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1 of dopamine neurotransmission in response to methamphetamine.
2 f locating and localizing the manufacture of methamphetamine.
3  by alcohol, opiate, cocaine, oxycodone, and methamphetamine.
4 administration of MDMA, its enantiomers, and methamphetamine.
5 male/female rhesus monkeys self-administered methamphetamine (0.03 mg/kg/injection, i.v.) under a fix
6 for drug reinforcement and self-administered methamphetamine (0.05 mg/kg) at control levels.
7  male Sprague-Dawley rats to self-administer methamphetamine (0.1 mg/kg/infusion) for 9 hours daily f
8  pellets for 6 days (6 h per day) for either methamphetamine (0.1 mg/kg/infusion) or heroin (0.1 mg/k
9           We trained rats to self-administer methamphetamine (10 days; 9 h/day, 0.1 mg/kg/infusion) a
10 ster palatable food pellets (6 d, 6 h/d) and methamphetamine (12 d, 6 h/d).
11 lanted radiotelemetry, following exposure to methamphetamine (12.5 and 100 mg/ml), MDPV (25, 50, and
12 speech task after MDMA (0.75 and 1.5 mg/kg), methamphetamine (20 mg), or placebo.
13 optimized for quantification of amphetamine, methamphetamine, 3,4-methylenedioxyamphetamine (MDA), 3,
14 drocodone) and five stimulants (amphetamine, methamphetamine, 3,4-methylenedioxymethamphetamine (MDMA
15 s highly dysregulated by the psychostimulant methamphetamine, a substrate for the dopamine transporte
16               Although a direct link between methamphetamine abuse and HIV-1 pathogenesis remains to
17                                              Methamphetamine abuse is associated with increased risk
18 cal profiles in animal models of cocaine and methamphetamine abuse.
19 se results are contrary to the paradigm that methamphetamine accentuates HIV-1 pathogenesis by increa
20 neural responses to drug cues in humans with methamphetamine addiction and suggests that naltrexone m
21                                              Methamphetamine addiction is mimicked in rats that self-
22 1R as a putative target for the treatment of methamphetamine addiction.
23 ct has been shown for alcohol, nicotine, and methamphetamine addictions, but not for heroin or cocain
24 gy may be helpful for recovering cocaine and methamphetamine addicts.
25 men who had recently used cocaine, crack, or methamphetamine (adjusted odds ratio (OR) = 2.13, 95% co
26 ad been exposed to different combinations of methamphetamine, alcohol, nicotine and marijuana prior t
27  or been offered opioids, cocaine, cannabis, methamphetamine, alcohol, or tobacco.
28                                              Methamphetamine also interacts with the sigma-1 receptor
29 lower DAT levels secondary to the effects of methamphetamine and alcohol.
30 avioral and neurophysiological correlates of methamphetamine and amphetamine administration are uniqu
31  showed, for drugs with weekly cycles (MDMA, methamphetamine and cocaine in this sample), sampling st
32  in midbrain D2/D3 BPnd was detected between methamphetamine and control groups, midbrain D2/D3 BPnd
33  results demonstrate that incubation of both methamphetamine and food craving occur after punishment-
34 e (using a discrete choice procedure between methamphetamine and food; 20 trials/d) for 19 d.
35  UCP1 knockout (UCP1KO) mice exhibit blunted methamphetamine and fully inhibited noradrenaline thermo
36  transmitted infections, and higher rates of methamphetamine and gamma hydroxybutyrate use when compa
37 sing ToF-SIMS and DESI-MS, as the signals of methamphetamine and heroin were completely suppressed by
38 rogressively increases after withdrawal from methamphetamine and other drugs, a phenomenon termed 'in
39 eved via a discrete choice procedure between methamphetamine and palatable food; 20 trials per day) f
40 little is known about the effect of comorbid methamphetamine and tobacco use on human fetal brain dev
41 ular application of amphetamine > dopamine > methamphetamine and was DAT-dependent.
42 ific genes, including Azi2 in sensitivity to methamphetamine and Zmynd11 in anxiety-like behavior.
43 rmalities reported in children with prenatal methamphetamine and/or tobacco exposure are present at b
44                       Collectively, prenatal methamphetamine and/or tobacco exposure may lead to dela
45                          Exposures: Prenatal methamphetamine and/or tobacco exposure.
46                                              Methamphetamine and/or tobacco use by pregnant women rem
47 UCP3 (UCP3KO) have impaired sympathomimetic (methamphetamine) and completely abrogated lipopolysaccha
48 tyrate or gamma-butyrolactone, 175 (8%) used methamphetamine, and 162 (7%) used mephedrone.
49 rated from inkjet-printed arrays of cocaine, methamphetamine, and heroin with a deposited-mass rangin
50 that repeated exposure to alcohol, nicotine, methamphetamine, and morphine upregulates alpha2delta-1
51 ul T-maze task following a binge exposure to methamphetamine, and no such changes in effort following
52 rkers in the mesocorticolimbic pathway after methamphetamine, and their relationship with animals' re
53 g behavior, is a major target of cocaine and methamphetamines, and has been assumed to be conserved a
54  relapse tests, both punished and unpunished methamphetamine- and food-trained rats showed significan
55 hold values for the detection of cocaine and methamphetamine as proof-of-principle with 0.25 and 0.75
56  reactivity, and attentional bias toward the methamphetamine-associated cue, compared with the placeb
57                                              Methamphetamine-associated psychosis (MAP) involves wide
58 sed methamphetamine choice, whereas removing methamphetamine availability decreased methamphetamine c
59 (gp120+ and gp120-) were exposed to either a methamphetamine binge (METH+) or saline (METH-), then te
60             The intracellular application of methamphetamine, but not amphetamine, prevented the dopa
61 urthermore, NAc core MeCP2 knockdown reduces methamphetamine, but not saccharin, SA on a progressive
62 d activity-based sleep measures disrupted by methamphetamine by decreasing sleep latency and increasi
63 e, whereas bupropion treatment did not alter methamphetamine choice up to doses that decreased rates
64                                              Methamphetamine choice was not significantly altered whe
65              Risperidone treatment increased methamphetamine choice, whereas bupropion treatment did
66         Removing food availability increased methamphetamine choice, whereas removing methamphetamine
67 oving methamphetamine availability decreased methamphetamine choice.
68 analysis was performed for four drugs (MDMA, methamphetamine, cocaine, methadone) collected through a
69 cur much more rapidly during withdrawal from methamphetamine compared with cocaine.
70 ently developed a rat model of incubation of methamphetamine craving after choice-based voluntary abs
71  introduced an animal model of incubation of methamphetamine craving after choice-based voluntary abs
72 osteric modulator, AZD8529, on incubation of methamphetamine craving after forced or voluntary abstin
73 ine D1 and D2 receptors in the incubation of methamphetamine craving after voluntary abstinence and t
74          Our results show that incubation of methamphetamine craving after voluntary abstinence gener
75 rate a role for the NAc in the incubation of methamphetamine craving and describe adaptations in syna
76 trate a critical role of DS in incubation of methamphetamine craving and that this incubation is asso
77                                  Cue-induced methamphetamine craving increases after prolonged forced
78       Here, we studied whether incubation of methamphetamine craving is observed after suppression of
79                                Incubation of methamphetamine craving was associated with CP-AMPAR acc
80 relationship between midbrain D2/D3 BPnd and methamphetamine craving was not detected.
81 ut mechanisms underlying this 'incubation of methamphetamine craving' are unknown.
82       We first confirmed that 'incubation of methamphetamine craving' occurs under our experimental c
83 ly increases after withdrawal (incubation of methamphetamine craving), but the underlying mechanisms
84 sed) abstinence from the drug (incubation of methamphetamine craving).
85 tinence days than after 1 day (incubation of methamphetamine craving).
86  than during early withdrawal (incubation of methamphetamine craving).
87 d abstinence than after 1 day (incubation of methamphetamine craving).
88 ary abstinence than after 1 d (incubation of methamphetamine craving).
89 ced seeking tests demonstrated incubation of methamphetamine craving.
90 similar plasticity accompanies incubation of methamphetamine craving.
91 cocaine and heroin craving, in incubation of methamphetamine craving.
92 wal interval when animals show incubation of methamphetamine craving.
93 within-subject design and underwent a visual methamphetamine cue-reactivity task during two blood-oxy
94 tic resonance imaging (fMRI) measures during methamphetamine cue-reactivity to elucidate the role of
95 y HIV serostatus (HIV+ or HIV-) and lifetime methamphetamine dependence (METH+ or METH-).
96  data from 4739 cases of alcohol, opioid, or methamphetamine dependence and 4924 controls.
97 lt men and women who met DSM-IV criteria for methamphetamine dependence and were enrolled in a reside
98        The extent to which these features of methamphetamine dependence are interrelated, however, is
99                                              Methamphetamine dependence is a common comorbid conditio
100 s from patients diagnosed with MAP (N = 12), methamphetamine dependence without psychosis (MA; N = 14
101 n be used to predict relapse among abstinent methamphetamine-dependent (MD) individuals.
102              Abstinent alcohol- (n = 30) and methamphetamine-dependent (n = 23) subjects, recreationa
103      In Study 2, an independent sample of 20 methamphetamine-dependent and 18 control subjects comple
104                               In Study 1, 19 methamphetamine-dependent and 26 healthy control subject
105 academic institution, this study involved 25 methamphetamine-dependent and 27 control participants.
106 nctional magnetic resonance imaging study of methamphetamine-dependent and healthy comparison partici
107                           Here, we show that methamphetamine-dependent attenuation of GABABR-GIRK cur
108                                          The methamphetamine-dependent decrease in GABABR-GIRK curren
109  the GIRK3 subunit appeared critical for the methamphetamine-dependent decrease of GABABR-GIRK curren
110 nk that may help explain high impulsivity in methamphetamine-dependent individuals.
111                                              Methamphetamine-dependent participants also exhibited gr
112 ventral striatum but weaker in the rDLPFC in methamphetamine-dependent participants than control indi
113 striatum, orbitofrontal cortex and insula in methamphetamine-dependent participants, but a positive r
114                                           In methamphetamine-dependent research participants, impulsi
115                            In this study, 21 methamphetamine-dependent subjects and 23 healthy contro
116 mpared with healthy volunteers, alcohol- and methamphetamine-dependent subjects and cannabis users sh
117                                              Methamphetamine-dependent subjects had greater nonlinear
118 n of subjective value similar to that of the methamphetamine-dependent subjects.
119                                              Methamphetamine-dependent users exhibited a positive rel
120                         Prenatal exposure to methamphetamine did not influence visual function.
121 ferred over no methamphetamine or small unit methamphetamine doses (0.01-0.032 mg/kg/injection).
122                                       Larger methamphetamine doses resulted in greater methamphetamin
123 trong preference for the palatable food over methamphetamine during the choice-based voluntary abstin
124                                              Methamphetamine exposure also increased repressor elemen
125 ice impaired learning, and that a history of methamphetamine exposure increased the susceptibility to
126             Lever presses in the cue-induced methamphetamine extinction tests were higher during late
127           We trained rats to self-administer methamphetamine for 20 days.
128 re, we show that stimulants like cocaine and methamphetamine greatly increase HS content and sulfatio
129 eceptor availability (BPnd) was lower in the methamphetamine group, as shown previously, the groups d
130 l mean cortical gray-matter thickness in the methamphetamine group, but no association was found betw
131                                       In the methamphetamine group, mean cortical gray-matter thickne
132 ty to differences in reward magnitude in the methamphetamine group: animals were more likely to overc
133                 Our results demonstrate that methamphetamine had a minimal effect on HIV-1 replicatio
134  social behaviors and vocalizations, whereas methamphetamine had only modest effects on affiliative b
135 nesis remains to be established in patients, methamphetamine has been shown to increase HIV-1 replica
136 the more methamphetamine users chose to view methamphetamine images, specifically vs pleasant images
137 nedioxymethamphetamine (MDMA; 'ecstasy') and methamphetamine in 13 ecstasy users.
138 in primary CD4(+) T cells in the presence of methamphetamine in a dose-dependent manner.
139               Through interactions with DAT, methamphetamine increases extracellular dopamine levels
140 us, both receptor subtypes may contribute to methamphetamine-induced alterations in cortical morpholo
141 at or near the plasma membrane and decreases methamphetamine-induced Ca(2+) signaling, providing pote
142 acellular Na(+) ions blocked amphetamine and methamphetamine-induced DAT-mediated inward current simi
143       The results reveal a new mechanism for methamphetamine-induced dysregulation of dopaminergic ne
144 lacking both UCP1 and 3 (UCPDK) fail to show methamphetamine-induced hyperthermia, and have a markedl
145 these findings, sigma1R activation decreases methamphetamine-induced locomotion, motivated behavior,
146  anti-MA immune response in mice, decreasing methamphetamine-induced locomotor activity.
147                          Exercise attenuates methamphetamine-induced neurochemical damage in the rat
148 ine release and that dopamine is involved in methamphetamine-induced neurotoxicity, associations betw
149    We found that sigma1R activation prevents methamphetamine-induced, DAT-mediated increases in firin
150 xed-ratio (FR) 100 schedule) and intravenous methamphetamine injections (0-0.32 mg/kg/injection, FR10
151 parison, effects of removing food pellets or methamphetamine injections and FR response requirement m
152                                  Importance: Methamphetamine is a common illicit drug used worldwide.
153                   Released from neurons when methamphetamine is self-administered, dopamine binds to
154  of psychostimulants such as amphetamine and methamphetamine is the dopamine transporter (DAT), the m
155                                              Methamphetamine is the second most frequently used illic
156  CR4056-associated lever responding included methamphetamine, ketamine, the endogenous imidazoline li
157          The high prevalence and severity of methamphetamine (MA) abuse demands greater neurobiologic
158 dynamics of cognitive control instability in methamphetamine (MA) abuse, as well its relationship to
159                                              Methamphetamine (MA) addiction is a serious public healt
160  The psychostimulants amphetamine (AMPH) and methamphetamine (MA) are widely abused illicit drugs.
161                                              Methamphetamine (MA) use disorder is a serious psychiatr
162                                    Continued methamphetamine (MA) use is dependent on a positive MA e
163                                              Methamphetamine (MA) users are assumed to have a high bu
164 ing the frontostriatal system, in adolescent methamphetamine (MA) users compared with adult users.
165 , male Wistar rats were exposed to vaporized methamphetamine (MA), 3,4-methylenedioxypyrovalerone (MD
166 d by chronic exposure to the psychostimulant methamphetamine (MA), the current study sought to use co
167 hetamine preference and 0.32 mg/kg/injection methamphetamine maintained near exclusive preference.
168 detect atmospheric effluent from clandestine methamphetamine manufacture is a useful tool for law enf
169                             The high rate of methamphetamine (METH) abuse among young adults and wome
170                          HIV-1 infection and methamphetamine (METH) abuse frequently occur simultaneo
171     Since CUMS is a model for depression and methamphetamine (METH) abuse induced psychosis recapitul
172                                              Methamphetamine (METH) abuse is a serious social and hea
173                                              Methamphetamine (METH) abuse is a worldwide threat, with
174 ctive medications to aid in the treatment of methamphetamine (METH) abuse, we used a nanotechnology a
175 to examine oxytocin as a pharmacotherapy for methamphetamine (meth) addiction.
176                                              Methamphetamine (meth) addicts often exhibit enduring co
177  hypothesized that Glycoprotein 120 (gp120), methamphetamine (METH) and nicotine (NT) can enhance amy
178  rhythmically pairing 2 reinforcing stimuli [methamphetamine (Meth) and running wheel (RW)] restores
179                                  Exposure to methamphetamine (meth) can produce lasting memory impair
180                                              Methamphetamine (METH) competes with dopamine uptake, in
181                                      Chronic methamphetamine (METH) exposure causes neuroadaptations
182  to segregate rats that reduce or stop their methamphetamine (METH) intake (nonaddicted) from those t
183  (nor-BNI) would attenuate the escalation of methamphetamine (METH) intake in an extended-access self
184                                              Methamphetamine (METH) is a drug with a high addictive p
185                                              Methamphetamine (Meth) is a psychomotor stimulant strong
186                                              Methamphetamine (METH) is a substrate for the dopamine t
187             Rats that have self-administered methamphetamine (meth) under long access, but not short
188     We investigated the associations between methamphetamine (meth) use, immune function, and the dyn
189 dopamine and serotonin terminals produced by methamphetamine (METH), but how ammonia contributes to t
190 ing that storage of memories associated with methamphetamine (METH), but not memories for fear or foo
191 ries associated with drugs of abuse, such as methamphetamine (METH), increase relapse vulnerability t
192 ries associated with drugs of abuse, such as methamphetamine (METH), increase relapse vulnerability t
193 as previously demonstrated in male rats that methamphetamine (Meth), when administered concurrently w
194 clades to exert differential effects and the methamphetamine (METH)-associated dopaminergic dysfuncti
195                                              Methamphetamine (METH)-induced neurotoxicity results in
196 ve therapy is available for the treatment of methamphetamine (METH)-induced neurotoxicity, aerobic ex
197 nds such as cocaine, amphetamine (AMPH), and methamphetamine (METH).
198 ta-phenylethylamine, amphetamine (AMPH), and methamphetamine (METH).
199 m 4% (norcocaine) to 99% (cocaine); however, methamphetamine, methadone, and EDDP showed a negative r
200 ine, cannabidiol, cocaine, codeine, heroine, methamphetamine, morphine, phentermine, L-phenylepherine
201 es to other abused drugs, including cocaine, methamphetamine, nicotine, and alcohol, and is also obse
202 omprehensive understanding of the effects of methamphetamine on HIV-1 pathogenesis.
203                 Intriguingly, the effects of methamphetamine on HIV-1 replication in human CD4(+) T c
204 parate and combined effects of HIV/gp120 and methamphetamine on learning and executive functions in b
205 cal intervention to attenuate the effects of methamphetamine on nighttime activity under well-control
206 igated the long-lasting, off-drug effects of methamphetamine on reward choices in a novel effortful m
207 racellular and intracellular amphetamine and methamphetamine on the spontaneous firing of cultured mi
208 ingent punishment suppressed extended-access methamphetamine or food self-administration; surprisingl
209 ccur after punishment-induced suppression of methamphetamine or palatable food self-administration.
210 withdrawal in rats following extended-access methamphetamine or saline (control condition) self-admin
211 tment conditions, food was preferred over no methamphetamine or small unit methamphetamine doses (0.0
212 mine, the psychostimulants d-amphetamine and methamphetamine, or to cocaine and cocaine analogues.
213 gm in which they received a moderate dose of methamphetamine paired with one stimulus and placebo wit
214                             The synthesis of methamphetamine precursors was performed, and the impuri
215 er methamphetamine doses resulted in greater methamphetamine preference and 0.32 mg/kg/injection meth
216 e unique mechanisms by which amphetamine and methamphetamine regulate DAT function and dopamine neuro
217 nence blocked compulsive-like context-driven methamphetamine reinstatement.
218 ls, methamphetamine users chose to view more methamphetamine-related images on one task, with a simil
219  both natural (binge eating) and artificial (methamphetamine) rewards, and obsessive-compulsive disor
220 tion, we show new evidence that both ELS and methamphetamine SA alter the expression of the epigeneti
221  genome-wide transcriptional consequences of methamphetamine SA and footshocks in the rat brain.
222  core MeCP2 may be recruited by both ELS and methamphetamine SA and promote the development of certai
223 gether, functional interactions between ELS, methamphetamine SA, and the expression of MeCP2 in the N
224  of MeCP2 expression in the NAc core reduces methamphetamine SA, as well as saccharin intake.
225  of AZD8529 (20 and 40 mg/kg) on cue-induced methamphetamine seeking 1 day or 21 days after forced or
226 SCH39166 or raclopride selectively decreased methamphetamine seeking after 21 abstinence days.
227 s induction, decreased incubated cue-induced methamphetamine seeking after prolonged withdrawal.
228 striatum in context-induced reinstatement of methamphetamine seeking and that this reinstatement is a
229 imol+baclofen, 0.03+0.3 nmol) on cue-induced methamphetamine seeking during early and late withdrawal
230 s into CeA but not BLA decreased cue-induced methamphetamine seeking during late but not early withdr
231 C, dmPFC, and OFC on 'incubated' cue-induced methamphetamine seeking during late withdrawal.
232                 We then assessed cue-induced methamphetamine seeking in extinction tests after 1 or 2
233 ining and abstinence conditions, cue-induced methamphetamine seeking in the extinction tests was high
234                               In both sexes, methamphetamine seeking in the relapse tests was higher
235  found that context-induced reinstatement of methamphetamine seeking increased expression of the neur
236                AZD8529 decreased cue-induced methamphetamine seeking on day 21 but not day 1 of force
237 DMS on abstinence day 18 decreased incubated methamphetamine seeking on day 21.
238                                  Cue-induced methamphetamine seeking progressively increases after wi
239                                  Cue-induced methamphetamine seeking progressively increases after wi
240                  We then assessed relapse to methamphetamine seeking under extinction conditions afte
241 kg/infusion) and tested them for cue-induced methamphetamine seeking under extinction conditions duri
242                                              Methamphetamine seeking was higher after 21 d of volunta
243 s, and DMS neuronal ensembles in "incubated" methamphetamine seeking, using selective dopamine recept
244 ocaine, and alcohol seeking, but not yet for methamphetamine seeking.
245 rawal had no effect on incubated cue-induced methamphetamine seeking.
246 olonged maternal separation, increases adult methamphetamine self-administration (SA) in male rats as
247 bred Wistar rats experienced extended access methamphetamine self-administration and individual diffe
248     In the present paper, we used a model of methamphetamine self-administration during which we used
249 tigraphy-based sleep parameters disrupted by methamphetamine self-administration in non-human primate
250               We found no sex differences in methamphetamine self-administration or in the strong pre
251                                Subsequently, methamphetamine self-administration rats were punished b
252 y comorbid and we have previously shown that methamphetamine self-administration significantly disrup
253 longed voluntary abstinence from intravenous methamphetamine self-administration, to demonstrate that
254 nimals that continue to press a lever to get methamphetamine (shock-resistant) and those that signifi
255                                Amphetamine > methamphetamine similarly enhanced DAT-mediated inward c
256 s revealed that sigma1R activation decreases methamphetamine-stimulated dopamine efflux without affec
257  play a role in the regulation of compulsive methamphetamine taking by rats.
258 -contingent punishment significantly reduced methamphetamine taking in some rats (shock-sensitive, SS
259 of 195 qualified neonates were evaluated (36 methamphetamine/tobacco exposed, 32 tobacco exposed, and
260          Conclusions and Relevance: Prenatal methamphetamine/tobacco exposure may lead to delays in m
261                                         Only methamphetamine/tobacco- and tobacco-exposed girls showe
262 ly less coherent ACR fibers were observed in methamphetamine/tobacco- and tobacco-exposed girls, poss
263                                         Only methamphetamine/tobacco-exposed boys had lower FA (group
264                                              Methamphetamine/tobacco-exposed infants showed delayed d
265 showed greater resistance to punishment than methamphetamine-trained rats.
266             Rats self-administered saline or methamphetamine under extended-access conditions.
267 ood (6 sessions) and then to self-administer methamphetamine under two conditions: 12 sessions (9 hou
268                      We also discovered that methamphetamine up-regulated the cellular anti-HIV-1 mic
269  sex partners (11 vs. 8; P < 0.001) and more methamphetamine use (15% vs. 8%; P < 0.001) than men who
270 ss was negatively associated with cumulative methamphetamine use and craving for the drug.
271 tion, non-treatment seeking individuals with methamphetamine use disorder (N=23; 74% male, mean age=3
272                                              Methamphetamine use disorder is associated with striatal
273  to an inpatient behavioral intervention for methamphetamine use disorder reverses deficits in striat
274                                      Chronic methamphetamine use poses potentially devastating conseq
275                         A 2-fold increase of methamphetamine use was detected, associated with a chea
276  receptors in cortical adaptation to chronic methamphetamine use.
277                              Thirteen of the methamphetamine users and 10 controls also underwent [(1
278                   Fifteen recently abstinent methamphetamine users and 15 healthy controls completed
279 able uptake of the radiotracer, BPnd) (in 31 methamphetamine users and 37 control participants).
280 availability has been observed in striata of methamphetamine users as compared with controls, but an
281 nalyses showed that, on both tasks, the more methamphetamine users chose to view methamphetamine imag
282 nalyses showed that, compared with controls, methamphetamine users chose to view more methamphetamine
283 post-mortem material, with no differences in methamphetamine users from controls in the caudate nucle
284               Maladaptive decision making by methamphetamine users may reflect circuit-level dysfunct
285 tively, to measure gray-matter volume (in 58 methamphetamine users) and dopamine D2/D3 receptor avail
286 eliorate striatal D2/D3 receptor deficits in methamphetamine users, and warrants further evaluation a
287 , insula, hippocampus and temporal cortex in methamphetamine users, but not in control participants (
288 ter volume in mesocorticolimbic circuitry in methamphetamine users, possibly reflecting greater dopam
289 d gray-matter volume have been unexplored in methamphetamine users.
290 rd-driven behavior over cognitive control in methamphetamine users.
291  dissociation in mechanisms of incubation of methamphetamine vs cocaine craving.
292 t the potential validity of this preclinical methamphetamine vs food choice model.
293     The aims of this study were to develop a methamphetamine vs food choice procedure and determine t
294 about the neuropharmacological mechanisms of methamphetamine vs food choice.
295                        Conversely, speech on methamphetamine was further from compassion than placebo
296                                              Methamphetamine was more strongly associated with higher
297                                  Craving for methamphetamine was negatively associated with gray-matt
298                       Morphine, cocaine, and methamphetamine were chosen as test analytes for this st
299                Thereafter, lever-presses for methamphetamine were punished by mild footshocks for 5 d
300  with 88% accuracy, and MDMA (1.5 mg/kg) and methamphetamine with 84% accuracy.

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