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3 This phenomenon was corroborated when WT mice were treated with a CB2-specific antagonist that caused
4 cted NSG mice, no hyperinfection was observed even when the mice were treated with a monoclonal antibody to eliminate res
6 fore, wild-type (WT) and AMPK-beta1-knockout (AMPK-beta1KO) mice were treated with a salsalate dose resulting in clinical
9 We used a murine model of simulated menses; mice were treated with a single dose of the nonaromatizable a
11 Six weeks after Pkd1 global gene knockout, the mice were treated with Agt-ASO (66 mg/kg/wk), lisinopril (100
14 meninges is influenced by interactions with CD11c(+) cells, mice were treated with anti-LFA-1 Abs to reduce the number of
20 ation, 2',3'-cyclic-nucleotide 3'-phosphodiesterase-EGFP(+) mice were treated with cuprizone, and OPCs were sorted from t
21 period, at a time when spontaneous seizures were frequent, mice were treated with diphtheria toxin to ablate peri-insult
22 months of age, tet-off amyloid precursor protein transgenic mice were treated with doxycycline (dox) to suppress further
23 Cholesterol-fed, LDL receptor-deficient mice were treated with either an anti-Apob antisense oligonuc
27 of BH4 deficiency in disease phenotypes, 12-month-old Fabry mice were treated with gene transfer-mediated ERT or substrat
31 3 failed to protect and even exacerbated liver injury after mice were treated with lipopolysaccharide/GalN and a pan-casp
32 Utilizing a murine calvarial model, Mk2(+/+) and Mk2(-/-) mice were treated with live A. actinomycetemcomitans bacteria
34 264.7, mouse primary lung monocytes/macrophages, and BALB/c mice were treated with LPS/IFN-gamma, in the presence and abs
35 the role of miR-497 on angiogenesis, VEGFR2-luc transgenic mice were treated with miR-497 mimic and applied to monitor t
38 (0.054 g +/- 0.041 vs 0.223 g +/- 0.112, respectively; six mice were treated with pazopanib and seven were control mice;
39 immune responses to sequential IAV infections, adult female mice were treated with placebo or one of two progestins, P4 o
41 Female KO and congenic wild type (WT) mice were treated with racemic PCB 136, and levels and chiral
43 In this study, newborn CBS-knockout (KO) mice were treated with recombinant polyethyleneglycolylated h
44 ovariectomized (OVX) female mice, and estrogen-treated male mice were treated with silica, and lung injury was assessed.
47 To further evaluate these findings, CaMKIIdeltaC transgenic mice were treated with the beta1-AR blocker metoprolol (270 m
49 Immediately after the surgery, these mice were treated with the Tgfbr2 inhibitor losartan for 8 we
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