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   1 lso to salinomycin and the alkylating agent, methylnitrosourea.                                      
     2 effects of methyl methanesulfonate (MMS) and methylnitrosourea.                                      
     3 ecies generator or the methylating chemical, methylnitrosourea.                                      
     4  subsequent mammary tumorigenesis induced by methylnitrosourea.                                      
     5 o seen with the S(N)1-type methylating agent methylnitrosourea.                                      
     6 r to detectable lesions after treatment with methylnitrosourea, a condition that was not detected in 
     7 ibit by approximately 60% the incidence of N-methylnitrosourea and approximately 50% of 7,12-dimethyl
     8 nd to be very similar to each other [in both methylnitrosourea and dimethylbenz(a)anthracene rat mamm
     9 pe are tolerant to methylating damage from N-methylnitrosourea and N-methyl-N'-nitro-N-nitrosoguanine
    10 ls are known to be hypersensitive to MMS and methylnitrosourea, and in the presence of MX (i.e. under
    11 fter low-dose treatment with the carcinogens methylnitrosourea, benzo(a)pyrene-diolepoxide 1, or both
  
    13 wn to display significant protection against methylnitrosourea-induced mammary tumorigenesis followin
    14 o identify differentially expressed genes in methylnitrosourea-induced rat mammary adenocarcinomas.  
    15  a potent in vivo inhibitor of the growth of methylnitrosourea-induced rat mammary tumors without any
  
    17 inogenesis model with CD-1 mice and in the N-methylnitrosourea mammary carcinogenesis model with Spra
    18 and MIAPaCa-2 could not be eradicated with N-methylnitrosourea (MNU) at concentrations as high as 2 m
  
    20 were exposed to the simple methylating agent methylnitrosourea (MNU) without previous treatment with 
    21  DNA targets: methylmethane sulfonate (MMS), methylnitrosourea (MNU), ethylmethane sulfonate (EMS), a
    22  We have examined the effects of estrogen on methylnitrosourea (MNU)-induced cataractogenesis in Spra
    23 parately and combined, on the development of methylnitrosourea (MNU)-induced rat mammary gland carcin
    24 f lymphomas by the methylating carcinogen, N-methylnitrosourea [MNU], in transgenic mice expressing b
  
    26 entrosomes/cell, whereas comparable cells in methylnitrosourea-treated animals displayed significantl
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