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1 ne led to [N-Me,des-Sar]Gal-B2, containing N-methyltryptophan.
2 y-4-methyltryptophan are the products from 5-methyltryptophan.
3 reefold and allowed threefold induction by 1-methyltryptophan.
4 th inhibition by the tryptophan analog alpha-methyltryptophan.
5 c mice were treated with the IDO inhibitor 1-methyltryptophan (1-MT) and monitored for the extent of
6 wo positions various tryptophan analogues (5-methyltryptophan, 5-fluorotryptophan, 1-methyltryptophan
8 of the transformed E. coli strains toward 5-methyltryptophan, a false-feedback inhibitor of AS, was
10 dendritic cells (pDC) were neutralized by 1-methyltryptophan and Abs against TGF-beta and pDC, respe
11 a series of analogues in which the key alpha-methyltryptophan and adamantyloxycarbonyl moieties, requ
12 was assessed in vivo by administration of 1-methyltryptophan and clinical and histologic evaluation
13 site of the indole rings of tryptophan and 5-methyltryptophan and of 3-indolepropionate and 3-indolea
14 binding of the corepressors tryptophan and 5-methyltryptophan and of the inducers 3-indolepropionate,
15 s (5-methyltryptophan, 5-fluorotryptophan, 1-methyltryptophan, and 2-naphthylalanine) and assessing t
16 5-Hydroxymethyltryptophan and 5-hydroxy-4-methyltryptophan are the products from 5-methyltryptopha
18 compstatin analogues, peptides containing 1-methyltryptophan at position 4 exhibited the highest bin
20 a-methylphenylalanine (beta-MePhe)7 and beta-methyltryptophan (beta-MeTrp)9 and the two isomers of 1,
22 nging the corepressor from L-tryptophan to 5-methyltryptophan causes effects at residues 52, 60, 61,
24 eatment with the competitive IDO inhibitor 1-methyltryptophan fully blocked BCG-induced depressive-li
26 3 and 4 with literature values of related N-methyltryptophan natural products was used to determine
27 th 1:1 and 2:1 complexes are observed with 5-methyltryptophan or if glycerol is present in the gel.
29 the indoleamine-2,3-dioxygenase antagonist 1-methyltryptophan or neutralizing anti-IL-10R from days 1
30 Monomeric sarcosine oxidase (MSOX) and N-methyltryptophan oxidase (MTOX) are homologous bacterial
31 Monomeric sarcosine oxidase (MSOX) and N-methyltryptophan oxidase (MTOX) are homologous enzymes t
37 und FAD in monomeric sarcosine oxidase and N-methyltryptophan oxidase, enzymes that exhibit sequence
38 lays a role in holoenzyme biosynthesis and N-methyltryptophan oxidation and (ii) MTOX contains separa
39 i) MTOX contains separate active sites for N-methyltryptophan oxidation and oxygen reduction on oppos
40 e abnormal leaf development, resistance to 5-methyltryptophan, reduced growth of the generalist lepid
41 The contents of the remaining compounds, 5-methyltryptophan, tryptamine, melatonin, indoleacetonitr
42 us fruiting bodies contained l-tryptophan, 5-methyltryptophan, tryptamine, melatonin, indoleacetonitr
43 background activity during incubations of 4-methyltryptophan with dimethylallyl diphosphate, we foun
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