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1 ns and this phenotype is mediated by mGluR5 (metabotropic glutamate receptor 5).
2 blockade of either cellular prion protein or metabotropic glutamate receptor 5.
3 of non-N-methyl-D-aspartate receptors or the metabotropic glutamate receptor 5.
6 mutant hippocampus, heightened expression of metabotropic glutamate receptor 5 and constitutively act
7 functional hyperemia depended on astrocytic metabotropic glutamate receptor 5 and cyclooxygenase act
8 process that requires stimulation of mGluR5 (metabotropic glutamate receptor 5) and activation of mit
9 induced in GABA cells that was dependent on metabotropic glutamate receptor 5, and cannabinoid recep
10 MPEP [2-methyl-6-(phenylethynyl)pyridine], a metabotropic glutamate receptor 5 antagonist that blocks
11 study, we tested whether the highly specific metabotropic glutamate receptor 5 antagonist, 3-[(2-meth
12 demonstrate that chronic treatment with the metabotropic glutamate receptor 5 antagonist, 3-[(2-meth
16 gamma-aminobutyric acidergic dysfunction and metabotropic glutamate receptor 5-associated long-term d
17 ory postsynaptic current frequency through a metabotropic glutamate receptor 5-dependent mechanism.
18 docannabinoid signalosome), is disrupted and metabotropic glutamate receptor-5-dependent 2-arachidono
19 etardation protein deletion in mice enhances metabotropic glutamate receptor-5-dependent long-term de
22 ts are corrected by treatments that modulate metabotropic glutamate receptor 5 in opposite directions
27 Negative allosteric modulators (NAMs) of metabotropic glutamate receptor 5 (mGlu5) have potential
31 regulator of synaptic protein synthesis, the metabotropic glutamate receptor 5 (mGlu5) protein, is si
32 Positive allosteric modulators (PAMs) of metabotropic glutamate receptor 5 (mGlu5) represent a pr
33 Positive allosteric modulators (PAMs) of metabotropic glutamate receptor 5 (mGlu5) represent a pr
34 entified a novel role of GluD1 in regulating metabotropic glutamate receptor 5 (mGlu5) signaling in t
35 e allosteric modulators (NAMs) targeting the metabotropic glutamate receptor 5 (mGlu5) subtype are cu
39 lnesses, including disorders associated with metabotropic glutamate receptor 5 (mGluR5) and dopaminer
40 Here we investigated how Gq-protein-coupled metabotropic glutamate receptor 5 (mGluR5) and oxytocin
41 ly (within 30 min) and require activation of metabotropic glutamate receptor 5 (mGluR5) and protein s
43 sts with or without prior treatment with the metabotropic glutamate receptor 5 (mGluR5) antagonist 2-
53 d the interaction between NMDA receptors and metabotropic glutamate receptor 5 (mGluR5) in the integr
54 Recent studies indicate a critical role for metabotropic glutamate receptor 5 (mGluR5) in the reinst
55 revealed that activation of the G(q)-coupled metabotropic glutamate receptor 5 (mGluR5) induces phosp
60 While abnormal signaling mediated through metabotropic glutamate receptor 5 (mGluR5) is involved i
61 ministration, the glutamate-receptor protein metabotropic glutamate receptor 5 (mGluR5) is phosphoryl
66 over of synaptic glutamate, which stimulates metabotropic glutamate receptor 5 (mGluR5) on a small po
67 cally, neurons release glutamate to activate metabotropic glutamate receptor 5 (mGluR5) on astrocytes
69 clinical data suggest that inhibition of the metabotropic glutamate receptor 5 (mGluR5) receptor migh
72 ligation induces a re-emergence of immature metabotropic glutamate receptor 5 (mGluR5) signaling in
75 are potent selective negative modulators of metabotropic glutamate receptor 5 (mGluR5) were identifi
78 The most recently identified include the metabotropic glutamate receptor 5 (mGluR5), dipeptidyl-p
80 mouse model of Rett syndrome (Mecp2 KO) that metabotropic glutamate receptor 5 (mGluR5)- and protein-
82 ive literature shows that astrocytes exhibit metabotropic glutamate receptor 5 (mGluR5)-dependent inc
83 esult from glutamate spillover, initiating a metabotropic glutamate receptor 5 (mGluR5)-dependent inc
84 h the same genetic deficiency, we found that metabotropic glutamate receptor 5 (mGluR5)-dependent syn
93 In the CA1 region of the rat hippocampus, metabotropic glutamate receptor-5 (mGluR5) and cannabino
95 GluR1, GABABR1, and GABABR2 levels, whereas metabotropic glutamate receptor 5, NMDA receptor 2B, Glu
96 on domain containing protein 12, mitofusin2, metabotropic glutamate receptor 5, p21-activated kinase
97 ocytes contain metabotropic receptors of the metabotropic glutamate receptor 5 subtype and the AMPA r
98 tants, the macromolecular complex that links metabotropic glutamate receptor-5 to the 2-arachidonoyl-
99 r prion protein associates via transmembrane metabotropic glutamate receptor 5 with the intracellular
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