ライフサイエンスコーパス: methamphetamine

1 of dopamine neurotransmission in response to methamphetamine.

2 f locating and localizing the manufacture of methamphetamine.

3 by alcohol, opiate, cocaine, oxycodone, and methamphetamine.

4 administration of MDMA, its enantiomers, and methamphetamine.

5 male/female rhesus monkeys self-administered methamphetamine (0.03 mg/kg/injection, i.v.) under a fix

6 for drug reinforcement and self-administered methamphetamine (0.05 mg/kg) at control levels.

7 male Sprague-Dawley rats to self-administer methamphetamine (0.1 mg/kg/infusion) for 9 hours daily f

8 pellets for 6 days (6 h per day) for either methamphetamine (0.1 mg/kg/infusion) or heroin (0.1 mg/k

9 We trained rats to self-administer methamphetamine (10 days; 9 h/day, 0.1 mg/kg/infusion) a

10 ster palatable food pellets (6 d, 6 h/d) and methamphetamine (12 d, 6 h/d).

11 lanted radiotelemetry, following exposure to methamphetamine (12.5 and 100 mg/ml), MDPV (25, 50, and

12 speech task after MDMA (0.75 and 1.5 mg/kg), methamphetamine (20 mg), or placebo.

13 optimized for quantification of amphetamine, methamphetamine, 3,4-methylenedioxyamphetamine (MDA), 3,

14 drocodone) and five stimulants (amphetamine, methamphetamine, 3,4-methylenedioxymethamphetamine (MDMA

15 s highly dysregulated by the psychostimulant methamphetamine, a substrate for the dopamine transporte

16 Although a direct link between methamphetamine abuse and HIV-1 pathogenesis remains to

17 Methamphetamine abuse is associated with increased risk

18 cal profiles in animal models of cocaine and methamphetamine abuse.

19 se results are contrary to the paradigm that methamphetamine accentuates HIV-1 pathogenesis by increa

20 neural responses to drug cues in humans with methamphetamine addiction and suggests that naltrexone m

21 Methamphetamine addiction is mimicked in rats that self-

22 1R as a putative target for the treatment of methamphetamine addiction.

23 ct has been shown for alcohol, nicotine, and methamphetamine addictions, but not for heroin or cocain

24 gy may be helpful for recovering cocaine and methamphetamine addicts.

25 men who had recently used cocaine, crack, or methamphetamine (adjusted odds ratio (OR) = 2.13, 95% co

26 ad been exposed to different combinations of methamphetamine, alcohol, nicotine and marijuana prior t

27 or been offered opioids, cocaine, cannabis, methamphetamine, alcohol, or tobacco.

28 Methamphetamine also interacts with the sigma-1 receptor

29 lower DAT levels secondary to the effects of methamphetamine and alcohol.

30 avioral and neurophysiological correlates of methamphetamine and amphetamine administration are uniqu

31 showed, for drugs with weekly cycles (MDMA, methamphetamine and cocaine in this sample), sampling st

32 in midbrain D2/D3 BPnd was detected between methamphetamine and control groups, midbrain D2/D3 BPnd

33 results demonstrate that incubation of both methamphetamine and food craving occur after punishment-

34 e (using a discrete choice procedure between methamphetamine and food; 20 trials/d) for 19 d.

35 UCP1 knockout (UCP1KO) mice exhibit blunted methamphetamine and fully inhibited noradrenaline thermo

36 transmitted infections, and higher rates of methamphetamine and gamma hydroxybutyrate use when compa

37 sing ToF-SIMS and DESI-MS, as the signals of methamphetamine and heroin were completely suppressed by

38 rogressively increases after withdrawal from methamphetamine and other drugs, a phenomenon termed 'in

39 eved via a discrete choice procedure between methamphetamine and palatable food; 20 trials per day) f

40 little is known about the effect of comorbid methamphetamine and tobacco use on human fetal brain dev

41 ular application of amphetamine > dopamine > methamphetamine and was DAT-dependent.

42 ific genes, including Azi2 in sensitivity to methamphetamine and Zmynd11 in anxiety-like behavior.

43 rmalities reported in children with prenatal methamphetamine and/or tobacco exposure are present at b

44 Collectively, prenatal methamphetamine and/or tobacco exposure may lead to dela

45 Exposures: Prenatal methamphetamine and/or tobacco exposure.

46 Methamphetamine and/or tobacco use by pregnant women rem

47 UCP3 (UCP3KO) have impaired sympathomimetic (methamphetamine) and completely abrogated lipopolysaccha

48 tyrate or gamma-butyrolactone, 175 (8%) used methamphetamine, and 162 (7%) used mephedrone.

49 rated from inkjet-printed arrays of cocaine, methamphetamine, and heroin with a deposited-mass rangin

50 that repeated exposure to alcohol, nicotine, methamphetamine, and morphine upregulates alpha2delta-1

51 ul T-maze task following a binge exposure to methamphetamine, and no such changes in effort following

52 rkers in the mesocorticolimbic pathway after methamphetamine, and their relationship with animals' re

53 g behavior, is a major target of cocaine and methamphetamines, and has been assumed to be conserved a

54 relapse tests, both punished and unpunished methamphetamine- and food-trained rats showed significan

55 hold values for the detection of cocaine and methamphetamine as proof-of-principle with 0.25 and 0.75

56 reactivity, and attentional bias toward the methamphetamine-associated cue, compared with the placeb

57 Methamphetamine-associated psychosis (MAP) involves wide

58 sed methamphetamine choice, whereas removing methamphetamine availability decreased methamphetamine c

59 (gp120+ and gp120-) were exposed to either a methamphetamine binge (METH+) or saline (METH-), then te

60 The intracellular application of methamphetamine, but not amphetamine, prevented the dopa

61 urthermore, NAc core MeCP2 knockdown reduces methamphetamine, but not saccharin, SA on a progressive

62 d activity-based sleep measures disrupted by methamphetamine by decreasing sleep latency and increasi

63 e, whereas bupropion treatment did not alter methamphetamine choice up to doses that decreased rates

64 Methamphetamine choice was not significantly altered whe

65 Risperidone treatment increased methamphetamine choice, whereas bupropion treatment did

66 Removing food availability increased methamphetamine choice, whereas removing methamphetamine

67 oving methamphetamine availability decreased methamphetamine choice.

68 analysis was performed for four drugs (MDMA, methamphetamine, cocaine, methadone) collected through a

69 cur much more rapidly during withdrawal from methamphetamine compared with cocaine.

70 ently developed a rat model of incubation of methamphetamine craving after choice-based voluntary abs

71 introduced an animal model of incubation of methamphetamine craving after choice-based voluntary abs

72 osteric modulator, AZD8529, on incubation of methamphetamine craving after forced or voluntary abstin

73 ine D1 and D2 receptors in the incubation of methamphetamine craving after voluntary abstinence and t

74 Our results show that incubation of methamphetamine craving after voluntary abstinence gener

75 rate a role for the NAc in the incubation of methamphetamine craving and describe adaptations in syna

76 trate a critical role of DS in incubation of methamphetamine craving and that this incubation is asso

77 Cue-induced methamphetamine craving increases after prolonged forced

78 Here, we studied whether incubation of methamphetamine craving is observed after suppression of

79 Incubation of methamphetamine craving was associated with CP-AMPAR acc

80 relationship between midbrain D2/D3 BPnd and methamphetamine craving was not detected.

81 ut mechanisms underlying this 'incubation of methamphetamine craving' are unknown.

82 We first confirmed that 'incubation of methamphetamine craving' occurs under our experimental c

83 ly increases after withdrawal (incubation of methamphetamine craving), but the underlying mechanisms

84 sed) abstinence from the drug (incubation of methamphetamine craving).

85 tinence days than after 1 day (incubation of methamphetamine craving).

86 than during early withdrawal (incubation of methamphetamine craving).

87 d abstinence than after 1 day (incubation of methamphetamine craving).

88 ary abstinence than after 1 d (incubation of methamphetamine craving).

89 ced seeking tests demonstrated incubation of methamphetamine craving.

90 similar plasticity accompanies incubation of methamphetamine craving.

91 cocaine and heroin craving, in incubation of methamphetamine craving.

92 wal interval when animals show incubation of methamphetamine craving.

93 within-subject design and underwent a visual methamphetamine cue-reactivity task during two blood-oxy

94 tic resonance imaging (fMRI) measures during methamphetamine cue-reactivity to elucidate the role of

95 y HIV serostatus (HIV+ or HIV-) and lifetime methamphetamine dependence (METH+ or METH-).

96 data from 4739 cases of alcohol, opioid, or methamphetamine dependence and 4924 controls.

97 lt men and women who met DSM-IV criteria for methamphetamine dependence and were enrolled in a reside

98 The extent to which these features of methamphetamine dependence are interrelated, however, is

99 Methamphetamine dependence is a common comorbid conditio

100 s from patients diagnosed with MAP (N = 12), methamphetamine dependence without psychosis (MA; N = 14

101 n be used to predict relapse among abstinent methamphetamine-dependent (MD) individuals.

102 Abstinent alcohol- (n = 30) and methamphetamine-dependent (n = 23) subjects, recreationa

103 In Study 2, an independent sample of 20 methamphetamine-dependent and 18 control subjects comple

104 In Study 1, 19 methamphetamine-dependent and 26 healthy control subject

105 academic institution, this study involved 25 methamphetamine-dependent and 27 control participants.

106 nctional magnetic resonance imaging study of methamphetamine-dependent and healthy comparison partici

107 Here, we show that methamphetamine-dependent attenuation of GABABR-GIRK cur

108 The methamphetamine-dependent decrease in GABABR-GIRK curren

109 the GIRK3 subunit appeared critical for the methamphetamine-dependent decrease of GABABR-GIRK curren

110 nk that may help explain high impulsivity in methamphetamine-dependent individuals.

111 Methamphetamine-dependent participants also exhibited gr

112 ventral striatum but weaker in the rDLPFC in methamphetamine-dependent participants than control indi

113 striatum, orbitofrontal cortex and insula in methamphetamine-dependent participants, but a positive r

114 In methamphetamine-dependent research participants, impulsi

115 In this study, 21 methamphetamine-dependent subjects and 23 healthy contro

116 mpared with healthy volunteers, alcohol- and methamphetamine-dependent subjects and cannabis users sh

117 Methamphetamine-dependent subjects had greater nonlinear

118 n of subjective value similar to that of the methamphetamine-dependent subjects.

119 Methamphetamine-dependent users exhibited a positive rel

120 Prenatal exposure to methamphetamine did not influence visual function.

121 ferred over no methamphetamine or small unit methamphetamine doses (0.01-0.032 mg/kg/injection).

122 Larger methamphetamine doses resulted in greater methamphetamin

123 trong preference for the palatable food over methamphetamine during the choice-based voluntary abstin

124 Methamphetamine exposure also increased repressor elemen

125 ice impaired learning, and that a history of methamphetamine exposure increased the susceptibility to

126 Lever presses in the cue-induced methamphetamine extinction tests were higher during late

127 We trained rats to self-administer methamphetamine for 20 days.

128 re, we show that stimulants like cocaine and methamphetamine greatly increase HS content and sulfatio

129 eceptor availability (BPnd) was lower in the methamphetamine group, as shown previously, the groups d

130 l mean cortical gray-matter thickness in the methamphetamine group, but no association was found betw

131 In the methamphetamine group, mean cortical gray-matter thickne

132 ty to differences in reward magnitude in the methamphetamine group: animals were more likely to overc

133 Our results demonstrate that methamphetamine had a minimal effect on HIV-1 replicatio

134 social behaviors and vocalizations, whereas methamphetamine had only modest effects on affiliative b

135 nesis remains to be established in patients, methamphetamine has been shown to increase HIV-1 replica

136 the more methamphetamine users chose to view methamphetamine images, specifically vs pleasant images

137 nedioxymethamphetamine (MDMA; 'ecstasy') and methamphetamine in 13 ecstasy users.

138 in primary CD4(+) T cells in the presence of methamphetamine in a dose-dependent manner.

139 Through interactions with DAT, methamphetamine increases extracellular dopamine levels

140 us, both receptor subtypes may contribute to methamphetamine-induced alterations in cortical morpholo

141 at or near the plasma membrane and decreases methamphetamine-induced Ca(2+) signaling, providing pote

142 acellular Na(+) ions blocked amphetamine and methamphetamine-induced DAT-mediated inward current simi

143 The results reveal a new mechanism for methamphetamine-induced dysregulation of dopaminergic ne

144 lacking both UCP1 and 3 (UCPDK) fail to show methamphetamine-induced hyperthermia, and have a markedl

145 these findings, sigma1R activation decreases methamphetamine-induced locomotion, motivated behavior,

146 anti-MA immune response in mice, decreasing methamphetamine-induced locomotor activity.

147 Exercise attenuates methamphetamine-induced neurochemical damage in the rat

148 ine release and that dopamine is involved in methamphetamine-induced neurotoxicity, associations betw

149 We found that sigma1R activation prevents methamphetamine-induced, DAT-mediated increases in firin

150 xed-ratio (FR) 100 schedule) and intravenous methamphetamine injections (0-0.32 mg/kg/injection, FR10

151 parison, effects of removing food pellets or methamphetamine injections and FR response requirement m

152 Importance: Methamphetamine is a common illicit drug used worldwide.

153 Released from neurons when methamphetamine is self-administered, dopamine binds to

154 of psychostimulants such as amphetamine and methamphetamine is the dopamine transporter (DAT), the m

155 Methamphetamine is the second most frequently used illic

156 CR4056-associated lever responding included methamphetamine, ketamine, the endogenous imidazoline li

157 The high prevalence and severity of methamphetamine (MA) abuse demands greater neurobiologic

158 dynamics of cognitive control instability in methamphetamine (MA) abuse, as well its relationship to

159 Methamphetamine (MA) addiction is a serious public healt

160 The psychostimulants amphetamine (AMPH) and methamphetamine (MA) are widely abused illicit drugs.

161 Methamphetamine (MA) use disorder is a serious psychiatr

162 Continued methamphetamine (MA) use is dependent on a positive MA e

163 Methamphetamine (MA) users are assumed to have a high bu

164 ing the frontostriatal system, in adolescent methamphetamine (MA) users compared with adult users.

165 , male Wistar rats were exposed to vaporized methamphetamine (MA), 3,4-methylenedioxypyrovalerone (MD

166 d by chronic exposure to the psychostimulant methamphetamine (MA), the current study sought to use co

167 hetamine preference and 0.32 mg/kg/injection methamphetamine maintained near exclusive preference.

168 detect atmospheric effluent from clandestine methamphetamine manufacture is a useful tool for law enf

169 The high rate of methamphetamine (METH) abuse among young adults and wome

170 HIV-1 infection and methamphetamine (METH) abuse frequently occur simultaneo

171 Since CUMS is a model for depression and methamphetamine (METH) abuse induced psychosis recapitul

172 Methamphetamine (METH) abuse is a serious social and hea

173 Methamphetamine (METH) abuse is a worldwide threat, with

174 ctive medications to aid in the treatment of methamphetamine (METH) abuse, we used a nanotechnology a

175 to examine oxytocin as a pharmacotherapy for methamphetamine (meth) addiction.

176 Methamphetamine (meth) addicts often exhibit enduring co

177 hypothesized that Glycoprotein 120 (gp120), methamphetamine (METH) and nicotine (NT) can enhance amy

178 rhythmically pairing 2 reinforcing stimuli [methamphetamine (Meth) and running wheel (RW)] restores

179 Exposure to methamphetamine (meth) can produce lasting memory impair

180 Methamphetamine (METH) competes with dopamine uptake, in

181 Chronic methamphetamine (METH) exposure causes neuroadaptations

182 to segregate rats that reduce or stop their methamphetamine (METH) intake (nonaddicted) from those t

183 (nor-BNI) would attenuate the escalation of methamphetamine (METH) intake in an extended-access self

184 Methamphetamine (METH) is a drug with a high addictive p

185 Methamphetamine (Meth) is a psychomotor stimulant strong

186 Methamphetamine (METH) is a substrate for the dopamine t

187 Rats that have self-administered methamphetamine (meth) under long access, but not short

188 We investigated the associations between methamphetamine (meth) use, immune function, and the dyn

189 dopamine and serotonin terminals produced by methamphetamine (METH), but how ammonia contributes to t

190 ing that storage of memories associated with methamphetamine (METH), but not memories for fear or foo

191 ries associated with drugs of abuse, such as methamphetamine (METH), increase relapse vulnerability t

192 ries associated with drugs of abuse, such as methamphetamine (METH), increase relapse vulnerability t

193 as previously demonstrated in male rats that methamphetamine (Meth), when administered concurrently w

194 clades to exert differential effects and the methamphetamine (METH)-associated dopaminergic dysfuncti

195 Methamphetamine (METH)-induced neurotoxicity results in

196 ve therapy is available for the treatment of methamphetamine (METH)-induced neurotoxicity, aerobic ex

197 nds such as cocaine, amphetamine (AMPH), and methamphetamine (METH).

198 ta-phenylethylamine, amphetamine (AMPH), and methamphetamine (METH).

199 m 4% (norcocaine) to 99% (cocaine); however, methamphetamine, methadone, and EDDP showed a negative r

200 ine, cannabidiol, cocaine, codeine, heroine, methamphetamine, morphine, phentermine, L-phenylepherine

201 es to other abused drugs, including cocaine, methamphetamine, nicotine, and alcohol, and is also obse

202 omprehensive understanding of the effects of methamphetamine on HIV-1 pathogenesis.

203 Intriguingly, the effects of methamphetamine on HIV-1 replication in human CD4(+) T c

204 parate and combined effects of HIV/gp120 and methamphetamine on learning and executive functions in b

205 cal intervention to attenuate the effects of methamphetamine on nighttime activity under well-control

206 igated the long-lasting, off-drug effects of methamphetamine on reward choices in a novel effortful m

207 racellular and intracellular amphetamine and methamphetamine on the spontaneous firing of cultured mi

208 ingent punishment suppressed extended-access methamphetamine or food self-administration; surprisingl

209 ccur after punishment-induced suppression of methamphetamine or palatable food self-administration.

210 withdrawal in rats following extended-access methamphetamine or saline (control condition) self-admin

211 tment conditions, food was preferred over no methamphetamine or small unit methamphetamine doses (0.0

212 mine, the psychostimulants d-amphetamine and methamphetamine, or to cocaine and cocaine analogues.

213 gm in which they received a moderate dose of methamphetamine paired with one stimulus and placebo wit

214 The synthesis of methamphetamine precursors was performed, and the impuri

215 er methamphetamine doses resulted in greater methamphetamine preference and 0.32 mg/kg/injection meth

216 e unique mechanisms by which amphetamine and methamphetamine regulate DAT function and dopamine neuro

217 nence blocked compulsive-like context-driven methamphetamine reinstatement.

218 ls, methamphetamine users chose to view more methamphetamine-related images on one task, with a simil

219 both natural (binge eating) and artificial (methamphetamine) rewards, and obsessive-compulsive disor

220 tion, we show new evidence that both ELS and methamphetamine SA alter the expression of the epigeneti

221 genome-wide transcriptional consequences of methamphetamine SA and footshocks in the rat brain.

222 core MeCP2 may be recruited by both ELS and methamphetamine SA and promote the development of certai

223 gether, functional interactions between ELS, methamphetamine SA, and the expression of MeCP2 in the N

224 of MeCP2 expression in the NAc core reduces methamphetamine SA, as well as saccharin intake.

225 of AZD8529 (20 and 40 mg/kg) on cue-induced methamphetamine seeking 1 day or 21 days after forced or

226 SCH39166 or raclopride selectively decreased methamphetamine seeking after 21 abstinence days.

227 s induction, decreased incubated cue-induced methamphetamine seeking after prolonged withdrawal.

228 striatum in context-induced reinstatement of methamphetamine seeking and that this reinstatement is a

229 imol+baclofen, 0.03+0.3 nmol) on cue-induced methamphetamine seeking during early and late withdrawal

230 s into CeA but not BLA decreased cue-induced methamphetamine seeking during late but not early withdr

231 C, dmPFC, and OFC on 'incubated' cue-induced methamphetamine seeking during late withdrawal.

232 We then assessed cue-induced methamphetamine seeking in extinction tests after 1 or 2

233 ining and abstinence conditions, cue-induced methamphetamine seeking in the extinction tests was high

234 In both sexes, methamphetamine seeking in the relapse tests was higher

235 found that context-induced reinstatement of methamphetamine seeking increased expression of the neur

236 AZD8529 decreased cue-induced methamphetamine seeking on day 21 but not day 1 of force

237 DMS on abstinence day 18 decreased incubated methamphetamine seeking on day 21.

238 Cue-induced methamphetamine seeking progressively increases after wi

239 Cue-induced methamphetamine seeking progressively increases after wi

240 We then assessed relapse to methamphetamine seeking under extinction conditions afte

241 kg/infusion) and tested them for cue-induced methamphetamine seeking under extinction conditions duri

242 Methamphetamine seeking was higher after 21 d of volunta

243 s, and DMS neuronal ensembles in "incubated" methamphetamine seeking, using selective dopamine recept

244 ocaine, and alcohol seeking, but not yet for methamphetamine seeking.

245 rawal had no effect on incubated cue-induced methamphetamine seeking.

246 olonged maternal separation, increases adult methamphetamine self-administration (SA) in male rats as

247 bred Wistar rats experienced extended access methamphetamine self-administration and individual diffe

248 In the present paper, we used a model of methamphetamine self-administration during which we used

249 tigraphy-based sleep parameters disrupted by methamphetamine self-administration in non-human primate

250 We found no sex differences in methamphetamine self-administration or in the strong pre

251 Subsequently, methamphetamine self-administration rats were punished b

252 y comorbid and we have previously shown that methamphetamine self-administration significantly disrup

253 longed voluntary abstinence from intravenous methamphetamine self-administration, to demonstrate that

254 nimals that continue to press a lever to get methamphetamine (shock-resistant) and those that signifi

255 Amphetamine > methamphetamine similarly enhanced DAT-mediated inward c

256 s revealed that sigma1R activation decreases methamphetamine-stimulated dopamine efflux without affec

257 play a role in the regulation of compulsive methamphetamine taking by rats.

258 -contingent punishment significantly reduced methamphetamine taking in some rats (shock-sensitive, SS

259 of 195 qualified neonates were evaluated (36 methamphetamine/tobacco exposed, 32 tobacco exposed, and

260 Conclusions and Relevance: Prenatal methamphetamine/tobacco exposure may lead to delays in m

261 Only methamphetamine/tobacco- and tobacco-exposed girls showe

262 ly less coherent ACR fibers were observed in methamphetamine/tobacco- and tobacco-exposed girls, poss

263 Only methamphetamine/tobacco-exposed boys had lower FA (group

264 Methamphetamine/tobacco-exposed infants showed delayed d

265 showed greater resistance to punishment than methamphetamine-trained rats.

266 Rats self-administered saline or methamphetamine under extended-access conditions.

267 ood (6 sessions) and then to self-administer methamphetamine under two conditions: 12 sessions (9 hou

268 We also discovered that methamphetamine up-regulated the cellular anti-HIV-1 mic

269 sex partners (11 vs. 8; P < 0.001) and more methamphetamine use (15% vs. 8%; P < 0.001) than men who

270 ss was negatively associated with cumulative methamphetamine use and craving for the drug.

271 tion, non-treatment seeking individuals with methamphetamine use disorder (N=23; 74% male, mean age=3

272 Methamphetamine use disorder is associated with striatal

273 to an inpatient behavioral intervention for methamphetamine use disorder reverses deficits in striat

274 Chronic methamphetamine use poses potentially devastating conseq

275 A 2-fold increase of methamphetamine use was detected, associated with a chea

276 receptors in cortical adaptation to chronic methamphetamine use.

277 Thirteen of the methamphetamine users and 10 controls also underwent [(1

278 Fifteen recently abstinent methamphetamine users and 15 healthy controls completed

279 able uptake of the radiotracer, BPnd) (in 31 methamphetamine users and 37 control participants).

280 availability has been observed in striata of methamphetamine users as compared with controls, but an

281 nalyses showed that, on both tasks, the more methamphetamine users chose to view methamphetamine imag

282 nalyses showed that, compared with controls, methamphetamine users chose to view more methamphetamine

283 post-mortem material, with no differences in methamphetamine users from controls in the caudate nucle

284 Maladaptive decision making by methamphetamine users may reflect circuit-level dysfunct

285 tively, to measure gray-matter volume (in 58 methamphetamine users) and dopamine D2/D3 receptor avail

286 eliorate striatal D2/D3 receptor deficits in methamphetamine users, and warrants further evaluation a

287 , insula, hippocampus and temporal cortex in methamphetamine users, but not in control participants (

288 ter volume in mesocorticolimbic circuitry in methamphetamine users, possibly reflecting greater dopam

289 d gray-matter volume have been unexplored in methamphetamine users.

290 rd-driven behavior over cognitive control in methamphetamine users.

291 dissociation in mechanisms of incubation of methamphetamine vs cocaine craving.

292 t the potential validity of this preclinical methamphetamine vs food choice model.

293 The aims of this study were to develop a methamphetamine vs food choice procedure and determine t

294 about the neuropharmacological mechanisms of methamphetamine vs food choice.

295 Conversely, speech on methamphetamine was further from compassion than placebo

296 Methamphetamine was more strongly associated with higher

297 Craving for methamphetamine was negatively associated with gray-matt

298 Morphine, cocaine, and methamphetamine were chosen as test analytes for this st

299 Thereafter, lever-presses for methamphetamine were punished by mild footshocks for 5 d

300 with 88% accuracy, and MDMA (1.5 mg/kg) and methamphetamine with 84% accuracy.