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1  regions are partly under the control of the mesostriatal and mesoaccumbens dopamine inputs, changes
2 h some of the known hormone sensitivities of mesostriatal and mesolimbic DA systems in rats and provi
3  influences for androgens and estrogens over mesostriatal and mesolimbic DA systems.
4       Later during development, the separate mesostriatal and mesolimbic pathways differentiate throu
5 factory tubercle, respectively, constituting mesostriatal and mesolimbic pathways.
6 us accumbens, respectively, establishing the mesostriatal and the mesolimbic pathways.
7           However, the VGCCs that operate on mesostriatal axons are incompletely defined; previous st
8 c, and fast spiking GABAergic neurons of the mesostriatal circuit, imbalance of cholinergic and dopam
9 ns occur at the level of corticostriatal and mesostriatal circuitry or within striatal neurons.
10 en dopamine (DA) neurons may explain why the mesostriatal DA neurons in the A9 region preferentially
11 ad received unilateral 6-OHDA lesions of the mesostriatal DA pathway 8 weeks earlier.
12 of neurodegenerative disorders involving the mesostriatal DA system.
13 (4, 17, and 24-26 months old) and history of mesostriatal dopamine (DA) depletion (1 or 14 months).
14 es have proposed that glutamate corelease by mesostriatal dopamine (DA) neurons regulates behavioral
15 Because of the involvement of mesolimbic and mesostriatal dopamine (DA) pathways in locomotion and st
16  may reflect abnormalities in mesolimbic and mesostriatal dopamine (DA).
17      Corticostriatal glutamate afferents and mesostriatal dopamine afferents commonly converge onto t
18                                          The mesostriatal dopamine system is prominently implicated i
19 a unilateral 6-hydroxydopamine lesion of the mesostriatal dopamine system reduced amphetamine- but no
20 on (focused stereotypy) known to involve the mesostriatal dopamine system.
21                                              Mesostriatal dopaminergic neurons (DANs) and striatal ch
22 hat neuregulins can modulate the activity of mesostriatal dopaminergic neurons.
23 tions from the orbitofrontal, limbic, and/or mesostriatal dopaminergic pathways, which conceivably ma
24 2 (FGF2) has been shown to interact with the mesostriatal dopaminergic system, and has been implicate
25 l for the development and maintenance of the mesostriatal dopaminergic system.
26 urites rostrally along the trajectory of the mesostriatal fiber system.
27 are linked to abnormal mesocorticolimbic and mesostriatal function.
28 and positive reinforcement are influenced by mesostriatal, mesocortical, and mesolimbic dopamine syst
29 th established dopaminergic circuitry, e.g., mesostriatal, mesolimbic, and mesocortical pathways.
30             Repeated alcohol intake leads to mesostriatal neuroadaptations, resulting in drinking esc
31 ation's origin via axon collaterals from the mesostriatal pathway.
32 l intake may lead to neuroadaptations in the mesostriatal reward system, resulting in addiction pheno
33                             A well developed mesostriatal system was revealed formed by conspicuous g
34 ormal cell development in the mesolimbic and mesostriatal systems.
35 d neuronal development in the mesolimbic and mesostriatal systems.
36  dopaminergic transmission in mesolimbic and mesostriatal systems.

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