ライフサイエンスコーパス: mice treated with

1 h intermediate or high specific activity and none of 6 (0%) mice treated with low specific activity or formulation demons

2 Four of 6 (67%) mice treated with intermediate or high specific activity and

3 In addition, mice treated with ANDRO showed reduced expression of inflamma

4 P2X7(-/-) mice and mice treated with a P2X7 inhibitor were protected from diabet

5 Finally, CD4+ T cells from arthritic mice treated with CM-MSC showed increases in FOXP3 and IL-4 e

6 T cells isolated from spleens and lymph nodes of arthritic mice treated with CM-MSC or MSCs.

7 3(+) LSCs derived from preneoplastic livers of beta2SP(+/-) mice treated with interleukin-6 (pIL6; (IL6) beta2SP(+/-) LSC

8 BALB/c mice treated with adeno-associated virus encoding the BL6 BAG

9 Moreover, CMs isolated from transverse aortic constriction mice treated with MR-409 showed improved contractility and re

10 Furthermore, CRS mice treated with LAC show resilience of the CRS-induced stru

11 Lineage tracing using Rosa-td tomato (Col2-Cre-ERT2) mice treated with tamoxifen indicated that the same Col2 expr

12 Furthermore, TAMs isolated from IH-exposed mice treated with celecoxib reduced the proliferation of LLC1

13 Analysis of plasma and lung tissues from SEB-exposed mice treated with abatacept demonstrated significantly lower

14 Oxygen consumption in freshly isolated mitochondria from mice treated with Honokiol showed enhanced mitochondrial resp

15 Furthermore, mice treated with alphavbeta3-MP-docetaxel exhibited a signif

16 In xenograft models of EOC using SKOV3ip1 or HeyA8, mice treated with the combination of itraconazole and paclita

17 The reduction in urinary oxalate excretion in hyperoxaluric mice treated with O. formigenes CM reflects the in vivo reten

18 In Trip13(Gt/Gt)hypomorph mice treated with unilateral renal IRI, persistent tubular ep

19 Human AML cells from terminally ill mice treated with chemotherapy (chemoAML) had higher lipid co

20 In mice treated with nephrotoxic serum to induce crescentic neph

21 ogether with a decreased expression of cleaved caspase-3 in mice treated with 6-OHDA or rotenone.

22 osphorylation, and synaptic pathology, which were absent in mice treated with zileuton.

23 In addition, Trim32 protein levels were elevated in mice treated with IMQ.

24 cells, but not progenitor proliferation, is interrupted in mice treated with a hedgehog (Hh) pathway inhibitor (HPI), an

25 wal in mice treated chronically with WIN55,212-2 but not in mice treated with GAT211.

26 accumulated glycogen, a phenotype that was also observed in mice treated with TFV and ADV.

27 was consistent with the restoration of the CHS response in mice treated with the DNA demethylating agent, 5-aza-2'-deoxy

28 termine the stability of reference gene expression (RGE) in mice treated with DNBS.

29 that lipogenesis is dispensable for liver tumorigenesis in mice treated with DEN, and identifies an important role for A

30 ing CRISPR-Cas9 genome editing in transplantable tumours in mice treated with immunotherapy to discover previously undesc

31 NOD mice treated with AZD1480 were protected from autoimmune diab

32 Despite the absence of diabetes, NOD mice treated with anti-CSF-1 receptor starting at 3 or 10 wk

33 Finally, PC3 xenograft nude mice treated with NLS in vivo showed a significant decrease i

34 n vitro efficacy of anti-CD20 mAbs and improves survival of mice treated with rituximab.

35 We also determined the renal and cardiac transcriptome of mice treated with the different diets.

36 hTNF-Tg mice treated with infliximab demonstrated significant clinica

37 We found that mice treated with these lifespan-extending interventions were

38 Here we show that mice treated with anakinra, an antagonist of the interleukin

39 Skin inflammation significantly improved in KC-Tie2 mice treated with each of the antibodies targeting IL-23, IL-

40 Tb4-overexpressing transgenic mice treated with CCl4 were susceptible to the development he

41 obtained from Cbx3/HP1gamma-insufficient mice or wild type mice treated with Cbx3/HP1gamma-insufficient CD8(+) T cells,

42 Wild type mice treated with rhCD55 immediately after reperfusion were a

43 group 2 innate lymphoid cells (ILC2), and C57BL/6 wild-type mice treated with anti-IL5 antibody.

44 was not observed in Ig-treated Rag1(-/-) mice or wild-type mice treated with anti-type I IFNR alone.

45 ne response element of Gpr64 gene in the uteri of wild-type mice treated with P4.

46 When mice treated with AAV9/MIS were paired with male breeders, th

47 y tumorigenic and metastatic, whereas those derived from WT mice treated with pIL6 ((IL6) WT LSCs) had significantly less

48 ER stress markers were increased in the lungs of healthy WT mice treated with recombinant murine CIRP, but not in the lun

49 Here, we demonstrate that WT mice treated with CLP for 2 weeks had significantly reduced t

50 er binding to CNS synaptosomes isolated from wild type (wt) mice treated with NIR light was significantly reduced and the