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1 ate acting through a phospholipase D-coupled metabotropic glutamate receptor.
2 ough the system Xc- antiporter to activate a metabotropic glutamate receptor.
3 ent inhibition via the activation of group I metabotropic glutamate receptors.
4 nsmission, through the activation of type 1a metabotropic glutamate receptors.
5 vagal afferent fibre-dependent activation of metabotropic glutamate receptors.
6 ribute to the production of 2-AG via group I metabotropic glutamate receptors.
7 with previous reports in the literature for metabotropic glutamate receptors.
8 ng variable effects on different subtypes of metabotropic glutamate receptors.
9 ssed postsynaptically and depends on group I metabotropic glutamate receptors.
10 Ca(2+) -permeable AMPA receptors and group I metabotropic glutamate receptors.
11 f glutamate release by presynaptic Group III metabotropic glutamate receptors.
12 esponses were depressed by the activation of metabotropic glutamate receptors.
13 strocytic glutamate release, and presynaptic metabotropic glutamate receptors.
14 fficiency reduces melanoma initiation in the metabotropic glutamate receptor 1 (Grm1(Tg)) transgenic
15 elanoma due to ectopic overexpression of the metabotropic glutamate receptor 1 (Grm1) in melanocytes.
16 he etiological role of ectopically expressed metabotropic glutamate receptor 1 (Grm1/mGluR1) in mouse
17 muscarinic acetylcholine receptor (mAChR) or metabotropic glutamate receptor 1 (mGluR1) agonists on t
19 an excellent PET radioligand for quantifying metabotropic glutamate receptor 1 (mGluR1) in monkey bra
22 well as the emergence of glutamate-activated metabotropic glutamate receptor 1 (mGluR1) signaling, ar
24 utamate to the ionotropic AMPA receptors and metabotropic glutamate receptor 1 and members of group 2
25 iated protein 29, glutamate decarboxylase 1, metabotropic glutamate receptor 1, and excitatory amino
28 finity, selective antibody antagonist of the metabotropic glutamate receptor-1 (BBB-mGluR1), a widely
29 roteins: ankyrin-R, cell adhesion molecules, metabotropic glutamate receptor-1 (mGluR1), voltage-gate
30 n receptor alpha (ERalpha) signaling through metabotropic glutamate receptor 1a (mGluR1a) leads to ER
31 nctional evidence that ERbeta interacts with metabotropic glutamate receptor 1a (mGluR1a) signaling t
33 ogenous beta-III spectrin interacts with the metabotropic glutamate receptor 1alpha (mGluR1alpha) and
35 L-ITCcs are GABAergic, and strongly express metabotropic glutamate receptor 1alpha and GABAA recepto
38 hat genetic variation of Grm2, which encodes metabotropic glutamate receptor 2 (mGluR2), alters alcoh
40 We also determined the effect of the novel metabotropic glutamate receptor 2 positive allosteric mo
41 ggest that positive allosteric modulators of metabotropic glutamate receptor 2 should be considered f
42 ynaptic long-term depression mediated by the metabotropic glutamate receptor 2/3 (mGluR2/3-LTD) remai
44 n in the NAc via bilateral injections of the metabotropic glutamate receptor-2/3 agonist LY379268 red
45 o LAC responsiveness (leptin receptors Lepr, metabotropic glutamate receptors-2 mGlu2, neuropeptide-Y
47 ies have revealed that genetic variations in metabotropic glutamate receptor 3 (mGlu3) affect perform
51 Negative allosteric modulators (NAMs) of metabotropic glutamate receptor 5 (mGlu5) have potential
54 regulator of synaptic protein synthesis, the metabotropic glutamate receptor 5 (mGlu5) protein, is si
55 Positive allosteric modulators (PAMs) of metabotropic glutamate receptor 5 (mGlu5) represent a pr
56 Positive allosteric modulators (PAMs) of metabotropic glutamate receptor 5 (mGlu5) represent a pr
57 entified a novel role of GluD1 in regulating metabotropic glutamate receptor 5 (mGlu5) signaling in t
58 e allosteric modulators (NAMs) targeting the metabotropic glutamate receptor 5 (mGlu5) subtype are cu
60 lnesses, including disorders associated with metabotropic glutamate receptor 5 (mGluR5) and dopaminer
61 Here we investigated how Gq-protein-coupled metabotropic glutamate receptor 5 (mGluR5) and oxytocin
62 ly (within 30 min) and require activation of metabotropic glutamate receptor 5 (mGluR5) and protein s
64 sts with or without prior treatment with the metabotropic glutamate receptor 5 (mGluR5) antagonist 2-
71 Recent studies indicate a critical role for metabotropic glutamate receptor 5 (mGluR5) in the reinst
74 While abnormal signaling mediated through metabotropic glutamate receptor 5 (mGluR5) is involved i
75 ministration, the glutamate-receptor protein metabotropic glutamate receptor 5 (mGluR5) is phosphoryl
77 over of synaptic glutamate, which stimulates metabotropic glutamate receptor 5 (mGluR5) on a small po
78 cally, neurons release glutamate to activate metabotropic glutamate receptor 5 (mGluR5) on astrocytes
79 clinical data suggest that inhibition of the metabotropic glutamate receptor 5 (mGluR5) receptor migh
82 ligation induces a re-emergence of immature metabotropic glutamate receptor 5 (mGluR5) signaling in
86 The most recently identified include the metabotropic glutamate receptor 5 (mGluR5), dipeptidyl-p
87 mouse model of Rett syndrome (Mecp2 KO) that metabotropic glutamate receptor 5 (mGluR5)- and protein-
88 ive literature shows that astrocytes exhibit metabotropic glutamate receptor 5 (mGluR5)-dependent inc
89 esult from glutamate spillover, initiating a metabotropic glutamate receptor 5 (mGluR5)-dependent inc
90 h the same genetic deficiency, we found that metabotropic glutamate receptor 5 (mGluR5)-dependent syn
98 mutant hippocampus, heightened expression of metabotropic glutamate receptor 5 and constitutively act
101 r prion protein associates via transmembrane metabotropic glutamate receptor 5 with the intracellular
102 induced in GABA cells that was dependent on metabotropic glutamate receptor 5, and cannabinoid recep
103 GluR1, GABABR1, and GABABR2 levels, whereas metabotropic glutamate receptor 5, NMDA receptor 2B, Glu
104 on domain containing protein 12, mitofusin2, metabotropic glutamate receptor 5, p21-activated kinase
105 gamma-aminobutyric acidergic dysfunction and metabotropic glutamate receptor 5-associated long-term d
108 ed from photoreceptors in the dark activates metabotropic glutamate receptor 6 (mGluR6) receptors in
109 bipolar cell glutamatergic transmission, the metabotropic glutamate receptor 6 and voltage-dependent
110 as normal in the Rs1-KO retina; however, the metabotropic glutamate receptor 6/transient receptor pot
114 y, reducing glutamate release by the group 2 metabotropic glutamate receptor agonist LY379268 amelior
116 ed signaling pathways, activated via Group I metabotropic glutamate receptors and cholecystokinin 2 r
118 ranscriptionally dysregulated ionotropic and metabotropic glutamate receptors and glutamate transport
119 gh a signaling cascade that involves group I metabotropic glutamate receptors and intracellular Ca(2+
120 n the structural dynamics and drug action at metabotropic glutamate receptors and validate an approac
121 c to connections with VLEs, requires group I metabotropic glutamate receptors, and has a presynaptic
122 ntagonists targeting multiple ionotropic and metabotropic glutamate receptors, and intracellular casc
123 d by systemic administration of the group II metabotropic glutamate receptor antagonist LY341495.
124 om a therapeutic standpoint because numerous metabotropic glutamate receptor antagonists are availabl
126 g by stimulating release-regulating group II metabotropic glutamate receptor autoreceptors to inhibit
128 hancement, which is caused by an increase in metabotropic glutamate receptor-dependent Ca(2+) signali
129 absence of FMRP in neurons abolishes group 1 metabotropic glutamate receptor-dependent DGK activity c
130 oreover, hippocampal NMDAR-dependent but not metabotropic glutamate receptor-dependent plasticity is
134 (P </= 2.40E-09, 1.8-fold enrichment) in the metabotropic glutamate receptor (GRM) GFIN, previously o
140 umption in a manner requiring intact group 1 metabotropic glutamate receptors, Homer2, phospholipase
141 tingly, eCB-LTD in PE animals was rescued by metabotropic glutamate receptor I activation, suggesting
142 ublished for the transmembrane domain of two metabotropic glutamate receptors in complex with negativ
143 pileptiform bursting induced by agonists for metabotropic glutamate receptors in the hippocampal CA1
144 Serotonin 7 receptor activation reverses metabotropic glutamate receptor-induced AMPA receptor in
145 DISC1 disruption resulted in an increase of metabotropic glutamate receptor-induced intracellular ca
146 and behavioral measures to demonstrate that metabotropic glutamate receptor-induced sensitization of
147 e absence of FMRP, signaling through group 1 metabotropic glutamate receptors is elevated and insensi
148 Activation of the FMRP pathway by group I metabotropic glutamate receptors is involved in regulati
149 atergic transmission, through ionotropic and metabotropic glutamate receptors, is necessary for the c
150 tudy demonstrates that expression of GRM3, a metabotropic glutamate receptor mainly expressed in mamm
152 ivity through the loss of cAMP regulation of metabotropic glutamate receptor-mediated intracellular C
155 Purkinje cells (PCs), where it mediates slow metabotropic glutamate receptor-mediated synaptic respon
156 e AMPA-type glutamate receptor GLR-1 and the metabotropic glutamate receptor MGL-1 in one of the prim
159 ssive mRNA translation downstream of group I metabotropic glutamate receptors (mGlu1/5) is a core pat
161 ippocampal subfields, we speculated that the metabotropic glutamate receptor mGlu5 may regulate infor
162 sponses, some receptors, such as the group 1 metabotropic glutamate receptor, mGlu5, are also localiz
167 genic phenotype that is triggered by group I metabotropic glutamate receptor (mGluR) activation.
168 d plasma insulin following microinjection of metabotropic glutamate receptor (mGluR) agonists and exe
170 ing inhibition was abolished by group II/III metabotropic glutamate receptor (mGluR) antagonists in w
171 e impairments were rescued by treatment with metabotropic glutamate receptor (mGluR) antagonists or l
172 combination of P2 purinergic and group I/II metabotropic glutamate receptor (mGluR) antagonists redu
174 es in cortical activities induced by group I metabotropic glutamate receptor (mGluR) stimulation incl
175 whether activation of one particular GPCR, a metabotropic glutamate receptor (mGluR), can reduce cone
176 )-knockout (KO) mice, and treatment with the metabotropic glutamate receptor (mGluR)-5 antagonist MTE
178 how increases in basal protein synthesis and metabotropic glutamate receptor (mGluR)-dependent long-t
179 eta burst-induced long-term potentiation and metabotropic glutamate receptor (mGluR)-dependent long-t
180 ddition to the spine changes at basal state, metabotropic glutamate receptor (mGluR)-induced dendriti
181 eceptors (CP-AMPARs) and a switch in group I metabotropic glutamate receptor (mGluR)-mediated suppres
182 Normally, FMRP binds to RNA and regulates metabotropic glutamate receptor (mGluR)-mediated synapti
188 on-induced sensitization models, we assessed metabotropic glutamate receptors (mGluR) signaling and r
189 owever, two forms of LTD, involving NMDA and metabotropic glutamate receptors (mGluR), are both great
190 am and downstream signaling specificities of metabotropic glutamate receptors (mGluR), we have examin
192 SCA28 mice, partial genetic silencing of the metabotropic glutamate receptor mGluR1 decreased Ca(2)(+
193 show that agonist activation of the group I metabotropic glutamate receptor mGluR1 increases the str
195 MPARs from synapses is contingent on group 1 metabotropic glutamate receptor (mGluR1) and PKC signali
200 alizes and modulates the activity of group I metabotropic glutamate receptors (mGluR1 and mGluR5).
202 Glutamate directs GAD67 expression via the metabotropic glutamate receptor mGluR1beta on GABApre te
206 markedly inhibited by 3-10 mum MPEP (blocks metabotropic glutamate receptor mGluR5) and 10 mum LY341
208 Here, we examine the significance of type 5 metabotropic glutamate receptors (mGluR5s) for behaviora
210 s, synaptic transmission is initiated by the metabotropic glutamate receptor, mGluR6, that signals vi
212 ith movement execution, EAAC1 limits group I metabotropic glutamate receptor (mGluRI) activation, fac
213 indings show that EAAC1 limits activation of metabotropic glutamate receptors (mGluRIs) in the striat
216 quires co-activation of postsynaptic group I metabotropic glutamate receptors (mGluRs) and Ca(2+) -pe
217 ines additionally requires signaling through metabotropic glutamate receptors (mGluRs) and inositol 1
218 regulation of SPN activity via activation of metabotropic glutamate receptors (mGluRs) and muscarinic
219 ings in rat hippocampal slices, that group I metabotropic glutamate receptors (mGluRs) and muscarinic
220 e, is a potent agonist against the group III metabotropic glutamate receptors (mGluRs) and, thus, is
229 evoked responses, we show that activation of metabotropic glutamate receptors (mGluRs) by general and
232 cordings, we show that activation of Group I metabotropic glutamate receptors (mGluRs) induces long-t
233 hippocampal synapses, activation of group I metabotropic glutamate receptors (mGluRs) induces long-t
236 gonists of ionotropic glutamate receptors or metabotropic glutamate receptors (mGluRs) or orthosteric
239 st synapse in the visual system, presynaptic metabotropic glutamate receptors (mGluRs) regulate cone
245 diverse ligand response specificities among metabotropic glutamate receptors (mGluRs), we combined c
246 single-molecule subunit counting on class C metabotropic glutamate receptors (mGluRs), we map dimeri
247 ed in adult mice with antagonists of group I metabotropic glutamate receptors (mGluRs), which have be
248 on boosts activation of perisynaptic group I metabotropic glutamate receptors (mGluRs), which in turn
249 processed by different receptors, including metabotropic glutamate receptors (mGluRs), which in turn
257 ologically inhibited and genetically ablated metabotropic glutamate receptors (mGluRs, especially mGl
258 c and allosteric antagonists of the group II metabotropic glutamate receptors (mGlus) have been used
259 glutamate induces modulatory actions via the metabotropic glutamate receptors (mGlus), which are clas
263 the tonic activation of presynaptic group II metabotropic glutamate receptors on inhibitory nerve ter
264 glutamate receptor 1 and members of group 2 metabotropic glutamate receptors on the plasma membrane.
265 Mouse cortical slice astrocyte activation by metabotropic glutamate receptors or photolysis of caged
266 t comprehensive structural comparison of all metabotropic glutamate receptors, placing selective nega
267 when coupled with concomitant activation of metabotropic glutamate receptors postsynaptic to cortica
269 h-power state because blocking ionotropic or metabotropic glutamate receptors results in high-power L
270 ment, we detect excess "gain of function" of metabotropic glutamate receptor signaling at an importan
271 n human patients support the hypothesis that metabotropic glutamate receptor signaling is a valuable
272 hypothesized that the activation of group I metabotropic glutamate receptor signaling though the fra
273 lso highlight emerging evidence that altered metabotropic glutamate receptor signalling and disrupted
274 Altered function of the Gq-coupled, Group 1 metabotropic glutamate receptors, specifically mGlu5, is
275 hese neurons exhibit a pronounced deficit in metabotropic glutamate receptor subtype 2 (mGluR(2)).
276 glutamate, binds to the presynaptic group II metabotropic glutamate receptor subtype 3 (mGluR3) and s
282 rameshift substitution of GRM5, encoding the metabotropic glutamate receptor subtype 5 (mGluR5), whic
283 is a potent and specific radioligand for the metabotropic glutamate receptor subtype 5 (mGluR5).
286 dvance the novel concept that a breakdown of metabotropic glutamate receptor subtype mGluR5 and endoc
287 cohol may be related, in part, to changes in metabotropic glutamate receptors-subtype 5 (mGluR5) in t
288 s in the discovery of allosteric ligands for metabotropic glutamate receptor subtypes 1-5 and 7 (mGlu
289 ts on the pancreas involves Group II and III metabotropic glutamate receptors that are located presyn
291 f extinction is impaired by locally blocking metabotropic glutamate receptor type 5 (mGluR5) activati
292 recently showed marked global reductions in metabotropic glutamate receptor type 5 (mGluR5) binding
293 ng cocaine exposure, including a decrease in metabotropic glutamate receptor type 5 (mGluR5) expressi
294 It was widely thought that activation of metabotropic glutamate receptor type 5 (mGluR5) on inter
296 ation of safe and valid PET radioligands for metabotropic glutamate receptor, type 5 (mGluR5), is ess
297 trasynaptic signaling through ionotropic and metabotropic glutamate receptors, ultimately resulting i
298 ons was abolished with antagonists of type I metabotropic glutamate receptor, validating the glutamat
300 logical changes, we find the localization of metabotropic glutamate receptors within cone bipolar, bu
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