory introduced the rat mesentery culture model as an ex vivo experimental platf

56 rs, with most staple lines oversewn, and all mesentery defects closed.

57 The dorsal mesentery (DM) is the major conduit for blood and lympha

58 symmetrical cellular behaviors in the dorsal mesentery (DM) of the early mid-gut, a structure connect

59 asymmetric cell behaviors within the dorsal mesentery (DM), which suspends the gut tube, and is down

60 ing the phenotype of new microvessels in the mesentery during induction of vascular remodelling by ov

61 ide synthase in the fat pad of the adult rat mesentery during inhibition of angiopoietin signalling w

62 We find that the dorsal mesentery ECM is indeed left-right asymmetric and moreov

63 determined that Bmp2 expressed in the dorsal mesentery establishes differential elongation rates betw

64 teraction in the microcirculation of the rat mesentery exposed to 25 mmol/l D-glucose for 12 h.

65 bstruction, hemorrhage, cancer and thickened mesentery; extensive disease; the presence of short gut;

66 l foster future investigation of the role of mesentery fat in colorectal diseases.

67 lect the larger sizes of rdh1-null mice, but mesentery, femoral, and inguinal fat pads grow dispropor

68 Second, in the E9.0-E9.5 period, before the mesentery forms, PGCs very rapidly exit the gut, but do

69 ral echinoderm tissues, including esophagus, mesenteries, gonads, respiratory trees, hemal system, te

70 re exposure to VEGF and 24 h later after the mesentery had been replaced in the abdominal cavity.

71 Diaphragm disease and large bowel mesentery implants were the only CT predictors of subopt

72 tion of tumors on the peritoneal surface and mesentery in an i.p. ovarian xenograft model.

73 ructural evidence in the capillaries of frog mesentery indicates a regularity in the structure of the

74 ellate cells), and splanchnic organs (liver, mesentery, intestine, colon, and spleen) were isolated f

75 neutropenic mice, ATAK cells spread from the mesentery into visceral organs on days 1-3.

76 e of the vascular anatomy of the root of the mesentery is necessary for the performance of complex su

77 lthough VEGF(165)b is anti-angiogenic in the mesentery, it does signal in endothelial cells in vivo r

78 afted RV-ATL cells developed lymphoma in the mesentery, liver, thymus, lungs, and spleen.

79 y observed, with occasional viremia; tonsil, mesentery lymph nodes, and intestinal mucosa served as m

80 In rat mesentery microvessels after occlusion, circulating leuk

81 ultifunctional intravital videomicroscopy in mesentery microvessels with hydroethidine, an oxidant-se

82 l and Phillips on individually perfused frog mesentery microvessels.

83 ior aspect of the pancreatic head or root of mesentery (mid gut carcinoid) may involve one of the 2 p

84 Using a rat mesentery model of inflammation-induced angiogenesis and

85 of various organs including the heart, lung, mesentery, muscle, and eye of different species.

86 o apply to the vascular systems of the lung, mesentery, muscle, eye, and so on.

87 is (n = 7), hepatoduodenal ligament (n = 3), mesentery (n = 2), mediastinum (n = 4), portal venous sy

88 ty in vascularized structures located in the mesentery near the pancreas, intestines, and spleen.

89 ith control tissue, as well as in livers and mesenteries of rats and mice with cirrhosis or/and porta

90 AF increased IOI to comparable values in the mesenteries of wild-type mice and those lacking the gene

91 ntery of wild-type mice but much less in the mesentery of eNOS-/- mice.

92 al cell lines established from the ovary and mesentery of female H-2K(b)-tsA58 mice were tested for r

93 in individually perfused microvessels in the mesentery of frogs and rats.

94 Xenotransplantation in mesentery of pig pancreatic primordia obtained very earl

95 ent intra-abdominal shunts) of BDL rats, and mesentery of sham and BDL rats.

96 polymers, and implanted into the small bowel mesentery of syngeneic animals.

97 hat typically affects the adipose tissue and mesentery of the small intestine but may also affect the

98 Similarly, PAF increased IOI in the mesentery of wild-type mice but much less in the mesente

99 ndrome, causing selective hypertrophy of the mesentery, omentum and other lymphoid tissue-containing

100 superior mesenteric vein at the level of the mesentery or head of the pancreas.

101 y disease involvement at the cut edge of the mesentery or nonserosalized portions of the colon.

102 e peritoneum (P < .05), bowel (P < .01), and mesentery (P < .05).

103 unit C fibre afferents innervating duodenum, mesentery, pancreas, portal hepatis, bile duct, gall bla

104 is involved in tumor cell attachment to the mesentery possibly via interaction with tumor cell surfa

105 re performed excising the envelope of rectal mesentery posteriorly and the supporting tissues lateral

106 ed and large-sized vessels of the serosa and mesentery preferentially demonstrated histologic changes

107 orcine thymus into either the mediastinum or mesentery provides earlier and more efficient reconstitu

108 In the mesentery, Prox1 expression is high in valve-forming lym

109 ed from the microbiota maintain inflammatory mesentery remodeling and consequently, transient ablatio

110 performed using an appropriately wide rectal mesentery resection technique if the tumor was high; if

111 fferent vascular beds (lung, heart, stomach, mesentery, small intestine, large intestine, and muscle)

112 volves vessels of the submucosa, serosa, and mesentery, some mucosal alterations have been suggested

113 Embryologic development of the dorsal mesenteries suggests the existence of retromesenteric pl

114 ll relative to parenchymal tissue in the rat mesentery suggests that in addition to serving as a cond

115 transmigrated leukocytes (P<0.01 vs. control mesenteries superfused with Krebs-Henseleit buffer).

116 immunofluorescence staining of ear skin and mesentery that lymphatic vessels in Ang2(-/-) mice fail

117 echanism of VEGF overexpression in liver and mesentery that promotes pathologic, but not physiologic,

118 s in the cellular architecture of the dorsal mesentery, the structure that connects the primitive gut

119 al elongation rates between the gut tube and mesentery, thereby regulating the compressive forces tha

120 force interstitial osmotic pressure in frog mesenteries, this was assessed in synovium.

121 In vivo, in the rat mesentery, thrombin (0.5 U/ml) or N(G)-nitro-L-arginine-

122 ological materials--including blood vessels, mesentery tissue, lung parenchyma, cornea and blood clot

123 a indicate that, at least in the CD-affected mesentery, TLOs are positioned along collecting lymphati

124 fferences in cellular organization cause the mesentery to assume a trapezoidal shape, tilting the pri

125 ntravital video microscopy in exposed rabbit mesentery to investigate the potential role of fibrinoge

126 1.2 to 35 dyne/cm2) of the exteriorized rat mesentery using high-resolution intravital microscopy.

127 tra- or retroperitoneal solid organs, bowel, mesentery, vascular structures, diaphragm, and urinary t

128 Similar phosphorylations were observed when mesentery was exposed to VPF/VEGF in vitro, or when mese

129 e tumor was in the middle or low rectum, all mesentery was resected.

130 In this model, the exteriorized mesentery was superfused with ferric chloride and the ac

131 Using intravital microscopy of the rat mesentery, we measured leukocyte-endothelium interaction

132 sly with E28 pig pancreatic primordia in the mesentery, we show normalization of glucose tolerance in

133 ry was exposed to VPF/VEGF in vitro, or when mesenteries were harvested from mice bearing the mouse o

134 At intervals after injecting VPF/VEGF i.p., mesenteries were harvested, extracted, and immunoprecipi

135 served due to idiopathic inflammation of the mesentery were identified.

136 f lymph nodes inside the intussusception and mesentery were noted.

137 TA3/St cells that initially attached to the mesentery were strongly CD44 positive.

138 dually perfused venular microvessels in frog mesentery when the perfusate contained albumin.

139 single perfused venular microvessels in rat mesentery, which enabled direct observation of permeabil

140 ing ventral to the dorsal aorta and into the mesentery, which is the portal to the gut.

141 rats and vasorelaxation in the isolated rat mesentery, which was blocked by the NO synthase inhibito

142 isappear from the regenerating intestine and mesentery, while fibronectin labeling and 4G7 (an echino

143 Concomitant superfusion of the rat mesentery with 10 nmol/liter of each of three lipoxin an

144 -glucose was inhibited by superfusion of the mesentery with 30 nmol/l bisindolylmaleimide-I, a potent

145 Superfusion of the rat mesentery with 50 micromol/liter NG-nitro-L-arginine met

146 Superfusion of IL-1beta-stimulated rabbit mesentery with C3a resulted in a rapid and stable adhesi

147 Superfusion of the rat mesentery with either 0.5 U/ml thrombin or 50 microM L-N

148 Superfusion of the rat mesentery with either thrombin or L-NAME consistently an

149 We demonstrated that superfusion of rat mesentery with LPS resulted in significant increases in

150 Thus, in vivo superfusion of the rat mesentery with stable lipoxin analogs at 10 nmol/liter r