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1 inhibitor hydroxyurea or DNA damaging agent methylmethane sulfonate.
2 DNA-damaging agents such as UV radiation and methylmethane sulfonate.
3 the zinc fingers themselves are sensitive to methylmethane sulfonate and have reduced ability to indu
4 reatment of cells with DNA alkylating agents methylmethane sulfonate and N-methyl-N'-nitro-N-nitrosog
5 tion in vitro, sensitize yeast to killing by methylmethane sulfonate and reduce spontaneous gene conv
7 are sensitive both to the DNA damaging agent methylmethane sulfonate and to exposure to UV radiation.
9 DNA-damaging reagents including hydroxyurea, methylmethane sulfonate, and bleocin, demonstrating a ro
10 ons are hyperrecombinogenic and sensitive to methylmethane sulfonate, and they become inviable upon i
11 ain was sensitive to UV irradiation, X-rays, methylmethane sulfonate, and thiabendazole, and these se
13 UV irradiation and to the DNA damaging agent methylmethane sulfonate, implying that DNA repair pathwa
14 l Fpg conferred Tpp1-dependent resistance to methylmethane sulfonate in yeast lacking the abasic endo
16 therapeutic agents, including UV radiation, methylmethane sulfonate, ionizing radiation, etoposide,
18 as PYK2, CADTK) is activated specifically by methylmethane sulfonate (MMS) and hyperosmolarity but no
19 ons and in response to the methylating-agent methylmethane sulfonate (MMS) and ionizing radiation.
20 ensitivity to the replication-blocking agent methylmethane sulfonate (MMS) in smc6 mutants, with doub
21 induced in 32D cl3 myeloid cells exposed to methylmethane sulfonate (MMS), a DNA alkylating agent.
22 iminished G1 checkpoint after treatment with methylmethane sulfonate (MMS), a DNA base-damaging agent
24 ferent mechanisms of action and DNA targets: methylmethane sulfonate (MMS), methylnitrosourea (MNU),
30 found that oxidation (H2O2) and alkylation (methylmethane sulfonate, MMS) stresses induced nearly id
32 mage with H(2)O(2) or alkylation damage with methylmethane sulfonate, or rat lung fibroblasts after o
35 e-strand break and partially complements the methylmethane sulfonate sensitivity of the mutant cells.
37 ellular resistance to the DNA damaging agent methylmethane sulfonate, suggesting that nucleotide bind
38 e found that Tpp1 could confer resistance to methylmethane sulfonate when expressed in bacteria that
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