ライフサイエンスコーパス: microM

1 with 40-100 microM glutamate or with 0.5-1.0 microM NMDA robustly activated Akt and ERK1/2.

2 croM) in a wide range of detection (0.01-1.0 microM) and a response time of 250 s for bisphenol A (BP

3 tive RyR2 channels to subphysiologic (</=1.0 microM) luminal [Ca(2+)].

4 e wide concentration range from 5.0 to 300.0 microM obtained by amperometric measurement.

5 auIII selectively inhibited SOAT1 (IC50; 5.0 microM (SOAT1) vs >90 microM (SOAT2)), while in those tr

6 the concentration of ds-DNA from 1.2 to 6.0 microM.

7 s desirable sensitivity (0.02340+/-0.0001) microM(-1), excellent stability for 7 weeks and good r

8 1)cm(-2) and a low detection limit of 0.0014 microM.

9 031 +/- 0.002 microM; Cox-1, 0.012 +/- 0.002 microM; and Cox-2, 0.43 +/- 0.025 microM) but does not s

10 ibitory concentration: FAAH, 0.031 +/- 0.002 microM; Cox-1, 0.012 +/- 0.002 microM; and Cox-2, 0.43 +

11 significantly high sensitivity (0.002microA/microM and 0.007microA/microM) and detection limit (22nM

12 sitivity (0.002microA/microM and 0.007microA/microM) and detection limit (22nM and 60nM) respectively

13 The system could detect FA as low as 0.01 microM with a linear range of 1-600 microM.

14 vivo the limit of detection was 0.04+/-0.02 microM, which permitted real-time monitoring of the basa

15 +/- 0.002 microM; and Cox-2, 0.43 +/- 0.025 microM) but does not significantly interact with a panel

16 inhibited RyR2 with an IC50 of 0.16 +/- 0.03 microM.

17 of 0.45 +/- 0.05 microM versus 0.22 +/- 0.03 microM; P = 0.006), demonstrating that modulation of i2

18 nt in vitro cytotoxicity (GI50 0.55 +/- 0.04 microM) and in vivo antitumor effects in a MIA PaCa-2 xe

19 M(-1)cm(-2)) and a low detection limit (0.04 microM) for H2O2.

20 +/- 4%) and amplitude (IC50 = 0.19 +/- 0.04 microM, Emax = 66 +/- 4%) in the presence of 100 nM CaM.

21 icroM(-1)s(-1)) than NPP (k cat /K m = 0.044 microM(-1)s(-1)).

22 ould detect cholesterol ester as low as 0.05 microM (S/N=3).

23 ug-induced cell death (IC50 of 0.45 +/- 0.05 microM versus 0.22 +/- 0.03 microM; P = 0.006), demonstr

24 nsitive (lower limit of quantitation of 0.05 microM), selective, accurate (</= 15% RE) and precise (<

25 centrations (IC50 values) on day 6 were 6.06 microM for imatinib, 3.72 microM for dasatinib, and 81.3

26 0.2-500 microM with detection limit of 0.063 microM for hydrogen peroxide was achieved.

27 evoked more potent UDB of INaP (IC50 = 0.07 microM) than ranolazine (16 microM) and lidocaine (17 mi

28 .63-140.61) microM to 190.52 (136.68-226.07) microM (P = .004).

29 .2 to 9.8mM, with sensitivity of 0.087microA/microM/cm(2) and a detection limit of 0.12microM (S/N=3)

30 ty results (limit of detection of around 0.1 microM) were obtained with an organic light emitting dio

31 e enterobactin (Kd approximately 0.4 +/- 0.1 microM), preferentially binds the Fe(III) complex of the

32 ponding limit of detection (LOD) of 16 +/- 1 microM (S/N=3), which is included in NAP physiological r

33 mal inhibitory concentration of 36.3 +/- 4.1 microM.

34 t 10-fold selectivity over TRPC3 (IC50 = 9.1 microM) and TRPC6 (IC50 = 11.3 microM).

35 g an assay comprised of 200 nM APTS-MT and 1 microM ConA, the fluorescence anisotropy capably tracks

36 8- and 9.9-fold for 1 microM CGP 12177 and 1 microM propranolol, respectively) and abolished in beta1

37 in the presence of 1 microM CGP 12177 and 1 microM propranolol, respectively.

38 for the immobilization at 50 microg/mL and 1 microM, respectively.

39 ticular, for low staurosporine doses below 1 microM, we showed that recording the dissipation shift a

40 tion was achieved through a range (blank - 1 microM Diuron herbicide solution) covering the limit of

41 Clinically achievable concentrations (1 microM) progressively mobilized cellular iron from hepat

42 nce complementation (9.8- and 9.9-fold for 1 microM CGP 12177 and 1 microM propranolol, respectively)

43 sponse is linear to its concentration from 1 microM to 2000 microM with a sensitivity of 121.5 microA

44 lpha2beta2 nAChRs that, at concentrations >1 microM, also inhibits these receptors and alpha7 nAChRs.

45 Exposure to concentrations >/=1 microM resulted in ERK(1/2)-dependent apoptosis and disr

46 reatment of cells with paclitaxel (250 nM, 1 microM) and cisplatin (250 nM, 1 microM) would result in

47 (250 nM, 1 microM) and cisplatin (250 nM, 1 microM) would result in elevated cellular levels of 1-de

48 anced 2.2- and 2.1-fold in the presence of 1 microM CGP 12177 and 1 microM propranolol, respectively.

49 ines, administered in combination with SP (1 microM), markedly increase (by 1,000-fold) TNF gene expr

50 itor, and we revealed that low-dose YC-1 (10 microM) suppressed HIF-1alpha expression, and induced hy

51 Depleting SR Ca(2+) using thapsigargin (2-10 microM) changed neither the STIM1 distribution pattern n

52 s obtained for drug treated samples above 10 microM dose indicating prominent effect of ZD6474 which

53 icate concentrations reached 19, 1.4, and 10 microM, respectively, at a water depth of 27 m.

54 nduced currents with IC50 values of 5 and 10 microM, respectively.

55 At 10 microM, the cholera-autoinducer CAI-1 stimulates activit

56 0.2 microM and maximal cGMP formation at 10 microM.

57 At ~10 microM all four APs impaired the ability of HBMECs to re

58 rameters such as the Kd value being below 10 microM (both methods), k(on) = 0.46 x 10(4) M(-1) s(-1)

59 with an inhibition of growth of 50% below 10 microM; four of the six molecules-ciclopirox olamine (CP

60 Furthermore, gedunin (10 microM) inhibited LPS-induced prostaglandin E2 productio

61 r-like knockout macrophages with gedunin (10 microM) significantly inhibited LPS (50 ng/ml)-induced t

62 d wide linear concentration range (5.0 nM-10 microM).

63 The apoptotic effect of 10 microM YC-1 was enhanced by additional glucose (G) and i

64 ponse relationship to KCl in the range of 10 microM-1 mM.

65 Here, we show that resveratrol (10 microM, 48 hr) induces both a cell growth arrest and a m

66 RT-PCR demonstrated that 10 microM YC-1 reduced hypoxia-induced expression of HIF-1a

67 amounts of uranium (U) after exposure to 10 microM natural U, with preferential uptake of the (235)U

68 PUA doses ranging from 1 to 10 microM stimulated respiration, organic matter hydrolysis

69 tability of the resulting chemosensor was 10 microM 6TG for the 0.5 M KF carrier solution in FIA.

70 M3-ACh receptor stimulation with 10 microM acetylcholine resulted in distinct changes in FRE

71 ere alleviated after preconditioning with 10 microM-RS.

72 were assessed using a wide dose range (0-100 microM) in vitro.

73 type (WT) macrophages with gedunin (0.01-100 microM, noncytotoxic concentrations) inhibited LPS (50 n

74 Treatment of SCs with 40-100 microM glutamate or with 0.5-1.0 microM NMDA robustly ac

75 H-GA3) were diluted with unlabelled ABA (100 microM) and GA3 (50 microM), respectively, and applied a

76 ne bone marrow-derived MC with ARL67156 (100 microM) increased ATP-induced renin release (>/=2-fold),

77 ndantly present in our bodies, at around 100 microM under normal conditions.

78 d comparative PARP inhibitory effects at 100 microM compared to carnosic acid and gallic acid, but th

79 s very weak at -120 mV (3% inhibition by 100 microM lacosamide) but greatly enhanced at -80 mV (43% i

80 ly enhanced at -80 mV (43% inhibition by 100 microM lacosamide), where there is partial fast inactiva

81 also enhanced cell motility mediated by 100 microM-H2O2, while higher RS-doses augmented the H2O2-in

82 e less cytotoxic on normal cells (IC50 > 100 microM).

83 females, 42.89 microM, 9.8 microM, and >100 microM, respectively.

84 PUA dosages near 100 microM appeared to be toxic, resulting in decreased bact

85 Here we show that 100 microM of ascorbate induced apoptosis in A2058 melanoma

86 sal root ganglia at concentrations up to 100 microM.

87 d was in the concentration range of 125-1000 microM with detection limit of 125 microM.

88 wever, the deleterious effects of H2O2 (1000 microM) on cell migration were alleviated after precondi

89 oncentrations in the ranges from 0.5 to 1000 microM, and from 0.2 to 1000 microM, with detection limi

90 rom 0.5 to 1000 microM, and from 0.2 to 1000 microM, with detection limits of 70 nM and 49 nM, respec

91 roM Trolox/100g of vegetables) and 2.66-103 (microM Fe(2+)/100g of vegetables), for vegetables after

92 The limit of detection was 11 microM (S/N=3).

93 Ls (IMP-1 and BcII, respectively), and 10-12 microM for the B3 enzyme L1.

94 sensor achieved an apparent Kd of 45 +/- 12 microM, and a limit of detection of 0.9 microM.

95 and PM chemosensing was as low as 40 and 12 microM, respectively.

96 L) fraction throughout the oxycline (< 1-120 microM O2 ) and upper anoxic layer.

97 th Km AMT1;1 = 89 microM and Km AMT2;1 = 123 microM, respectively.

98 linearly over the concentration range 1-125 microM, with a limit of detection of 0.5 microM and a re

99 125-1000 microM with detection limit of 125 microM.

100 metal ions and has a detection limit of 135 microM (3.1 ppm).

101 se in oligomer concentrations over a 0.5-140-microM range of alphaS.

102 (r(2)=0.98+/-0.01, concentration range=0-15 microM, limit of detection =0.96+/-0.5 microM).

103 f all MBL classes in vitro, with Kis of 6-15 microM or 36-84 microM for subclass B1 MBLs (IMP-1 and B

104 A DeltaspeC mutant required >/= 15 microM exogenous putrescine to grow and could not grow a

105 ing, TCR-pMHC interactions with Kds of </=15 microM were needed to exclude CD45.

106 .0 microA mM(-1) and a detection limit of 15 microM were obtained.

107 DCA (10-150 microM) stimulated the release of both HbetaD1 and Hbeta

108 ection limit 2 nM; linear range: 2 nM to 150 microM) was better than the anti-cyt c/CNT/PPy (detectio

109 ant that is sensed via TlpC with a K D = 155 microM.

110 NaP (IC50 = 0.07 microM) than ranolazine (16 microM) and lidocaine (17 microM).

111 product, with K m and k cat of 3.69 +/- 1.17 microM and 2.01 s(-1) respectively.

112 han ranolazine (16 microM) and lidocaine (17 microM).

113 Ca(2+) wave frequency (IC50 = 0.42 +/- 0.18 microM, Emax = 47 +/- 4%) and amplitude (IC50 = 0.19 +/-

114 e kinase domain contributing to a 156 +/- 18 microM Kd interaction: a hydrophobic pocket lined by 4 c

115 ce, showing a slightly higher LOD (33 +/- 18 microM).

116 tection, and lower limit of detection (0.186 microM) than NiO nanosheet (NS)-Ni foam electrode patter

117 infinite inactivator concentration are 2.19 microM and 0.0056 minute(-1) for dronedarone and 5.45 mi

118 nner with an EC50 value of approximately 0.2 microM and maximal cGMP formation at 10 microM.

119 (bisDHBS)](2-) (Kd approximately 0.5 +/- 0.2 microM).

120 a marked pre-bloom silicate decline of 1.5-2 microM throughout the winter mixed layer during the last

121 NPs with lower affinity (80 nM, 37 nM and 2 microM, respectively) with the data fitting better to th

122 xpressing ABACUS1-2micro (Kd approximately 2 microM) and ABACUS1-80micro (Kd approximately 80 microM)

123 The Kd was approximately 2 microM.

124 We found that MSA treatments at 2 microM increased cell adherence, while inhibiting cell m

125 nstrate that MSA inhibited angiogenesis at 2 microM, which falls in the range of moderate Se nutritio

126 e bi-enzyme sensor was 30.6+/-2.7microAcm(-2)microM(-1) and the limit of detection and limit of quant

127 ADP (4-20 microM)-induced aggregation of patients' platelets was m

128 lastrol (IC50 versus TbFPPS approximately 20 microM).

129 xhibited about 10-fold lower IC50values (<20 microM) relative to previous analogs against all four ca

130 Changes of 20 microM glutamate or acetylcholine triggered diffusive de

131 ro In contrast, ursodeoxycholic acid (50-200 microM) inhibited both basal and DCA-induced defensin re

132 ere, respectively, 5.68 Omega/microm and 200 microM.

133 stituent: IC50values ranged from 4.8 to >200 microM; and they correlated with the rates of uptake, ex

134 r to its concentration from 1 microM to 2000 microM with a sensitivity of 121.5 microA microM(-1)cm(-

135 method shows a maximum sensitivity of 205nm/microM near ERY concentration of 0.01microM.

136 mediated cADPR hydrolysis (IC50 7 muM and 21 microM respectively) with 8-Br-L-cIDPR over 20-fold more

137 equilibrium dissociation constant (KD) of 21 microM, measured by surface plasmon resonance.

138 chaelis-Menten constant Kapp(M) value was 21 microM.

139 t males, 41.6 microM, 3.87 microM, and 68.22 microM, respectively; and for adult females, 42.89 micro

140 oncentration range of 0.15-2.5mM with the 22 microM limit of detection (LOD) and 7.01(+/-0.15) mVmM(-

141 es were obtained with the range of 7.47-235 (microM Trolox/100g of vegetables) and 2.66-103 (microM F

142 58) microA pM(-1)cm(-2) and (2) 0.25 nM-2.24 microM with sensitivity of 0.037 (+/- 0.081) microA nM(-

143 -1), respectively, for dronedarone, and 6.24 microM and 0.099 minute(-1), respectively, for NDBD.

144 n microsomes incubated with 200, 2, and 0.25 microM Ca(2+) and with the half-maximal K201 inhibitory

145 At concentrations </=0.25 microM, Gal-1 increased eosinophil adhesion to vascular

146 thy, gout or hyperuricemia, and can reach 25 microM in certain cases.

147 ; glutamate concentrations that exceeded 250 microM failed to activate cell signaling.

148 eer's law in a concentration range of 20-260 microM.

149 920) with a detection limit (S/N=3) of 0.264 microM.

150 5 microM for nilotinib; for L3 larvae, 11.27 microM, 13.64 microM, and 70.98 microM, respectively; fo

151 2 binds to the consensus THP with a KD of 28 microM measured in solution, but shows a higher affinity

152 er Kis (0.26-0.36 microM) than d-BTZs (26-29 microM).

153 lectrode exhibits high sensitivity of 60.2nA/microM in linear detection range (2-100muM) with a detec

154 sses a remarkably good sensitivity of 26.2nm/microM in addition to linear range from 0 to 3microM wit

155 n with a 50% inhibitory concentration of 1-3 microM, without affecting other major kidney tubule tran

156 mantadine or HMA (IC50 = 0.2 vs. 0.6 and 1.3 microM, respectively).

157 3 (IC50 = 9.1 microM) and TRPC6 (IC50 = 11.3 microM).

158 ncentration of adenosine by 120% (5.6-->12.3 microM) [P<0.001].

159 enza virus encoding wild-type M2 (EC50 = 2.3 microM), both 27: and tert-butyl 4'-(carbamimidoylcarbam

160 C50 = 3.7 microM) and guanosine (IC50 = 21.3 microM) had the highest affinities.

161 200 nL: 6.8+/-1.8 microM; 400 nL: 19.4+/-5.3 microM) [P<0.001].

162 7 receptors (P2X7R) blockade with A740003 (3 microM) prevented it.

163 We found that MB (<3 microM) readily accepted electrons from NAPQI-altered, s

164 found to be 1.62x10(-3)microM and 6.14x10(-3)microM, respectively.

165 mit of the sensor are found to be 1.62x10(-3)microM and 6.14x10(-3)microM, respectively.

166 ibitor and has no cytotoxic effects up to 30 microM; 2) inhibits CAR-mediated gene expression in prim

167 s included in NAP physiological range (9-300 microM).

168 ttle effect on hNav1.5 Na(+) currents at 300 microM when cells were held at -140 mV.

169 c detection of NAP had an upper limit of 300 microM with a corresponding limit of detection (LOD) of

170 retreatment with 300 microM lidocaine or 300 microM carbamazepine, suggesting that lacosamide, lidoca

171 Acetaldehyde, an I/R product (300 microM), elicited an 80% increase in ATP release from HM

172 was greatly reduced by pretreatment with 300 microM lidocaine or 300 microM carbamazepine, suggesting

173 ax 307 pmol/mg per minute) and HeLa (Km 0.32 microM and Vmax 42 pmol/mg per minute) cells.

174 Optimized conditions were found to be 1.34 microM for capture probe concentration and 3.15 mM for m

175 atinib, 3.72 microM for dasatinib, and 81.35 microM for nilotinib; for L3 larvae, 11.27 microM, 13.64

176 logy with a detection limit at a level of 35 microM.

177 iomers exhibit 100-fold lower Kis (0.26-0.36 microM) than d-BTZs (26-29 microM).

178 with an apparent dissociation constant of 37 microM, and steady-state kinetics reveal a competitive m

179 R. solanacearum, was enriched 76-fold to 37 microM in R. solanacearum-infected sap.

180 (31.20-54.95) microM to 79.37 (50.29-106.37) microM (P = .002), leucine levels increased from 69.21 (

181 ent eradication activities (MBEC = 0.59-9.38 microM) against MRSA, MRSE and VRE biofilms while showin

182 y, prothrombin fragment 1.2 (F1.2) (1.36-2.4 microM), thrombin-antithrombin complex (14.5-50 microg/L

183 mantadine and HMA (IC50 = 0.6 microM and 4.4 microM, respectively).

184 alue of Hb at the modified electrode is 55.4 microM, showing its high affinity.

185 ctivity for TRPC5 over TRPC4beta (IC50 = 6.4 microM), the closest structural relative of TRPC5, and a

186 te-monocyte co-incubation studies across a 4 microM semipermeable membrane suggested that L. infantum

187 ne levels increased from 20.43 (10.92-27.41) microM to 48.72 (36.28-64.84) microM (P < .001), and val

188 d 0.0056 minute(-1) for dronedarone and 5.45 microM and 0.056 minute(-1) for NDBD.

189 125 microM, with a limit of detection of 0.5 microM and a response time of 15s.

190 ed with IGF-1 (50 ng/ml) with or without 0.5 microM CX-5461 (CX), an inhibitor of RNA polymerase I.

191 ear concentration range of ara-CTP (0.05-0.5 microM), and detection is independent of reaction volume

192 =0-15 microM, limit of detection =0.96+/-0.5 microM).

193 AZD4547 (1-5 microM) demonstrated potent anti-proliferative effects,

194 ing M2(S31N) (respective EC50 = 18.0 and 1.5 microM).

195 centration in rat cerebral cortex (4.3+/-1.5 microM).

196 ide range of concentrations from 5 nM to 1.5 microM.

197 modulin-dependent protein kinase II with 2.5 microM of KN93 prevented the decrease in cell shortening

198 ign strategy to guide lead optimization, a 5 microM virtual screening hit was transformed to a series

199 noacetate (CAS 4608-49-5; larixyl acetate; 5 microM) prevented acute hypoxia-induced vasoconstriction

200 9 microM (cardiac muscle) and 104, 13, and 5 microM (SkM SR).

201 relatively weak complex (Kd approximately 5 microM).

202 systolic Ca(2+) conditions ( approximately 5 microM; channels at Po approximately 0.3) but not under

203 alpha/beta1:C binding with IC50s as low as 5 microM.

204 At 5 microM, the small molecule annotated 3F1 dispersed 50% o

205 K201 (>/=5 microM) increased RyR1-mediated Ca(2+) release from SkM

206 K201 (>/=5 microM) increased the open probability (Po) of very acti

207 reen produced 15 correctors with an EC50 < 5 microM.

208 tire range of studied concentrations (1 nM-5 microM), are nontoxic, and showed efficacy in reducing t

209 rity and became exceptionally resistant to 5 microM BTX, aside from a small residual BTX effect.

210 roA nM(-1) cm(-2)), wider linearity (0.01-50 microM) with good selectivity towards BPA.

211 Mn(2+) (50 microM) decreased the activation energy of RNA-IRP1 bind

212 Mn(2+) (50 microM, 25 degrees C) increased IRE-RNA/IRP1 binding (K

213 nstants (K(D)) ranging between 100 nM and 50 microM.

214 e expression (98%) and secretion (64%) at 50 microM and phosphorylation of p-IKB-alpha at 1 muM when

215 ound that none of the tested compounds at 50 microM significantly inhibited or increased AQP1 water p

216 with unlabelled ABA (100 microM) and GA3 (50 microM), respectively, and applied at mid-grain developm

217 ficient=0.995), with a detection limit of 50 microM.

218 ction limit 10 nM; linear range: 10 nM to 50 microM).

219 A linear response from 0.2-500 microM with detection limit of 0.063 microM for hydrogen

220 the water samples that were spiked with 500 microM phosphate.

221 48.36-99.89) microM to 114.16 (92.89-143.52) microM (P = .004), isoleucine levels increased from 20.4

222 lues are from 0.34 +/- 0.21 to 3.54 +/- 0.54 microM in 10 cancer cell lines including KBV200 cells wi

223 430, with submicromolar potency (IC50 = 0.54 microM).

224 r ten fold higher for GPP (k cat /K m = 0.56 microM(-1)s(-1)) than NPP (k cat /K m = 0.044 microM(-1)

225 C50 range of 4.43 +/- 0.35 to 49.63 +/- 3.59 microM, but they are less cytotoxic on normal cells (IC5

226 0.2 to 27.0muM with high sensitivity (1.5muA/microM) and the limit of detection was obtained about 80

227 efficacy over amantadine and HMA (IC50 = 0.6 microM and 4.4 microM, respectively).

228 ant TRPC6 by larixyl acetate (IC50 = 0.1-0.6 microM) was confirmed for native TRPC6-like [Ca(2+)]i si

229 that a labile heme pool ( approximately 1.6 microM) is stably maintained throughout parasite develop

230 resulted in a lower limit of detection (~1.6 microM) than previous reports.

231 or of GABArho1 receptors with an IC50 of 1.6 microM, an enhancer of alpha7 nicotinic receptors at rel

232 p to 300 muM, with a detection limit of 15.6 microM.

233 imit of detection (based on 3-sigma) of 22.6 microM and exhibits a sensitivity of 3417 +/- 131 microA

234 esicles had a high Km value for As(GS)3 (3-6 microM), regardless of the cell line.

235 microM, respectively; for adult males, 41.6 microM, 3.87 microM, and 68.22 microM, respectively; and

236 and inhibited Akt kinase activity (IC50 = 6 microM) and downstream signaling.

237 ors of UGM against NADPH with Ki values of 6 microM and 74 microM, respectively.

238 as 0.01 microM with a linear range of 1-600 microM.

239 levels increased from 122.56 (95.63-140.61) microM to 190.52 (136.68-226.07) microM (P = .004).

240 very-LDLs (27.8 mg/dl), and fatty acids (632 microM).

241 tration imposed by Canadian government (0.64 microM), using a halogen white light source for the stim

242 ilotinib; for L3 larvae, 11.27 microM, 13.64 microM, and 70.98 microM, respectively; for adult males,

243 tables after the sous-vide process 4.45-648 (microM Trolox/100g of vegetables) and 3.06-99.9 (microM

244 ralvinallae was grown in a chemostat with 65 microM of added H2(aq) .

245 r the conventional cooking process 6.15-657 (microM Trolox/100g of vegetables) and 3.03-99.9 (microM

246 Inosine (IC50 = 3.7 microM) and guanosine (IC50 = 21.3 microM) had the highe

247 on day 6 were 6.06 microM for imatinib, 3.72 microM for dasatinib, and 81.35 microM for nilotinib; fo

248 ation of penta-N-acetyl-chitopentaose are 72 microM and 1.4 s(-1), respectively.

249 tly higher in the higher IAA group (IAA>3.73 microM) than in the lower IAA group (IAA<3.73 microM).

250 icroM) than in the lower IAA group (IAA<3.73 microM).

251 inst NADPH with Ki values of 6 microM and 74 microM, respectively.

252 against MRSA, MRSE and VRE (MIC = 0.003-0.78 microM).

253 apparent binding affinity (Kd = 10.2 +/- 0.8 microM) is comparable to that of cAMP and nonfluorescent

254 bilized), BeauIII inhibited SOAT1 (IC50, 1.8 microM) and SOAT2 (5.9 microM).

255 tracellular concentration (200 nL: 6.8+/-1.8 microM; 400 nL: 19.4+/-5.3 microM) [P<0.001].

256 sumoylation inhibitor in vitro (IC50 = 12.8 microM) and in cells.

257 rom human embryonic kidney 293 (HEK) (Km 3.8 microM and Vmax 307 pmol/mg per minute) and HeLa (Km 0.3

258 ion range=0-4mM, limit of detection=31 +/- 8 microM) in a linear fashion.

259 ly; and for adult females, 42.89 microM, 9.8 microM, and >100 microM, respectively.

260 s in physiologically relevant ranges of 5-80 microM, showing linear response above 20 microm with app

261 (50% inhibitory concentration) around 70-80 microM.

262 oM) and ABACUS1-80micro (Kd approximately 80 microM) respond to perfusion with ABA in a concentration

263 Maximal K201 doses at 80 microM produced approximately 37% of the increase in SkM

264 and biphasic, reaching maximum effect at 800 microM.

265 Scopolamine (800 microM) also had an anxiolytic effect in a group behavio

266 icroM (S/N=3) and sensitivity of 13.80microA/microM/cm(2).

267 s in vitro, with Kis of 6-15 microM or 36-84 microM for subclass B1 MBLs (IMP-1 and BcII, respectivel

268 (10.92-27.41) microM to 48.72 (36.28-64.84) microM (P < .001), and valine levels increased from 122.

269 al performance with a sensitivity of 11.86nm/microM and 28nM as the limit of detection (LOD).

270 infinite inactivator concentration are 0.87 microM and 0.039 minute(-1), respectively, for dronedaro

271 ectively; for adult males, 41.6 microM, 3.87 microM, and 68.22 microM, respectively; and for adult fe

272 e transporters saturated with Km AMT1;1 = 89 microM and Km AMT2;1 = 123 microM, respectively.

273 , respectively; and for adult females, 42.89 microM, 9.8 microM, and >100 microM, respectively.

274 ne levels increased from 69.21 (48.36-99.89) microM to 114.16 (92.89-143.52) microM (P = .004), isole

275 - 12 microM, and a limit of detection of 0.9 microM.

276 identified compounds varied from 2.1 to 19.9 microM, of which 7 of 9 molecules did not appreciably in

277 in body fluids which are in the range 0-2.9 microM for patients with diagnosed diabetic nephropathy,

278 er the concentration range of 30.4 and 243.9 microM in phosphate buffer solution, with a correspondin

279 of CYP71AN24 for phenylacetaldoxime were 3.9 microM and 46.3 min(-1), respectively.

280 ited SOAT1 (IC50, 1.8 microM) and SOAT2 (5.9 microM).

281 ory doses (IC50) estimated at 130, 19, and 9 microM (cardiac muscle) and 104, 13, and 5 microM (SkM S

282 oM Trolox/100g of vegetables) and 3.03-99.9 (microM Fe(2+)/100g of vegetables), for vegetables after

283 oM Trolox/100g of vegetables) and 3.06-99.9 (microM Fe(2+)/100g of vegetables).

284 bited SOAT1 (IC50; 5.0 microM (SOAT1) vs >90 microM (SOAT2)), while in those treated with saponin (pl

285 s saturated with a voltage-dependent Km = 90 microM at -80 mV.

286 ne levels increased from 42.03 (31.20-54.95) microM to 79.37 (50.29-106.37) microM (P = .002), leucin

287 nd L-nAChRs activated by levamisole (Kb 0.96 microM) and bephenium.

288 arvae, 11.27 microM, 13.64 microM, and 70.98 microM, respectively; for adult males, 41.6 microM, 3.87

289 ly given as mM and should have been given as microM.

290 ly given as mM and should have been given as microM.

291 conversion and higher catalytic currents at microM glucose concentrations.

292 with one able to reduce VEEV replication at microM concentration, concomitant with reduced C nuclear

293 econstituted in Xenopus oocytes are gated by microM concentrations of betaine and mM concentrations o

294 ods were used: free radical scavenging DPPH (microM Trolox) and the reduction of Fe(3+)to Fe(2+) - FR

295 and the reduction of Fe(3+)to Fe(2+) - FRAP (microM Fe(2+)).

296 ibits a comparable sensitivity (361.9 kOmega/microM) in a wide range of detection (0.01-1.0 microM) a

297 00 microM with a sensitivity of 121.5 microA microM(-1)cm(-2) and a low detection limit of 0.0014 mic

298 3 nM and highest sensitivity of 476.2 microA/microM/cm(2), for UA, which are orders of magnitude high

299 response above 20 microm with approx. 0.1 nA/microM slope.

300 rformance with a sensitivity of 2.4+/-1.8 nA/microM, a response time of 20+/-13 s and a lower detecti