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2 splasia, and CAC as well as in dysplastic tumors of C57BL/6 mice treated with azoxymethane-dextran sulfate sodium.
4 survival/proliferation, and survival in brain tumor-bearing mice treated with PT2385 alone and in combination with standa
5 red blood cells and urine, and skin photosensitivity in CEP mice treated with deferiprone (1 or 3 mg/mL in drinking water
7 omach and liver were collected from Mdr2KO and FVBN control mice treated with Ghr, des-octanoyl-ghrelin (DG) or vehicle.
11 NA-sequencing was performed on liver samples collected from mice treated with a reference chemical (phenobarbital) or veh
15 222 expression were shown in MLN-M isolated from 7 Gy GIARS mice treated with GL, and these macrophages did not display a
21 an facilitate rapid recovery of hematopoietic components in mice treated with the endoradiotherapeutic agent (153)Sm-EDTM
22 r erythroid 2-related factor 2 expression was also found in mice treated with MIA-690 and MR-409.
23 y weight, we found that the decrease in body weight gain in mice treated with metformin is not directly attributable to i
24 produced in mice lacking Bruton tyrosine kinase, but not in mice treated with 4 weeks of acalabrutinib, a more specific B
26 nflammation and tissue damage were significantly reduced in mice treated with metronidazole when combined with anti-E. hi
27 Histological sections of lung tumors in mice treated with MSU-42011 exhibited reduced cell density an
29 Compared to hypercholesterolemic Ldlr(-/-) mice treated with vehicle or 4-HOBA, a nonreactive analogue,
31 mpared to their controls and decrease in serum of Mdr2(-/-) mice treated with 5HTR2A/2B/2C antagonists compared to untrea
32 (G609G/G609G)Mmp13(-/-) mice or Lmna(G609G/G609G)Mmp13(+/+) mice treated with a MMP inhibitor show lower SMC loss in the
33 Npr1(-/-) ), 2-copy (Npr1(+/+) ), and 4-copy (Npr1(++/++) ) mice treated with GC inhibitor, A71915 and cGMP-dependent pro
34 activation on gene expression and phenotype of the liver of mice treated with valproic acid (VPA), or 2-propylpentanoic a
35 Finally, expression of Ahr in the enteric neurons of mice treated with antibiotics partially restores intestinal m
37 We performed electrophysiology studies on mice treated with ibrutinib to assess inducibility of AF.
40 levated in the placenta of pregnant ZIKV-infected Rag1(-/-) mice treated with an anti-IFNAR Ab, primarily at the end of p
43 to sort the glial and vascular cells from the brains of the mice treated with LPS at different time points, and RNA-seq w
48 However, reduction was incomplete compared with mice treated with human FVIII concentrate, and no difference
50 ivermectin had exacerbated renal IR injury whereas P2X4 WT mice treated with a selective P2X4 antagonist (5-BDBD) were p