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1 of dopamine neurotransmission in response to methamphetamine.
2 f locating and localizing the manufacture of methamphetamine.
3 by alcohol, opiate, cocaine, oxycodone, and methamphetamine.
4 administration of MDMA, its enantiomers, and methamphetamine.
5 male/female rhesus monkeys self-administered methamphetamine (0.03 mg/kg/injection, i.v.) under a fix
7 male Sprague-Dawley rats to self-administer methamphetamine (0.1 mg/kg/infusion) for 9 hours daily f
8 pellets for 6 days (6 h per day) for either methamphetamine (0.1 mg/kg/infusion) or heroin (0.1 mg/k
11 lanted radiotelemetry, following exposure to methamphetamine (12.5 and 100 mg/ml), MDPV (25, 50, and
13 optimized for quantification of amphetamine, methamphetamine, 3,4-methylenedioxyamphetamine (MDA), 3,
14 drocodone) and five stimulants (amphetamine, methamphetamine, 3,4-methylenedioxymethamphetamine (MDMA
15 s highly dysregulated by the psychostimulant methamphetamine, a substrate for the dopamine transporte
19 se results are contrary to the paradigm that methamphetamine accentuates HIV-1 pathogenesis by increa
20 neural responses to drug cues in humans with methamphetamine addiction and suggests that naltrexone m
23 ct has been shown for alcohol, nicotine, and methamphetamine addictions, but not for heroin or cocain
25 men who had recently used cocaine, crack, or methamphetamine (adjusted odds ratio (OR) = 2.13, 95% co
26 ad been exposed to different combinations of methamphetamine, alcohol, nicotine and marijuana prior t
30 avioral and neurophysiological correlates of methamphetamine and amphetamine administration are uniqu
31 showed, for drugs with weekly cycles (MDMA, methamphetamine and cocaine in this sample), sampling st
32 in midbrain D2/D3 BPnd was detected between methamphetamine and control groups, midbrain D2/D3 BPnd
33 results demonstrate that incubation of both methamphetamine and food craving occur after punishment-
35 UCP1 knockout (UCP1KO) mice exhibit blunted methamphetamine and fully inhibited noradrenaline thermo
36 transmitted infections, and higher rates of methamphetamine and gamma hydroxybutyrate use when compa
37 sing ToF-SIMS and DESI-MS, as the signals of methamphetamine and heroin were completely suppressed by
38 rogressively increases after withdrawal from methamphetamine and other drugs, a phenomenon termed 'in
39 eved via a discrete choice procedure between methamphetamine and palatable food; 20 trials per day) f
40 little is known about the effect of comorbid methamphetamine and tobacco use on human fetal brain dev
42 ific genes, including Azi2 in sensitivity to methamphetamine and Zmynd11 in anxiety-like behavior.
43 rmalities reported in children with prenatal methamphetamine and/or tobacco exposure are present at b
47 UCP3 (UCP3KO) have impaired sympathomimetic (methamphetamine) and completely abrogated lipopolysaccha
49 rated from inkjet-printed arrays of cocaine, methamphetamine, and heroin with a deposited-mass rangin
50 that repeated exposure to alcohol, nicotine, methamphetamine, and morphine upregulates alpha2delta-1
51 ul T-maze task following a binge exposure to methamphetamine, and no such changes in effort following
52 rkers in the mesocorticolimbic pathway after methamphetamine, and their relationship with animals' re
53 g behavior, is a major target of cocaine and methamphetamines, and has been assumed to be conserved a
54 relapse tests, both punished and unpunished methamphetamine- and food-trained rats showed significan
55 hold values for the detection of cocaine and methamphetamine as proof-of-principle with 0.25 and 0.75
56 reactivity, and attentional bias toward the methamphetamine-associated cue, compared with the placeb
58 sed methamphetamine choice, whereas removing methamphetamine availability decreased methamphetamine c
59 (gp120+ and gp120-) were exposed to either a methamphetamine binge (METH+) or saline (METH-), then te
61 urthermore, NAc core MeCP2 knockdown reduces methamphetamine, but not saccharin, SA on a progressive
62 d activity-based sleep measures disrupted by methamphetamine by decreasing sleep latency and increasi
63 e, whereas bupropion treatment did not alter methamphetamine choice up to doses that decreased rates
68 analysis was performed for four drugs (MDMA, methamphetamine, cocaine, methadone) collected through a
70 ently developed a rat model of incubation of methamphetamine craving after choice-based voluntary abs
71 introduced an animal model of incubation of methamphetamine craving after choice-based voluntary abs
72 osteric modulator, AZD8529, on incubation of methamphetamine craving after forced or voluntary abstin
73 ine D1 and D2 receptors in the incubation of methamphetamine craving after voluntary abstinence and t
75 rate a role for the NAc in the incubation of methamphetamine craving and describe adaptations in syna
76 trate a critical role of DS in incubation of methamphetamine craving and that this incubation is asso
83 ly increases after withdrawal (incubation of methamphetamine craving), but the underlying mechanisms
93 within-subject design and underwent a visual methamphetamine cue-reactivity task during two blood-oxy
94 tic resonance imaging (fMRI) measures during methamphetamine cue-reactivity to elucidate the role of
97 lt men and women who met DSM-IV criteria for methamphetamine dependence and were enrolled in a reside
100 s from patients diagnosed with MAP (N = 12), methamphetamine dependence without psychosis (MA; N = 14
103 In Study 2, an independent sample of 20 methamphetamine-dependent and 18 control subjects comple
105 academic institution, this study involved 25 methamphetamine-dependent and 27 control participants.
106 nctional magnetic resonance imaging study of methamphetamine-dependent and healthy comparison partici
109 the GIRK3 subunit appeared critical for the methamphetamine-dependent decrease of GABABR-GIRK curren
112 ventral striatum but weaker in the rDLPFC in methamphetamine-dependent participants than control indi
113 striatum, orbitofrontal cortex and insula in methamphetamine-dependent participants, but a positive r
116 mpared with healthy volunteers, alcohol- and methamphetamine-dependent subjects and cannabis users sh
121 ferred over no methamphetamine or small unit methamphetamine doses (0.01-0.032 mg/kg/injection).
123 trong preference for the palatable food over methamphetamine during the choice-based voluntary abstin
125 ice impaired learning, and that a history of methamphetamine exposure increased the susceptibility to
128 re, we show that stimulants like cocaine and methamphetamine greatly increase HS content and sulfatio
129 eceptor availability (BPnd) was lower in the methamphetamine group, as shown previously, the groups d
130 l mean cortical gray-matter thickness in the methamphetamine group, but no association was found betw
132 ty to differences in reward magnitude in the methamphetamine group: animals were more likely to overc
134 social behaviors and vocalizations, whereas methamphetamine had only modest effects on affiliative b
135 nesis remains to be established in patients, methamphetamine has been shown to increase HIV-1 replica
136 the more methamphetamine users chose to view methamphetamine images, specifically vs pleasant images
140 us, both receptor subtypes may contribute to methamphetamine-induced alterations in cortical morpholo
141 at or near the plasma membrane and decreases methamphetamine-induced Ca(2+) signaling, providing pote
142 acellular Na(+) ions blocked amphetamine and methamphetamine-induced DAT-mediated inward current simi
144 lacking both UCP1 and 3 (UCPDK) fail to show methamphetamine-induced hyperthermia, and have a markedl
145 these findings, sigma1R activation decreases methamphetamine-induced locomotion, motivated behavior,
148 ine release and that dopamine is involved in methamphetamine-induced neurotoxicity, associations betw
149 We found that sigma1R activation prevents methamphetamine-induced, DAT-mediated increases in firin
150 xed-ratio (FR) 100 schedule) and intravenous methamphetamine injections (0-0.32 mg/kg/injection, FR10
151 parison, effects of removing food pellets or methamphetamine injections and FR response requirement m
154 of psychostimulants such as amphetamine and methamphetamine is the dopamine transporter (DAT), the m
156 CR4056-associated lever responding included methamphetamine, ketamine, the endogenous imidazoline li
158 dynamics of cognitive control instability in methamphetamine (MA) abuse, as well its relationship to
160 The psychostimulants amphetamine (AMPH) and methamphetamine (MA) are widely abused illicit drugs.
164 ing the frontostriatal system, in adolescent methamphetamine (MA) users compared with adult users.
165 , male Wistar rats were exposed to vaporized methamphetamine (MA), 3,4-methylenedioxypyrovalerone (MD
166 d by chronic exposure to the psychostimulant methamphetamine (MA), the current study sought to use co
167 hetamine preference and 0.32 mg/kg/injection methamphetamine maintained near exclusive preference.
168 detect atmospheric effluent from clandestine methamphetamine manufacture is a useful tool for law enf
171 Since CUMS is a model for depression and methamphetamine (METH) abuse induced psychosis recapitul
174 ctive medications to aid in the treatment of methamphetamine (METH) abuse, we used a nanotechnology a
177 hypothesized that Glycoprotein 120 (gp120), methamphetamine (METH) and nicotine (NT) can enhance amy
178 rhythmically pairing 2 reinforcing stimuli [methamphetamine (Meth) and running wheel (RW)] restores
182 to segregate rats that reduce or stop their methamphetamine (METH) intake (nonaddicted) from those t
183 (nor-BNI) would attenuate the escalation of methamphetamine (METH) intake in an extended-access self
188 We investigated the associations between methamphetamine (meth) use, immune function, and the dyn
189 dopamine and serotonin terminals produced by methamphetamine (METH), but how ammonia contributes to t
190 ing that storage of memories associated with methamphetamine (METH), but not memories for fear or foo
191 ries associated with drugs of abuse, such as methamphetamine (METH), increase relapse vulnerability t
192 ries associated with drugs of abuse, such as methamphetamine (METH), increase relapse vulnerability t
193 as previously demonstrated in male rats that methamphetamine (Meth), when administered concurrently w
194 clades to exert differential effects and the methamphetamine (METH)-associated dopaminergic dysfuncti
196 ve therapy is available for the treatment of methamphetamine (METH)-induced neurotoxicity, aerobic ex
199 m 4% (norcocaine) to 99% (cocaine); however, methamphetamine, methadone, and EDDP showed a negative r
200 ine, cannabidiol, cocaine, codeine, heroine, methamphetamine, morphine, phentermine, L-phenylepherine
201 es to other abused drugs, including cocaine, methamphetamine, nicotine, and alcohol, and is also obse
204 parate and combined effects of HIV/gp120 and methamphetamine on learning and executive functions in b
205 cal intervention to attenuate the effects of methamphetamine on nighttime activity under well-control
206 igated the long-lasting, off-drug effects of methamphetamine on reward choices in a novel effortful m
207 racellular and intracellular amphetamine and methamphetamine on the spontaneous firing of cultured mi
208 ingent punishment suppressed extended-access methamphetamine or food self-administration; surprisingl
209 ccur after punishment-induced suppression of methamphetamine or palatable food self-administration.
210 withdrawal in rats following extended-access methamphetamine or saline (control condition) self-admin
211 tment conditions, food was preferred over no methamphetamine or small unit methamphetamine doses (0.0
212 mine, the psychostimulants d-amphetamine and methamphetamine, or to cocaine and cocaine analogues.
213 gm in which they received a moderate dose of methamphetamine paired with one stimulus and placebo wit
215 er methamphetamine doses resulted in greater methamphetamine preference and 0.32 mg/kg/injection meth
216 e unique mechanisms by which amphetamine and methamphetamine regulate DAT function and dopamine neuro
218 ls, methamphetamine users chose to view more methamphetamine-related images on one task, with a simil
219 both natural (binge eating) and artificial (methamphetamine) rewards, and obsessive-compulsive disor
220 tion, we show new evidence that both ELS and methamphetamine SA alter the expression of the epigeneti
222 core MeCP2 may be recruited by both ELS and methamphetamine SA and promote the development of certai
223 gether, functional interactions between ELS, methamphetamine SA, and the expression of MeCP2 in the N
225 of AZD8529 (20 and 40 mg/kg) on cue-induced methamphetamine seeking 1 day or 21 days after forced or
227 s induction, decreased incubated cue-induced methamphetamine seeking after prolonged withdrawal.
228 striatum in context-induced reinstatement of methamphetamine seeking and that this reinstatement is a
229 imol+baclofen, 0.03+0.3 nmol) on cue-induced methamphetamine seeking during early and late withdrawal
230 s into CeA but not BLA decreased cue-induced methamphetamine seeking during late but not early withdr
233 ining and abstinence conditions, cue-induced methamphetamine seeking in the extinction tests was high
235 found that context-induced reinstatement of methamphetamine seeking increased expression of the neur
241 kg/infusion) and tested them for cue-induced methamphetamine seeking under extinction conditions duri
243 s, and DMS neuronal ensembles in "incubated" methamphetamine seeking, using selective dopamine recept
246 olonged maternal separation, increases adult methamphetamine self-administration (SA) in male rats as
247 bred Wistar rats experienced extended access methamphetamine self-administration and individual diffe
248 In the present paper, we used a model of methamphetamine self-administration during which we used
249 tigraphy-based sleep parameters disrupted by methamphetamine self-administration in non-human primate
252 y comorbid and we have previously shown that methamphetamine self-administration significantly disrup
253 longed voluntary abstinence from intravenous methamphetamine self-administration, to demonstrate that
254 nimals that continue to press a lever to get methamphetamine (shock-resistant) and those that signifi
256 s revealed that sigma1R activation decreases methamphetamine-stimulated dopamine efflux without affec
258 -contingent punishment significantly reduced methamphetamine taking in some rats (shock-sensitive, SS
259 of 195 qualified neonates were evaluated (36 methamphetamine/tobacco exposed, 32 tobacco exposed, and
262 ly less coherent ACR fibers were observed in methamphetamine/tobacco- and tobacco-exposed girls, poss
267 ood (6 sessions) and then to self-administer methamphetamine under two conditions: 12 sessions (9 hou
269 sex partners (11 vs. 8; P < 0.001) and more methamphetamine use (15% vs. 8%; P < 0.001) than men who
271 tion, non-treatment seeking individuals with methamphetamine use disorder (N=23; 74% male, mean age=3
273 to an inpatient behavioral intervention for methamphetamine use disorder reverses deficits in striat
279 able uptake of the radiotracer, BPnd) (in 31 methamphetamine users and 37 control participants).
280 availability has been observed in striata of methamphetamine users as compared with controls, but an
281 nalyses showed that, on both tasks, the more methamphetamine users chose to view methamphetamine imag
282 nalyses showed that, compared with controls, methamphetamine users chose to view more methamphetamine
283 post-mortem material, with no differences in methamphetamine users from controls in the caudate nucle
285 tively, to measure gray-matter volume (in 58 methamphetamine users) and dopamine D2/D3 receptor avail
286 eliorate striatal D2/D3 receptor deficits in methamphetamine users, and warrants further evaluation a
287 , insula, hippocampus and temporal cortex in methamphetamine users, but not in control participants (
288 ter volume in mesocorticolimbic circuitry in methamphetamine users, possibly reflecting greater dopam
293 The aims of this study were to develop a methamphetamine vs food choice procedure and determine t
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