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1 genes (e.g., cystathionine-beta-synthase and methionine synthase reductase).
2 ein, which is comparable with that seen with methionine synthase reductase.
3 e P450 reductase, nitric oxide synthase, and methionine synthase reductase.
4 component of the nitric-oxide synthases and methionine-synthase reductase.
6 s, the electron is thought to be provided by methionine synthase reductase, a protein containing a do
7 cloned and expressed the cDNA encoding human methionine synthase reductase and demonstrate that it is
8 and cblG classes of patients with defects in methionine synthase reductase and methionine synthase, r
9 ptophan in related diflavin reductases (e.g. methionine synthase reductase and novel reductase 1), an
10 of mutations in the gene encoding a putative methionine synthase reductase in the cblE class of patie
11 PH is 2.6 +/- 0.5 microm, and the K(act) for methionine synthase reductase is 80.7 +/- 13.7 nm for NA
14 also showed that purified recombinant human methionine synthase reductase (MSR) in combination with
17 d A1298C), methionine synthase (MTR A2756G), methionine synthase reductase (MTRR A66G), cystathionine
18 te reductase (MTHFR) 677C-->T and 1298A-->C, methionine synthase reductase (MTRR) 66A-->G, and cystat
19 thesis--methionine synthase (MTR) A2756G and methionine synthase reductase (MTRR) A66G--provided evid
22 hylenetetrahydrofolate reductase (MTHFR) and methionine synthase reductase (MTRR), have generated con
24 ytotoxicity was observed in cells expressing methionine synthase reductase (MTRR), novel diflavin oxi
25 tathionine beta-synthase [CBS] 844ins68, and methionine synthase reductase [MTRR] 66A>G) in 452 young
26 oxidoreductase with significant homology to methionine synthase reductase, NR1, has been described r
28 he soluble dual flavoprotein oxidoreductase, methionine synthase reductase, serves as a redox partner
29 thionine synthase is required for NR1 versus methionine synthase reductase, suggesting that it may re
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