ライフサイエンスコーパス: methionine enkephalin

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1 of a-, b-, and y-type fragments of [D-Ala2,3]methionine enkephalin.

2 ponding increase in tissue concentrations of methionine enkephalin.

3 ponsive to stimulation by the opioid peptide methionine enkephalin.

4 3 but not antisera against beta-endorphin or methionine-enkephalin.

5 s may contain other opioid peptides, such as methionine-enkephalin.

6 /- 1 vs. 4 +/- 1, 7 +/- 1, and 11 +/- 1% for methionine enkephalin 10(-10), 10(-8), 10(-6) M in the a

7 +/- 1 vs. 2 +/- 1, 4 +/- 1, and 7 +/- 1% for methionine enkephalin 10(-10), 10(-8), and 10(-6) M in t

8 Coadministration of NOC/oFQ (10(-10) M) with methionine enkephalin (10(-10), 10(-8), 10(-6) M) attenu

9 Methionine enkephalin (10(-10), 10(-8), 10(-6) M)-induce

10 ted peptides as substrates: neurotransmitter methionine enkephalin (Ac-Tyr-Gly-Gly-Phe-Met) and synth

11 Methionine enkephalin, an endogenous mu agonist, elicite

12 increased CSF concentrations of the opioids methionine enkephalin and leucine enkephalin during hypo

13 ioids (503+/-61 vs. 1488+/-186 pg/mg protein methionine enkephalin before and after hypoxia, (PO2 app

14 ioids (471+/-58 vs. 1181+/-148 pg/mg protein methionine enkephalin before and after SNP 10-6 M).

15 ide bonds cleaved were the Gly3-Phe4 bond in methionine enkephalin, Gly4-Gly5 bond in Met-peptide, Gl

16 Furthermore, methionine-enkephalin immunolabeled fiber densities with

17 that N2O selectively stimulates synthesis of methionine enkephalin in the diencephalic region of the

18 Methionine enkephalin-induced dilation was attenuated by

19 Moreover, FPI attenuated PACAP, methionine enkephalin, leucine enkephalin, and dynorphin

20 Methionine-enkephalin (M-ENK) and leucine-enkephalin (L-

21 in sections that were also dual labeled for methionine-enkephalin (M-ENK), an opioid peptide known t

22 currents (IPSCs) were reversibly reduced by methionine enkephalin (ME) with an IC(50) value of 1.1 +

23 ic vagal deafferentation, the opioid agonist methionine-enkephalin (ME) inhibited the amplitude of ev

24 d for immunoreactivity to the opioid peptide methionine enkephalin (mENK).

25 ignificance of such interactions, we exposed methionine-enkephalin (MENK) to PN and identified the ma

26 munoreactive (ir) for galanin, GABA, TRH, or methionine-enkephalin (mENK) were dense in the ventrocau

27 Methionine enkephalin (Met) vasodilation was blunted aft

28 at least in part, from decreased release of methionine enkephalin (Met), an opioid known to contribu

29 Following immunocytochemistry for either methionine enkephalin (met-enkephalin) or leucine enkeph

30 the application of adenosine, serotonin, or methionine enkephalin (met-enkephalin).

31 ked by receptor-saturating concentrations of methionine-enkephalin (Met-Enk), [d-Ala(2), N-MePhe(4),

32 e to exogenous leucine-enkephalin (Leu-Enk), methionine-enkephalin (Met-Enk), and the opioid antagoni

33 urons in the LH activate spinally projecting methionine enkephalin neurons, as well as two population

34 nin gene-related peptide, iron, substance P, methionine enkephalin, nicotinamide adenine dinucleotide

35 We found that ILC2s produce methionine-enkephalin peptides that can act directly on

36 etabolically active in cleaving leucine- and methionine-enkephalins peptides by releasing the N-termi

37 s a Km of 95 microM and a kcat of 7.8 s-1 on methionine-enkephalin, releasing only the N-terminal tyr

38 ol/ml for control, 10(-10), 10(-8), 10(-6) M methionine enkephalin, respectively).

39 Glial cell cultures produced more methionine enkephalin than leucine enkephalin under basa

40 leucine enkephalin (Tyr-Gly-Gly-Phe-Leu) and methionine enkephalin (Tyr-Gly-Gly-Phe-Met).

41 Dilation in response to methionine enkephalin was accompanied by increased CSF c

42 Similarly, the concentration of methionine enkephalin was significantly higher after 24

43 Dilation to other opioids (e.g., methionine enkephalin) was unchanged by [F/G] NOC/oFQ (1

44 c levels of preproenkephalin mRNA as well as methionine enkephalin were analyzed.

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