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1 spiration of a right axillary node confirmed metastatic carcinoma.
2 of the three sentinel lymph nodes contained metastatic carcinoma.
3 asive adenocarcinoma to castration-resistant metastatic carcinoma.
4 paradigm for clinical intervention of liver metastatic carcinoma.
5 Two of the 11 axillary lymph nodes contained metastatic carcinoma.
6 differentiation from cholangiocarcinoma and metastatic carcinoma.
7 carcinoma, and in metastatic cell lines and metastatic carcinoma.
8 ary for active cell migration in invasive or metastatic carcinoma.
9 ken for an intraparotid lymph node harboring metastatic carcinoma.
10 plasms that progress to locally invasive and metastatic carcinomas.
11 r-reaching implications for the treatment of metastatic carcinomas.
12 or macrophage-activating factors secreted by metastatic carcinomas.
13 adhesion kinase (FAK) are expressed in colon metastatic carcinomas.
14 of non-invasive tumor cells into malignant, metastatic carcinomas.
15 protease associated with rapidly growing and metastatic carcinomas.
16 ents a testable therapeutic approach against metastatic carcinomas.
17 s but delayed their development to invasive, metastatic carcinomas.
18 mas, 18 of 28 primary carcinomas, and 5 of 5 metastatic carcinomas.
19 A1, a human gene with elevated expression in metastatic carcinomas.
20 n reaction in 30 of 33 EBVaGC cases, 8 of 10 metastatic carcinomas, 14 non-neoplastic tissues from 27
21 ary tumor growth and lung dissemination of a metastatic carcinoma (4T1), but not of a nonmetastatic c
22 nulosa cell (GC) tumors that can progress to metastatic carcinoma and thus provide a model system for
23 on of BKV large T antigen in the primary and metastatic carcinoma, but not in the nonneoplastic uroth
24 concurrently with the transition to invasive metastatic carcinoma, but they were expressed in differe
25 ntous G proteins, which are overexpressed in metastatic carcinomas, but their functions in epithelial
27 r actin polymerization in the invadopodia of metastatic carcinoma cells and define four distinct stag
29 In addition, silencing alphaB-crystallin in metastatic carcinoma cells reduced the number of viable
32 he splenic lesion was performed and revealed metastatic carcinoma, consistent with the lung primary.
33 epithelial neoplasia (HGPIN) to invasive and metastatic carcinoma could facilitate study of the regul
35 hyperplasias, papillomas, and dysplasias to metastatic carcinomas developed in squamous epithelia of
36 patients in the radical group, only 10% had metastatic carcinoma in the resected retroperitoneal lym
37 ation, lymphoproliferative inflammation, and metastatic carcinomas in the lung after a period of late
38 e been linked to various diseases, including metastatic carcinomas in which the 9 carbon sialic acid
40 biopsy of the gastric antrum later showed a metastatic carcinoma of breast origin with typical tumor
42 18 years) diagnosed with locally advanced or metastatic carcinoma of the pancreas confirmed by cytolo
43 Identifying the primary site in cases of metastatic carcinoma of unknown origin has profound clin
45 d 18 years or older initially diagnosed with metastatic carcinoma or who subsequently developed metas
49 promotes cell migration and is a hallmark of metastatic carcinomas, we asked whether it increases FA
50 hat develop extensive HGPIN and invasive and metastatic carcinoma with neuroendocrine (NE) differenti
51 t inhibitor, in 41 patients with progressive metastatic carcinoma with or without mismatch-repair def
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