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1 e crop production systems in a world without methyl bromide.
2 the distinctiveness of industrially produced methyl bromide.
3 ents, rats received infusions of scopolamine methyl bromide (10 microg/0.5 microl) into the nucleus a
4 osylation of 5 with (1,3-dihydroxy-2-propoxy)methyl bromide (22) followed by a ring annulation with h
6 epithelium by inhalation of the olfactotoxin methyl bromide activates a population of multipotent glo
7 implies that about 1 percent of atmospheric methyl bromide and 5 percent of methyl iodide arise from
10 By observing emissions of methyl chloride, methyl bromide, and methyl iodide from flooded Californi
11 information system to estimate the amount of methyl bromide applied within 5 km of a woman's residenc
15 luding methane (CH(4)), isoprene (C(5)H(8)), methyl bromide (CH(3)Br), dimethyl sulfide, and oxygen (
17 need for technologies to capture and destroy methyl bromide (CH3Br) emissions from postharvest fumiga
22 tution after direct experimental injury with methyl bromide, expression of the GBC-1 antigen overlaps
23 r is utilized for the generation of volatile methyl bromide from aqueous solutions of bromide and bro
27 the bulb are reconstituted after ablation by methyl bromide gas, and some measure of olfactory functi
28 of the anterior bulb in the food-restricted, methyl bromide gas-lesioned animals indicates that the m
31 nstitution of the OE, rats were subjected to methyl bromide-induced OE lesion and their OE and OBs ex
33 ion following injury by the olfactotoxic gas methyl bromide is coincident with the down-regulation of
35 t epithelium that recover as olfactory after methyl bromide lesion with those undergoing respiratory
41 marker-labeled cells into the epithelium of methyl bromide-lesioned, unmarked host mice to dissect p
43 as a stratospheric ozone-depleting compound, methyl bromide (MeBr) is being phased out despite being
45 1 day after exposure to the olfactotoxic gas methyl bromide (MeBr) to assess the lineage relationship
46 llent soil fumigant for killing plant pests, methyl bromide (MeBr) was phased out in 2005 in the USA,
48 This is related to a different gas product (methyl bromide, MeBr) removal mechanism, changing from c
49 may have prostate-specific biologic effects (methyl bromide (odds ratio = 1.62, 95% confidence interv
50 hed and abandoning gas fumigation containing methyl bromide resulted in the recurrence of an almost f
56 s among applicators over 50 years of age and methyl bromide use were significantly associated with pr
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