ライフサイエンスコーパス: methemoglobinemia

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1 cytochrome b(5) reductase mutant that causes methemoglobinemia.

2 ons that give rise to the hereditary disease methemoglobinemia.

3 ead to clinically significant hypotension or methemoglobinemia.

4 associated with 3-AP administration include methemoglobinemia.

5 of gastrointestinal cancer and, in infants, methemoglobinemia.

6 nt systemic vasoconstriction without causing methemoglobinemia.

7 municipal tap water as a potential cause of methemoglobinemia and monitor for excessive levels of ox

8 polycythemias, including hemoglobin mutants, methemoglobinemias and congenital 2,3-bisphosphoglycerat

9 ysis systems resulted in no further cases of methemoglobinemia at our institution.

10 To evaluate the root cause of a series of methemoglobinemia cases in a medical ICU.

11 Congenital methemoglobinemia caused by an erythrocytic deficiency o

12 The positions of the methemoglobinemia-causing mutations are scattered throug

13 subacute toxicity testing, 4 did not produce methemoglobinemia in rats (400 mg/kg po daily for 9 days

14 the first description of familial idiopathic methemoglobinemia in the United Kingdom was reported in

15 Recessive congenital methemoglobinemia (RCM) is caused by a deficiency of red

16 We report a sentinel case of methemoglobinemia that was associated with dialysis sess

17 Methemoglobinemia, the first hereditary disease to be id

18 described for the human enzyme or any of the methemoglobinemia variants.

19 S127P, a mutant that causes type II methemoglobinemia, was the first to be positively identi

20 interfering factors, including pigment from methemoglobinemia, we found the assay had to be modified

21 ta indicate the t-BuBpT ligands may minimize methemoglobinemia, which is a marked advantage over 3-AP

22 on of four additional patients who developed methemoglobinemia while undergoing portable dialysis.

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