ライフサイエンスコーパス: methyl jasmonate

1 salicylic acid and jasmonic acid (applied as methyl jasmonate).

2 herbivory, uprooting, physical wounding, and methyl jasmonate.

3 of M. truncatula cell suspension cultures to methyl jasmonate.

4 mulate in response to exogenous cytokinin or methyl jasmonate.

5 mutant that exhibits reduced sensitivity to methyl jasmonate.

6 ntent was not modified by the application of methyl jasmonate.

7 0002930) which was specifically activated by methyl jasmonate.

8 , but the gene can be induced in leaves with methyl jasmonate.

9 2 retains aphid resistance when treated with methyl jasmonate.

10 wing treatment of roots with F. oxysporum or methyl jasmonate.

11 tified several potential targets for COR and methyl jasmonate.

12 as induced by wounding and by treatment with methyl jasmonate.

13 ssion in response to mechanical wounding and methyl-jasmonate.

14 High concentrations of methyl jasmonate (2-4.5 mol per disc surface) suppressed

15 els, effects that were overcome by exogenous methyl jasmonate (45 nmol per disc surface).

16 ichomes under normal conditions, addition of methyl jasmonate, a biotic stress hormone, induced expre

17 Methyl jasmonate, a common plant secondary compound, whe

18 cript variants in Col-0 after treatment with methyl jasmonate, a condition known to "induce ESP", it

19 te downstream SA defenses, were treated with methyl jasmonate, a dramatic delay in nymph development

20 on in potato tissue following treatment with methyl jasmonate, a lipoxygenase product of linolenic ac

21 Interestingly, methyl jasmonate, a repellent derived from the nonvolati

22 of M. lanceolata shoot cultures treated with methyl jasmonate, a well-known elicitor of plant special

23 a infection, and pretreatment of plants with methyl jasmonate alleviated pathogen damage to the mutan

24 to wounding, systemin, oligosaccharides, and methyl jasmonate also was inhibited by DPI.

25 n response to wounding followed by ethylene, methyl jasmonate and ABA treatment.

26 st, mutants compromised in responsiveness to methyl jasmonate and ethylene developed the same resista

27 ed that SLW1 RNAs accumulated in response to methyl jasmonate and ethylene, whereas SLW3 was not regu

28 functioned independently of salicylic acid, methyl jasmonate and ethylene.

29 growth in jar1-1 was equally insensitive to methyl jasmonate and jasmonic acid, indicating that the

30 in the leaves of intact plants sprayed with methyl jasmonate and methyl salicylate and in excised le

31 ar weight volatile hormone analogues such as methyl jasmonate and methyl salicylate can trigger defen

32 of expression was observed with the hormones methyl jasmonate and naphthalene acetic acid and diterpe

33 owed that foliar treatments carried out with methyl jasmonate and yeast extract achieved the best res

34 The results of this study indicate that methyl jasmonate and yeast extract applications could be

35 ine resolution and collection of both major (methyl jasmonates) and minor (epi-methyl jasmonates) ste

36 crograms of jasmonic acid, 1.3 micrograms of methyl jasmonate, and 31.0 micrograms of salicylic acid.

37 uced in response to UV-B, dehydration, NaCl, methyl jasmonate, and abscisic acid treatments indicatin

38 a small gene that is inducible by wounding, methyl jasmonate, and ethylene.

39 enzoic acid, 2,4-dichlorophenoxyacetic acid, methyl jasmonate, and hydrogen peroxide, whereas treatme

40 Jasmonic acid, methyl jasmonate, and salicylic acid have been reported

41 cells in response to wounding, systemin, and methyl jasmonate, and the nascent protein is sequestered

42 In response to methyl jasmonate application or phosphate starvation, At

43 is genes and exogenous methyl salicylate and methyl jasmonate applications showed that plant defense

44 nts and found that auxin, salicylic acid and methyl-jasmonate are three effective inducers of the as-

45 isplayed a higher level of jasmonic acid and methyl jasmonate, as well as the oxylipin-biosynthetic i

46 ed ethylene precursor, and by treatment with methyl jasmonate, but disappeared upon treatment of seed

47 The results (i) suggest that methyl jasmonate can function as a signal for hmg1 expre

48 l of sink tissues or exposure to atmospheric methyl jasmonate caused increased levels of SVL in leave

49 Methyl jasmonate, chitosan, and a commercial yeast extra

50 atment but was not significantly affected by methyl jasmonate, dehydration or heat shock stress.

51 A similar treatment with methyl jasmonate did not protect the jasmonate-insensiti

52 ed for this compound (or a related compound, methyl jasmonate) during plant development and in respon

53 corresponding gene transcript is induced in methyl jasmonate-elicited seedlings, along with the othe

54 genes in response to the signaling molecules methyl jasmonate, ethylene and salicylic acid differed m

55 Several phytohormones, including methyl jasmonate, ethylene, and abscisic acid, regulated

56 amino acids was increased as consequence of methyl jasmonate foliar application, i.e., histidine, se

57 ated from Taxus cuspidata cells induced with methyl jasmonate for Taxol production) was screened.

58 Further, transcript profiling under methyl jasmonate, gibberellic acid, and yeast extract el

59 abscisic acid; and signal molecules such as methyl jasmonate, H(2)O(2), and salicylic acid.

60 Elicitation, particularly with methyl jasmonate, has been effective at increasing the p

61 screens based on insensitivity to COR and/or methyl jasmonate identified several potential targets fo

62 In conclusion, foliar application of methyl jasmonate improved must nitrogen composition.

63 temisia tridentata, a plant shown to possess methyl jasmonate in leaf surface structures, is incubate

64 The presence of methyl jasmonate in the atmosphere of chambers containin

65 Treatment of suspension-cultured cells with methyl jasmonate increased the total binding of (125)I-T

66 Treatment with SA and methyl jasmonate induced defense gene expression only in

67 Neither exogenous salicylic acid nor methyl jasmonate induced GUS expression; and H2O2 induce

68 Jasmonates, including jasmonic acid (JA) and methyl jasmonate, induced the formation of tyloses, wher

69 library constructed from mRNA isolated from methyl jasmonate-induced Taxus cells, from which several

70 library constructed from mRNA isolated from methyl jasmonate-induced Taxus cells, from which the ful

71 apain of the CysPIs encoded by the wound and methyl jasmonate-inducible genes (57 and 21 nM for N2 an

72 Both mRNA species are wound- and methyl jasmonate-inducible.

73 s VSP2 (AtVSP2), a gene induced by wounding, methyl jasmonate, insect feeding, and phosphate deprivat

74 response mutants, etr1 and ein2, and in the methyl jasmonate-insensitive mutant, jar1, suggesting th

75 ignaling deficiencies were complemented with methyl jasmonate, JA-Ile, and its functional homolog, co

76 d N2 transcripts were induced by wounding or methyl jasmonate (M-JA) treatment in local and systemic

77 Methyl jasmonate (Me-JA) pretreatment of Cvi-0 decreased

78 oliar applications of phenylalanine (Phe) or methyl jasmonate (MeJ) could improve the synthesis of se

79 as to study the effect of the elicitation of methyl jasmonate (MeJ) supported by phenylalanine (Phe)

80 herefore, this work studied elicitation with methyl jasmonate (MeJ), supported by precursor feeding w

81 postharvest life, preharvest applications of methyl jasmonate (MeJA) and chitosan were evaluated duri

82 Exposure to methyl jasmonate (MeJA) and methyl salicylate (MeSA) vap

83 Methyl jasmonate (MeJA) elicits stomatal closure in many

84 Arabidopsis mutants that were insensitive to methyl jasmonate (MeJA) in an assay for seedling root gr

85 Treatment of plants with methyl jasmonate (MeJA) increased the total binding of [

86 Methyl jasmonate (MeJA) induced AtAO1 gene expression in

87 Wounding and methyl jasmonate (MeJA) induced Chi9 expression, whereas

88 Methyl jasmonate (MeJA) is commonly used to elicit plant

89 The effects of methyl jasmonate (MeJA) on leaf growth regulation were i

90 Foliar application of methyl jasmonate (MeJA) or brassinazole (BRZ) resulted i

91 rbivory with the well-known defense elicitor methyl jasmonate (MeJA) to young leaves of Arabidopsis (

92 Methyl jasmonate (MeJA) treatment increased betacyanin s

93 tebark pine needle transcriptomes, following methyl jasmonate (MeJA) treatment using RNA-seq.

94 transcriptional changes in sweet basil after methyl jasmonate (MeJA) treatment, which is considered a

95 Methyl jasmonate (MeJA) was applied in a vineyard on lea

96 redox in response to abscisic acid (ABA) or methyl jasmonate (MeJA) were identified by complementary

97 the signaling compounds salicylic acid (SA), methyl jasmonate (MeJA), and the ethylene precursor amin

98 Jasmonic acid and its methyl ester, methyl jasmonate (MeJA), are plant signaling molecules t

99 se to treatment with various combinations of methyl jasmonate (MeJA), auxin (AUX), and ethylene (ETH)

100 ponsiveness, in root tissue, to ethylene and methyl jasmonate (MeJA), important cues during the estab

101 unding, and this induction was suppressed by methyl jasmonate (MeJA), in agreement with results previ

102 timize the treatment doses of the elicitors: methyl jasmonate (MeJA), jasmonic acid (JA) and DL-methi

103 eactions of (3)C* and (1)O2* with five GLVs: methyl jasmonate (MeJa), methyl salicylate (MeSa), cis-3

104 s collection responsive to greenbug feeding, methyl jasmonate (MeJA), or SA application.

105 ication of phytohormone salicylic acid (SA), methyl jasmonate (MeJA), phytopathogenic bacteria, and f

106 The effects of methyl jasmonate (MeJA), spermine (Spm), epibrassinolide

107 effects on gene expression are transduced by methyl jasmonate (MeJA)-dependent and oligogalacturonide

108 functionally characterized a leaf-specific, methyl jasmonate (MeJA)-responsive monoterpene synthase

109 on whitebark pine needles demonstrated that methyl jasmonate (MeJA)-triggered transcriptome re-progr

110 responses occur when the plant is exposed to methyl jasmonate (MeJA).

111 hat are strongly induced by the phytohormone methyl jasmonate (MeJA).

112 ated by treatment with ethylene precursor or methyl jasmonate (MeJA).

113 are differentially regulated in response to methyl jasmonate (MeJA).

114 ultures responding to yeast elicitor (YE) or methyl jasmonate (MeJA).

115 s partially insensitive to gene induction by Methyl-Jasmonate (MeJA), whereas the constitutively acti

116 f Glyma18g48580 was not induced by wounding, methyl jasmonate, methyl salicylate, or ethephon, synthe

117 Systemin, OGA, chitosan, and methyl jasmonate (MJ) all induce the accumulation of H2O

118 oybean seedlings were exposed to atmospheric methyl jasmonate (MJ) to determine if low levels of this

119 no major increase in jasmonic acid (JA) and methyl jasmonate (MJ) was detected after the activation

120 Recently, methyl jasmonate (MJ) was shown to induce conifer resin

121 ack currant cultivars after elicitation with methyl jasmonate (MJ) were examined.

122 reoisomers present in a commercial sample of methyl jasmonate (MJ) were isolated at semi-preparative

123 medicarpin in response to yeast elicitor or methyl jasmonate (MJ), accompanied by decreased levels o

124 ntrations (0.45-45 nmol per disc surface) of methyl jasmonate nearly doubled the wound-induced accumu

125 luate the influence of foliar application of methyl jasmonate on must amino acid content.

126 ropane-L-carboxylic acid, salicylic acid and methyl jasmonate) on the phytochemical composition of br

127 ed in potato leaves treated with ethylene or methyl jasmonate or infected with either virulent or avi

128 after wounding, or treatment with systemin, methyl jasmonate, or linolenic acid.

129 ) with caterpillar herbivory, application of methyl jasmonate, or mechanical damage during vegetative

130 tionally induced by wounding, treatment with methyl jasmonate, Pep peptides, and pathogen-associated

131 By contrast, treatment with methyl jasmonate promoted an increase of all analyzed At

132 h as jasmonate, 12-oxo-phytodienoic acid, or methyl-jasmonate, promotes physical interaction between

133 KEY MESSAGE: A methyl jasmonate responsive 3-carene synthase (Li3CARS)

134 n addition, treatment of soybean plants with methyl jasmonate resulted in higher levels of both LOX7

135 Furthermore, the elicitations in the form of methyl jasmonate, salicylic acid, ultraviolet B light, a

136 es are expressed in response to wounding and methyl jasmonate, similar to the expression of the syste

137 atment of red raspberry fruits with the pure methyl jasmonate stereoisomers isolated proved that (-)-

138 oth major (methyl jasmonates) and minor (epi-methyl jasmonates) stereoisomers.

139 Application of exogenous methyl jasmonate substantially protected mutant plants,

140 Application of methyl jasmonate to leaves of intact tomato plants, or s

141 that the defect was not in the conversion of methyl jasmonate to the acid.

142 chloroplast and total protein extracts from methyl jasmonate-treated leaves indicated that a small p

143 on was highest in leaves, and was induced by methyl jasmonate treatment and wounding.

144 r with induction of MusaSAP1 by wounding and methyl jasmonate treatment indicated possible involvemen

145 This finding suggests that methyl jasmonate treatment might be conducive to obtain

146 the two mRNAs was unaffected by wounding or methyl jasmonate treatment, conditions that increase the

147 In response to methyl jasmonate treatment, LIF2 was rapidly recruited t

148 , MtFNSII-2 was inducible by S. meliloti and methyl jasmonate treatment, whereas MtFNSII-1 was not.

149 two (N2 and R1) were induced by wounding or methyl jasmonate treatment.

150 er induced after F. oxysporum inoculation or methyl jasmonate treatment.

151 We found that upon wounding and methyl jasmonate treatments, MYB11 and ZML2 proteins are

152 to 50 min following wounding or systemin or methyl jasmonate treatments, the TomLoxD mRNA level incr

153 erea infection or after benzothiadiazole and methyl jasmonate treatments.

154 but is induced in leaves by alamethicin and methyl jasmonate treatments.

155 ion cultures treated with fungal elicitor or methyl jasmonate, treatments which did not affect tdc1 e

156 Exposing intact plants to methyl jasmonate vapors induced the accumulation of PLPI

157 ystemin more than threefold, suggesting that methyl jasmonate was activating transcription of the gen

158 ere analyzed in a negative-ion mode, whereas methyl jasmonate was analyzed in a positive-ion mode.

159 in response to the exogenous application of methyl jasmonate was associated with increased bark conc

160 Flower opening induced by exogenous applied methyl jasmonate was impaired in osjar1 plants and was r

161 Salicylic acid, but not methyl-jasmonate was able to replace the stress treatmen

162 cells (induced for taxoid biosynthesis with methyl jasmonate) was undertaken.

163 data cells induced for Taxol production with methyl jasmonate, were individually expressed in Escheri

164 also inducible by auxin, salicylic acid and methyl-jasmonate with similar kinetics.

165 and 99.9%, and 0.18 mg for (-)- and (+)-epi-methyl jasmonates, with 98.6% and 91.6% respective purit

166 solated amounts were 3.56 mg for (-) and (+)-methyl jasmonates, with respective purities of 96.1% and

167 immature cotyledons but is highly induced by methyl jasmonate, wounding and Aspergillus infections in