ライフサイエンスコーパス: mesothelial cell

1 lite exposures have similar effects on human mesothelial cells.

2 vide a novel mechanism for the generation of mesothelial cells.

3 n implicated previously in transformation of mesothelial cells.

4 PRRs) in bone marrow-derived macrophages and mesothelial cells.

5 elioma tumor cells in situ but not by normal mesothelial cells.

6 ted in asbestos-induced oncogenesis of human mesothelial cells.

7 in MM cell lines compared with non-malignant mesothelial cells.

8 signaling pathway compared with normal human mesothelial cells.

9 f neutrophils via IL-1R/MyD88 on neighboring mesothelial cells.

10 tumor 1 gene (Wt1) is expressed only in the mesothelial cells.

11 -6 from macrophages but robust production in mesothelial cells.

12 assayed for in vitro cytotoxicity to murine mesothelial cells.

13 ar matrix proteins, as well as to peritoneal mesothelial cells.

14 of chemokine and antimicrobial responses in mesothelial cells.

15 pancreatic tissue but not in melanocytes and mesothelial cells.

16 stimulated by angiogenic factors produced by mesothelial cells.

17 red chick embryonic and rat adult epicardial mesothelial cells.

18 lly expressed in MM cells compared to normal mesothelial cells.

19 in MM cells but had minimal effect on normal mesothelial cells.

20 We found that JCV did not infect human mesothelial cells.

21 l of a cell line derived from rat epicardial mesothelial cells.

22 required for the growth of SV40-transformed mesothelial cells.

23 signaling enhanced collagen I deposition by mesothelial cells.

24 ristically associated with SV40 infection of mesothelial cells.

25 xpression was restricted to human peritoneal mesothelial cells.

26 ers and is critical to the transformation of mesothelial cells.

27 rease in VEGF production from murine pleural mesothelial cells.

28 ely 40-fold higher levels of hDio2 mRNA than mesothelial cells.

29 g the adhesion of ovarian carcinoma cells to mesothelial cells.

30 interaction of ovarian carcinoma cells with mesothelial cells.

31 its limited expression in normal peritoneal mesothelial cells.

32 5) in a dose-dependent manner in rat pleural mesothelial cells.

33 in both rat lung epithelial and rat pleural mesothelial cells.

34 elial cells, follicular dendritic cells, and mesothelial cells.

35 been named mesothelin because it is made by mesothelial cells.

36 th factor or insulin-like growth factor-1 in mesothelial cells.

37 contribute to the tumorigenic conversion of mesothelial cells.

38 in 7 patients did not show TF expression by mesothelial cells.

39 assays, we find that apCAFs are derived from mesothelial cells.

40 an-specific signature genes of pericytes and mesothelial cells.

41 logy is obscured by inflammation or reactive mesothelial cells.

42 on of IL-1beta and IL-18 production in human mesothelial cells.

43 ne marrow derived macrophages, as well as by mesothelial cells.

44 orphologies and function compared to control mesothelial cells.

45 expression and TGF-beta1 expression in human mesothelial cells.

46 FA mRNA (P < 0.05) and protein (P < 0.05) in mesothelial cells.

47 sed concentrations of ID1 mRNA (P < 0.05) in mesothelial cells.

48 he O-GlcNAc signal primarily originates from mesothelial cells.

49 ion in normal human tissues is restricted to mesothelial cells.

50 ntum, which are covered by a single layer of mesothelial cells.

51 al effects and transforming actions in human mesothelial cells.

52 al steps of OvCa metastasis and suggest that mesothelial cells actively contribute to metastasis.

53 ometastatic spheroids that display increased mesothelial cell adhesion and submesothelial invasion.

54 Mesothelial cells also produced chemokines in response t

55 e relationships between epicardium, arterial mesothelial cells (AMCs), and the coronary vasculature.

56 Furthermore, mesothelial cell and macrophage expression of VEGF-C inc

57 Ectopic hTERT expression immortalized normal mesothelial cells and a premalignant, p16(INK4a)-negativ

58 host-derived Wnt5a, expressed by peritoneal mesothelial cells and adipocytes, as a primary regulator

59 Mesotheliomas are tumors arising from mesothelial cells and are associated with asbestos expos

60 SV40 did not lyse mesothelial cells and caused a high rate of transformati

61 lls associated with the basal lamina beneath mesothelial cells and expressing activated leukocyte cel

62 ulture containing primary human fibroblasts, mesothelial cells and extracellular matrix can be adapte

63 reased phosphorylation of Tyr-216 in pleural mesothelial cells and GSK-3beta mobilization from the cy

64 th increased DNA damage both in Met-5A human mesothelial cells and human DPM cell lines.

65 f innate immune mediators from primary mouse mesothelial cells and human monocytic MonoMac6 cells, an

66 rt their ability to colonize and multiply in mesothelial cells and in both resident and recruited leu

67 However, normal mesothelial cells and mesothelioma cells from Bap1(+/-)

68 TNT-mediated interaction between peritoneal mesothelial cells and OvCa cells was enhanced under comp

69 Immortalized human mesothelial cells and primary mesothelial cells, culture

70 y resident macrophages and then amplified by mesothelial cells and probably other cells of the perito

71 data identify distinctive fates for injured mesothelial cells and submesothelial fibroblasts during

72 ty, adhesion to and retraction of peritoneal mesothelial cells and subsequent anchoring.

73 t isolates may retain properties of reactive mesothelial cells and suggests that targets in addition

74 Both production of LPA by peritoneal mesothelial cells and the chemotactic activity in the co

75 d PDF promoted morphological preservation of mesothelial cells and the submesothelial zone in a mouse

76 submesothelial cells is higher than that of mesothelial cells and transitional cells.

77 n tumor microenvironment, notably peritoneal mesothelial cells and visceral adipose, secreted Wnt5a.

78 asked whether asbestos induced apoptosis in mesothelial cells and whether reactive oxygen species we

79 synthesis and secretion by peritoneal-lining mesothelial cells and/or fibroblasts in response to stim

80 h factor (EGF) is a potent mitogen for human mesothelial cells, and autophosphorylation of the EGF re

81 two epithelial cell types, keratinocytes and mesothelial cells, and determined the effect on prolifer

82 tracellular matrix components, human primary mesothelial cells, and full-thickness human peritoneum a

83 emical analysis, transcriptomic profiling of mesothelial cells, and in vitro mechanical stretch exper

84 Mesothelial cells are an important cellular component of

85 Accumulating evidence suggests that mesothelial cells are an important component of the meta

86 In conclusion, mesothelial cells are an important source of VEGF.

87 In tuberculous pleurisy pleural mesothelial cells are exposed to mycobacteria in the ple

88 Mesothelial cells are generally thought to be "bystander

89 chanisms by which asbestos induces injury in mesothelial cells are not known.

90 Mesothelial cells are uniformly infected but not lysed b

91 Collectively, our results highlight mesothelial cells as a key driver of ovarian cancer chem

92 on, this study identified O-GlcNAcylation in mesothelial cells as a potentially important molecular m

93 These results define mesothelial cells as microbial sensors through TLRs and

94 ed ovarian tumor cell adhesion to peritoneal mesothelial cells as well as migration and invasion, lea

95 vasculogenic niche composed of AMCs and sub-mesothelial cells at the base of the pulmonary artery.

96 ressed by submesothelial fibroblasts but not mesothelial cells, attenuated the peritoneal fibrosis bu

97 ally in the developing mesothelium reveals a mesothelial cell-autonomous role for Ezh2 in repression

98 s to telomerase activity before the infected mesothelial cells become transformed and immortalized.

99 by this approach was abundant in normal rat mesothelial cells but not expressed in rat MM cell lines

100 induced telomerase activity in primary human mesothelial cells, but not in primary fibroblasts.

101 not control particles, induced apoptosis in mesothelial cells by all assays and induction of apoptos

102 We found that infection of human mesothelial cells by SV40 is very different from the sem

103 1, Tcf21) and Tbx18, can be induced in naive mesothelial cells by the liver bud, both in vitro and in

104 The heart is lined by a single layer of mesothelial cells called the epicardium that provides im

105 Indeed, knockdown of CD157 in Met-5A mesothelial cells changed their morphology and cytoskele

106 e, for the first time, that human peritoneal mesothelial cells constitutively produce bioactive lipid

107 We show here that human peritoneal mesothelial cells constitutively produce LPA, which acco

108 Pleural mesothelial cells contribute to pleural rind formation b

109 Thus, specialized omental mesothelial cells coordinate the recruitment and aggrega

110 ms of interactions of pathogenic fibers with mesothelial cells, crucial signaling pathways, and genet

111 rtalized human mesothelial cells and primary mesothelial cells, cultured from human omentum or clinic

112 a induced MIP-1alpha and MCP-1 expression in mesothelial cells, demonstrating that mesothelial cell-d

113 ion in mesothelial cells, demonstrating that mesothelial cell-derived C-C chemokines play a biologica

114 These data suggest that downregulation of mesothelial cell-derived ITLN1 in the omental tumor micr

115 We show that SV40 infection of human mesothelial cells directly causes overexpression of Notc

116 ) three Ca(2+) shuttling pathways in primary mesothelial cells during Ca(2+) oscillations: Ca(2+) shu

117 enesis and indicate that signals controlling mesothelial cell entry are organ specific.

118 We visualized WT1(+) mesothelial cell entry into the lung by live imaging and

119 e vertebrate heart originates from migratory mesothelial cells (epicardium) that give rise to coronar

120 Changes in p65 expression in pleural mesothelial cells exposed to asbestos in inhalation expe

121 Mesothelial cells exposed to asbestos or bleomycin for 9

122 nuclear translocation of p65 in rat pleural mesothelial cells exposed to asbestos.

123 also shown to efficiently rupture peritoneal mesothelial cells, exposing the submesothelial collagen

124 Mesothelial cells expressed podoplanin and ALCAM.

125 BKV and SV40 infected mesothelial cells, expressed viral oncoproteins, and cau

126 lly devoid of a mesothelium but that serosal mesothelial cells expressing the Wilm's tumor protein (W

127 31 was mainly derived from the local tissue (mesothelial cells, fibroblasts).

128 During pancreatic cancer progression, mesothelial cells form apCAFs by downregulating mesothel

129 Adenoviral-Cre treatment of normal mesothelial cells from Bap1;Nf2;Cdkn2a CKO mice, but not

130 spheroid formation in vitro, suggesting that mesothelial cells from Bap1;Nf2;Cdkn2a mice have stem ce

131 ll lines than they were in normal peritoneal mesothelial cells from surgical specimens, in organ cult

132 Sub-mesothelial cells from this niche differentiate into lym

133 wth gene, may encode a negative regulator of mesothelial cell growth.

134 sion of ovarian cancer cells into peritoneal mesothelial cells has also been analyzed and shown to re

135 marker Wt1, appeared to show that peritoneal mesothelial cells have a range of differentiative capaci

136 Here we show that malignant mesothelial cells have an elevated Notch signaling pathw

137 y assays for PLA(2) indicate that peritoneal mesothelial cells have strong constitutive PLA(2) activi

138 alysis of global gene expression of isolated mesothelial cells highlighted mesothelin (Msln) and its

139 ly, we reported that SV40 infection of human mesothelial cells (HM) causes aberrant methylation of th

140 SV40 infection of human mesothelial cells (HM) causes early cellular immortaliza

141 Asbestos is cytotoxic to human mesothelial cells (HM), which appears counterintuitive f

142 er mesothelial cells (SHM) and primary human mesothelial cells (HM).

143 s not induce transformation of primary human mesothelial cells (HM); instead, asbestos is very cytoto

144 1 production were measured in vitro in human mesothelial cells (HMC) in the presence or absence of me

145 Human peritoneal mesothelial cells (HPMC) constitutively expressed ICAM-1

146 We have shown that human peritoneal mesothelial cells (HPMCs) recovered from the pelvic peri

147 ted in TGF-beta1-stimulated human peritoneal mesothelial cells (HPMCs), non-adherent cells obtained f

148 Human peritoneal mesothelial cells (HPMCs), the main source of IL-6 and V

149 ning vesicle transportation in human pleural mesothelial cells (HPMCs).

150 ffluents, peritoneal tissues, and peritoneal mesothelial cells (HPMCs).

151 SV40 may be related to the very high rate of mesothelial cell immortalization that is characteristica

152 Treatment of mesothelial cells in culture with carboplatin resulted i

153 uption is repaired and replaced by surviving mesothelial cells in peritoneal injury, and not by subme

154 esive nanoparticles (BNPs) can interact with mesothelial cells in the abdominal cavity and significan

155 highlight an important role for hypoxia and mesothelial cells in the modification of the extracellul

156 of transformation in infected primary human mesothelial cells in tissue culture, leading to the form

157 achment of ovarian tumor cells to peritoneal mesothelial cells in vitro and increases the numbers of

158 overexpression of ET-1 induced MMT in human mesothelial cells in vitro and promoted the early cellul

159 Blocking fibronectin production in primary mesothelial cells in vitro or in murine models, either g

160 roduction in pleural tissues in vivo, and by mesothelial cells in vitro.

161 TGF-beta increases the VEGF production by mesothelial cells in vivo and in vitro.

162 on of Wilms tumor 1 (WT1), a known marker of mesothelial cells, in various cell types in normal and f

163 r genetic mapping of Wilms' tumor-1-positive mesothelial cells indicated that peritoneal membrane dis

164 Activation of LPA1 on mesothelial cells induced these cells to express connect

165 this activity was undetectable or minimal in mesothelial cells infected and/or transformed by SV40 ta

166 tos did not influence telomerase activity in mesothelial cells infected with SV40.

167 acterization of icodextrin-based PDF-induced mesothelial cell injury identified aB-crystallin as the

168 ory effects of DLX4 on CD44 levels and tumor-mesothelial cell interactions were abrogated when IL-1be

169 gated the ability of DLX4 to stimulate tumor-mesothelial cell interactions.

170 Phenotype conversion of mesothelial cells into myofibroblasts, the so-called mes

171 RK1/2 activation in pulmonary epithelial and mesothelial cells is unclear.

172 ormal tissues, mesothelin is present only on mesothelial cells, it represents a good target for antib

173 etastasis, potentiating invasion through the mesothelial cell layer and colonization of the submesoth

174 its ability to recruit an entirely exogenous mesothelial cell layer during development.

175 The epicardium, a mesothelial cell layer enveloping the myocardium, is act

176 The epicardium is a mesothelial cell layer essential for vertebrate heart de

177 tant contribution of somatic mesoderm to the mesothelial cell layer of the PE.

178 ovarian cancer metastasis, clearance of the mesothelial cell layer, to examine the clearance ability

179 cin promoted intercalation of filopodia into mesothelial cell layers and cell invasion.

180 s solution promotes glycolysis in peritoneal mesothelial cells, leading to mesothelial-to-mesenchymal

181 A human mesothelial cell line (LP9/TERT-1) and isolated human pl

182 documented by immunocytochemistry in a human mesothelial cell line (MET5A) exposed to various concent

183 AR5 as well as LP9 cells, a human peritoneal mesothelial cell line, were analyzed by flow cytometry f

184 imens (n = 5), and MPM and SV40-immortalized mesothelial cell lines (n = 5).

185 SV40 oncoprotein expression in murine mesothelial cell lines enhanced spontaneous and asbestos

186 Murine mesothelial cell lines lacking wild-type p53 due to a po

187 addition, stress-induced senescence in human mesothelial cell lines was impaired by SV40 oncoprotein

188 immortal cell lines (SV40-transformed human mesothelial cell lines, S-HML).

189 the invasion of tumor cell clusters into the mesothelial cell lining of peritoneal cavity organs; how

190 mas (MMs) are aggressive tumors derived from mesothelial cells lining the lungs, pericardium and peri

191 g through the mesothelium, a single layer of mesothelial cells lining the peritoneal cavity.

192 eads by implantation of tumor cells onto the mesothelial cells lining the peritoneal cavity.

193 Endogenous peritoneal macrophages and mesothelial cells lining the peritoneum contain MCP-1, w

194 Binding of mesothelin on normal mesothelial cells lining the pleura or peritoneum to the

195 Activation of mesothelial cell LPA1 induced CTGF expression by inducin

196 BKV replicated faster than SV40 and caused mesothelial cell lysis, not cellular transformation.

197 er, treatment with an antibody targeting the mesothelial cell marker mesothelin can effectively inhib

198 Taken together, our study elucidates how mesothelial cells may contribute to immune evasion in pa

199 n the developing liver, lung, and intestine, mesothelial cells (MCs) differentiate into specific mese

200 Mesothelial cells (MCs) form a single epithelial layer a

201 Mesothelial cells (MCs) line the peritoneal cavity and h

202 onstrated that MesP1+ mesoderm gives rise to mesothelial cells (MCs), which differentiate into HSCs a

203 itol-linked glycoprotein highly expressed in mesothelial cells, mesotheliomas, and ovarian cancer, bu

204 0-kDa glycoprotein present on the surface of mesothelial cells, mesotheliomas, and ovarian cancers.

205 13), and primary peritoneal and immortalized mesothelial cells (MeT5A) by immunohistochemistry, qRT-P

206 rom this organism, adhered to primary murine mesothelial cells (MMCs) in vitro.

207 e adherence of human PMN to human peritoneal mesothelial cell monolayers and examines the importance

208 cancer cell death during adhesion to normal mesothelial cell monolayers.

209 loss-of-function mice, we demonstrated that mesothelial cell movement into the lung requires the dir

210 le for this vascular defect was the yolk sac mesothelial cells, not the cardiomyocytes or the VSMC.

211 Epithelium, stroma and proximal mesothelial cells of endometriomas showed dysregulation

212 mesothelioma (MPeM) is a rare cancer of the mesothelial cells of the peritoneum.

213 es apoptosis in MMC without affecting normal mesothelial cells of the pleura.

214 Novel findings include the following: mesothelial cells of the serosa transduce Hedgehog signa

215 proliferation in this tissue, rabbit ovarian mesothelial cells (OMC) were transfected in vitro with a

216 l ester "mixed isomers" (CCFSE) dye to label mesothelial cells on the surface of the embryonic lung.

217 single-chain urokinase-bound rabbit pleural mesothelial cells or lung fibroblasts with kinetics simi

218 Although primary cultures of mesothelial cells or submesothelial fibroblasts each exp

219 podoplanin was also expressed by peritoneal mesothelial cells, osteocytes, glandular myoepithelial c

220 ggregation and increases in EGF-R protein in mesothelial cells phagocytizing long asbestos fibers wer

221 wth factor-beta1 (TGF-beta1) induces pleural mesothelial cell (PMC) transformation into myofibroblast

222 n of interleukin (IL)-8 in activated pleural mesothelial cells (PMC) and the migration of PMNL across

223 investigate whether talc stimulates pleural mesothelial cells (PMC) to release C-X-C and/or C-C chem

224 t mesothelioma cells (MMC) or normal pleural mesothelial cells (PMC).

225 Results indicate pleural mesothelial cells (PMCs) express ICAM-1 in tuberculous p

226 ies using NIH 3T3 fibroblasts and peritoneal mesothelial cells (PMCs) showed that CTGF blockade suppr

227 rogression of pleural injury, normal pleural mesothelial cells (PMCs) undergo a transition, termed me

228 this study we demonstrate that mouse pleural mesothelial cells (PMCs), when stimulated with BCG or IF

229 Pleural mesothelial cells (PMCs), which are derived from the mes

230 ary vessels arise from a unique extracardiac mesothelial cell population, the proepicardium, which de

231 ic grafting and subsequent identification of mesothelial cell populations, we demonstrate that a diff

232 anation for the ability of SV40 to transform mesothelial cells preferentially and indicate that asbes

233 ese findings indicate that cancer-associated mesothelial cells promote colonization during the initia

234 However, the mechanisms by which mesothelial cells promote metastasis are unclear.

235 e, we investigated whether cancer-associated mesothelial cells promote ovarian cancer chemoresistance

236 cell injury identified aB-crystallin as the mesothelial cell protein most consistently counter-regul

237 gates the role of dynamic O-GlcNAcylation of mesothelial cell proteins in cell survival during exposu

238 Pleural mesothelial cells (rabbit or human) were exposed to asbe

239 PR expression predominantly to pericytes and mesothelial cells rather than to adipocytes.

240 Fas expression rendering transformed ovarian mesothelial cells resistant to apoptosis.

241 ophils (RNase 3), macrophages (RNase 6), and mesothelial cells (RNase 7).

242 Upon exposure to bacterial products, mesothelial cells secrete chemokines, but the signaling

243 pic 3D cultures, we found that primary human mesothelial cells secrete fibronectin in the presence of

244 Together, our data provide evidence that mesothelial cells serve as a source of vascular smooth m

245 P-1) activity in both primary Syrian hamster mesothelial cells (SHM) and primary human mesothelial ce

246 line (LP9/TERT-1) and isolated human pleural mesothelial cells showed rapid and protracted asbestos-i

247 A subset of mesothelial cells situated atop the immune aggregates wa

248 In contrast, areas of reactive mesothelial cells stained positively for these enzymes.

249 Conditioned medium from peritoneal mesothelial cells stimulate migration, adhesion, and inv

250 nd, VCAM-1, inhibits EMT of chick epicardial mesothelial cells stimulated by TGFbeta isoforms.

251 In vitro, pleural mesothelial cells stimulated with bacille Calmette-Gueri

252 ve loads stimulated remodeling of peritoneal mesothelial cell surface ultrastructure via induction of

253 LiCl improved mesothelial cell survival in a dose-dependent manner.

254 In cultured mesothelial cells, TGF-beta1 upregulated the expression

255 ovarian tumor cells as well as in peritoneal mesothelial cells that are in direct contact with dissem

256 ember proto-oncogenes in lung epithelial and mesothelial cells that are linked to proliferation and c

257 ereby favoring survival and proliferation of mesothelial cells that have sustained DNA damage.

258 inflammasome, a component of macrophages or mesothelial cells that leads to production of chemotacti

259 Mesothelial cells that line the serous cavities and oute

260 RAC1/SMAD-dependent signaling pathway in the mesothelial cells that promotes a mesenchymal phenotype

261 We observed that, in primary mesothelial cells, the plasmalemmal Ca(2+) influx played

262 Mesothelial cells, the progenitor cell of the asbestos-i

263 /apyrimidinic (AP)-endonuclease, in isolated mesothelial cells, the progenitor cells of malignant mes

264 s are up-regulated in asbestos-exposed human mesothelial cells through an epidermal growth factor rec

265 uced osteopontin expression and secretion by mesothelial cells through TGF-B signaling.

266 in peritoneal macrophages (TLR2/4, C5aR) and mesothelial cells (TLR2, C5aR).

267 ll marker mesothelin can effectively inhibit mesothelial cell to apCAF transition and Treg formation

268 hese data demonstrate an intrinsic origin of mesothelial cells to a coelomic organ and provide a nove

269 Exposure of mesothelial cells to asbestos complemented SV40 mutants

270 of rat lung epithelial cells and rat pleural mesothelial cells to asbestos increased binding of nucle

271 Recent studies have proposed mesothelial cells to be an important source of myofibrob

272 hesize that SV40 oncoproteins will sensitize mesothelial cells to DNA damage induced by asbestos or c

273 ty of the conditioned medium from peritoneal mesothelial cells to ovarian cancer cells.

274 ccompanied by transport of mitochondria from mesothelial cells to OvCa cells.

275 the unusual susceptibility of primary human mesothelial cells to SV40 carcinogenesis.

276 s of erionite, and examined the hallmarks of mesothelial cell transformation in vitro and in vivo.

277 Here, we demonstrate that asbestos-induced mesothelial cell transformation is linked to increases i

278 There was evidence of mesothelial cell transition to a mesenchymal phenotype w

279 We characterize two distinct mesothelial cell types as well as early hepatic stellate

280 monstrate that EGCs interact with immune and mesothelial cells under homeostasis and helminth infecti

281 monstrate that EGCs interact with immune and mesothelial cells under homeostasis and helminth infecti

282 hese observations support a scenario whereby mesothelial cells undergo a series of chronic injury, in

283 The mesothelial cell VEGF production was significantly reduc

284 eport that stimulation of primary peritoneal mesothelial cells via nucleotide-binding oligomerization

285 conclude that asbestos induces apoptosis in mesothelial cells via reactive oxygen species.

286 Yet, Nod1 stimulation of mesothelial cells via RICK enhanced chemokine secretion

287 uced inflammasome/inflammation activation in mesothelial cells was CREB dependent, further supporting

288 2, TLR4, TRIF, or inflammasome components in mesothelial cells was critical for the production of CXC

289 ng anti-ALCAM antibodies, submesothelial and mesothelial cells were isolated by FACS.

290 lls (visSMCs) of the foetal mid-gut, but not mesothelial cells, were labelled after tamoxifen adminis

291 cer cell apoptosis during adhesion to normal mesothelial cells which line the peritoneum.

292 because its normal expression is limited to mesothelial cells, which are dispensable.

293 he direct interaction of the OvCa cells with mesothelial cells, which cover the surface of the omentu

294 nment of chronic IL-1beta signaling in human mesothelial cells, which may promote mesothelial to fibr

295 he surface of FALCs were covered by CXCL1(+) mesothelial cells, which we termed FALC cover cells.

296 entified in the allantois: an outer layer of mesothelial cells, whose distal portion will become tran

297 sbestos induces a fibroblastic transition of mesothelial cells with a gain of mesenchymal markers (vi

298 pathway could allow the abnormal survival of mesothelial cells with asbestos-induced mutations.

299 Finally, infection of mesothelial cells with Listeria monocytogenes induced pr

300 Stimulation of human peritoneal mesothelial cells with OSM induced phosphorylation of gp