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1 rols, the mast cells that lack FAK were less metachromatic and by electron microscopy had granules th
4 acteristic staining of the same tissues with metachromatic dyes such as crystal violet or with the co
5 synovium were embedded in cartilage-specific metachromatic extracellular matrix and costained positiv
6 muscle that normally contain heparin lacked metachromatic granules and failed to store appreciable a
7 cluding proliferative responsiveness to SCF, metachromatic granules, mRNA expression for mast cell pr
8 ngliosidosis (Tay-Sachs and Sandhoff forms), metachromatic leucodystrophy, mucolipidosis type IV, Nie
10 uction of the myelin sheath and resulting in metachromatic leukodystrophy (MLD), a neurodegenerative
11 tase A (ASA) activity found in patients with metachromatic leukodystrophy (MLD), a progressive neurod
12 ilable during the last 20 years for juvenile metachromatic leukodystrophy (MLD), reported with variab
13 rovement of cerebral white matter changes in metachromatic leukodystrophy after treatment with hemato
14 ApoE-derived peptide appears useful to treat metachromatic leukodystrophy and possibly other neurolog
16 developed for X-linked adrenoleukodystrophy, metachromatic leukodystrophy and Zellweger syndrome and
17 h correction is possible in a mouse model of metachromatic leukodystrophy by the transplantation of h
18 T with cord blood in 1 patient with juvenile metachromatic leukodystrophy diagnosed before frank dege
19 iminated between healthy subject samples and metachromatic leukodystrophy patient samples, and, there
20 he first to document neurological outcome of metachromatic leukodystrophy treated by umbilical cord b
22 stem cell transplant, if performed early in metachromatic leukodystrophy, can not only stabilize but
26 d in KcsA tetramers, but not in monomers, by metachromatic reaction to o-toluidine blue stain on SDS-
28 nterfere dramatically with these assays; the metachromatic response was also clearly influenced by di
29 nflammatory cells (lymphocytes, eosinophils, metachromatic staining cells, and neutrophils) in bronch
30 ted by higher proteoglycan content, stronger metachromatic staining, and altered ultrastructure in co
31 hat the absence of Ctr2 results in increased metachromatic staining, the latter indicating an impact
32 ositive cells (lymphocytes, eosinophils, and metachromatic-staining cells) in the group treated with
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