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1 regions are partly under the control of the mesostriatal and mesoaccumbens dopamine inputs, changes
2 h some of the known hormone sensitivities of mesostriatal and mesolimbic DA systems in rats and provi
8 c, and fast spiking GABAergic neurons of the mesostriatal circuit, imbalance of cholinergic and dopam
10 en dopamine (DA) neurons may explain why the mesostriatal DA neurons in the A9 region preferentially
13 (4, 17, and 24-26 months old) and history of mesostriatal dopamine (DA) depletion (1 or 14 months).
14 es have proposed that glutamate corelease by mesostriatal dopamine (DA) neurons regulates behavioral
15 Because of the involvement of mesolimbic and mesostriatal dopamine (DA) pathways in locomotion and st
19 a unilateral 6-hydroxydopamine lesion of the mesostriatal dopamine system reduced amphetamine- but no
23 tions from the orbitofrontal, limbic, and/or mesostriatal dopaminergic pathways, which conceivably ma
24 2 (FGF2) has been shown to interact with the mesostriatal dopaminergic system, and has been implicate
28 and positive reinforcement are influenced by mesostriatal, mesocortical, and mesolimbic dopamine syst
29 th established dopaminergic circuitry, e.g., mesostriatal, mesolimbic, and mesocortical pathways.
32 l intake may lead to neuroadaptations in the mesostriatal reward system, resulting in addiction pheno
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