ライフサイエンスコーパス: metabolic alkalosis

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1 by renal salt loss, marked hypokalemia, and metabolic alkalosis.

2 , respectively, autosomal recessive forms of metabolic alkalosis.

3 is are closely similar to those operative in metabolic alkalosis.

4 ses numerous mechanisms that help to prevent metabolic alkalosis.

5 h respiratory alkalosis (5%--> 3% CO(2)) and metabolic alkalosis (22 mm--> 35 mm HCO(3)(-)), DeltapH(

6 (almitrine; -5.9 mm Hg) and decreasing with metabolic alkalosis (-3.7 mm Hg).

7 kidney caused hypotension, hypokalemia, and metabolic alkalosis, an exact mirror image of PHA-II.

8 was diagnosed with CF after presenting with metabolic alkalosis and hypokalemia.

9 ion, and electrolyte imbalance (hypokalemia, metabolic alkalosis, and hypomagnesemia).

10 ines but had more frequent hypophosphatemia, metabolic alkalosis, and thrombocytopenia.

11 consisting of a hypokalemic, hypomagnesemic, metabolic alkalosis associated with seizures, sensorineu

12 0.5 mEq/L; P < .001) and number of days with metabolic alkalosis (between-group difference, -1; 95% C

13 nic obstructive pulmonary disease (COPD) and metabolic alkalosis, but no large randomized placebo-con

14 In addition, PCO2 higher than 30 mm Hg or metabolic alkalosis did not have an effect on this proce

15 Maintenance of metabolic alkalosis for any length of time means that re

16 Maintenance of metabolic alkalosis generated by chloride depletion is o

17 ectrolyte homeostasis including hypokalaemic metabolic alkalosis; Gitelman's syndrome represents the

18 Previous studies in metabolic alkalosis have demonstrated that two factors a

19 These data suggest that hypocapnic but not metabolic alkalosis impairs alveolar fluid reabsorption.

20 ered intravenously in cases of pure or mixed metabolic alkalosis, initiated within 48 hours of ICU ad

21 Although metabolic alkalosis is frequently not dangerous, in cert

22 Metabolic alkalosis is maintained by the excessive sweat

23 requently not dangerous, in certain settings metabolic alkalosis may contribute to mortality and shou

24 The metabolic alkalosis of congenital chloride-losing diarrh

25 ined the effects of alkalosis (hypocapnic or metabolic alkalosis) on alveolar fluid reabsorption in t

26 microdose (15 microg/kg) pretreatment or by metabolic alkalosis (pH 7.60).

27 process is greatly upregulated in models of metabolic alkalosis, such as following aldosterone admin

28 specifically volume overload, hyponatremia, metabolic alkalosis, uremia, and hyperglycemia, than tho

29 ders manifested by hypokalemic hypochloremic metabolic alkalosis with normotensive hyperreninemic hyp

30 rofound volume depletion, renal failure, and metabolic alkalosis without hypokalemia, which were all

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