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4 ft assay, site-directed mutagenesis, and DNA methylation interference analyses, we show that three di
9 gulated PscpA and Psic promoters, as well as methylation interference analysis of PscpA, establish th
11 ion electrophoretic mobility shift assay and methylation interference analysis revealed that C'BP-2 i
14 Here, we extended this earlier work by using methylation interference analysis to identify and charac
16 of cellular factors were identified by using methylation interference and electrophoretic mobility sh
20 mobility-shift assay, DNase I footprinting, methylation interference, and ethylation interference.
21 n mutants of HMG I(Y), DNase I footprinting, methylation interference, and in vivo transcriptional as
23 Electrophoretic mobility shift assay and methylation interference assay revealed that the GST fus
25 phoretic mobility gel shift assay (EMSA) and methylation interference assay, GKLF was found to bind B
28 In vitro gel mobility shift analyses and methylation interference assays demonstrated that NFIL-6
33 ility shift assay, DNase I footprinting, and methylation interference assays, we demonstrate that Sp1
34 el mobility shift, DNase I footprinting, and methylation interference assays, we demonstrated that th
40 ns were examined by DNase I footprinting and methylation-interference assays, and are very similar, i
42 ding assays, DNase 1 footprint analysis, and methylation interference demonstrate that the binding is
47 adical footprinting, missing nucleoside, and methylation interference experiments to investigate the
48 proposed minor-groove binding model based on methylation interference experiments, our structure clea
50 Electrophoretic mobility shift assays and methylation interference footprinting demonstrated that
52 B sites, which are here shown by DNase I and methylation interference footprinting to flank a novel b
55 n organello footprinting techniques based on methylation interference have been utilized to investiga
57 he affinity increase, DNase I protection and methylation interference (MI) assays were performed.
58 at of the Dfd HD, the missing nucleoside and methylation interference patterns resemble those of the
60 rinting using exonucleaseIII and DNaseI, and methylation interference show no asymmetry, with both DN
62 DNA binding specificities by competition and methylation interference studies and are immunologically
63 sequence, by using DNase I footprinting and methylation interference studies and electrophoretic mob
65 xamined the contacts between EVI1 and DNA by methylation interference studies, which revealed extensi
67 not C) determined by sequence comparison and methylation interference, we predicted that HNF-6 will b
68 an electrophoretic mobility shift assay and methylation interference, we show that IL-6 induced reci
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