ライフサイエンスコーパス: mesocortical

1 tures may be part of the neurobiology giving mesocortical afferents their hormone sensitivities and/o

2 ted with trait anxiety, as well as increased mesocortical and decreased mesohippocampal coupling.

3 hese 5-HT1A-mediated actions of 5-HT include mesocortical and mesolimbic dopaminergic neurons having

4 ntipsychotics that may differentially target mesocortical and mesolimbic dopaminergic neurons having

5 g revealed that kappa2 receptors localize to mesocortical and subcortical limbic areas, including the

6 einforcement are influenced by mesostriatal, mesocortical, and mesolimbic dopamine systems.

7 These findings reveal that mesocortical DA can facilitate dissociable components of

8 ulation secondary to sustained deficiency in mesocortical DA function.

9 t this impairment relates to a deficiency in mesocortical DA function.

10 ereas the adjacent A10 region mesolimbic and mesocortical DA neurons are relatively spared.

11 In contrast, short burst stimulation of mesocortical DA neurons that produced transient (<4 s) D

12 sequences, and the delayed maturation of the mesocortical DA pathway.

13 eal an additional level of complexity in how mesocortical DA regulates different forms of cognition v

14 We conclude that while baseline mesocortical DA synthesis is indeed dependent on tyrosin

15 ) receptors potentiate the phasic release of mesocortical DA.

16 Mesocortical DAT immunoreactivity in motor, premotor, an

17 s at early Parkisnon's disease stages, while mesocortical degeneration mediated increases of ineffici

18 A) receptors modulate the functioning of the mesocortical dopamine (DA) pathway.

19 Mesocortical dopamine (DA) regulates a variety of cognit

20 rontal cortex (PFC) and are modulated by the mesocortical dopamine (DA) system.

21 However, why and how mesocortical dopamine axon density increases across adol

22 hese findings suggest that the depression of mesocortical dopamine axons that follows gonadectomy is

23 st in part, from a paucity of DAT content in mesocortical dopamine axons, as well as a distribution o

24 Disrupted mesocortical dopamine contributes to cognitive symptoms

25 in cognition, the specific contributions of mesocortical dopamine depletion and striatal dysfunction

26 e present data suggest that the influence of mesocortical dopamine neurons on the dopamine projection

27 task-related changes previously observed in mesocortical dopamine neurons, which have been implicate

28 uction of sensitization, suggesting that the mesocortical dopamine pathway is not involved in the acu

29 f cortical/prefrontal cortical function, the mesocortical dopamine system.

30 from animal studies that the mesolimbic and mesocortical dopamine systems are sensitive to circulati

31 ion who study the role of the mesolimbic and mesocortical dopamine systems that originate in the vent

32 resentations of reward to the mesolimbic and mesocortical dopamine systems, thereby initiating motiva

33 oss of CB1R-mediated retrograde signaling on mesocortical dopamine transmission, and, in turn, altere

34 es in corticocortical, corticobasal ganglia, mesocortical dopamine, and cerebellar-thalamic-prefronta

35 nous input to both regions, for example from mesocortical dopamine, or separate and consecutive stage

36 eads to selective abnormalities in postnatal mesocortical dopaminergic maturation and behavioral abno

37 pecific period of maturation and function of mesocortical dopaminergic neurons and their sensitivity

38 The epigenetic changes in the mesocortical dopaminergic neurons were prevented when an

39 r impact on cognitive functions modulated by mesocortical dopaminergic neurons.

40 epressants, which act through enhancement of mesocortical dopaminergic signaling.

41 ntral tegmental area, and activation of this mesocortical dopaminergic system decreases spontaneous a

42 c projections to motor cortices, whereas the mesocortical dopaminergic system facilitates working mem

43 In contrast, mesocortical dopaminergic transmission appears well pres

44 function as a direct consequence of impaired mesocortical dopaminergic transmission or an indirect co

45 with a parallel shift toward mesolimbic and mesocortical function.

46 an animal model that affects mesolimbic and mesocortical function.

47 tion that paralleled established patterns of mesocortical hormone sensitivity, including the androgen

48 inergic cell group, reminiscent of recurrent mesocortical loops described in mammals.

49 on of dopamine signaling may be an important mesocortical mechanism contributing to its ability to ai

50 estimates of DA utilization and synthesis in mesocortical, mesolimbic and nigrostriatal DA terminal r

51 correlated with increased activation in the mesocortical-mesolimbic reward circuitry encompassing th

52 ely labeled dopaminergic and nondopaminergic mesocortical neurons projecting to prefrontal, premotor,

53 ning mesoaccumbens neurons and DA-containing mesocortical neurons suggests novel mechanisms through w

54 , have been linked to changes in mesolimbic, mesocortical neurotransmitter systems utilizing biogenic

55 and Lewis rats have selective differences in mesocortical, nigrostriatal and mesolimbic DA neuronal r

56 related to hypodopaminergic activity in the mesocortical pathway and prefrontal cortex, are predicti

57 development of the DA system, especially the mesocortical pathway involved in action adaptation to ru

58 hich form the nigrostriatal, mesolimbic, and mesocortical pathways.

59 rcuitry, e.g., mesostriatal, mesolimbic, and mesocortical pathways.

60 th origins of nigrostriatal, mesolimbic, and mesocortical projections are consistent with identified

61 he proportions of dopamine neurons making up mesocortical projections were approximately 30% in males

62 oral systems, progressing from mesolimbic to mesocortical regions and from latent to patent behaviors

63 l rats point to the regulatory influences on mesocortical serotonin circuits in highly aggressive ani

64 sterior bed nucleus of the stria terminalis, mesocortical structures and the hippocampal formation.

65 m neurons may implicate VPvm in facilitating mesocortical structures with information related to the

66 We suggest that the mesocortical system might transmit fast signals about re

67 nd electrochemical measures to show that the mesocortical system produces a fast non-DA-mediated post

68 RAIC projections to mesolimbic/mesocortical ventral forebrain circuits are likely to pa