ライフサイエンスコーパス: microaneurysm

1 opathy lesions (retinal blot hemorrhages and microaneurysms).

2 r to those seen in human DR including ME and microaneurysms.

3 tailed characterization of perfused diabetic microaneurysms.

4 n sensitivities of new, static and regressed microaneurysms.

5 infiltrated with macrophages, forming pseudo-microaneurysms.

6 hemosiderin deposits, vessel tortuosity, and microaneurysms.

7 ppearance of pericyte ghosts or formation of microaneurysms.

8 ntrol) and was preceded by mesangiolysis and microaneurysms.

9 ll death, leukocyte plugging of vessels, and microaneurysms.

10 ers (15/173; 9%) and the outermost extent of microaneurysms (113/173; 68%) were localized to the oute

11 0) for any retinopathy, 3.11 (1.71-5.65) for microaneurysms, 3.08 (1.42-6.68) for soft exudates, 2.55

12 Over half of closed microaneurysms (45/86, 52.3%) left hyperreflective spots

13 as able to identify a mean (SD) of 6.4 (4.0) microaneurysms (95% CI, 4.4-8.5), while FA identified a

14 while FA identified a mean (SD) of 10 (6.9) microaneurysms (95% CI, 6.4-13.5).

15 cantly inhibiting the development of retinal microaneurysms, acellular capillaries, and pericyte ghos

16 ng clinical pathologic fundus lesions (e.g., microaneurysms and focal edema), were markedly delayed (

17 ading of secondary vascular effects, such as microaneurysms and hemorrhages, by clinical examination

18 lar to those observed in diabetes, including microaneurysms and increased vascular permeability, sugg

19 Smaller microaneurysms and those with heterogeneous lumen were p

20 n nodular glomerulosclerosis, mesangiolysis, microaneurysms, and arteriolar hyalinosis associated wit

21 sangial matrix expansion, mesangiolysis with microaneurysms, and Kimmelstiel-Wilson nodules.

22 illary length, width, density, the number of microaneurysms, and the percent of capillary length invo

23 e superior field than in the inferior field, microaneurysms are more frequent in the superior than in

24 Retinal thickness through microaneurysms as well as the presence of adjacent hypor

25 External diameter of microaneurysms averaged 104 mum (range 43-266 mum).

26 Closure rate of microaneurysms by both FA and SD-OCT was 69.9% (84/123),

27 iabetic retinopathy (DR) is characterized by microaneurysms, capillary nonperfusion, and ischemia wit

28 Some microaneurysm centers (15/173; 9%) and the outermost ext

29 reflectivity were positively associated with microaneurysm closure at 12 months (P < .0001, P < .001,

30 Microaneurysm closure rate increased at 6 and 12 months

31 eneous lumen were positively associated with microaneurysm closure.

32 or DRIL extent, cysts, hyperreflective foci, microaneurysms, cone outer segment tip visibility, and e

33 Microaneurysm dimensions, percent depth within the retin

34 ing of atheromatous plaques, atherosclerotic microaneurysms extending into periaortic vascular channe

35 Characterization of microaneurysms following focal laser photocoagulation re

36 ce of pericyte ghosts, vascular leakage, and microaneurysm formation.

37 ejection with glomerulitis, microthrombosis, microaneurysms, glomerular hypertrophy, podocyte loss, g

38 To evaluate detection of hemorrhage and/or microaneurysm (H/Ma) using ultrawide field (UWF) retinal

39 Most microaneurysms had an internal lumen with homogeneous re

40 d from another 15 dogs after 31 months, when microaneurysms had previously been observed to develop.

41 presence of pathologic conditions, including microaneurysms, hemorrhages, exudates, neovascularizatio

42 tio [OR] 2.47 [95% CI 1.42-4.31]) or retinal microaneurysms/hemorrhages (2.28 [1.24-4.18]) were signi

43 r narrowing, arteriovenous (AV) nicking, and microaneurysms/hemorrhages were evaluated on digital ret

44 etinal microvascular lesions (AV nicking and microaneurysms/hemorrhages) are more likely to have mult

45 , and histological analysis showed incipient microaneurysms in retinas of gal-fed marmosets.

46 tion at month 1 and again every 3 months for microaneurysm leakage.

47 signaling leads to the formation of abundant microaneurysms, leaky capillaries, and retinal hemorrhag

48 The worst changes histologically were microaneurysms, leukocyte and platelet plugging of aneur

49 An automated system for the measurement of microaneurysm (MA) turnover was developed and compared w

50 To correlate the appearance of microaneurysms (MAs) on structural spectral-domain optic

51 haracterized by microangiopathies, including microaneurysms, microhemorrhages, and nerve layer infarc

52 Leaking microaneurysms (n = 123) were analyzed in eyes (n = 29)

53 s manifested by an increase in the number of microaneurysms, neovascular tufts, and preretinal nuclei

54 The exact intraretinal depth of microaneurysms on OCTA was localized in all cases (100%)

55 nal hemorrhages only, 3) presence of retinal microaneurysms only, and 4) presence of moderate or wors

56 nts with diabetes had retinal defects (e.g., microaneurysms or exudates or both) within the field of

57 age, larger haemorrhage, fibrinoid necrosis, microaneurysms, perivascular space dilation, perivascula

58 Increased FA leakage of diabetic microaneurysms positively correlated with perianeurysm f

59 Perfused microaneurysms seen by SD-OCT were localized deeper than

60 here was not complete correspondence between microaneurysms shown on FA and PV-OCT images.

61 Almost all microaneurysms spanned more than 1 retinal layer (157/17

62 layers (DRIL), cysts, epiretinal membranes, microaneurysms, subretinal fluid, and outer layer disrup

63 It identified fewer microaneurysms than FA, but located their exact intraret

64 ted system to investigate some properties of microaneurysm turnover, in particular the differing dete

65 Closure of diabetic microaneurysms was characterized in detail following foc

66 A total of 173 microaneurysms were analyzed in 50 eyes (14 mild nonprol

67 inal microvascular abnormalities (IRMAs) and microaneurysms were associated with the areas of nonperf

68 The characteristics of microaneurysms were evaluated by 2 masked observers usin

69 blocked the development of mesangiolysis and microaneurysms, whereas tubulointerstitial injury was no

70 teriopathy include the "string of beads" and microaneurysms which are indistinguishable from those of