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1 mGluR1alpha orthosteric antagonist (S)-alpha-methyl-4-carboxyphenylglycine.
2 he group I mGluR antagonist MCPG [(RS)-alpha-methyl-4-carboxyphenylglycine, 50-500 mum] on persistent
3 e group I/II-specific antagonist (R,S)-alpha-methyl-4-carboxyphenylglycine and the group III-specific
5 the metabotropic receptors 1 (mGluR1), +/-2-methyl-4-carboxyphenylglycine (LY367385), and mGluR5, 2-
7 ibited by the metabotropic antagonists alpha-methyl-4-carboxyphenylglycine (MCPG) and 1-aminoindan-1,
9 antagonism of group 1 mGluRs with (S)-alpha-methyl-4-carboxyphenylglycine (MCPG) did not result in s
10 he APB-insensitive receptor antagonist alpha-methyl-4-carboxyphenylglycine (MCPG) does not block the
11 Over the past few years, the compound alpha-methyl-4-carboxyphenylglycine (MCPG) has been widely use
12 glutamate receptor (mGluR) antagonist alpha-methyl-4-carboxyphenylglycine (MCPG) strongly attenuated
14 ulfamoylbenzo[f]quinoxaline (NBQX) and alpha-methyl-4-carboxyphenylglycine (MCPG) were evaluated agai
15 ed the effect of the infusion of (+/-)-alpha-methyl-4-carboxyphenylglycine (MCPG), a metabotropic glu
16 a non-selective mGluR antagonist, (S)-alpha-methyl-4-carboxyphenylglycine (MCPG), into the dorsal st
19 liminated by the mGluR antagonist (RS)-alpha-methyl-4-carboxyphenylglycine (MCPG; 500 microM) and by
21 opic glutamate receptor antagonist (+)-alpha-methyl-4-carboxyphenylglycine ((+)-MCPG; 1000 microM) bu
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