コーパス検索結果 (1語後でソート)
通し番号をクリックするとPubMedの該当ページを表示します
1 coitum (dpc) on the ventromedial side of the mesonephros.
2 1(+) mesenchymal cells that migrate from the mesonephros.
3 by mature structures such as the somites or mesonephros.
4 ds arise on the ventromedial surface of each mesonephros.
5 rs that are repressed by the presence of the mesonephros.
6 y coelomic epithelium invagination along the mesonephros.
7 lockade on the WD cultured in the absence of mesonephros.
8 to become the nephric duct, pronephros, and mesonephros.
9 ation in the absence of a developing pro- or mesonephros.
10 of homeotic transformation of metanephros to mesonephros.
11 ue and Lmx-1 mRNA expression as a marker for mesonephros.
12 umber of endothelial cells from the adjacent mesonephros, a mechanism totally absent in XX gonads.
13 the PGE2-cAMP-PKA pathway in the aorta-gonad-mesonephros (AGM) abolished enhancement in hematopoietic
14 c stem cells (HSCs) arise in the aorta-gonad-mesonephros (AGM) and mature as they transit through the
15 c stem cell emergence, viz., the aorta-gonad-mesonephros (AGM) and the fetal liver at 10.5-11.5 dpc,
17 tive direct Notch targets in the aorta-gonad-mesonephros (AGM) embryonic tissue by chromatin precipit
24 (CD34(+)c-kit(+)) cells from the aorta-gonad-mesonephros (AGM) region of the developing mouse are mul
26 mogenic endothelium within the aorta, gonad, mesonephros (AGM) region of the mammalian embryo is cruc
27 fates concurrently occur in the aorta-gonad-mesonephros (AGM) region prior to haematopoietic stem ce
28 inactivated SCL in yolk sac, the aortagonad-mesonephros (AGM) region, and fetal liver hematopoietic
29 HSCs were not released from the aorta-gonad-mesonephros (AGM) region, as evidenced by the accumulati
30 tem cells (HSCs) emerge from the aorta-gonad-mesonephros (AGM) region, but the molecular regulation o
31 an intraembryonic location, the aorta-gonad-mesonephros (AGM) region, is a site of residence and, po
32 t human HSCs emerge first in the aorta-gonad-mesonephros (AGM) region, specifically in the dorsal aor
33 such as the dorsal aorta of the aorta-gonad-mesonephros (AGM) region, suggesting that signals from t
34 lly dissect HSC emergence in the aorta-gonad-mesonephros (AGM) region, we screened a collection of in
42 vested from embryonic day 9 (E9) aorta-gonad-mesonephros (AGM) regions of GATA2 null embryos showed r
43 topoietic stem cells (HSCs) from aorta/gonad/mesonephros (AGM) regions of midgestation mouse embryos
44 Rare endothelial cells in the aorta-gonad-mesonephros (AGM) transition into hematopoietic stem cel
47 mice lacking RA synthesis and signalling in mesonephros and adjacent gonad and reveal that Stra8 exp
48 embryonic day 10.5 by the thickening of the mesonephros and consist of somatic cells and migratory p
52 We also show that the region between the mesonephros and the gonad harbors steroidogenic cell pre
55 ube derived from an epithelial anlage at the mesonephros anterior end, which then segregates from the
56 d/or para-aorta-splanchno-pleura/aorta-gonad-mesonephros are hypothesized to colonize the fetal liver
57 anterior structures, the pronephros and the mesonephros, are transitory and largely non-functional,
58 ated embryos cultured for 3 days in ovo, the mesonephros as well as the pronephros failed to develop
59 TH-responsive cells are present at the gonad/mesonephros border and seem to migrate into the XY but n
61 opoietic stem cells (HSC) in the aorta-gonad-mesonephros by abrogating Smad1 expression and the conse
67 ack, unilateral anomalous development of the mesonephros in males causes atresia of the homolateral e
69 elopment, endothelial cells migrate from the mesonephros into the gonad to form a coelomic blood vess
78 the para-aortic splanchnopleura/aorta-gonads-mesonephros of mouse embryos and that abrogation of nitr
79 -aortic splanchnopleura/aorta-genital ridges-mesonephros (P-Sp/AGM) region are the main sites of haem
80 ting cells from murine yolk sac, aorta-gonad-mesonephros, placenta, fetal liver, and bone marrow with
82 undergoes dramatic growth in the aorta-gonad-mesonephros region and by E11.5 reaches the size that ma
83 ls (HSCs) are first found in the aorta-gonad-mesonephros region and vitelline and umbilical arteries
84 itors are first generated in the aorta-gonad-mesonephros region between days 27 and 40 of human embry
85 c stem cells (HSCs) in the mouse aorta-gonad-mesonephros region emerge between embryonic days 10.5 an
86 genitor cells derived from the aorto gonadal mesonephros region of day 11 mouse embryos on the Jagged
87 (HSCs) that first appear in the aorta-gonado-mesonephros region of the fetus on embryonic day (E) 10.
89 ived during embryogenesis in the aorta-gonad-mesonephros region subsequently colonize fetal and adult
90 on and atria earlier than in the aorta-gonad-mesonephros region, and is transient and definitive in n
91 CX3CR1(+) cells localized to the aorta-gonad-mesonephros region, and visualized at embryonic day (E)9
92 they are first generated in the aorta-gonad-mesonephros region, but at later developmental stages, i
93 od vessel walls in the yolk sac, aorta-gonad-mesonephros region, embryonic liver, and fetal bone marr
94 os developed cd41(+) HSCs in the aorta-gonad-mesonephros region, which later migrated to the kidney,
97 poietic cluster formation in the aorta-gonad-mesonephros region; embryonic-to-adult transplantation s
99 endent induction of Stra8, but only when the mesonephros remains attached, pointing to a non-RA signa
100 pression pattern of the transgene in the pro/mesonephros suggest an intraembryonic site of developmen
101 tached, pointing to a non-RA signal from the mesonephros that induces Stra8 in the adjacent gonad.
102 sult from failure of cell migration from the mesonephros, thought to be a possible source of Leydig c
106 d11 is ectopically activated in the anterior mesonephros, we observe a partial transformation to a me
WebLSDに未収録の専門用語(用法)は "新規対訳" から投稿できます。